Association between dopamine transporter gene polymorphism and susceptibility to Parkinson's disease in Japan
Identifieur interne : 001299 ( Istex/Corpus ); précédent : 001298; suivant : 001300Association between dopamine transporter gene polymorphism and susceptibility to Parkinson's disease in Japan
Auteurs : Masataka Nishimura ; Ryuji Kaji ; Mitsuhiro Ohta ; Ikuko Mizuta ; Sadako KunoSource :
- Movement Disorders [ 0885-3185 ] ; 2002-07.
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Abstract
We studied a polymorphism in the dopamine transporter (DAT) gene in 236 Japanese patients with Parkinson's disease (PD) and compared the results with 220 controls. The 1215A/G genotype of the DAT gene was significantly different between PD patients and controls, suggesting a possible involvement of DAT in genetic susceptibility to PD. © 2002 Movement Disorder Society
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DOI: 10.1002/mds.10187
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ISTEX:B4B8CC9AB3FA6FF6F56543EF779ADCCE33FA0F69Le document en format XML
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The 1215A/G genotype of the DAT gene was significantly different between PD patients and controls, suggesting a possible involvement of DAT in genetic susceptibility to PD. © 2002 Movement Disorder Society</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Association between dopamine transporter gene polymorphism and susceptibility to Parkinson's disease in Japan</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>DAT and Parkinson's Disease</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Association between dopamine transporter gene polymorphism and susceptibility to Parkinson's disease in Japan</title></titleInfo><name type="personal"><namePart type="given">Masataka</namePart><namePart type="family">Nishimura</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Clinical Neuroscience, Tokushima University Hospital, Tokushima, Japan</affiliation><description>Correspondence: Department of Clinical Neuroscience, Tokushima University Hospital, Tokushima 770‐8503, Japan</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Ryuji</namePart><namePart type="family">Kaji</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Clinical Neuroscience, Tokushima University Hospital, Tokushima, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Mitsuhiro</namePart><namePart type="family">Ohta</namePart><namePart type="termsOfAddress">PhD</namePart><affiliation>Department of Clinical Chemistry, Kobe Pharmaceutical University, Kobe, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Ikuko</namePart><namePart type="family">Mizuta</namePart><namePart type="termsOfAddress">PhD</namePart><affiliation>Department of Clinical Research Center, Utano National Hospital, Ukyo‐ku, Kyoto, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Sadako</namePart><namePart type="family">Kuno</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Clinical Research Center, Utano National Hospital, Ukyo‐ku, Kyoto, Japan</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="brief communication">shortCommunication</genre><originInfo><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><place><placeTerm type="text">New York</placeTerm></place><dateIssued encoding="w3cdtf">2002-07</dateIssued><dateCaptured encoding="w3cdtf">2001-07-12</dateCaptured><dateValid encoding="w3cdtf">2002-01-11</dateValid><copyrightDate encoding="w3cdtf">2002</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="tables">1</extent><extent unit="references">10</extent><extent unit="words">1180</extent></physicalDescription><abstract lang="en">We studied a polymorphism in the dopamine transporter (DAT) gene in 236 Japanese patients with Parkinson's disease (PD) and compared the results with 220 controls. The 1215A/G genotype of the DAT gene was significantly different between PD patients and controls, suggesting a possible involvement of DAT in genetic susceptibility to PD. © 2002 Movement Disorder Society</abstract><subject lang="en"><genre>Keywords</genre><topic>Parkinson's disease</topic><topic>dopamine transporter</topic><topic>polymorphism</topic><topic>case‐control study</topic><topic>early‐onset Parkinson's disease</topic></subject><relatedItem type="host"><titleInfo><title>Movement Disorders</title></titleInfo><titleInfo type="abbreviated"><title>Mov. 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