Serveur d'exploration H2N2 - Checkpoint (PubMed)

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Transcription, Genetic < Transfection < Treatment Outcome  Facettes :

List of bibliographic references

Number of relevant bibliographic references: 8.
Ident.Authors (with country if any)Title
000232 (2008) Nigel J. Dimmock [Royaume-Uni] ; Edward W. Rainsford ; Paul D. Scott ; Anthony C. MarriottInfluenza virus protecting RNA: an effective prophylactic and therapeutic antiviral.
000288 (2005) Takashi Suzuki [Japon] ; Tadanobu Takahashi ; Chao-Tan Guo ; Kazuya I-P Jwa Hidari ; Daisei Miyamoto ; Hideo Goto ; Yoshihiro Kawaoka [États-Unis] ; Yasuo SuzukiSialidase activity of influenza A virus in an endocytic pathway enhances viral replication.
000334 (2000) S. Duhaut [Royaume-Uni] ; N J DimmockApproximately 150 nucleotides from the 5' end of an influenza A segment 1 defective virion RNA are needed for genome stability during passage of defective virus in infected cells.
000388 (1992) M L Clements [États-Unis] ; E K Subbarao ; L F Fries ; R A Karron ; W T London ; B R MurphyUse of single-gene reassortant viruses to study the role of avian influenza A virus genes in attenuation of wild-type human influenza A virus for squirrel monkeys and adult human volunteers.
000393 (1992) S Q Li [États-Unis] ; J L Schulman ; T. Moran ; C. Bona ; P. PaleseInfluenza A virus transfectants with chimeric hemagglutinins containing epitopes from different subtypes.
000465 (1984) A R Townsend ; A J Mcmichael ; N P Carter ; J A Huddleston ; G G BrownleeCytotoxic T cell recognition of the influenza nucleoprotein and hemagglutinin expressed in transfected mouse L cells.
000476 (1983) G L Smith ; B R Murphy ; B. MossConstruction and characterization of an infectious vaccinia virus recombinant that expresses the influenza hemagglutinin gene and induces resistance to influenza virus infection in hamsters.
000614 (????) B R Murphy [États-Unis] ; E J Park ; P. Gottlieb ; K. SubbaraoAn influenza A live attenuated reassortant virus possessing three temperature-sensitive mutations in the PB2 polymerase gene rapidly loses temperature sensitivity following replication in hamsters.

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