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Influenza type A in humans, mammals and birds: Determinants of virus virulence, host‐range and interspecies transmission

Identifieur interne : 001724 ( Main/Exploration ); précédent : 001723; suivant : 001725

Influenza type A in humans, mammals and birds: Determinants of virus virulence, host‐range and interspecies transmission

Auteurs : Susan J. Baigent [Royaume-Uni] ; John W. Mccauley [Royaume-Uni]

Source :

RBID : ISTEX:DB449994C91F97C5B10D2CF0D58DC90131860E0E

English descriptors

Abstract

The virulence of a virus is determined by its ability to adversely affect the host cell, host organism or population of host organisms. Influenza A viruses have been responsible for four pandemics of severe human respiratory disease this century. Avian species harbour a large reservoir of influenza virus strains, which can contribute genes to potential new pandemic human strains. The fundamental importance of understanding the role of each of these genes in determining virulence in birds and humans was dramatically emphasised by the recent direct transmission of avian influenza A viruses to humans, causing fatal infection but not community spread. An understanding of the factors involved in transmission between avian and mammalian species should assist in the development of better surveillance strategies for early recognition of influenza A virus strains having human pandemic potential, and possibly in the design of anti‐viral strategies. BioEssays 25:657–671, 2003. © 2003 Wiley Periodicals, Inc.

Url:
DOI: 10.1002/bies.10303


Affiliations:


Links toward previous steps (curation, corpus...)


Le document en format XML

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<term>Amino acid</term>
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<term>Avian viruses</term>
<term>Better surveillance strategies</term>
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<term>Cell nuclei virion</term>
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<term>Cytokine</term>
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<term>Diagram summarises</term>
<term>Duck intestine</term>
<term>Early recognition</term>
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<term>Fatal disease</term>
<term>Functional compatibility</term>
<term>Gene</term>
<term>Genetic analysis</term>
<term>Glycosylation</term>
<term>Growth factor</term>
<term>Haemagglutinin</term>
<term>High levels</term>
<term>High pathogenicity</term>
<term>Hong kong</term>
<term>Hong kong poultry markets</term>
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<term>Host cell membranes</term>
<term>Host cells</term>
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<term>Host organism</term>
<term>Host organisms</term>
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<term>Host range restriction</term>
<term>Host species</term>
<term>Host specificity</term>
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<term>Human disease</term>
<term>Human influenza</term>
<term>Human influenza viruses</term>
<term>Human pandemic</term>
<term>Human population</term>
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<term>Influenza</term>
<term>Influenza virus</term>
<term>Influenza virus haemagglutinin</term>
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<term>Influenza viruses</term>
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<term>Internal genes</term>
<term>Interspecies transmission</term>
<term>Kawaoka</term>
<term>Ketosidic linkage</term>
<term>Krauss</term>
<term>Large reservoir</term>
<term>Life cycle</term>
<term>Lineage</term>
<term>Linkage type</term>
<term>Major determinant</term>
<term>Major histocompatibility</term>
<term>Major pandemics</term>
<term>Mammal</term>
<term>Mammalian influenza viruses</term>
<term>Mammalian virus</term>
<term>Matrix</term>
<term>Matrix protein</term>
<term>Molecular basis</term>
<term>Multibasic cleavage site</term>
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<term>Review articles</term>
<term>Review articles figure</term>
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<term>Seroepidemiologic data</term>
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<term>Sialic acids</term>
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<term>Squirrel monkeys</term>
<term>Substrate specificity</term>
<term>Subtype viruses</term>
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<term>Target cells</term>
<term>Tissue culture</term>
<term>Tissue tropism</term>
<term>Transcription factors</term>
<term>Transmembrane span</term>
<term>Tumour necrosis factor</term>
<term>Vaccine</term>
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<div type="abstract" xml:lang="en">The virulence of a virus is determined by its ability to adversely affect the host cell, host organism or population of host organisms. Influenza A viruses have been responsible for four pandemics of severe human respiratory disease this century. Avian species harbour a large reservoir of influenza virus strains, which can contribute genes to potential new pandemic human strains. The fundamental importance of understanding the role of each of these genes in determining virulence in birds and humans was dramatically emphasised by the recent direct transmission of avian influenza A viruses to humans, causing fatal infection but not community spread. An understanding of the factors involved in transmission between avian and mammalian species should assist in the development of better surveillance strategies for early recognition of influenza A virus strains having human pandemic potential, and possibly in the design of anti‐viral strategies. BioEssays 25:657–671, 2003. © 2003 Wiley Periodicals, Inc.</div>
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