Systemic bone diseases and reduction of the residual ridge of the mandible; primary hyperparathyroidism. A preliminary report.
Identifieur interne : 004C21 ( PubMed/Corpus ); précédent : 004C20; suivant : 004C22Systemic bone diseases and reduction of the residual ridge of the mandible; primary hyperparathyroidism. A preliminary report.
Auteurs : C. LekkasSource :
- The Journal of prosthetic dentistry [ 0022-3913 ] ; 1989.
English descriptors
- KwdEn :
- MESH :
- chemical , blood : Parathyroid Hormone.
- blood : Hyperparathyroidism.
- complications : Hyperparathyroidism.
- etiology : Bone Diseases, Metabolic, Mandibular Diseases.
- pathology : Jaw, Edentulous, Mandible.
- Atrophy, Female, Humans, Male, Vertical Dimension.
Abstract
Twenty-five edentulous patients with established hyperparathyroidism were evaluated to establish a possible relation between extreme reduction of the height of the mandible and systemic bone loss. As an important parameter of the severity of the systemic bone loss, the level of the intact biologically active molecule of the parathormone was used. In this study no relation between hyperparathyroidism and reduction of the residual alveolar ridge could be established. In only two patients the reduction could be classified as "severe." Furthermore the level of the active, intact, circulating parathormone or the duration of the hyperparathyroidism could not be identified as an important factor in relation to the local phenomenon of reduction of the alveolar ridges.
PubMed: 2607479
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pubmed:2607479Le document en format XML
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A preliminary report.</title><author><name sortKey="Lekkas, C" sort="Lekkas, C" uniqKey="Lekkas C" first="C" last="Lekkas">C. Lekkas</name><affiliation><nlm:affiliation>University of Leiden, The Netherlands.</nlm:affiliation></affiliation></author></analytic><series><title level="j">The Journal of prosthetic dentistry</title><idno type="ISSN">0022-3913</idno><imprint><date when="1989" type="published">1989</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Atrophy</term><term>Bone Diseases, Metabolic (etiology)</term><term>Female</term><term>Humans</term><term>Hyperparathyroidism (blood)</term><term>Hyperparathyroidism (complications)</term><term>Jaw, Edentulous (pathology)</term><term>Male</term><term>Mandible (pathology)</term><term>Mandibular Diseases (etiology)</term><term>Parathyroid Hormone (blood)</term><term>Vertical Dimension</term></keywords><keywords scheme="MESH" type="chemical" qualifier="blood" xml:lang="en"><term>Parathyroid Hormone</term></keywords><keywords scheme="MESH" qualifier="blood" xml:lang="en"><term>Hyperparathyroidism</term></keywords><keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Hyperparathyroidism</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Bone Diseases, Metabolic</term><term>Mandibular Diseases</term></keywords><keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Jaw, Edentulous</term><term>Mandible</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Atrophy</term><term>Female</term><term>Humans</term><term>Male</term><term>Vertical Dimension</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Twenty-five edentulous patients with established hyperparathyroidism were evaluated to establish a possible relation between extreme reduction of the height of the mandible and systemic bone loss. As an important parameter of the severity of the systemic bone loss, the level of the intact biologically active molecule of the parathormone was used. In this study no relation between hyperparathyroidism and reduction of the residual alveolar ridge could be established. In only two patients the reduction could be classified as "severe." Furthermore the level of the active, intact, circulating parathormone or the duration of the hyperparathyroidism could not be identified as an important factor in relation to the local phenomenon of reduction of the alveolar ridges.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">2607479</PMID><DateCompleted><Year>1990</Year><Month>02</Month><Day>13</Day></DateCompleted><DateRevised><Year>2004</Year><Month>11</Month><Day>17</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0022-3913</ISSN><JournalIssue CitedMedium="Print"><Volume>62</Volume><Issue>5</Issue><PubDate><Year>1989</Year><Month>Nov</Month></PubDate></JournalIssue><Title>The Journal of prosthetic dentistry</Title><ISOAbbreviation>J Prosthet Dent</ISOAbbreviation></Journal><ArticleTitle>Systemic bone diseases and reduction of the residual ridge of the mandible; primary hyperparathyroidism. A preliminary report.</ArticleTitle><Pagination><MedlinePgn>546-50</MedlinePgn></Pagination><Abstract><AbstractText>Twenty-five edentulous patients with established hyperparathyroidism were evaluated to establish a possible relation between extreme reduction of the height of the mandible and systemic bone loss. As an important parameter of the severity of the systemic bone loss, the level of the intact biologically active molecule of the parathormone was used. In this study no relation between hyperparathyroidism and reduction of the residual alveolar ridge could be established. In only two patients the reduction could be classified as "severe." Furthermore the level of the active, intact, circulating parathormone or the duration of the hyperparathyroidism could not be identified as an important factor in relation to the local phenomenon of reduction of the alveolar ridges.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Lekkas</LastName><ForeName>C</ForeName><Initials>C</Initials><AffiliationInfo><Affiliation>University of Leiden, The Netherlands.</Affiliation></AffiliationInfo></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>United States</Country><MedlineTA>J Prosthet Dent</MedlineTA><NlmUniqueID>0376364</NlmUniqueID><ISSNLinking>0022-3913</ISSNLinking></MedlineJournalInfo><ChemicalList><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D010281">Parathyroid Hormone</NameOfSubstance></Chemical></ChemicalList><CitationSubset>D</CitationSubset><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D001284" MajorTopicYN="N">Atrophy</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D001851" MajorTopicYN="N">Bone Diseases, Metabolic</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D006961" MajorTopicYN="N">Hyperparathyroidism</DescriptorName><QualifierName UI="Q000097" MajorTopicYN="N">blood</QualifierName><QualifierName UI="Q000150" MajorTopicYN="Y">complications</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D007575" MajorTopicYN="Y">Jaw, Edentulous</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="N">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008334" MajorTopicYN="N">Mandible</DescriptorName><QualifierName UI="Q000473" MajorTopicYN="N">pathology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008336" MajorTopicYN="N">Mandibular Diseases</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D010281" MajorTopicYN="N">Parathyroid Hormone</DescriptorName><QualifierName UI="Q000097" MajorTopicYN="N">blood</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D014716" MajorTopicYN="N">Vertical Dimension</DescriptorName></MeshHeading></MeshHeadingList></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>1989</Year><Month>11</Month><Day>1</Day></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>1989</Year><Month>11</Month><Day>1</Day><Hour>0</Hour><Minute>1</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>1989</Year><Month>11</Month><Day>1</Day><Hour>0</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">2607479</ArticleId></ArticleIdList></PubmedData></pubmed></record>Pour manipuler ce document sous Unix (Dilib)
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