The soft tissue response to osseointegrated dental implants.
Identifieur interne : 003C48 ( PubMed/Corpus ); précédent : 003C47; suivant : 003C49The soft tissue response to osseointegrated dental implants.
Auteurs : H P Weber ; D L CochranSource :
- The Journal of prosthetic dentistry [ 0022-3913 ] ; 1998.
English descriptors
- KwdEn :
- Alveolar Bone Loss (etiology), Animals, Bacteria (growth & development), Biomechanical Phenomena, Collagen, Connective Tissue (anatomy & histology), Connective Tissue (physiology), Dental Implantation, Endosseous (adverse effects), Dental Implants (adverse effects), Dental Plaque (complications), Dental Plaque (microbiology), Dental Restoration Failure, Epithelial Attachment (anatomy & histology), Epithelial Attachment (physiology), Epithelium (anatomy & histology), Epithelium (physiology), Follow-Up Studies, Gingiva (anatomy & histology), Gingiva (physiology), Humans, Jaw (pathology), Jaw (physiopathology), Mouth Mucosa (anatomy & histology), Mouth Mucosa (microbiology), Mouth Mucosa (physiology), Mouth, Edentulous (rehabilitation), Mouth, Edentulous (surgery), Periodontal Diseases (etiology), Periodontal Diseases (microbiology), Periodontium (anatomy & histology), Periodontium (microbiology), Periodontium (physiology), Treatment Outcome, Wound Healing.
- MESH :
- chemical , adverse effects : Dental Implants.
- chemical : Collagen.
- adverse effects : Dental Implantation, Endosseous.
- anatomy & histology : Connective Tissue, Epithelial Attachment, Epithelium, Gingiva, Mouth Mucosa, Periodontium.
- complications : Dental Plaque.
- etiology : Alveolar Bone Loss, Periodontal Diseases.
- growth & development : Bacteria.
- microbiology : Dental Plaque, Mouth Mucosa, Periodontal Diseases, Periodontium.
- pathology : Jaw.
- physiology : Connective Tissue, Epithelial Attachment, Epithelium, Gingiva, Mouth Mucosa, Periodontium.
- physiopathology : Jaw.
- rehabilitation : Mouth, Edentulous.
- surgery : Mouth, Edentulous.
- Animals, Biomechanical Phenomena, Dental Restoration Failure, Follow-Up Studies, Humans, Treatment Outcome, Wound Healing.
Abstract
The use of dental implants in the treatment of fully edentulous patients has become an important addition in oral/dental rehabilitation. The fact that these implants penetrate the oral mucosa can lead to the assumption that peri-implant tissues, similar to the periodontal tissues, are fulfilling an important function as a barrier to protect the bony anchorage underneath. It has been shown that insufficient plaque removal may lead to peri-implant tissue disease with bone loss similar to teeth. However, it is unclear how important this cause is as a source of implant failure compared with other factors, such as inadequate bone healing, unfavorable quantity and quality of bone, or (bio)mechanical and functional problems. It is also not understood if peri-implant epithelium and connective tissue are equally needed and/or qualified to slow down or prevent tissue breakdown as their periodontal counterparts. The scientific work focusing on peri-implant soft tissues has dramatically increased in the past few years. Most studies to date have examined and described their structure but little data exist on their true biologic function. This review analyzes the current understanding of morphologic and clinical features of the peri-implant soft tissues. Furthermore, evidence shall be provided that peri-implant soft tissues do not interfere with the current favorable results obtained when treating the edentulous patient with osseointegrated implants.
PubMed: 9474546
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pubmed:9474546Le document en format XML
<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">The soft tissue response to osseointegrated dental implants.</title>
<author><name sortKey="Weber, H P" sort="Weber, H P" uniqKey="Weber H" first="H P" last="Weber">H P Weber</name>
<affiliation><nlm:affiliation>Department of Restorative Dentistry, Harvard School of Dental Medicine, Boston, Mass., USA.</nlm:affiliation>
</affiliation>
</author>
<author><name sortKey="Cochran, D L" sort="Cochran, D L" uniqKey="Cochran D" first="D L" last="Cochran">D L Cochran</name>
</author>
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<sourceDesc><biblStruct><analytic><title xml:lang="en">The soft tissue response to osseointegrated dental implants.</title>
<author><name sortKey="Weber, H P" sort="Weber, H P" uniqKey="Weber H" first="H P" last="Weber">H P Weber</name>
<affiliation><nlm:affiliation>Department of Restorative Dentistry, Harvard School of Dental Medicine, Boston, Mass., USA.</nlm:affiliation>
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<author><name sortKey="Cochran, D L" sort="Cochran, D L" uniqKey="Cochran D" first="D L" last="Cochran">D L Cochran</name>
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<series><title level="j">The Journal of prosthetic dentistry</title>
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Alveolar Bone Loss (etiology)</term>
<term>Animals</term>
<term>Bacteria (growth & development)</term>
<term>Biomechanical Phenomena</term>
<term>Collagen</term>
<term>Connective Tissue (anatomy & histology)</term>
<term>Connective Tissue (physiology)</term>
<term>Dental Implantation, Endosseous (adverse effects)</term>
<term>Dental Implants (adverse effects)</term>
<term>Dental Plaque (complications)</term>
<term>Dental Plaque (microbiology)</term>
<term>Dental Restoration Failure</term>
<term>Epithelial Attachment (anatomy & histology)</term>
<term>Epithelial Attachment (physiology)</term>
<term>Epithelium (anatomy & histology)</term>
<term>Epithelium (physiology)</term>
<term>Follow-Up Studies</term>
<term>Gingiva (anatomy & histology)</term>
<term>Gingiva (physiology)</term>
<term>Humans</term>
<term>Jaw (pathology)</term>
<term>Jaw (physiopathology)</term>
<term>Mouth Mucosa (anatomy & histology)</term>
<term>Mouth Mucosa (microbiology)</term>
<term>Mouth Mucosa (physiology)</term>
<term>Mouth, Edentulous (rehabilitation)</term>
<term>Mouth, Edentulous (surgery)</term>
<term>Periodontal Diseases (etiology)</term>
<term>Periodontal Diseases (microbiology)</term>
<term>Periodontium (anatomy & histology)</term>
<term>Periodontium (microbiology)</term>
<term>Periodontium (physiology)</term>
<term>Treatment Outcome</term>
<term>Wound Healing</term>
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<keywords scheme="MESH" type="chemical" qualifier="adverse effects" xml:lang="en"><term>Dental Implants</term>
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<keywords scheme="MESH" qualifier="adverse effects" xml:lang="en"><term>Dental Implantation, Endosseous</term>
</keywords>
<keywords scheme="MESH" qualifier="anatomy & histology" xml:lang="en"><term>Connective Tissue</term>
<term>Epithelial Attachment</term>
<term>Epithelium</term>
<term>Gingiva</term>
<term>Mouth Mucosa</term>
<term>Periodontium</term>
</keywords>
<keywords scheme="MESH" qualifier="complications" xml:lang="en"><term>Dental Plaque</term>
</keywords>
<keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Alveolar Bone Loss</term>
<term>Periodontal Diseases</term>
</keywords>
<keywords scheme="MESH" qualifier="growth & development" xml:lang="en"><term>Bacteria</term>
</keywords>
<keywords scheme="MESH" qualifier="microbiology" xml:lang="en"><term>Dental Plaque</term>
<term>Mouth Mucosa</term>
<term>Periodontal Diseases</term>
<term>Periodontium</term>
</keywords>
<keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Jaw</term>
</keywords>
<keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>Connective Tissue</term>
<term>Epithelial Attachment</term>
<term>Epithelium</term>
<term>Gingiva</term>
<term>Mouth Mucosa</term>
<term>Periodontium</term>
</keywords>
<keywords scheme="MESH" qualifier="physiopathology" xml:lang="en"><term>Jaw</term>
</keywords>
<keywords scheme="MESH" qualifier="rehabilitation" xml:lang="en"><term>Mouth, Edentulous</term>
</keywords>
<keywords scheme="MESH" qualifier="surgery" xml:lang="en"><term>Mouth, Edentulous</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Animals</term>
<term>Biomechanical Phenomena</term>
<term>Dental Restoration Failure</term>
<term>Follow-Up Studies</term>
<term>Humans</term>
<term>Treatment Outcome</term>
<term>Wound Healing</term>
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<front><div type="abstract" xml:lang="en">The use of dental implants in the treatment of fully edentulous patients has become an important addition in oral/dental rehabilitation. The fact that these implants penetrate the oral mucosa can lead to the assumption that peri-implant tissues, similar to the periodontal tissues, are fulfilling an important function as a barrier to protect the bony anchorage underneath. It has been shown that insufficient plaque removal may lead to peri-implant tissue disease with bone loss similar to teeth. However, it is unclear how important this cause is as a source of implant failure compared with other factors, such as inadequate bone healing, unfavorable quantity and quality of bone, or (bio)mechanical and functional problems. It is also not understood if peri-implant epithelium and connective tissue are equally needed and/or qualified to slow down or prevent tissue breakdown as their periodontal counterparts. The scientific work focusing on peri-implant soft tissues has dramatically increased in the past few years. Most studies to date have examined and described their structure but little data exist on their true biologic function. This review analyzes the current understanding of morphologic and clinical features of the peri-implant soft tissues. Furthermore, evidence shall be provided that peri-implant soft tissues do not interfere with the current favorable results obtained when treating the edentulous patient with osseointegrated implants.</div>
</front>
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<Title>The Journal of prosthetic dentistry</Title>
<ISOAbbreviation>J Prosthet Dent</ISOAbbreviation>
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<ArticleTitle>The soft tissue response to osseointegrated dental implants.</ArticleTitle>
<Pagination><MedlinePgn>79-89</MedlinePgn>
</Pagination>
<Abstract><AbstractText>The use of dental implants in the treatment of fully edentulous patients has become an important addition in oral/dental rehabilitation. The fact that these implants penetrate the oral mucosa can lead to the assumption that peri-implant tissues, similar to the periodontal tissues, are fulfilling an important function as a barrier to protect the bony anchorage underneath. It has been shown that insufficient plaque removal may lead to peri-implant tissue disease with bone loss similar to teeth. However, it is unclear how important this cause is as a source of implant failure compared with other factors, such as inadequate bone healing, unfavorable quantity and quality of bone, or (bio)mechanical and functional problems. It is also not understood if peri-implant epithelium and connective tissue are equally needed and/or qualified to slow down or prevent tissue breakdown as their periodontal counterparts. The scientific work focusing on peri-implant soft tissues has dramatically increased in the past few years. Most studies to date have examined and described their structure but little data exist on their true biologic function. This review analyzes the current understanding of morphologic and clinical features of the peri-implant soft tissues. Furthermore, evidence shall be provided that peri-implant soft tissues do not interfere with the current favorable results obtained when treating the edentulous patient with osseointegrated implants.</AbstractText>
</Abstract>
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<QualifierName UI="Q000502" MajorTopicYN="Y">physiology</QualifierName>
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