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Clinical management for epidermolysis bullosa dystrophica.

Identifieur interne : 002252 ( PubMed/Corpus ); précédent : 002251; suivant : 002253

Clinical management for epidermolysis bullosa dystrophica.

Auteurs : Thais M. Oliveira ; Vivien T. Sakai ; Liliani A. Candido ; Salete M B. Silva ; Maria Aparecida A M. Machado

Source :

RBID : pubmed:19089295

English descriptors

Abstract

Epidermolysis bullosa (EB) consists of a group of genetic hereditary disorders in which patients frequently present fragile skin and mucosa that form blisters following minor trauma. More than 20 subtypes of EB have been recognized in the literature. Specific genetic mutations are well characterized for most the different EB subtypes and variants. The most common oral manifestations of EB are painful blisters affecting all the oral surfaces. Dental treatment for patients with EB consists of palliative therapy for its oral manifestations along with typical restorative and periodontal procedures. The aim of this article is to describe two dental clinical treatments of recessive dystrophic EB cases and their specific clinical manifestations. The psychological intervention required during the dental treatment of these patients is also presented.

PubMed: 19089295

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pubmed:19089295

Le document en format XML

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<name sortKey="Machado, Maria Aparecida A M" sort="Machado, Maria Aparecida A M" uniqKey="Machado M" first="Maria Aparecida A M" last="Machado">Maria Aparecida A M. Machado</name>
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<div type="abstract" xml:lang="en">Epidermolysis bullosa (EB) consists of a group of genetic hereditary disorders in which patients frequently present fragile skin and mucosa that form blisters following minor trauma. More than 20 subtypes of EB have been recognized in the literature. Specific genetic mutations are well characterized for most the different EB subtypes and variants. The most common oral manifestations of EB are painful blisters affecting all the oral surfaces. Dental treatment for patients with EB consists of palliative therapy for its oral manifestations along with typical restorative and periodontal procedures. The aim of this article is to describe two dental clinical treatments of recessive dystrophic EB cases and their specific clinical manifestations. The psychological intervention required during the dental treatment of these patients is also presented.</div>
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<RefSource>Oral Dis. 2003 Sep;9(5):264-8</RefSource>
<PMID Version="1">14628894</PMID>
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<RefSource>Spec Care Dentist. 1995 Jul-Aug;15(4):144-8</RefSource>
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<CommentsCorrections RefType="Cites">
<RefSource>Ann Chir Main Memb Super. 1997;16(2):91-100; discussion 101</RefSource>
<PMID Version="1">9259950</PMID>
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<CommentsCorrections RefType="Cites">
<RefSource>Expert Opin Biol Ther. 2006 Apr;6(4):367-78</RefSource>
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<RefSource>J Anesth. 2006;20(2):138-40</RefSource>
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<RefSource>Ann Plast Surg. 1999 Nov;43(5):555-9</RefSource>
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<CommentsCorrections RefType="Cites">
<RefSource>Pediatr Dermatol. 2006 Mar-Apr;23(2):167-8</RefSource>
<PMID Version="1">16650229</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites">
<RefSource>Pediatr Dent. 1994 Nov-Dec;16(6):427-32</RefSource>
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<RefSource>Pediatr Dent. 2001 Sep-Oct;23(5):438-43</RefSource>
<PMID Version="1">11699172</PMID>
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<CommentsCorrections RefType="Cites">
<RefSource>Pediatr Clin North Am. 2006 Apr;53(2):279-92</RefSource>
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<CommentsCorrections RefType="Cites">
<RefSource>J Wound Care. 2005 Nov;14(10):490-1, 494-6</RefSource>
<PMID Version="1">16304926</PMID>
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<CommentsCorrections RefType="Cites">
<RefSource>J Periodontol. 1999 Nov;70(11):1392-6</RefSource>
<PMID Version="1">10588504</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites">
<RefSource>Spec Care Dentist. 2004 Jan-Feb;24(1):22-7</RefSource>
<PMID Version="1">15157056</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites">
<RefSource>Oral Surg Oral Med Oral Pathol. 1993 Jan;75(1):54-7</RefSource>
<PMID Version="1">8419876</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites">
<RefSource>Spec Care Dentist. 1991 Jul-Aug;11(4):148-50</RefSource>
<PMID Version="1">1835172</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites">
<RefSource>Pediatr Dent. 2000 Sep-Oct;22(5):385-8</RefSource>
<PMID Version="1">11048306</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites">
<RefSource>Acta Dermatovenerol Alp Pannonica Adriat. 2005 Dec;14(4):127-35</RefSource>
<PMID Version="1">16435041</PMID>
</CommentsCorrections>
<CommentsCorrections RefType="Cites">
<RefSource>Arch Oral Biol. 1993 Nov;38(11):945-55</RefSource>
<PMID Version="1">8297258</PMID>
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<RefSource>J Med Genet. 2006 Aug;43(8):641-52</RefSource>
<PMID Version="1">16473856</PMID>
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<CommentsCorrections RefType="Cites">
<RefSource>J Clin Pediatr Dent. 2005 Fall;30(1):59-65</RefSource>
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<CommentsCorrections RefType="Cites">
<RefSource>Clin Dermatol. 2005 Jul-Aug;23(4):430-6</RefSource>
<PMID Version="1">16023940</PMID>
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<CommentsCorrections RefType="Cites">
<RefSource>Aust Dent J. 1997 Feb;42(1):1-7</RefSource>
<PMID Version="1">9078638</PMID>
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