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<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Influence of Helicobacter pylori, sex, and age on serum gastrin and pepsinogen concentrations in subjects without symptoms and patients with duodenal ulcers.</title>
<author><name sortKey="Mossi, S" sort="Mossi, S" uniqKey="Mossi S" first="S" last="Mossi">S. Mossi</name>
</author>
<author><name sortKey="Meyer Wyss, B" sort="Meyer Wyss, B" uniqKey="Meyer Wyss B" first="B" last="Meyer-Wyss">B. Meyer-Wyss</name>
</author>
<author><name sortKey="Renner, E L" sort="Renner, E L" uniqKey="Renner E" first="E L" last="Renner">E L Renner</name>
</author>
<author><name sortKey="Merki, H S" sort="Merki, H S" uniqKey="Merki H" first="H S" last="Merki">H S Merki</name>
</author>
<author><name sortKey="Gamboni, G" sort="Gamboni, G" uniqKey="Gamboni G" first="G" last="Gamboni">G. Gamboni</name>
</author>
<author><name sortKey="Beglinger, C" sort="Beglinger, C" uniqKey="Beglinger C" first="C" last="Beglinger">C. Beglinger</name>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">PMC</idno>
<idno type="pmid">8314506</idno>
<idno type="pmc">1374256</idno>
<idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1374256</idno>
<idno type="RBID">PMC:1374256</idno>
<date when="1993">1993</date>
<idno type="wicri:Area/Pmc/Corpus">001520</idno>
<idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Corpus" wicri:corpus="PMC">001520</idno>
</publicationStmt>
<sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">Influence of Helicobacter pylori, sex, and age on serum gastrin and pepsinogen concentrations in subjects without symptoms and patients with duodenal ulcers.</title>
<author><name sortKey="Mossi, S" sort="Mossi, S" uniqKey="Mossi S" first="S" last="Mossi">S. Mossi</name>
</author>
<author><name sortKey="Meyer Wyss, B" sort="Meyer Wyss, B" uniqKey="Meyer Wyss B" first="B" last="Meyer-Wyss">B. Meyer-Wyss</name>
</author>
<author><name sortKey="Renner, E L" sort="Renner, E L" uniqKey="Renner E" first="E L" last="Renner">E L Renner</name>
</author>
<author><name sortKey="Merki, H S" sort="Merki, H S" uniqKey="Merki H" first="H S" last="Merki">H S Merki</name>
</author>
<author><name sortKey="Gamboni, G" sort="Gamboni, G" uniqKey="Gamboni G" first="G" last="Gamboni">G. Gamboni</name>
</author>
<author><name sortKey="Beglinger, C" sort="Beglinger, C" uniqKey="Beglinger C" first="C" last="Beglinger">C. Beglinger</name>
</author>
</analytic>
<series><title level="j">Gut</title>
<idno type="ISSN">0017-5749</idno>
<idno type="eISSN">1458-3288</idno>
<imprint><date when="1993">1993</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc><textClass></textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en"><p>The relation between Helicobacter pylori (H pylori) infection and fasting gastrin and pepsinogen-I and -II concentrations was evaluated in 278 volunteers without symptoms and the results were compared with the values obtained in 35 patients with duodenal ulcers. H pylori infection was determined with the 13C-urea breath test in subjects without symptoms and with endoscopy, biopsy (histology and culture), and quick urease test (CLO-test) in patients with duodenal ulcers. Gastrin and pepsinogen-I and -II concentrations were assayed with specific radioimmunoassay systems. The results clearly indicate that fasting gastrin and pepsinogen-I and -II concentrations were significantly higher in H pylori positive compared with H pylori negative subjects. Neither age nor sex affected basal gastrin and pepsinogen concentrations in H pylori negative subjects. Fasting gastrin, pepsinogen-I and -II concentrations in serum samples were similar in H pylori positive persons with no symptoms and those with duodenal ulcers suggesting that similar mechanisms are involved in increasing plasma concentrations of these variables in both populations. Hypergastrinaemia and hyperpepsinogenaemia are therefore probably secondary to active H pylori infection.</p>
</div>
</front>
</TEI>
<pmc article-type="research-article"><pmc-comment>The publisher of this article does not allow downloading of the full text in XML form.</pmc-comment>
<front><journal-meta><journal-id journal-id-type="nlm-ta">Gut</journal-id>
<journal-title>Gut</journal-title>
<issn pub-type="ppub">0017-5749</issn>
<issn pub-type="epub">1458-3288</issn>
</journal-meta>
<article-meta><article-id pub-id-type="pmid">8314506</article-id>
<article-id pub-id-type="pmc">1374256</article-id>
<article-categories><subj-group subj-group-type="heading"><subject>Research Article</subject>
</subj-group>
</article-categories>
<title-group><article-title>Influence of Helicobacter pylori, sex, and age on serum gastrin and pepsinogen concentrations in subjects without symptoms and patients with duodenal ulcers.</article-title>
</title-group>
<contrib-group><contrib contrib-type="author"><name><surname>Mossi</surname>
<given-names>S</given-names>
</name>
</contrib>
<contrib contrib-type="author"><name><surname>Meyer-Wyss</surname>
<given-names>B</given-names>
</name>
</contrib>
<contrib contrib-type="author"><name><surname>Renner</surname>
<given-names>E L</given-names>
</name>
</contrib>
<contrib contrib-type="author"><name><surname>Merki</surname>
<given-names>H S</given-names>
</name>
</contrib>
<contrib contrib-type="author"><name><surname>Gamboni</surname>
<given-names>G</given-names>
</name>
</contrib>
<contrib contrib-type="author"><name><surname>Beglinger</surname>
<given-names>C</given-names>
</name>
</contrib>
</contrib-group>
<aff>Department of Research, University Hospital, Basel, Switzerland.</aff>
<pub-date pub-type="ppub"><month>6</month>
<year>1993</year>
</pub-date>
<volume>34</volume>
<issue>6</issue>
<fpage>752</fpage>
<lpage>756</lpage>
<abstract><p>The relation between Helicobacter pylori (H pylori) infection and fasting gastrin and pepsinogen-I and -II concentrations was evaluated in 278 volunteers without symptoms and the results were compared with the values obtained in 35 patients with duodenal ulcers. H pylori infection was determined with the 13C-urea breath test in subjects without symptoms and with endoscopy, biopsy (histology and culture), and quick urease test (CLO-test) in patients with duodenal ulcers. Gastrin and pepsinogen-I and -II concentrations were assayed with specific radioimmunoassay systems. The results clearly indicate that fasting gastrin and pepsinogen-I and -II concentrations were significantly higher in H pylori positive compared with H pylori negative subjects. Neither age nor sex affected basal gastrin and pepsinogen concentrations in H pylori negative subjects. Fasting gastrin, pepsinogen-I and -II concentrations in serum samples were similar in H pylori positive persons with no symptoms and those with duodenal ulcers suggesting that similar mechanisms are involved in increasing plasma concentrations of these variables in both populations. Hypergastrinaemia and hyperpepsinogenaemia are therefore probably secondary to active H pylori infection.</p>
</abstract>
</article-meta>
</front>
</pmc>
</record>
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