POSTOPERATIVE HYPOPHOSPHATEMIA: A MULTIFACTORIAL PROBLEM
Identifieur interne : 00C455 ( Main/Merge ); précédent : 00C454; suivant : 00C456POSTOPERATIVE HYPOPHOSPHATEMIA: A MULTIFACTORIAL PROBLEM
Auteurs :Source :
- Nutrition Reviews [ 0029-6643 ] ; 1989-04.
Descripteurs français
English descriptors
- KwdEn :
- Anabolic steroids, Antacid, Arch intern, Boston veterans administration, Chronic alcoholics, Diabetic ketoacidosis, Dietary deficiency, Diuretic therapy, Glucose, Hypophosphatemia, Inorganic phosphate, Intestinal absorption, Intracellular compartment, Intracellular shift, Metabolic acidosis, Multiple risk factors, Nasogastric suction, Normal limits, Nutrition reviewsivol, Nutrition rniewsivol, Nutritional support, Other causes, Parenteral nutrition, Phosphate, Phosphaturia, Phosphorus, Postoperative, Refeeding syndrome, Risk factors, Serum concentrations, Serum phosphorus, Severe hypophosphatemia, Severe malnutrition, Symptomatic hypophosphatemia, Syndrome, Total parenteral nutrition, Transcellular shift, Urinary excretion, Volume overload, Wound site.
- Teeft :
- Anabolic steroids, Antacid, Arch intern, Boston veterans administration, Chronic alcoholics, Diabetic ketoacidosis, Dietary deficiency, Diuretic therapy, Glucose, Hypophosphatemia, Inorganic phosphate, Intestinal absorption, Intracellular compartment, Intracellular shift, Metabolic acidosis, Multiple risk factors, Nasogastric suction, Normal limits, Nutrition reviewsivol, Nutrition rniewsivol, Nutritional support, Other causes, Parenteral nutrition, Phosphate, Phosphaturia, Phosphorus, Postoperative, Refeeding syndrome, Risk factors, Serum concentrations, Serum phosphorus, Severe hypophosphatemia, Severe malnutrition, Symptomatic hypophosphatemia, Syndrome, Total parenteral nutrition, Transcellular shift, Urinary excretion, Volume overload, Wound site.
Abstract
This case study from the Boston Veterans Administration Medical Center emphasizes the signs, pathogenesis, and importance of hypophosphatemia in the setting of postoperative intensive nutritional support. Multiple contributing factors, including preoperative nutritional depletion, alcoholism, antacid therapy, and intracellular shifts induced by intravenous administration of glucose solutions were all involved in the pathogenesis of hypophosphatemia. Other risk factors are identified; familiarity with these should alert physicians to look for this potentially fatal but easily treatable, iatrogenic nutritional disorder.
Url:
DOI: 10.1111/j.1753-4887.1989.tb02809.x
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ISTEX:592EAEAB0B3DD8665AEAC40BFFAAAC875835E3AELe document en format XML
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Anabolic steroids</term>
<term>Antacid</term>
<term>Arch intern</term>
<term>Boston veterans administration</term>
<term>Chronic alcoholics</term>
<term>Diabetic ketoacidosis</term>
<term>Dietary deficiency</term>
<term>Diuretic therapy</term>
<term>Glucose</term>
<term>Hypophosphatemia</term>
<term>Inorganic phosphate</term>
<term>Intestinal absorption</term>
<term>Intracellular compartment</term>
<term>Intracellular shift</term>
<term>Metabolic acidosis</term>
<term>Multiple risk factors</term>
<term>Nasogastric suction</term>
<term>Normal limits</term>
<term>Nutrition reviewsivol</term>
<term>Nutrition rniewsivol</term>
<term>Nutritional support</term>
<term>Other causes</term>
<term>Parenteral nutrition</term>
<term>Phosphate</term>
<term>Phosphaturia</term>
<term>Phosphorus</term>
<term>Postoperative</term>
<term>Refeeding syndrome</term>
<term>Risk factors</term>
<term>Serum concentrations</term>
<term>Serum phosphorus</term>
<term>Severe hypophosphatemia</term>
<term>Severe malnutrition</term>
<term>Symptomatic hypophosphatemia</term>
<term>Syndrome</term>
<term>Total parenteral nutrition</term>
<term>Transcellular shift</term>
<term>Urinary excretion</term>
<term>Volume overload</term>
<term>Wound site</term>
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<term>Antacid</term>
<term>Arch intern</term>
<term>Boston veterans administration</term>
<term>Chronic alcoholics</term>
<term>Diabetic ketoacidosis</term>
<term>Dietary deficiency</term>
<term>Diuretic therapy</term>
<term>Glucose</term>
<term>Hypophosphatemia</term>
<term>Inorganic phosphate</term>
<term>Intestinal absorption</term>
<term>Intracellular compartment</term>
<term>Intracellular shift</term>
<term>Metabolic acidosis</term>
<term>Multiple risk factors</term>
<term>Nasogastric suction</term>
<term>Normal limits</term>
<term>Nutrition reviewsivol</term>
<term>Nutrition rniewsivol</term>
<term>Nutritional support</term>
<term>Other causes</term>
<term>Parenteral nutrition</term>
<term>Phosphate</term>
<term>Phosphaturia</term>
<term>Phosphorus</term>
<term>Postoperative</term>
<term>Refeeding syndrome</term>
<term>Risk factors</term>
<term>Serum concentrations</term>
<term>Serum phosphorus</term>
<term>Severe hypophosphatemia</term>
<term>Severe malnutrition</term>
<term>Symptomatic hypophosphatemia</term>
<term>Syndrome</term>
<term>Total parenteral nutrition</term>
<term>Transcellular shift</term>
<term>Urinary excretion</term>
<term>Volume overload</term>
<term>Wound site</term>
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<term>Phosphate</term>
<term>Phosphore</term>
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<front><div type="abstract" xml:lang="en">This case study from the Boston Veterans Administration Medical Center emphasizes the signs, pathogenesis, and importance of hypophosphatemia in the setting of postoperative intensive nutritional support. Multiple contributing factors, including preoperative nutritional depletion, alcoholism, antacid therapy, and intracellular shifts induced by intravenous administration of glucose solutions were all involved in the pathogenesis of hypophosphatemia. Other risk factors are identified; familiarity with these should alert physicians to look for this potentially fatal but easily treatable, iatrogenic nutritional disorder.</div>
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