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A case of acanthosis nigricans—an investigative approach

Identifieur interne : 006C48 ( Istex/Curation ); précédent : 006C47; suivant : 006C49

A case of acanthosis nigricans—an investigative approach

Auteurs : Shernaz Walton ; Lionel Krause ; Janet Marks

Source :

Clinical and Experimental Dermatology [ 0307-6938 ] ; 1984-01.

RBID : ISTEX:DAA2873466C93688F3EA12A84A24095EE5830952

Descripteurs français

Wicri :
- topic : Glucose.

English descriptors

KwdEn :
- Abdominal pain, Abnormality, Acanthosis, Acanthosis nigricans, Adenocarcinoma, Biochemical analysis, Birth rate, Cadaverine, Clinical biochemistry, Dermatology, Epidermal, Epidermal growth factor, Epidermal proliferation, Experimental dermatology, Glucagon, Glucose, Glucose load, Growth hormone, Histological, Histological appearance, Histological examination, Infirmary, Insulin, Insulin receptors, Insulin resistance, Lymph nodes, Malignant, Malignant disease, Newcastle, Nigricans, Oesophagus, Peptide, Receptor, Royal victoria infirmary, Seborrhoeic warts, Spermine, Tumour, Tyne, University department, Warty, Warty appearance, Warty skin.
Teeft :
- Abdominal pain, Abnormality, Acanthosis, Acanthosis nigricans, Adenocarcinoma, Biochemical analysis, Birth rate, Cadaverine, Clinical biochemistry, Dermatology, Epidermal, Epidermal growth factor, Epidermal proliferation, Experimental dermatology, Glucagon, Glucose, Glucose load, Growth hormone, Histological, Histological appearance, Histological examination, Infirmary, Insulin, Insulin receptors, Insulin resistance, Lymph nodes, Malignant, Malignant disease, Newcastle, Nigricans, Oesophagus, Peptide, Receptor, Royal victoria infirmary, Seborrhoeic warts, Spermine, Tumour, Tyne, University department, Warty, Warty appearance, Warty skin.

Abstract

A patient with acanthosis nigricans secondary to a metastatic jejunal adenocarcinoma is described. An attempt was made to define the basic epidermal defect and to examine such possible influences as somatotrophin, epidermal growth factor, intestinal peptides and insulin receptors. No evidence to support a hyperproliferative epidermal mechanism was found and the hormonal profile was normal. However, there was an increase in circulating α‐MSH and this may have contributed to the pigmentation seen in this patient.

Url:

https://api.istex.fr/document/DAA2873466C93688F3EA12A84A24095EE5830952/fulltext/pdf

DOI: 10.1111/j.1365-2230.1984.tb00756.x

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Shernaz Walton

<affiliation><mods:affiliation>Correspondence address: Dr Shernaz Walton, University Department of Dermatology, Royal Victoria Infirmary, Newcastle upon Tyne NE1 4LP.</mods:affiliation>
<wicri:noCountry code="subField">4LP.</wicri:noCountry>
</affiliation>

Lionel Krause

<affiliation><mods:affiliation>University Department of Dermatology, Royal Victoria Infirmary, Newcastle upon Tyne</mods:affiliation>
<wicri:noCountry code="subField">Tyne</wicri:noCountry>
</affiliation>

Janet Marks

<affiliation><mods:affiliation>University Department of Dermatology, Royal Victoria Infirmary, Newcastle upon Tyne</mods:affiliation>
<wicri:noCountry code="subField">Tyne</wicri:noCountry>
</affiliation>

Le document en format XML

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<term>Acanthosis nigricans</term>
<term>Adenocarcinoma</term>
<term>Biochemical analysis</term>
<term>Birth rate</term>
<term>Cadaverine</term>
<term>Clinical biochemistry</term>
<term>Dermatology</term>
<term>Epidermal</term>
<term>Epidermal growth factor</term>
<term>Epidermal proliferation</term>
<term>Experimental dermatology</term>
<term>Glucagon</term>
<term>Glucose</term>
<term>Glucose load</term>
<term>Growth hormone</term>
<term>Histological</term>
<term>Histological appearance</term>
<term>Histological examination</term>
<term>Infirmary</term>
<term>Insulin</term>
<term>Insulin receptors</term>
<term>Insulin resistance</term>
<term>Lymph nodes</term>
<term>Malignant</term>
<term>Malignant disease</term>
<term>Newcastle</term>
<term>Nigricans</term>
<term>Oesophagus</term>
<term>Peptide</term>
<term>Receptor</term>
<term>Royal victoria infirmary</term>
<term>Seborrhoeic warts</term>
<term>Spermine</term>
<term>Tumour</term>
<term>Tyne</term>
<term>University department</term>
<term>Warty</term>
<term>Warty appearance</term>
<term>Warty skin</term>
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<term>Abnormality</term>
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<term>Adenocarcinoma</term>
<term>Biochemical analysis</term>
<term>Birth rate</term>
<term>Cadaverine</term>
<term>Clinical biochemistry</term>
<term>Dermatology</term>
<term>Epidermal</term>
<term>Epidermal growth factor</term>
<term>Epidermal proliferation</term>
<term>Experimental dermatology</term>
<term>Glucagon</term>
<term>Glucose</term>
<term>Glucose load</term>
<term>Growth hormone</term>
<term>Histological</term>
<term>Histological appearance</term>
<term>Histological examination</term>
<term>Infirmary</term>
<term>Insulin</term>
<term>Insulin receptors</term>
<term>Insulin resistance</term>
<term>Lymph nodes</term>
<term>Malignant</term>
<term>Malignant disease</term>
<term>Newcastle</term>
<term>Nigricans</term>
<term>Oesophagus</term>
<term>Peptide</term>
<term>Receptor</term>
<term>Royal victoria infirmary</term>
<term>Seborrhoeic warts</term>
<term>Spermine</term>
<term>Tumour</term>
<term>Tyne</term>
<term>University department</term>
<term>Warty</term>
<term>Warty appearance</term>
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<front><div type="abstract" xml:lang="en">A patient with acanthosis nigricans secondary to a metastatic jejunal adenocarcinoma is described. An attempt was made to define the basic epidermal defect and to examine such possible influences as somatotrophin, epidermal growth factor, intestinal peptides and insulin receptors. No evidence to support a hyperproliferative epidermal mechanism was found and the hormonal profile was normal. However, there was an increase in circulating α‐MSH and this may have contributed to the pigmentation seen in this patient.</div>
</front>
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A case of acanthosis nigricans—an investigative approach

A case of acanthosis nigricans—an investigative approach

Source :

Descripteurs français

English descriptors

Abstract

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