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Idiopathic Headache as a Possible Risk Factor for Phantom Tooth Pain

Identifieur interne : 003F01 ( Istex/Curation ); précédent : 003F00; suivant : 003F02

Idiopathic Headache as a Possible Risk Factor for Phantom Tooth Pain

Auteurs : Federigo Sicuteri ; Maria Nicolodi ; Bruno Marcello Fusco ; Salvatore Orlando [Italie]

Source :

RBID : ISTEX:7F2A07124261735420822F1A3305643F4A0BB5A3

English descriptors

Abstract

SYNOPSIS Following tooth pulp extirpation, some subjects suffer from persistent pain which affects edentate sitesin absence of any local pathology. As regards this peculiar pain, called phantom tooth pain (PTP), what ispuzzling is the fact there is a low prevalence of PTP in a very large population showing identical conditionsof tooth pulp extirpation. The present investigation indicates that PTP mainly affects migraine (M) andcluster headache (CH) sufferers, whereas it does not affect subjects who have a negative personal andfamily history for idiopathic headache (IH). These results circumscribe the presence of PTP to a specificsection of the population. The present results, besides indicating that PTP may be the result of a peculiarneuronal predisposition relating to IH pathogenesis, suggests some practical therapeutic hints. In fact,successful anti‐ M and anti‐CH prophylactic treatment greatly improve PTP syndrome.

Url:
DOI: 10.1111/j.1526-4610.1991.hed3109577.x

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Federigo Sicuteri
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Maria Nicolodi
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Bruno Marcello Fusco
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<term>Pharmacological aspects</term>
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<term>Risk factor</term>
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<div type="abstract" xml:lang="en">SYNOPSIS Following tooth pulp extirpation, some subjects suffer from persistent pain which affects edentate sitesin absence of any local pathology. As regards this peculiar pain, called phantom tooth pain (PTP), what ispuzzling is the fact there is a low prevalence of PTP in a very large population showing identical conditionsof tooth pulp extirpation. The present investigation indicates that PTP mainly affects migraine (M) andcluster headache (CH) sufferers, whereas it does not affect subjects who have a negative personal andfamily history for idiopathic headache (IH). These results circumscribe the presence of PTP to a specificsection of the population. The present results, besides indicating that PTP may be the result of a peculiarneuronal predisposition relating to IH pathogenesis, suggests some practical therapeutic hints. In fact,successful anti‐ M and anti‐CH prophylactic treatment greatly improve PTP syndrome.</div>
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