Idiopathic Headache as a Possible Risk Factor for Phantom Tooth Pain
Identifieur interne : 003F01 ( Istex/Curation ); précédent : 003F00; suivant : 003F02Idiopathic Headache as a Possible Risk Factor for Phantom Tooth Pain
Auteurs : Federigo Sicuteri ; Maria Nicolodi ; Bruno Marcello Fusco ; Salvatore Orlando [Italie]Source :
- Headache: The Journal of Head and Face Pain [ 0017-8748 ] ; 1991-10.
English descriptors
- KwdEn :
- Atypical odontalgia, Cluster headache, Control group, Control subjects, Deafferentation, Dental avulsion, Dental extractions, Edentate, Edentate sites, Excerpta medica, Family history, Federigo sicuteri, Functional deafferentation, Headache, Idiopathic headache, Internal medicine, Limb pain, Migraine, Oral cavity, Oral surg, Pain transmission, Persistent pain, Phantom, Phantom pain, Phantom tooth pain, Pharmacological aspects, Present investigation, Primary trigeminal axons, Prophylactic treatment, Psychological factors, Risk factor, Sicuteri, Significant difference, Sufferer, Syndrome, Tooth extractions, Tooth pulp extirpation, Tract nucleus, Trigeminal, Trigeminal pain.
- Teeft :
- Atypical odontalgia, Cluster headache, Control group, Control subjects, Deafferentation, Dental avulsion, Dental extractions, Edentate, Edentate sites, Excerpta medica, Family history, Federigo sicuteri, Functional deafferentation, Headache, Idiopathic headache, Internal medicine, Limb pain, Migraine, Oral cavity, Oral surg, Pain transmission, Persistent pain, Phantom, Phantom pain, Phantom tooth pain, Pharmacological aspects, Present investigation, Primary trigeminal axons, Prophylactic treatment, Psychological factors, Risk factor, Sicuteri, Significant difference, Sufferer, Syndrome, Tooth extractions, Tooth pulp extirpation, Tract nucleus, Trigeminal, Trigeminal pain.
Abstract
SYNOPSIS Following tooth pulp extirpation, some subjects suffer from persistent pain which affects edentate sitesin absence of any local pathology. As regards this peculiar pain, called phantom tooth pain (PTP), what ispuzzling is the fact there is a low prevalence of PTP in a very large population showing identical conditionsof tooth pulp extirpation. The present investigation indicates that PTP mainly affects migraine (M) andcluster headache (CH) sufferers, whereas it does not affect subjects who have a negative personal andfamily history for idiopathic headache (IH). These results circumscribe the presence of PTP to a specificsection of the population. The present results, besides indicating that PTP may be the result of a peculiarneuronal predisposition relating to IH pathogenesis, suggests some practical therapeutic hints. In fact,successful anti‐ M and anti‐CH prophylactic treatment greatly improve PTP syndrome.
Url:
DOI: 10.1111/j.1526-4610.1991.hed3109577.x
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Federigo Sicuteri<affiliation><mods:affiliation>Institute of Internal Medicine and Therapeutics IV.</mods:affiliation>
<wicri:noCountry code="no comma">Institute of Internal Medicine and Therapeutics IV.</wicri:noCountry>
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<affiliation><mods:affiliation>Institute of Internal Medicine and Therapeutics IV.</mods:affiliation>
<wicri:noCountry code="no comma">Institute of Internal Medicine and Therapeutics IV.</wicri:noCountry>
</affiliation>
<affiliation><mods:affiliation>Institute of Internal Medicine and Therapeutics IV.</mods:affiliation>
<wicri:noCountry code="no comma">Institute of Internal Medicine and Therapeutics IV.</wicri:noCountry>
</affiliation>
Le document en format XML
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<term>Control group</term>
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<term>Deafferentation</term>
<term>Dental avulsion</term>
<term>Dental extractions</term>
<term>Edentate</term>
<term>Edentate sites</term>
<term>Excerpta medica</term>
<term>Family history</term>
<term>Federigo sicuteri</term>
<term>Functional deafferentation</term>
<term>Headache</term>
<term>Idiopathic headache</term>
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<term>Migraine</term>
<term>Oral cavity</term>
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<term>Persistent pain</term>
<term>Phantom</term>
<term>Phantom pain</term>
<term>Phantom tooth pain</term>
<term>Pharmacological aspects</term>
<term>Present investigation</term>
<term>Primary trigeminal axons</term>
<term>Prophylactic treatment</term>
<term>Psychological factors</term>
<term>Risk factor</term>
<term>Sicuteri</term>
<term>Significant difference</term>
<term>Sufferer</term>
<term>Syndrome</term>
<term>Tooth extractions</term>
<term>Tooth pulp extirpation</term>
<term>Tract nucleus</term>
<term>Trigeminal</term>
<term>Trigeminal pain</term>
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<term>Limb pain</term>
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<term>Oral surg</term>
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<term>Phantom pain</term>
<term>Phantom tooth pain</term>
<term>Pharmacological aspects</term>
<term>Present investigation</term>
<term>Primary trigeminal axons</term>
<term>Prophylactic treatment</term>
<term>Psychological factors</term>
<term>Risk factor</term>
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<front><div type="abstract" xml:lang="en">SYNOPSIS Following tooth pulp extirpation, some subjects suffer from persistent pain which affects edentate sitesin absence of any local pathology. As regards this peculiar pain, called phantom tooth pain (PTP), what ispuzzling is the fact there is a low prevalence of PTP in a very large population showing identical conditionsof tooth pulp extirpation. The present investigation indicates that PTP mainly affects migraine (M) andcluster headache (CH) sufferers, whereas it does not affect subjects who have a negative personal andfamily history for idiopathic headache (IH). These results circumscribe the presence of PTP to a specificsection of the population. The present results, besides indicating that PTP may be the result of a peculiarneuronal predisposition relating to IH pathogenesis, suggests some practical therapeutic hints. In fact,successful anti‐ M and anti‐CH prophylactic treatment greatly improve PTP syndrome.</div>
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