Dental and bone abnormalities in patients with familial polyposis coli
Identifieur interne : 005C15 ( Istex/Corpus ); précédent : 005C14; suivant : 005C16Dental and bone abnormalities in patients with familial polyposis coli
Auteurs : William Carl ; Lemuel HerreraSource :
- Seminars in Surgical Oncology [ 8756-0437 ] ; 1987.
English descriptors
- KwdEn :
- Abnormality, Bone abnormalities, Colectomy, Coli, Cuspid, Dental abnormalities, Diagnostic features, Distal roots, Familial, Familial polyposis coli, First cousin, Herrera, Ileoproctostomy, Lesion, Mandible, Mandibular, Mandibular molars, Maxilla, Maxillary, Maxillary molars, Mesial, Mesial root, Molar, Osteoma, Osteomatous, Osteomatous lesions, Panoramic, Panoramic radiograph, Polyp, Polyposis, Polyposis coli, Premolar, Radiograph, Radiopacity, Right mandibular, Second molar, Second molars, Second premolar, Subtotal, Third molars.
- Teeft :
- Abnormality, Bone abnormalities, Colectomy, Coli, Cuspid, Dental abnormalities, Diagnostic features, Distal roots, Familial, Familial polyposis coli, First cousin, Herrera, Ileoproctostomy, Lesion, Mandible, Mandibular, Mandibular molars, Maxilla, Maxillary, Maxillary molars, Mesial, Mesial root, Molar, Osteoma, Osteomatous, Osteomatous lesions, Panoramic, Panoramic radiograph, Polyp, Polyposis, Polyposis coli, Premolar, Radiograph, Radiopacity, Right mandibular, Second molar, Second molars, Second premolar, Subtotal, Third molars.
Abstract
Dental and bone abnormalities of the maxilla and mandible are present in approximately 80% of patients with familial polyposis coli. The dental abnormalities include impacted teeth (other than third molars), supernumerary teeth, congenitally missing teeth, fused roots of first and second molars, and unusually long and tapered roots of posterior teeth. The bone lesions consist mostly of osteomas, either isolated or in clusters, in the maxilla and mandible or of exostoses with lateral and/or lingual extensions. Since dental and bone abnormalities are already present early in life there is a strong suggestion that they may be used as diagnostic features in the recognition of familial polyposis coli.
Url:
DOI: 10.1002/ssu.2980030206
Links to Exploration step
ISTEX:B907470A9DA61FF06B6632A9DE4C16DBFF5D76EFLe document en format XML
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The dental abnormalities include impacted teeth (other than third molars), supernumerary teeth, congenitally missing teeth, fused roots of first and second molars, and unusually long and tapered roots of posterior teeth. The bone lesions consist mostly of osteomas, either isolated or in clusters, in the maxilla and mandible or of exostoses with lateral and/or lingual extensions. 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The dental abnormalities include impacted teeth (other than third molars), supernumerary teeth, congenitally missing teeth, fused roots of first and second molars, and unusually long and tapered roots of posterior teeth. The bone lesions consist mostly of osteomas, either isolated or in clusters, in the maxilla and mandible or of exostoses with lateral and/or lingual extensions. Since dental and bone abnormalities are already present early in life there is a strong suggestion that they may be used as diagnostic features in the recognition of familial polyposis coli.</p></abstract><textClass><keywords xml:lang="en"><term xml:id="kwd1">bone lesions</term></keywords><keywords rend="articleCategory"><term>Article</term></keywords><keywords rend="tocHeading1"><term>Articles</term></keywords></textClass><langUsage><language ident="en"></language></langUsage></profileDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/B907470A9DA61FF06B6632A9DE4C16DBFF5D76EF/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>John Wiley & Sons, Inc.</publisherName><publisherLoc>New York</publisherLoc></publisherInfo><doi registered="yes">10.1002/(ISSN)1098-2388</doi><issn type="print">8756-0437</issn><issn type="electronic">1098-2388</issn><idGroup><id type="product" value="SSU"></id></idGroup><titleGroup><title type="main" xml:lang="en" sort="SEMINARS IN SURGICAL ONCOLOGY">Seminars in Surgical Oncology</title><title type="short">Semin. 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The dental abnormalities include impacted teeth (other than third molars), supernumerary teeth, congenitally missing teeth, fused roots of first and second molars, and unusually long and tapered roots of posterior teeth. The bone lesions consist mostly of osteomas, either isolated or in clusters, in the maxilla and mandible or of exostoses with lateral and/or lingual extensions. Since dental and bone abnormalities are already present early in life there is a strong suggestion that they may be used as diagnostic features in the recognition of familial polyposis coli.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Dental and bone abnormalities in patients with familial polyposis coli</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>Dental Abnormalities in FPC</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Dental and bone abnormalities in patients with familial polyposis coli</title></titleInfo><name type="personal"><namePart type="given">William</namePart><namePart type="family">Carl</namePart><namePart type="termsOfAddress">DDS</namePart><affiliation>Department of Dentistry, Maxillofacial Prosthetics, Roswell Park Memorial Institute, Buffalo</affiliation><affiliation>Correspondence address: Roswell Park Memorial Institute, 666 Elm Street, Buffalo, NY 14263</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Lemuel</namePart><namePart type="family">Herrera</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>Department of Surgical Oncology, Roswell Park Memorial Institute, Buffalo</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article" authority="ISTEX" authorityURI="https://content-type.data.istex.fr" valueURI="https://content-type.data.istex.fr/ark:/67375/XTP-6N5SZHKN-D">article</genre><originInfo><publisher>John Wiley & Sons, Inc.</publisher><place><placeTerm type="text">New York</placeTerm></place><dateIssued encoding="w3cdtf">1987</dateIssued><copyrightDate encoding="w3cdtf">1987</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><extent unit="figures">7</extent><extent unit="tables">1</extent><extent unit="references">7</extent></physicalDescription><abstract lang="en">Dental and bone abnormalities of the maxilla and mandible are present in approximately 80% of patients with familial polyposis coli. The dental abnormalities include impacted teeth (other than third molars), supernumerary teeth, congenitally missing teeth, fused roots of first and second molars, and unusually long and tapered roots of posterior teeth. The bone lesions consist mostly of osteomas, either isolated or in clusters, in the maxilla and mandible or of exostoses with lateral and/or lingual extensions. Since dental and bone abnormalities are already present early in life there is a strong suggestion that they may be used as diagnostic features in the recognition of familial polyposis coli.</abstract><subject lang="en"><genre>keywords</genre><topic>bone lesions</topic></subject><relatedItem type="host"><titleInfo><title>Seminars in Surgical Oncology</title></titleInfo><titleInfo type="abbreviated"><title>Semin. Surg. Oncol.</title></titleInfo><genre type="journal" authority="ISTEX" authorityURI="https://publication-type.data.istex.fr" valueURI="https://publication-type.data.istex.fr/ark:/67375/JMC-0GLKJH51-B">journal</genre><subject><genre>article-category</genre><topic>Article</topic></subject><identifier type="ISSN">8756-0437</identifier><identifier type="eISSN">1098-2388</identifier><identifier type="DOI">10.1002/(ISSN)1098-2388</identifier><identifier type="PublisherID">SSU</identifier><part><date>1987</date><detail type="volume"><caption>vol.</caption><number>3</number></detail><detail type="issue"><caption>no.</caption><number>2</number></detail><extent unit="pages"><start>77</start><end>83</end><total>7</total></extent></part></relatedItem><identifier type="istex">B907470A9DA61FF06B6632A9DE4C16DBFF5D76EF</identifier><identifier type="ark">ark:/67375/WNG-X2LXFCW2-Q</identifier><identifier type="DOI">10.1002/ssu.2980030206</identifier><identifier type="ArticleID">SSU2980030206</identifier><accessCondition type="use and reproduction" contentType="copyright">Copyright © 1987 Wiley‐Liss, Inc., A Wiley Company</accessCondition><recordInfo><recordContentSource authority="ISTEX" authorityURI="https://loaded-corpus.data.istex.fr" valueURI="https://loaded-corpus.data.istex.fr/ark:/67375/XBH-L0C46X92-X">wiley</recordContentSource><recordOrigin>John Wiley & Sons, Inc.</recordOrigin></recordInfo></mods><json:item><extension>json</extension><original>false</original><mimetype>application/json</mimetype><uri>https://api.istex.fr/document/B907470A9DA61FF06B6632A9DE4C16DBFF5D76EF/metadata/json</uri></json:item></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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