Transmission of Periodontopathic Bacteria from Natural Teeth to Implants
Identifieur interne : 004D84 ( Istex/Corpus ); précédent : 004D83; suivant : 004D85Transmission of Periodontopathic Bacteria from Natural Teeth to Implants
Auteurs : Masanori Aoki ; Kiyotoshi Takanashi ; Takashi Matsukubo ; Yasutomo Yajima ; Katsuji Okuda ; Toru Sato ; Kazuyuki IshiharaSource :
- Clinical Implant Dentistry and Related Research [ 1523-0899 ] ; 2012-06.
English descriptors
- KwdEn :
- Actinomycetemcomitans, Adjacent teeth, Clin, Clin periodontol, Colonization, Contralateral, Contralateral teeth, Dental implants, Denticola, Detection rate, Detection rates, Forsythia, Fusobacterium, Fusobacterium nucleatum, Gingival, Gingival crevice, Gingival crevices, Gingivalis, Implant, Implant sulci, Implant sulcus, Implant treatment, Intermedia, Microorganism, Natural teeth, Nucleatum, Occluding, Osseointegrated, Periodontal, Periodontitis, Periodontol, Periodontopathic, Periodontopathic bacteria, Porphyromonas, Porphyromonas gingivalis, Prevotella, Prevotella intermedia, Subgingival, Sulcus, Tannerella, Tannerella forsythia, Treponema, Treponema denticola.
- Teeft :
- Actinomycetemcomitans, Adjacent teeth, Clin, Clin periodontol, Colonization, Contralateral, Contralateral teeth, Dental implants, Denticola, Detection rate, Detection rates, Forsythia, Fusobacterium, Fusobacterium nucleatum, Gingival, Gingival crevice, Gingival crevices, Gingivalis, Implant, Implant sulci, Implant sulcus, Implant treatment, Intermedia, Microorganism, Natural teeth, Nucleatum, Occluding, Osseointegrated, Periodontal, Periodontitis, Periodontol, Periodontopathic, Periodontopathic bacteria, Porphyromonas, Porphyromonas gingivalis, Prevotella, Prevotella intermedia, Subgingival, Sulcus, Tannerella, Tannerella forsythia, Treponema, Treponema denticola.
Abstract
Purpose: Prevention of peri‐implantitis is essential for the success of implant rehabilitation. Infection by periodontopathic bacteria is a major cause of peri‐implantitis. The aim of the present study was to identify the source of peri‐implant colonization by periodontopathic bacteria.
Url:
DOI: 10.1111/j.1708-8208.2009.00260.x
Links to Exploration step
ISTEX:9B7E160F5A4CADE9D7AC08A54270D21C3966F29FLe document en format XML
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Aoki</name></author><author><name sortKey="Takanashi, Kiyotoshi" sort="Takanashi, Kiyotoshi" uniqKey="Takanashi K" first="Kiyotoshi" last="Takanashi">Kiyotoshi Takanashi</name></author><author><name sortKey="Matsukubo, Takashi" sort="Matsukubo, Takashi" uniqKey="Matsukubo T" first="Takashi" last="Matsukubo">Takashi Matsukubo</name></author><author><name sortKey="Yajima, Yasutomo" sort="Yajima, Yasutomo" uniqKey="Yajima Y" first="Yasutomo" last="Yajima">Yasutomo Yajima</name></author><author><name sortKey="Okuda, Katsuji" sort="Okuda, Katsuji" uniqKey="Okuda K" first="Katsuji" last="Okuda">Katsuji Okuda</name></author><author><name sortKey="Sato, Toru" sort="Sato, Toru" uniqKey="Sato T" first="Toru" last="Sato">Toru Sato</name></author><author><name sortKey="Ishihara, Kazuyuki" sort="Ishihara, Kazuyuki" uniqKey="Ishihara K" first="Kazuyuki" last="Ishihara">Kazuyuki Ishihara</name></author></analytic><monogr></monogr><series><title level="j" type="main">Clinical Implant Dentistry and Related Research</title><title level="j" type="alt">CLINICAL IMPLANT DENTISTRY RELATED RESEARCH</title><idno type="ISSN">1523-0899</idno><idno type="eISSN">1708-8208</idno><imprint><biblScope unit="vol">14</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="406">406</biblScope><biblScope unit="page" to="411">411</biblScope><biblScope unit="page-count">6</biblScope><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="2012-06">2012-06</date></imprint><idno type="ISSN">1523-0899</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">1523-0899</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Actinomycetemcomitans</term><term>Adjacent teeth</term><term>Clin</term><term>Clin periodontol</term><term>Colonization</term><term>Contralateral</term><term>Contralateral teeth</term><term>Dental implants</term><term>Denticola</term><term>Detection 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teeth</term><term>Clin</term><term>Clin periodontol</term><term>Colonization</term><term>Contralateral</term><term>Contralateral teeth</term><term>Dental implants</term><term>Denticola</term><term>Detection rate</term><term>Detection rates</term><term>Forsythia</term><term>Fusobacterium</term><term>Fusobacterium nucleatum</term><term>Gingival</term><term>Gingival crevice</term><term>Gingival crevices</term><term>Gingivalis</term><term>Implant</term><term>Implant sulci</term><term>Implant sulcus</term><term>Implant treatment</term><term>Intermedia</term><term>Microorganism</term><term>Natural teeth</term><term>Nucleatum</term><term>Occluding</term><term>Osseointegrated</term><term>Periodontal</term><term>Periodontitis</term><term>Periodontol</term><term>Periodontopathic</term><term>Periodontopathic bacteria</term><term>Porphyromonas</term><term>Porphyromonas gingivalis</term><term>Prevotella</term><term>Prevotella 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Infection by periodontopathic bacteria is a major cause of peri‐implantitis. The aim of the present study was to identify the source of peri‐implant colonization by periodontopathic bacteria.</div></front></TEI><istex><corpusName>wiley</corpusName><keywords><teeft><json:string>implant</json:string><json:string>periodontopathic</json:string><json:string>periodontopathic bacteria</json:string><json:string>gingivalis</json:string><json:string>porphyromonas</json:string><json:string>natural teeth</json:string><json:string>sulcus</json:string><json:string>porphyromonas gingivalis</json:string><json:string>nucleatum</json:string><json:string>actinomycetemcomitans</json:string><json:string>denticola</json:string><json:string>treponema</json:string><json:string>intermedia</json:string><json:string>colonization</json:string><json:string>prevotella</json:string><json:string>contralateral</json:string><json:string>occluding</json:string><json:string>treponema denticola</json:string><json:string>adjacent teeth</json:string><json:string>gingival</json:string><json:string>prevotella intermedia</json:string><json:string>tannerella</json:string><json:string>forsythia</json:string><json:string>clin</json:string><json:string>subgingival</json:string><json:string>tannerella forsythia</json:string><json:string>periodontal</json:string><json:string>periodontitis</json:string><json:string>contralateral teeth</json:string><json:string>fusobacterium</json:string><json:string>periodontol</json:string><json:string>osseointegrated</json:string><json:string>microorganism</json:string><json:string>gingival crevices</json:string><json:string>fusobacterium nucleatum</json:string><json:string>clin periodontol</json:string><json:string>implant sulcus</json:string><json:string>detection rates</json:string><json:string>gingival crevice</json:string><json:string>implant treatment</json:string><json:string>detection rate</json:string><json:string>dental implants</json:string><json:string>implant sulci</json:string></teeft></keywords><author><json:item><name>Masanori Aoki DDS, PhD</name></json:item><json:item><name>Kiyotoshi Takanashi DDS, PhD</name></json:item><json:item><name>Takashi Matsukubo DDS, PhD</name></json:item><json:item><name>Yasutomo Yajima DDS, PhD</name></json:item><json:item><name>Katsuji Okuda DDS, PhD</name></json:item><json:item><name>Toru Sato DDS, PhD</name></json:item><json:item><name>Kazuyuki Ishihara DDS, PhD</name></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>implantitis</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>infection</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>periodontitis</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>periodontopathic bacteria</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>transmission</value></json:item></subject><articleId><json:string>CID260</json:string></articleId><arkIstex>ark:/67375/WNG-N4BD5S4X-M</arkIstex><language><json:string>eng</json:string></language><originalGenre><json:string>article</json:string></originalGenre><abstract>Purpose: Prevention of peri‐implantitis is essential for the success of implant rehabilitation. Infection by periodontopathic bacteria is a major cause of peri‐implantitis. The aim of the present study was to identify the source of peri‐implant colonization by periodontopathic bacteria.</abstract><qualityIndicators><score>5.352</score><pdfWordCount>2884</pdfWordCount><pdfCharCount>20433</pdfCharCount><pdfVersion>1.3</pdfVersion><pdfPageCount>6</pdfPageCount><pdfPageSize>585 x 783 pts</pdfPageSize><refBibsNative>true</refBibsNative><abstractWordCount>39</abstractWordCount><abstractCharCount>287</abstractCharCount><keywordCount>5</keywordCount></qualityIndicators><title>Transmission of Periodontopathic Bacteria from Natural Teeth to Implants</title><pmid><json:string>20002682</json:string></pmid><genre><json:string>article</json:string></genre><host><title>Clinical Implant Dentistry and Related Research</title><language><json:string>unknown</json:string></language><doi><json:string>10.1111/(ISSN)1708-8208</json:string></doi><issn><json:string>1523-0899</json:string></issn><eissn><json:string>1708-8208</json:string></eissn><publisherId><json:string>CID</json:string></publisherId><volume>14</volume><issue>3</issue><pages><first>406</first><last>411</last><total>6</total></pages><genre><json:string>journal</json:string></genre></host><namedEntities><unitex><date><json:string>2012</json:string><json:string>1-2-2</json:string><json:string>16s</json:string></date><geogName></geogName><orgName><json:string>Department of Microbiology Tokyo Dental College, Chiba, Japan Reprint</json:string><json:string>Wiley Periodicals, Inc.</json:string><json:string>Wako Pure Chemical Industries, Osaka, Japan</json:string><json:string>Department of Epidemiology and Public Health</json:string><json:string>Related Research, Volume</json:string><json:string>Department of Crown and Bridge</json:string><json:string>Department of Microbiology, Tokyo Dental College</json:string><json:string>High-Tech Research Center</json:string><json:string>Oral Health Science Center</json:string><json:string>Department of Oral</json:string><json:string>SAS Institute, Cary, NC, USA</json:string><json:string>Ethical Committee of Tokyo Dental College</json:string><json:string>Brånemark Systems</json:string></orgName><orgName_funder></orgName_funder><orgName_provider></orgName_provider><persName><json:string>K. 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Infection by periodontopathic bacteria is a major cause of peri‐implantitis. The aim of the present study was to identify the source of peri‐implant colonization by periodontopathic bacteria.</p><p><hi rend="bold">Materials and Methods:</hi> Twenty‐one patients with implants were enrolled in the study. Subgingival plaque samples from the adjacent, occluding, and contralateral natural teeth were collected prior to second‐stage surgery. Samples from implant sulci were then obtained 2 weeks later. Detection of periodontopathic bacteria was performed by the polymerase chain reaction.</p><p><hi rend="bold">Results:</hi> The detection rates for <hi rend="italic">Aggregatibacter actinomycetemcomitans, Prevotella intermedia, Porphyromonas gingivalis, Treponema denticola, Tannerella forsythia</hi>, and <hi rend="italic">Fusobacterium nucleatum</hi> in all subgingival samples from natural teeth were similar to that in the peri‐implant sulci. Multiple logistic regression analysis revealed an association between the detection of <hi rend="italic">A. actinomycetemcomitans</hi>, <hi rend="italic">Prevotella intermedia</hi>, <hi rend="italic">Porphyromonas gingivalis</hi>, <hi rend="italic">Treponema denticola</hi>, and <hi rend="italic">F. nucleatum</hi> in the gingival crevices of adjacent teeth and that of the peri‐implant sulcus, but no association for <hi rend="italic">Tannerella forsythia</hi>.</p><p><hi rend="bold">Conclusions:</hi> The present findings suggest that colonization by <hi rend="italic">A. actinomycetemcomitans</hi>, <hi rend="italic">Prevotella intermedia</hi>, <hi rend="italic">Porphyromonas gingivalis</hi>, <hi rend="italic">Treponema denticola</hi>, and <hi rend="italic">F. nucleatum</hi> at the implant sulcus was affected by these microorganisms in the gingival crevice of adjacent teeth rather than those on occluding and contralateral teeth.</p></abstract><textClass><keywords xml:lang="en"><term xml:id="k1">implantitis</term><term xml:id="k2">infection</term><term xml:id="k3">periodontitis</term><term xml:id="k4">periodontopathic bacteria</term><term xml:id="k5">transmission</term></keywords><keywords rend="tocHeading1"><term>ARTICLES</term></keywords></textClass><langUsage><language ident="en"></language></langUsage></profileDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/9B7E160F5A4CADE9D7AC08A54270D21C3966F29F/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1708-8208</doi><issn type="print">1523-0899</issn><issn type="electronic">1708-8208</issn><idGroup><id type="product" value="CID"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="CLINICAL IMPLANT DENTISTRY RELATED RESEARCH">Clinical Implant Dentistry and Related Research</title></titleGroup></publicationMeta><publicationMeta level="part" position="06103"><doi origin="wiley">10.1111/cid.2012.14.issue-3</doi><numberingGroup><numbering type="journalVolume" number="14">14</numbering><numbering type="journalIssue" number="3">3</numbering></numberingGroup><coverDate startDate="2012-06">June 2012</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="12" status="forIssue"><doi origin="wiley">10.1111/j.1708-8208.2009.00260.x</doi><idGroup><id type="unit" value="CID260"></id></idGroup><countGroup><count type="pageTotal" number="6"></count></countGroup><titleGroup><title type="tocHeading1">ARTICLES</title></titleGroup><copyright>© 2009 Wiley Periodicals, Inc.</copyright><eventGroup><event type="firstOnline" date="2009-12-11"></event><event type="publishedOnlineAcceptedOrEarlyUnpaginated" date="2009-12-11"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:3.1.5 mode:FullText" date="2012-06-01"></event><event type="publishedOnlineFinalForm" date="2012-06-01"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-01-15"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.3.4 mode:FullText" date="2015-02-25"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="406">406</numbering><numbering type="pageLast" number="411">411</numbering></numberingGroup><correspondenceTo>Kazuyuki Ishihara, Department of Microbiology, Tokyo Dental College, 1‐2‐2 Masago, Mihama‐ku, Chiba 261‐8502, Japan; e‐mail: <email>ishihara@tdc.ac.jp</email></correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:CID.CID260.pdf"></link></linkGroup></publicationMeta><contentMeta><countGroup><count type="figureTotal" number="1"></count><count type="tableTotal" number="3"></count><count type="formulaTotal" number="0"></count><count type="referenceTotal" number="38"></count><count type="wordTotal" number="3320"></count><count type="linksPubMed" number="0"></count><count type="linksCrossRef" number="0"></count></countGroup><titleGroup><title type="main">Transmission of Periodontopathic Bacteria from Natural Teeth to Implants</title><title type="shortAuthors">Clinical Implant Dentistry and Related Research, Volume *, Number *, 2009</title><title type="short">Transmission of Periodontopathic Bacteria from Natural Teeth to Implants</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" noteRef="#fn1"><personName><givenNames>Masanori</givenNames><familyName>Aoki</familyName><degrees>DDS, PhD</degrees></personName></creator><creator creatorRole="author" xml:id="cr2" noteRef="#fn1"><personName><givenNames>Kiyotoshi</givenNames><familyName>Takanashi</familyName><degrees>DDS, PhD</degrees></personName></creator><creator creatorRole="author" xml:id="cr3" noteRef="#fn2"><personName><givenNames>Takashi</givenNames><familyName>Matsukubo</familyName><degrees>DDS, PhD</degrees></personName></creator><creator creatorRole="author" xml:id="cr4" noteRef="#fn3"><personName><givenNames>Yasutomo</givenNames><familyName>Yajima</familyName><degrees>DDS, PhD</degrees></personName></creator><creator creatorRole="author" xml:id="cr5" noteRef="#fn4"><personName><givenNames>Katsuji</givenNames><familyName>Okuda</familyName><degrees>DDS, PhD</degrees></personName></creator><creator creatorRole="author" xml:id="cr6" noteRef="#fn5"><personName><givenNames>Toru</givenNames><familyName>Sato</familyName><degrees>DDS, PhD</degrees></personName></creator><creator creatorRole="author" xml:id="cr7" noteRef="#fn6"><personName><givenNames>Kazuyuki</givenNames><familyName>Ishihara</familyName><degrees>DDS, PhD</degrees></personName></creator></creators><keywordGroup xml:lang="en"><keyword xml:id="k1">implantitis</keyword><keyword xml:id="k2">infection</keyword><keyword xml:id="k3">periodontitis</keyword><keyword xml:id="k4">periodontopathic bacteria</keyword><keyword xml:id="k5">transmission</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><title type="main">ABSTRACT</title><p><b>Purpose:</b> Prevention of peri‐implantitis is essential for the success of implant rehabilitation. Infection by periodontopathic bacteria is a major cause of peri‐implantitis. The aim of the present study was to identify the source of peri‐implant colonization by periodontopathic bacteria.</p><p><b>Materials and Methods:</b> Twenty‐one patients with implants were enrolled in the study. Subgingival plaque samples from the adjacent, occluding, and contralateral natural teeth were collected prior to second‐stage surgery. Samples from implant sulci were then obtained 2 weeks later. Detection of periodontopathic bacteria was performed by the polymerase chain reaction.</p><p><b>Results:</b> The detection rates for <i>Aggregatibacter actinomycetemcomitans, Prevotella intermedia, Porphyromonas gingivalis, Treponema denticola, Tannerella forsythia</i>, and <i>Fusobacterium nucleatum</i> in all subgingival samples from natural teeth were similar to that in the peri‐implant sulci. Multiple logistic regression analysis revealed an association between the detection of <i>A. actinomycetemcomitans</i>, <i>Prevotella intermedia</i>, <i>Porphyromonas gingivalis</i>, <i>Treponema denticola</i>, and <i>F. nucleatum</i> in the gingival crevices of adjacent teeth and that of the peri‐implant sulcus, but no association for <i>Tannerella forsythia</i>.</p><p><b>Conclusions:</b> The present findings suggest that colonization by <i>A. actinomycetemcomitans</i>, <i>Prevotella intermedia</i>, <i>Porphyromonas gingivalis</i>, <i>Treponema denticola</i>, and <i>F. nucleatum</i> at the implant sulcus was affected by these microorganisms in the gingival crevice of adjacent teeth rather than those on occluding and contralateral teeth.</p></abstract></abstractGroup></contentMeta><noteGroup><note xml:id="fn1"><p>
Assistant professor, Department of Crown and Bridge Prosthodontics, Tokyo Dental College, Chiba, Japan; </p></note><note xml:id="fn2"><p>
professor and chairman, Department of Epidemiology and Public Health Tokyo Dental College, Chiba, Japan; </p></note><note xml:id="fn3"><p>
professor and chairman, Department of Oral and Maxillofacial Implantology, Tokyo Dental College, Chiba, Japan; </p></note><note xml:id="fn4"><p>
professor emeritus, </p></note><note xml:id="fn5"><p>
professor and chairman, Department of Crown and Bridge Prosthodontics, Tokyo Dental College, Chiba, Japan; </p></note><note xml:id="fn6"><p>
professor and chairman, Department of Microbiology Tokyo Dental College, Chiba, Japan</p></note></noteGroup></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Transmission of Periodontopathic Bacteria from Natural Teeth to Implants</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>Transmission of Periodontopathic Bacteria from Natural Teeth to Implants</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Transmission of Periodontopathic Bacteria from Natural Teeth to Implants</title></titleInfo><name type="personal"><namePart type="given">Masanori</namePart><namePart type="family">Aoki</namePart><namePart type="termsOfAddress">DDS, PhD</namePart><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Kiyotoshi</namePart><namePart type="family">Takanashi</namePart><namePart type="termsOfAddress">DDS, PhD</namePart><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Takashi</namePart><namePart type="family">Matsukubo</namePart><namePart type="termsOfAddress">DDS, PhD</namePart><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Yasutomo</namePart><namePart type="family">Yajima</namePart><namePart type="termsOfAddress">DDS, PhD</namePart><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Katsuji</namePart><namePart type="family">Okuda</namePart><namePart type="termsOfAddress">DDS, PhD</namePart><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Toru</namePart><namePart type="family">Sato</namePart><namePart type="termsOfAddress">DDS, PhD</namePart><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Kazuyuki</namePart><namePart type="family">Ishihara</namePart><namePart type="termsOfAddress">DDS, PhD</namePart><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article" authority="ISTEX" authorityURI="https://content-type.data.istex.fr" valueURI="https://content-type.data.istex.fr/ark:/67375/XTP-6N5SZHKN-D">article</genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">2012-06</dateIssued><copyrightDate encoding="w3cdtf">2012</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><extent unit="figures">1</extent><extent unit="tables">3</extent><extent unit="formulas">0</extent><extent unit="references">38</extent><extent unit="linksCrossRef">0</extent><extent unit="words">3320</extent></physicalDescription><abstract>Purpose: Prevention of peri‐implantitis is essential for the success of implant rehabilitation. Infection by periodontopathic bacteria is a major cause of peri‐implantitis. The aim of the present study was to identify the source of peri‐implant colonization by periodontopathic bacteria.</abstract><abstract>Materials and Methods: Twenty‐one patients with implants were enrolled in the study. Subgingival plaque samples from the adjacent, occluding, and contralateral natural teeth were collected prior to second‐stage surgery. Samples from implant sulci were then obtained 2 weeks later. Detection of periodontopathic bacteria was performed by the polymerase chain reaction.</abstract><abstract>Results: The detection rates for Aggregatibacter actinomycetemcomitans, Prevotella intermedia, Porphyromonas gingivalis, Treponema denticola, Tannerella forsythia, and Fusobacterium nucleatum in all subgingival samples from natural teeth were similar to that in the peri‐implant sulci. Multiple logistic regression analysis revealed an association between the detection of A. actinomycetemcomitans, Prevotella intermedia, Porphyromonas gingivalis, Treponema denticola, and F. nucleatum in the gingival crevices of adjacent teeth and that of the peri‐implant sulcus, but no association for Tannerella forsythia.</abstract><abstract>Conclusions: The present findings suggest that colonization by A. actinomycetemcomitans, Prevotella intermedia, Porphyromonas gingivalis, Treponema denticola, and F. nucleatum at the implant sulcus was affected by these microorganisms in the gingival crevice of adjacent teeth rather than those on occluding and contralateral teeth.</abstract><subject lang="en"><genre>keywords</genre><topic>implantitis</topic><topic>infection</topic><topic>periodontitis</topic><topic>periodontopathic bacteria</topic><topic>transmission</topic></subject><relatedItem type="host"><titleInfo><title>Clinical Implant Dentistry and Related Research</title></titleInfo><genre type="journal" authority="ISTEX" authorityURI="https://publication-type.data.istex.fr" valueURI="https://publication-type.data.istex.fr/ark:/67375/JMC-0GLKJH51-B">journal</genre><identifier type="ISSN">1523-0899</identifier><identifier type="eISSN">1708-8208</identifier><identifier type="DOI">10.1111/(ISSN)1708-8208</identifier><identifier type="PublisherID">CID</identifier><part><date>2012</date><detail type="volume"><caption>vol.</caption><number>14</number></detail><detail type="issue"><caption>no.</caption><number>3</number></detail><extent unit="pages"><start>406</start><end>411</end><total>6</total></extent></part></relatedItem><identifier type="istex">9B7E160F5A4CADE9D7AC08A54270D21C3966F29F</identifier><identifier type="ark">ark:/67375/WNG-N4BD5S4X-M</identifier><identifier type="DOI">10.1111/j.1708-8208.2009.00260.x</identifier><identifier type="ArticleID">CID260</identifier><accessCondition type="use and reproduction" contentType="copyright">© 2009 Wiley Periodicals, Inc.</accessCondition><recordInfo><recordContentSource authority="ISTEX" authorityURI="https://loaded-corpus.data.istex.fr" valueURI="https://loaded-corpus.data.istex.fr/ark:/67375/XBH-L0C46X92-X">wiley</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods><json:item><extension>json</extension><original>false</original><mimetype>application/json</mimetype><uri>https://api.istex.fr/document/9B7E160F5A4CADE9D7AC08A54270D21C3966F29F/metadata/json</uri></json:item></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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