Risk factors for periodontitis
Identifieur interne : 004368 ( Istex/Corpus ); précédent : 004367; suivant : 004369Risk factors for periodontitis
Auteurs : Mf Timmerman ; Ga Van Der WeijdenSource :
- International Journal of Dental Hygiene [ 1601-5029 ] ; 2006-02.
English descriptors
- KwdEn :
- Attachment loss, Calculus, Caries, Clin, Clin periodontol, Community dent, Dent, Dent assoc, Dent hygiene, Dental caries, Dental plaque, Dental research, Epidemiological, Epidemiology, Gingivitis, Hygiene, Indonesian adolescents, Longitudinal, Longitudinal studies, National institute, Natural development, Natural history, Older adults, Oral hygiene, Periodontal, Periodontal conditions, Periodontal destruction, Periodontal disease, Periodontal diseases, Periodontitis, Periodontol, Plaque, Progression, Public health dent, Public health service, Risk factor, Risk factors, Risk indicators, Subgingival calculus, Timmerman, Tooth loss, Weijden.
- Teeft :
- Attachment loss, Calculus, Caries, Clin, Clin periodontol, Community dent, Dent, Dent assoc, Dent hygiene, Dental caries, Dental plaque, Dental research, Epidemiological, Epidemiology, Gingivitis, Hygiene, Indonesian adolescents, Longitudinal, Longitudinal studies, National institute, Natural development, Natural history, Older adults, Oral hygiene, Periodontal, Periodontal conditions, Periodontal destruction, Periodontal disease, Periodontal diseases, Periodontitis, Periodontol, Plaque, Progression, Public health dent, Public health service, Risk factor, Risk factors, Risk indicators, Subgingival calculus, Timmerman, Tooth loss, Weijden.
Abstract
Abstract: This review presents a selected overview of the literature concerning risk factors for periodontitis. That in some individuals gingivitis develops into periodontitis is still a matter of extensive research. Cross‐sectional studies of clinical and microbiological factors can be meaningful. Longitudinal studies of the natural history allow analysis of potential factors and conditions that may have an impact on the disease process. At present, several possible risk factors for the initiation and progression of periodontitis have been identified: age, gender, plaque, calculus, existing attachment loss. A consistent finding appears to be genetic predisposition for the development of the disease. In terms of microbiology, several micro‐organisms have been identified. The results of the Java Project on natural development of Periodontal Disease clearly pinpoint Actinobacillus actinimycetemcomitans as being associated with the onset of disease. The presence of subgingival calculus was found to be associated with onset and dental plaque with progression of disease. Consistent with literature males are more susceptible to disease. The presence of pockets ≥5 mm appear to be a useful tool, since it was found to be a prognostic factor for disease progression.
Url:
DOI: 10.1111/j.1601-5037.2006.00168.x
Links to Exploration step
ISTEX:8836F02C6C609B56B5ED494A85250790C6017411Le document en format XML
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That in some individuals gingivitis develops into periodontitis is still a matter of extensive research. Cross‐sectional studies of clinical and microbiological factors can be meaningful. Longitudinal studies of the natural history allow analysis of potential factors and conditions that may have an impact on the disease process. At present, several possible risk factors for the initiation and progression of periodontitis have been identified: age, gender, plaque, calculus, existing attachment loss. A consistent finding appears to be genetic predisposition for the development of the disease. In terms of microbiology, several micro‐organisms have been identified. The results of the Java Project on natural development of Periodontal Disease clearly pinpoint Actinobacillus actinimycetemcomitans as being associated with the onset of disease. The presence of subgingival calculus was found to be associated with onset and dental plaque with progression of disease. Consistent with literature males are more susceptible to disease. The presence of pockets ≥5 mm appear to be a useful tool, since it was found to be a prognostic factor for disease progression.</div></front></TEI><istex><corpusName>wiley</corpusName><keywords><teeft><json:string>periodontal</json:string><json:string>periodontitis</json:string><json:string>periodontol</json:string><json:string>periodontal disease</json:string><json:string>risk factors</json:string><json:string>clin periodontol</json:string><json:string>clin</json:string><json:string>tooth loss</json:string><json:string>attachment loss</json:string><json:string>timmerman</json:string><json:string>weijden</json:string><json:string>calculus</json:string><json:string>risk indicators</json:string><json:string>caries</json:string><json:string>dent</json:string><json:string>dent hygiene</json:string><json:string>gingivitis</json:string><json:string>epidemiology</json:string><json:string>periodontal diseases</json:string><json:string>natural history</json:string><json:string>longitudinal</json:string><json:string>oral hygiene</json:string><json:string>community dent</json:string><json:string>periodontal conditions</json:string><json:string>dent assoc</json:string><json:string>public health service</json:string><json:string>progression</json:string><json:string>epidemiological</json:string><json:string>plaque</json:string><json:string>older adults</json:string><json:string>dental plaque</json:string><json:string>risk factor</json:string><json:string>national institute</json:string><json:string>dental research</json:string><json:string>dental caries</json:string><json:string>public health dent</json:string><json:string>natural development</json:string><json:string>periodontal destruction</json:string><json:string>subgingival calculus</json:string><json:string>longitudinal studies</json:string><json:string>indonesian adolescents</json:string><json:string>hygiene</json:string></teeft></keywords><author><json:item><name>MF Timmerman</name><affiliations><json:string>M.F. 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That in some individuals gingivitis develops into periodontitis is still a matter of extensive research. Cross‐sectional studies of clinical and microbiological factors can be meaningful. Longitudinal studies of the natural history allow analysis of potential factors and conditions that may have an impact on the disease process. At present, several possible risk factors for the initiation and progression of periodontitis have been identified: age, gender, plaque, calculus, existing attachment loss. A consistent finding appears to be genetic predisposition for the development of the disease. In terms of microbiology, several micro‐organisms have been identified. The results of the Java Project on natural development of Periodontal Disease clearly pinpoint Actinobacillus actinimycetemcomitans as being associated with the onset of disease. The presence of subgingival calculus was found to be associated with onset and dental plaque with progression of disease. Consistent with literature males are more susceptible to disease. 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Timmerman, G.A. van der Weijden, Department of Periodontology, Academic Centre for Dentistry Amsterdam, Amsterdam, The Netherlands<address><country key="NL"></country></address></affiliation></author><author xml:id="author-0001"><persName><forename type="first">GA</forename><surname>Weijden</surname></persName><affiliation>M.F. Timmerman, G.A. van der Weijden, Department of Periodontology, Academic Centre for Dentistry Amsterdam, Amsterdam, The Netherlands<address><country key="NL"></country></address></affiliation></author><idno type="istex">8836F02C6C609B56B5ED494A85250790C6017411</idno><idno type="ark">ark:/67375/WNG-3CN3KJ9W-C</idno><idno type="DOI">10.1111/j.1601-5037.2006.00168.x</idno><idno type="unit">IDH168</idno><idno type="toTypesetVersion">file:IDH.IDH168.pdf</idno></analytic><monogr><title level="j" type="main">International Journal of Dental Hygiene</title><title level="j" type="alt">INTERNATIONAL JOURNAL OF DENTAL HYGIENE</title><idno type="pISSN">1601-5029</idno><idno type="eISSN">1601-5037</idno><idno type="book-DOI">10.1111/(ISSN)1601-5037</idno><idno type="book-part-DOI">10.1111/idh.2006.4.issue-1</idno><idno type="product">IDH</idno><idno type="publisherDivision">ST</idno><imprint><biblScope unit="vol">4</biblScope><biblScope unit="issue">1</biblScope><biblScope unit="page" from="2">2</biblScope><biblScope unit="page" to="7">7</biblScope><biblScope unit="page-count">6</biblScope><publisher>Munksgaard International Publishers</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="2006-02"></date></imprint></monogr></biblStruct></sourceDesc></fileDesc><profileDesc><abstract xml:lang="en" style="main"><p><hi rend="bold">Abstract: </hi> This review presents a selected overview of the literature concerning risk factors for periodontitis. That in some individuals gingivitis develops into periodontitis is still a matter of extensive research. Cross‐sectional studies of clinical and microbiological factors can be meaningful. Longitudinal studies of the natural history allow analysis of potential factors and conditions that may have an impact on the disease process. At present, several possible risk factors for the initiation and progression of periodontitis have been identified: age, gender, plaque, calculus, existing attachment loss. A consistent finding appears to be genetic predisposition for the development of the disease. In terms of microbiology, several micro‐organisms have been identified. The results of the Java Project on natural development of Periodontal Disease clearly pinpoint <hi rend="italic">Actinobacillus actinimycetemcomitans</hi> as being associated with the onset of disease. The presence of subgingival calculus was found to be associated with onset and dental plaque with progression of disease. Consistent with literature males are more susceptible to disease. The presence of pockets ≥5 mm appear to be a useful tool, since it was found to be a prognostic factor for disease progression.</p></abstract><textClass><keywords xml:lang="en"><term xml:id="k1">epidemiology</term><term xml:id="k2">periodontal pathogens</term><term xml:id="k3">periodontitis</term><term xml:id="k4">risk factors</term></keywords><keywords rend="tocHeading1"><term>REVIEW ARTICLE</term></keywords></textClass><langUsage><language ident="en"></language></langUsage></profileDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/8836F02C6C609B56B5ED494A85250790C6017411/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Munksgaard International Publishers</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1601-5037</doi><issn type="print">1601-5029</issn><issn type="electronic">1601-5037</issn><idGroup><id type="product" value="IDH"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="INTERNATIONAL JOURNAL OF DENTAL HYGIENE">International Journal of Dental Hygiene</title></titleGroup></publicationMeta><publicationMeta level="part" position="02001"><doi origin="wiley">10.1111/idh.2006.4.issue-1</doi><numberingGroup><numbering type="journalVolume" number="4">4</numbering><numbering type="journalIssue" number="1">1</numbering></numberingGroup><coverDate startDate="2006-02">February 2006</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="2" status="forIssue"><doi origin="wiley">10.1111/j.1601-5037.2006.00168.x</doi><idGroup><id type="unit" value="IDH168"></id></idGroup><countGroup><count type="pageTotal" number="6"></count></countGroup><titleGroup><title type="tocHeading1">REVIEW ARTICLE</title></titleGroup><eventGroup><event type="firstOnline" date="2006-02-01"></event><event type="publishedOnlineFinalForm" date="2006-02-01"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:FullText result:FullText" date="2010-03-07"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-01-27"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-23"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="2">2</numbering><numbering type="pageLast" number="7">7</numbering></numberingGroup><correspondenceTo><i>M.F. Timmerman</i>
Department of Periodontology
Academic Centre for Dentistry Amsterdam
Louwesweg 1
1066 EA Amsterdam
The Netherlands
Tel.: +31 205 188 307
Fax: +31 205 188 512
E‐mail: <email>m.timmerman@acta.nl</email></correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:IDH.IDH168.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory><b>Dates:</b> Accepted 14 November 2005</unparsedEditorialHistory><countGroup><count type="figureTotal" number="0"></count><count type="tableTotal" number="0"></count></countGroup><titleGroup><title type="main">Risk factors for periodontitis</title><title type="shortAuthors">Timmerman and van der Weijden</title><title type="short">Risk factors for periodontitis</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#aff-1-1"><personName><givenNames>MF</givenNames><familyName>Timmerman</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#aff-1-1"><personName><givenNames>GA</givenNames><familyNamePrefix>van der</familyNamePrefix><familyName>Weijden</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="aff-1-1" countryCode="NL"><unparsedAffiliation><i>M.F. Timmerman, G.A. van der Weijden</i>, Department of Periodontology, Academic Centre for Dentistry Amsterdam, Amsterdam, The Netherlands</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">epidemiology</keyword><keyword xml:id="k2">periodontal pathogens</keyword><keyword xml:id="k3">periodontitis</keyword><keyword xml:id="k4">risk factors</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p><b>Abstract: </b> This review presents a selected overview of the literature concerning risk factors for periodontitis. That in some individuals gingivitis develops into periodontitis is still a matter of extensive research. Cross‐sectional studies of clinical and microbiological factors can be meaningful. Longitudinal studies of the natural history allow analysis of potential factors and conditions that may have an impact on the disease process. At present, several possible risk factors for the initiation and progression of periodontitis have been identified: age, gender, plaque, calculus, existing attachment loss. A consistent finding appears to be genetic predisposition for the development of the disease. In terms of microbiology, several micro‐organisms have been identified. The results of the Java Project on natural development of Periodontal Disease clearly pinpoint <i>Actinobacillus actinimycetemcomitans</i> as being associated with the onset of disease. The presence of subgingival calculus was found to be associated with onset and dental plaque with progression of disease. Consistent with literature males are more susceptible to disease. The presence of pockets ≥5 mm appear to be a useful tool, since it was found to be a prognostic factor for disease progression.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Risk factors for periodontitis</title></titleInfo><titleInfo type="abbreviated" lang="en"><title>Risk factors for periodontitis</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Risk factors for periodontitis</title></titleInfo><name type="personal"><namePart type="given">MF</namePart><namePart type="family">Timmerman</namePart><affiliation>M.F. Timmerman, G.A. van der Weijden, Department of Periodontology, Academic Centre for Dentistry Amsterdam, Amsterdam, The Netherlands</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">GA</namePart><namePart type="family">van der Weijden</namePart><affiliation>M.F. Timmerman, G.A. van der Weijden, Department of Periodontology, Academic Centre for Dentistry Amsterdam, Amsterdam, The Netherlands</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article" authority="ISTEX" authorityURI="https://content-type.data.istex.fr" valueURI="https://content-type.data.istex.fr/ark:/67375/XTP-6N5SZHKN-D">article</genre><originInfo><publisher>Munksgaard International Publishers</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">2006-02</dateIssued><edition>Dates: Accepted 14 November 2005</edition><copyrightDate encoding="w3cdtf">2006</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><extent unit="figures">0</extent><extent unit="tables">0</extent></physicalDescription><abstract>Abstract: This review presents a selected overview of the literature concerning risk factors for periodontitis. That in some individuals gingivitis develops into periodontitis is still a matter of extensive research. Cross‐sectional studies of clinical and microbiological factors can be meaningful. Longitudinal studies of the natural history allow analysis of potential factors and conditions that may have an impact on the disease process. At present, several possible risk factors for the initiation and progression of periodontitis have been identified: age, gender, plaque, calculus, existing attachment loss. A consistent finding appears to be genetic predisposition for the development of the disease. In terms of microbiology, several micro‐organisms have been identified. The results of the Java Project on natural development of Periodontal Disease clearly pinpoint Actinobacillus actinimycetemcomitans as being associated with the onset of disease. The presence of subgingival calculus was found to be associated with onset and dental plaque with progression of disease. Consistent with literature males are more susceptible to disease. The presence of pockets ≥5 mm appear to be a useful tool, since it was found to be a prognostic factor for disease progression.</abstract><subject lang="en"><genre>keywords</genre><topic>epidemiology</topic><topic>periodontal pathogens</topic><topic>periodontitis</topic><topic>risk factors</topic></subject><relatedItem type="host"><titleInfo><title>International Journal of Dental Hygiene</title></titleInfo><genre type="journal" authority="ISTEX" authorityURI="https://publication-type.data.istex.fr" valueURI="https://publication-type.data.istex.fr/ark:/67375/JMC-0GLKJH51-B">journal</genre><identifier type="ISSN">1601-5029</identifier><identifier type="eISSN">1601-5037</identifier><identifier type="DOI">10.1111/(ISSN)1601-5037</identifier><identifier type="PublisherID">IDH</identifier><part><date>2006</date><detail type="volume"><caption>vol.</caption><number>4</number></detail><detail type="issue"><caption>no.</caption><number>1</number></detail><extent unit="pages"><start>2</start><end>7</end><total>6</total></extent></part></relatedItem><identifier type="istex">8836F02C6C609B56B5ED494A85250790C6017411</identifier><identifier type="ark">ark:/67375/WNG-3CN3KJ9W-C</identifier><identifier type="DOI">10.1111/j.1601-5037.2006.00168.x</identifier><identifier type="ArticleID">IDH168</identifier><recordInfo><recordContentSource authority="ISTEX" authorityURI="https://loaded-corpus.data.istex.fr" valueURI="https://loaded-corpus.data.istex.fr/ark:/67375/XBH-L0C46X92-X">wiley</recordContentSource><recordOrigin>Munksgaard International Publishers</recordOrigin></recordInfo></mods><json:item><extension>json</extension><original>false</original><mimetype>application/json</mimetype><uri>https://api.istex.fr/document/8836F02C6C609B56B5ED494A85250790C6017411/metadata/json</uri></json:item></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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