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<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">The Rachitic Tooth</title><author><name sortKey="Foster, Brian L" sort="Foster, Brian L" uniqKey="Foster B" first="Brian L." last="Foster">Brian L. Foster</name></author><author><name sortKey="Nociti, Francisco H" sort="Nociti, Francisco H" uniqKey="Nociti F" first="Francisco H." last="Nociti">Francisco H. Nociti</name></author><author><name sortKey="Somerman, Martha J" sort="Somerman, Martha J" uniqKey="Somerman M" first="Martha J." last="Somerman">Martha J. Somerman</name></author></titleStmt><publicationStmt><idno type="wicri:source">PMC</idno><idno type="pmid">23939820</idno><idno type="pmc">3895863</idno><idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3895863</idno><idno type="RBID">PMC:3895863</idno><idno type="doi">10.1210/er.2013-1009</idno><date when="2013">2013</date><idno type="wicri:Area/Pmc/Corpus">000788</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Corpus" wicri:corpus="PMC">000788</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">The Rachitic Tooth</title><author><name sortKey="Foster, Brian L" sort="Foster, Brian L" uniqKey="Foster B" first="Brian L." last="Foster">Brian L. Foster</name></author><author><name sortKey="Nociti, Francisco H" sort="Nociti, Francisco H" uniqKey="Nociti F" first="Francisco H." last="Nociti">Francisco H. Nociti</name></author><author><name sortKey="Somerman, Martha J" sort="Somerman, Martha J" uniqKey="Somerman M" first="Martha J." last="Somerman">Martha J. Somerman</name></author></analytic><series><title level="j">Endocrine Reviews</title><idno type="ISSN">0163-769X</idno><idno type="eISSN">1945-7189</idno><imprint><date when="2013">2013</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en"><p>Teeth are mineralized organs composed of three unique hard tissues, enamel, dentin, and cementum, and supported by the surrounding alveolar bone. Although odontogenesis differs from osteogenesis in several respects, tooth mineralization is susceptible to similar developmental failures as bone. Here we discuss conditions fitting under the umbrella of rickets, which traditionally referred to skeletal disease associated with vitamin D deficiency but has been more recently expanded to include newly identified factors involved in endocrine regulation of vitamin D, phosphate, and calcium, including phosphate-regulating endopeptidase homolog, X-linked, fibroblast growth factor 23, and dentin matrix protein 1. Systemic mineral metabolism intersects with local regulation of mineralization, and factors including tissue nonspecific alkaline phosphatase are necessary for proper mineralization, where rickets can result from loss of activity of tissue nonspecific alkaline phosphatase. Individuals suffering from rickets often bear the additional burden of a defective dentition, and transgenic mouse models have aided in understanding the nature and mechanisms involved in tooth defects, which may or may not parallel rachitic bone defects. This report reviews dental effects of the range of rachitic disorders, including discussion of etiologies of hereditary forms of rickets, a survey of resulting bone and tooth mineralization disorders, and a discussion of mechanisms, known and hypothesized, involved in the observed dental pathologies. Descriptions of human pathology are augmented by analysis of transgenic mouse models, and new interpretations are brought to bear on questions of how teeth are affected under conditions of rickets. In short, the rachitic tooth will be revealed.</p></div></front></TEI><pmc article-type="review-article"><pmc-comment>The publisher of this article does not allow downloading of the full text in XML form.</pmc-comment>
<front><journal-meta><journal-id journal-id-type="nlm-ta">Endocr Rev</journal-id><journal-id journal-id-type="iso-abbrev">Endocr. Rev</journal-id><journal-id journal-id-type="hwp">edrv</journal-id><journal-id journal-id-type="publisher-id">endre</journal-id><journal-id journal-id-type="pmc">edrv</journal-id><journal-id journal-id-type="publisher-id">edrv</journal-id><journal-title-group><journal-title>Endocrine Reviews</journal-title></journal-title-group><issn pub-type="ppub">0163-769X</issn><issn pub-type="epub">1945-7189</issn><publisher><publisher-name>Endocrine Society</publisher-name><publisher-loc>Chevy Chase, MD</publisher-loc></publisher></journal-meta><article-meta><article-id pub-id-type="pmid">23939820</article-id><article-id pub-id-type="pmc">3895863</article-id><article-id pub-id-type="publisher-id">ER-13-1009</article-id><article-id pub-id-type="doi">10.1210/er.2013-1009</article-id><article-categories><subj-group subj-group-type="heading"><subject>Reviews</subject></subj-group></article-categories><title-group><article-title>The Rachitic Tooth</article-title></title-group><contrib-group><contrib contrib-type="author" corresp="yes"><name><surname>Foster</surname><given-names>Brian L.</given-names></name></contrib><contrib contrib-type="author"><name><surname>Nociti</surname><given-names>Francisco H.</given-names><suffix>Jr</suffix></name></contrib><contrib contrib-type="author"><name><surname>Somerman</surname><given-names>Martha J.</given-names></name></contrib><aff>National Institute for Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, Maryland 20892</aff></contrib-group><author-notes><corresp>Address requests for reprints to: Brian L. Foster, <addr-line>9000 Rockville Pike, Building 50, Room 4120, Bethesda, Maryland 20892.</addr-line> E-mail: <email>brian.foster@nih.gov</email>.</corresp></author-notes><pub-date pub-type="ppub"><month>2</month><year>2014</year></pub-date><pub-date pub-type="epub"><day>12</day><month>8</month><year>2013</year></pub-date><pub-date pub-type="pmc-release"><day>1</day><month>2</month><year>2015</year></pub-date><pmc-comment> PMC Release delay is 12 months and
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<volume>35</volume><issue>1</issue><fpage>1</fpage><lpage>34</lpage><history><date date-type="received"><day>31</day><month>1</month><year>2013</year></date><date date-type="accepted"><day>2</day><month>8</month><year>2013</year></date></history><permissions><copyright-statement>Copyright © 2014 by The Endocrine Society</copyright-statement><copyright-year>2014</copyright-year></permissions><self-uri xlink:title="pdf" xlink:type="simple" xlink:href="zef00114000001.pdf"></self-uri><abstract><p>Teeth are mineralized organs composed of three unique hard tissues, enamel, dentin, and cementum, and supported by the surrounding alveolar bone. Although odontogenesis differs from osteogenesis in several respects, tooth mineralization is susceptible to similar developmental failures as bone. Here we discuss conditions fitting under the umbrella of rickets, which traditionally referred to skeletal disease associated with vitamin D deficiency but has been more recently expanded to include newly identified factors involved in endocrine regulation of vitamin D, phosphate, and calcium, including phosphate-regulating endopeptidase homolog, X-linked, fibroblast growth factor 23, and dentin matrix protein 1. Systemic mineral metabolism intersects with local regulation of mineralization, and factors including tissue nonspecific alkaline phosphatase are necessary for proper mineralization, where rickets can result from loss of activity of tissue nonspecific alkaline phosphatase. Individuals suffering from rickets often bear the additional burden of a defective dentition, and transgenic mouse models have aided in understanding the nature and mechanisms involved in tooth defects, which may or may not parallel rachitic bone defects. This report reviews dental effects of the range of rachitic disorders, including discussion of etiologies of hereditary forms of rickets, a survey of resulting bone and tooth mineralization disorders, and a discussion of mechanisms, known and hypothesized, involved in the observed dental pathologies. Descriptions of human pathology are augmented by analysis of transgenic mouse models, and new interpretations are brought to bear on questions of how teeth are affected under conditions of rickets. In short, the rachitic tooth will be revealed.</p></abstract></article-meta></front></pmc></record>Pour manipuler ce document sous Unix (Dilib)
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