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Idiopathic hyperplasia of the sublingual glands in totally or partially edentulous individuals

Identifieur interne : 000390 ( PascalFrancis/Corpus ); précédent : 000389; suivant : 000391

Idiopathic hyperplasia of the sublingual glands in totally or partially edentulous individuals

Auteurs : Carina Domaneschi ; Ana R. Mauricio ; Filipe Modolo ; Dante A. Migliari

Source :

RBID : Pascal:08-0102536

Descripteurs français

English descriptors

Abstract

Objective. To determine the possible etiologic factors associated with the development of idiopathic hyperplasia of the sublingual gland (IHSG). Study design. Fifty partially or totally edentulous patients were divided into 2 groups, each with 25 subjects, matched by sex. Group 1 was made up of patients with IHSG, whereas group 2 consisted of patients without IHSG. Patients of both groups were investigated with respect to the use of prostheses, systemic medication, systemic diseases, smoking habits, alcohol consumption, and xerostomia. In addition, patients of group 1 were examined by occlusal radiography, and in 6 of them, a biopsy of the sublingual gland swelling was taken for histopathologic investigation. The Fisher exact test was used, with significance set for P ≤.05. Results. The comparative analysis revealed no statistical difference between the 2 groups concerning all factors investigated (P >.05). Radiographic examination did not detect any presence of salivary calculi; the histopathologic findings revealed either chronic sialadenitis (4 cases) or normal glandular tissue (2 cases). Conclusion. Our clinical and histopathologic analyses confirm that IHSG is a harmless condition for which no surgical treatment is required. The etiologic investigation did not find any local or systemic factors significantly associated with the development of IHSG. The absence of posterior teeth remains as the only known predisposing factor to be strongly linked to this condition.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

pA  
A01 01  1    @0 1079-2104
A03   1    @0 Oral surg. oral med. oral pathol. oral radiol. endo.
A05       @2 103
A06       @2 3
A08 01  1  ENG  @1 Idiopathic hyperplasia of the sublingual glands in totally or partially edentulous individuals
A11 01  1    @1 DOMANESCHI (Carina)
A11 02  1    @1 MAURICIO (Ana R.)
A11 03  1    @1 MODOLO (Filipe)
A11 04  1    @1 MIGLIARI (Dante A.)
A14 01      @1 UNIVERSITY OF S========Atilde;O PAULO @3 BRA @Z 1 aut. @Z 2 aut. @Z 3 aut. @Z 4 aut.
A20       @1 374-377
A21       @1 2007
A23 01      @0 ENG
A43 01      @1 INIST @2 5101 @5 354000173962930140
A44       @0 0000 @1 © 2008 INIST-CNRS. All rights reserved.
A45       @0 13 ref.
A47 01  1    @0 08-0102536
A60       @1 P
A61       @0 A
A64 01  1    @0 Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
A66 01      @0 USA
C01 01    ENG  @0 Objective. To determine the possible etiologic factors associated with the development of idiopathic hyperplasia of the sublingual gland (IHSG). Study design. Fifty partially or totally edentulous patients were divided into 2 groups, each with 25 subjects, matched by sex. Group 1 was made up of patients with IHSG, whereas group 2 consisted of patients without IHSG. Patients of both groups were investigated with respect to the use of prostheses, systemic medication, systemic diseases, smoking habits, alcohol consumption, and xerostomia. In addition, patients of group 1 were examined by occlusal radiography, and in 6 of them, a biopsy of the sublingual gland swelling was taken for histopathologic investigation. The Fisher exact test was used, with significance set for P ≤.05. Results. The comparative analysis revealed no statistical difference between the 2 groups concerning all factors investigated (P >.05). Radiographic examination did not detect any presence of salivary calculi; the histopathologic findings revealed either chronic sialadenitis (4 cases) or normal glandular tissue (2 cases). Conclusion. Our clinical and histopathologic analyses confirm that IHSG is a harmless condition for which no surgical treatment is required. The etiologic investigation did not find any local or systemic factors significantly associated with the development of IHSG. The absence of posterior teeth remains as the only known predisposing factor to be strongly linked to this condition.
C02 01  X    @0 002B10C02
C03 01  X  FRE  @0 Hyperplasie @5 01
C03 01  X  ENG  @0 Hyperplasia @5 01
C03 01  X  SPA  @0 Hiperplasia @5 01
C03 02  X  FRE  @0 Edentation @5 02
C03 02  X  ENG  @0 Edentulousness @5 02
C03 02  X  SPA  @0 Edentación @5 02
C03 03  X  FRE  @0 Idiopathique @5 07
C03 03  X  ENG  @0 Idiopathic @5 07
C03 03  X  SPA  @0 Idiopático @5 07
C03 04  X  FRE  @0 Glande sublinguale @5 08
C03 04  X  ENG  @0 Sublingual gland @5 08
C03 04  X  SPA  @0 Glándula sublingual @5 08
C03 05  X  FRE  @0 Homme @5 09
C03 05  X  ENG  @0 Human @5 09
C03 05  X  SPA  @0 Hombre @5 09
C07 01  X  FRE  @0 Pathologie dentaire @5 37
C07 01  X  ENG  @0 Dental disease @5 37
C07 01  X  SPA  @0 Diente patología @5 37
C07 02  X  FRE  @0 Stomatologie @5 38
C07 02  X  ENG  @0 Stomatology @5 38
C07 02  X  SPA  @0 Estomatología @5 38
N21       @1 056
N44 01      @1 OTO
N82       @1 OTO

Format Inist (serveur)

NO : PASCAL 08-0102536 INIST
ET : Idiopathic hyperplasia of the sublingual glands in totally or partially edentulous individuals
AU : DOMANESCHI (Carina); MAURICIO (Ana R.); MODOLO (Filipe); MIGLIARI (Dante A.)
AF : UNIVERSITY OF S========Atilde;O PAULO/Brésil (1 aut., 2 aut., 3 aut., 4 aut.)
DT : Publication en série; Niveau analytique
SO : Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics; ISSN 1079-2104; Etats-Unis; Da. 2007; Vol. 103; No. 3; Pp. 374-377; Bibl. 13 ref.
LA : Anglais
EA : Objective. To determine the possible etiologic factors associated with the development of idiopathic hyperplasia of the sublingual gland (IHSG). Study design. Fifty partially or totally edentulous patients were divided into 2 groups, each with 25 subjects, matched by sex. Group 1 was made up of patients with IHSG, whereas group 2 consisted of patients without IHSG. Patients of both groups were investigated with respect to the use of prostheses, systemic medication, systemic diseases, smoking habits, alcohol consumption, and xerostomia. In addition, patients of group 1 were examined by occlusal radiography, and in 6 of them, a biopsy of the sublingual gland swelling was taken for histopathologic investigation. The Fisher exact test was used, with significance set for P ≤.05. Results. The comparative analysis revealed no statistical difference between the 2 groups concerning all factors investigated (P >.05). Radiographic examination did not detect any presence of salivary calculi; the histopathologic findings revealed either chronic sialadenitis (4 cases) or normal glandular tissue (2 cases). Conclusion. Our clinical and histopathologic analyses confirm that IHSG is a harmless condition for which no surgical treatment is required. The etiologic investigation did not find any local or systemic factors significantly associated with the development of IHSG. The absence of posterior teeth remains as the only known predisposing factor to be strongly linked to this condition.
CC : 002B10C02
FD : Hyperplasie; Edentation; Idiopathique; Glande sublinguale; Homme
FG : Pathologie dentaire; Stomatologie
ED : Hyperplasia; Edentulousness; Idiopathic; Sublingual gland; Human
EG : Dental disease; Stomatology
SD : Hiperplasia; Edentación; Idiopático; Glándula sublingual; Hombre
LO : INIST-5101.354000173962930140
ID : 08-0102536

Links to Exploration step

Pascal:08-0102536

Le document en format XML

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<div type="abstract" xml:lang="en">Objective. To determine the possible etiologic factors associated with the development of idiopathic hyperplasia of the sublingual gland (IHSG). Study design. Fifty partially or totally edentulous patients were divided into 2 groups, each with 25 subjects, matched by sex. Group 1 was made up of patients with IHSG, whereas group 2 consisted of patients without IHSG. Patients of both groups were investigated with respect to the use of prostheses, systemic medication, systemic diseases, smoking habits, alcohol consumption, and xerostomia. In addition, patients of group 1 were examined by occlusal radiography, and in 6 of them, a biopsy of the sublingual gland swelling was taken for histopathologic investigation. The Fisher exact test was used, with significance set for P ≤.05. Results. The comparative analysis revealed no statistical difference between the 2 groups concerning all factors investigated (P >.05). Radiographic examination did not detect any presence of salivary calculi; the histopathologic findings revealed either chronic sialadenitis (4 cases) or normal glandular tissue (2 cases). Conclusion. Our clinical and histopathologic analyses confirm that IHSG is a harmless condition for which no surgical treatment is required. The etiologic investigation did not find any local or systemic factors significantly associated with the development of IHSG. The absence of posterior teeth remains as the only known predisposing factor to be strongly linked to this condition.</div>
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