Diabetes mellitus and periodontal diseases.
Identifieur interne : 000162 ( Ncbi/Checkpoint ); précédent : 000161; suivant : 000163Diabetes mellitus and periodontal diseases.
Auteurs : Corneliu Sima [Canada] ; Michael GlogauerSource :
- Current diabetes reports [ 1539-0829 ] ; 2013.
Descripteurs français
- KwdFr :
- MESH :
English descriptors
- KwdEn :
- MESH :
- complications : Diabetes Mellitus, Type 1, Diabetes Mellitus, Type 2.
- etiology : Periodontal Diseases.
- therapy : Hyperglycemia, Periodontal Diseases.
- Humans, Models, Biological, Risk Factors.
Abstract
A bidirectional relationship between diabetes mellitus (DM) and periodontal diseases (PDs) has been established. It is estimated that patients with poorly controlled DM are 3 times more likely to develop chronic PD compared with normoglycemic individuals despite similar composition in subgingival biofilms. Furthermore, these patients present with increased severity and rapid progression of attachment loss around teeth resulting in edentulism. Treatment of PD results in a modest but significant improvement in glycemic control in patients with DM reflected by a 0.4 % reduction in HbA1c-glycated hemoglobin levels. Compelling evidence from in vitro and animal studies supports a plausible biological explanation for the relationship between the 2 conditions centered on systemic low-grade inflammation. However, the limited number of comparable large randomized clinical trials is reflected in the limited specific guidelines offered by the international organizations for DM and PD regarding the management of the 2 diseases in an individual.
DOI: 10.1007/s11892-013-0367-y
PubMed: 23430581
Affiliations:
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pubmed:23430581Le document en format XML
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It is estimated that patients with poorly controlled DM are 3 times more likely to develop chronic PD compared with normoglycemic individuals despite similar composition in subgingival biofilms. Furthermore, these patients present with increased severity and rapid progression of attachment loss around teeth resulting in edentulism. Treatment of PD results in a modest but significant improvement in glycemic control in patients with DM reflected by a 0.4 % reduction in HbA1c-glycated hemoglobin levels. Compelling evidence from in vitro and animal studies supports a plausible biological explanation for the relationship between the 2 conditions centered on systemic low-grade inflammation. However, the limited number of comparable large randomized clinical trials is reflected in the limited specific guidelines offered by the international organizations for DM and PD regarding the management of the 2 diseases in an individual.</div></front></TEI><affiliations><list><country><li>Canada</li></country></list><tree><noCountry><name sortKey="Glogauer, Michael" sort="Glogauer, Michael" uniqKey="Glogauer M" first="Michael" last="Glogauer">Michael Glogauer</name></noCountry><country name="Canada"><noRegion><name sortKey="Sima, Corneliu" sort="Sima, Corneliu" uniqKey="Sima C" first="Corneliu" last="Sima">Corneliu Sima</name></noRegion></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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