Successful Rechallenge with Imatinib in a Patient with Chronic Myeloid Leukemia Who Previously Experienced Imatinib Mesylate Induced Pneumonitis
Identifieur interne : 001224 ( Main/Exploration ); précédent : 001223; suivant : 001225Successful Rechallenge with Imatinib in a Patient with Chronic Myeloid Leukemia Who Previously Experienced Imatinib Mesylate Induced Pneumonitis
Auteurs : Seong Woo Go [Corée du Sud] ; Boo Kyeong Kim [Corée du Sud] ; Sung Hak Lee [Corée du Sud] ; Tae-Jung Kim [Corée du Sud] ; Joo Yeon Huh [Corée du Sud] ; Jong Min Lee [Corée du Sud] ; Jick Hwan Hah [Corée du Sud] ; Dong Whi Kim [Corée du Sud] ; Min Jung Cho [Corée du Sud] ; Tae Wan Kim [Corée du Sud] ; Ji Young Kang [Corée du Sud]Source :
- Tuberculosis and Respiratory Diseases [ 1738-3536 ] ; 2013.
Abstract
Imatinib mesylate is a targeted therapy that acts by inhibiting tyrosine kinase of the bcr-abl fusion oncoprotein, which is specific to chronic myeloid leukemia (CML), and the c-transmembrane receptor, which is specific to gastrointestinal stromal tumors. Interstitial pneumonitis is a rare adverse event of imatinib therapy. It is clinically difficult to distinguish from infectious pneumonia, which can frequently occur due to the underlying disease. The standard treatment for imatinib-induced pneumonitis is to discontinue the medication and optionally administer corticosteroids. However, there are a few cases of successful retrial with imatinib. We describe a case of successful rechallenge of imatinib in a patient with imatinib-induced interstitial pneumonitis and CML without a recurrence of the underlying disease after 3 months of follow-up.
Url:
DOI: 10.4046/trd.2013.75.6.256
PubMed: 24416057
PubMed Central: 3884114
Affiliations:
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<front><div type="abstract" xml:lang="en"><p>Imatinib mesylate is a targeted therapy that acts by inhibiting tyrosine kinase of the bcr-abl fusion oncoprotein, which is specific to chronic myeloid leukemia (CML), and the c-transmembrane receptor, which is specific to gastrointestinal stromal tumors. Interstitial pneumonitis is a rare adverse event of imatinib therapy. It is clinically difficult to distinguish from infectious pneumonia, which can frequently occur due to the underlying disease. The standard treatment for imatinib-induced pneumonitis is to discontinue the medication and optionally administer corticosteroids. However, there are a few cases of successful retrial with imatinib. We describe a case of successful rechallenge of imatinib in a patient with imatinib-induced interstitial pneumonitis and CML without a recurrence of the underlying disease after 3 months of follow-up.</p>
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<tree><country name="Corée du Sud"><region name="Région capitale de Séoul"><name sortKey="Go, Seong Woo" sort="Go, Seong Woo" uniqKey="Go S" first="Seong Woo" last="Go">Seong Woo Go</name>
</region>
<name sortKey="Cho, Min Jung" sort="Cho, Min Jung" uniqKey="Cho M" first="Min Jung" last="Cho">Min Jung Cho</name>
<name sortKey="Hah, Jick Hwan" sort="Hah, Jick Hwan" uniqKey="Hah J" first="Jick Hwan" last="Hah">Jick Hwan Hah</name>
<name sortKey="Huh, Joo Yeon" sort="Huh, Joo Yeon" uniqKey="Huh J" first="Joo Yeon" last="Huh">Joo Yeon Huh</name>
<name sortKey="Kang, Ji Young" sort="Kang, Ji Young" uniqKey="Kang J" first="Ji Young" last="Kang">Ji Young Kang</name>
<name sortKey="Kim, Boo Kyeong" sort="Kim, Boo Kyeong" uniqKey="Kim B" first="Boo Kyeong" last="Kim">Boo Kyeong Kim</name>
<name sortKey="Kim, Dong Whi" sort="Kim, Dong Whi" uniqKey="Kim D" first="Dong Whi" last="Kim">Dong Whi Kim</name>
<name sortKey="Kim, Tae Jung" sort="Kim, Tae Jung" uniqKey="Kim T" first="Tae-Jung" last="Kim">Tae-Jung Kim</name>
<name sortKey="Kim, Tae Wan" sort="Kim, Tae Wan" uniqKey="Kim T" first="Tae Wan" last="Kim">Tae Wan Kim</name>
<name sortKey="Lee, Jong Min" sort="Lee, Jong Min" uniqKey="Lee J" first="Jong Min" last="Lee">Jong Min Lee</name>
<name sortKey="Lee, Sung Hak" sort="Lee, Sung Hak" uniqKey="Lee S" first="Sung Hak" last="Lee">Sung Hak Lee</name>
</country>
</tree>
</affiliations>
</record>
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