Abstract 36. In vivo and in vitro models of demyelinating disease: factors influencing the disease process caused by coronavirus infection of the rat
Identifieur interne : 001D80 ( Main/Exploration ); précédent : 001D79; suivant : 001D81Abstract 36. In vivo and in vitro models of demyelinating disease: factors influencing the disease process caused by coronavirus infection of the rat
Auteurs : O. Sorensen [Canada] ; S. Beushausen [Canada] ; M. Coulter-Mackie [Canada] ; S. Dales [Canada]Source :
- Progress in Neuropsychopharmacology & Biological Psychiatry [ 0278-5846 ] ; 1985.
English descriptors
- Teeft :
- Adoptive, Adoptive transfer, Alterin, Amyelinrelated, Amyelinrelated enzyme, Apparent neurologic disease, Astrocyte, Autoimmune, Autoimmune reaction, Beushausen, Brain antigen, Cdna, Cephalomyelitis, Cerebrospinal, Cerebrospinal fluid, Cess, Chronic disease, Clnd, Coincident, Corona virus antigen, Coronaations, Coronavirus, Coronavirus infection, Coronaviruses, Dales university, Demyelinating, Demyelinating disease, Demyelinative, Demyelinative disease process, Difcytes, Different stages, Different tissues, Disease process, Diseased rats, Ection, Encephalomyelitis, Enhigh, Enhigh cephalomyelitis, Explanted, Explanted cells, Ferences, Fresh demyelinating lesions, Genetic background, Glia, Histological, Histologically, Host cell, Host control, Immune, Immune system, Immunity clnd, Immunofluorescent, Immunofluorescent microscopy, Inbred, Incubation periods, Infection, Inoculated, Insusceptibility, Intrathecal, Jhmv, Jhmv cdna probes, Latent state, Latter interaction, Lesion, Local antibody production, Logic symptoms, Manifest predilthe murine viserotropic strain, Mhv3, Murine, Myelin, Neonatal rats, Neurologic, Neurological, Neurological changes, Neurological disease, Neurothe, Neurothe infection, Neurotropic, Oligodendrocyte, Oligodendrocyte differentiation, Oligodendrocytes influence, Oligodendrowith, Other thogenetic importance, Paralytic, Paralytic disease, Particular interest, Pathis, Pathis phenomenon, Persistent infection, Predilthe, Previous observations, Programme, Prothe, Provi, Rats jhmv, Recognizable tissue damage, Relapsing, Relapsing course, Remission, Replicative, Replicative strategy, Serotype, Serotype specificity, Slower prothe, Small difcytes, Sorensen, Spatial density, Spleen cells, Structural alterin coronaations, Subacute, Subacute demyelinating encephalomyelitis, Subsequent induction, Such cells causes, Temporal programme, Thogenetic, Tive, Tropism, Unambiguous tropism, Viral, Virus infection, Virus infections, Virus tive, Viserotropic, Western ontario.
Url:
DOI: 10.1016/0278-5846(85)90131-9
Affiliations:
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Sorensen</name></region><name sortKey="Beushausen, S" sort="Beushausen, S" uniqKey="Beushausen S" first="S." last="Beushausen">S. Beushausen</name><name sortKey="Coulter Mackie, M" sort="Coulter Mackie, M" uniqKey="Coulter Mackie M" first="M." last="Coulter-Mackie">M. Coulter-Mackie</name><name sortKey="Dales, S" sort="Dales, S" uniqKey="Dales S" first="S." last="Dales">S. Dales</name></country></tree></affiliations></record>Pour manipuler ce document sous Unix (Dilib)
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