Experimental autoimmune uveoretinitis (EAU) versus experimental allergic encephalomyelitis (EAE): a comparison of T cell-mediated mechanisms.
Identifieur interne : 000280 ( Pmc/Corpus ); précédent : 000279; suivant : 000281Experimental autoimmune uveoretinitis (EAU) versus experimental allergic encephalomyelitis (EAE): a comparison of T cell-mediated mechanisms.
Auteurs : V L Calder ; S L LightmanSource :
- Clinical and Experimental Immunology [ 0009-9104 ] ; 1992.
Abstract
EAU is a model of ocular inflammatory disease. EAU resembles another T cell-mediated autoimmune disease--experimental allergic encephalomyelitis--since both have increased expression of MHC class II molecules in the target tissue, can be adoptively transferred by activated CD4+ T cells and are inhibited by cyclosporin A. The immunological findings will be compared to find out if the same cellular mechanisms are involved in both diseases.
Url:
PubMed: 1638760
PubMed Central: 1554436
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PMC:1554436Le document en format XML
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<author><name sortKey="Lightman, S L" sort="Lightman, S L" uniqKey="Lightman S" first="S L" last="Lightman">S L Lightman</name>
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<series><title level="j">Clinical and Experimental Immunology</title>
<idno type="ISSN">0009-9104</idno>
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<front><div type="abstract" xml:lang="en"><p>EAU is a model of ocular inflammatory disease. EAU resembles another T cell-mediated autoimmune disease--experimental allergic encephalomyelitis--since both have increased expression of MHC class II molecules in the target tissue, can be adoptively transferred by activated CD4+ T cells and are inhibited by cyclosporin A. The immunological findings will be compared to find out if the same cellular mechanisms are involved in both diseases.</p>
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<pmc article-type="review-article"><pmc-comment>The publisher of this article does not allow downloading of the full text in XML form.</pmc-comment>
<front><journal-meta><journal-id journal-id-type="nlm-ta">Clin Exp Immunol</journal-id>
<journal-title>Clinical and Experimental Immunology</journal-title>
<issn pub-type="ppub">0009-9104</issn>
<issn pub-type="epub">1365-2249</issn>
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<article-meta><article-id pub-id-type="pmid">1638760</article-id>
<article-id pub-id-type="pmc">1554436</article-id>
<article-categories><subj-group subj-group-type="heading"><subject>Research Article</subject>
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<title-group><article-title>Experimental autoimmune uveoretinitis (EAU) versus experimental allergic encephalomyelitis (EAE): a comparison of T cell-mediated mechanisms.</article-title>
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<contrib-group><contrib contrib-type="author"><name><surname>Calder</surname>
<given-names>V L</given-names>
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<contrib contrib-type="author"><name><surname>Lightman</surname>
<given-names>S L</given-names>
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<aff>Department of Clinical Science, Institute of Ophthalmology, London, UK.</aff>
<pub-date pub-type="ppub"><month>8</month>
<year>1992</year>
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<volume>89</volume>
<issue>2</issue>
<fpage>165</fpage>
<lpage>169</lpage>
<abstract><p>EAU is a model of ocular inflammatory disease. EAU resembles another T cell-mediated autoimmune disease--experimental allergic encephalomyelitis--since both have increased expression of MHC class II molecules in the target tissue, can be adoptively transferred by activated CD4+ T cells and are inhibited by cyclosporin A. The immunological findings will be compared to find out if the same cellular mechanisms are involved in both diseases.</p>
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