Antimalarial lichenoid tissue reactions in patients with pre-existing lupus erythematosus
Identifieur interne : 002020 ( Main/Curation ); précédent : 002019; suivant : 002021Antimalarial lichenoid tissue reactions in patients with pre-existing lupus erythematosus
Auteurs : P. Geraminejad [États-Unis] ; M S Stone [États-Unis] ; R D Sontheimer [États-Unis]Source :
- Lupus [ 0961-2033 ] ; 2004-06.
English descriptors
- Teeft :
- Acad dermatol, Aminoquinolone antimalarials, Antimalarial, Antimalarial lichenoid tissue reactions, Apoptosis, Arch dermatol, Basal, Basal vacuolopathy, Cutaneous, Cutaneous lupus erythematosus, Dermatitis, Dermatol, Eruption, Erythematosus, Hydroxychloroquine, Idiopathic, Idiopathic cutaneous, Keratinocyte, Keratinocytes, Lesion, Lichenoid, Lichenoid drug eruptions, Lichenoid drug reactions, Lichenoid tissue reaction, Lupus, Medication, Other medications, Paucicellular interface dermatitis, Quinacrine, Skin disease, Skin lesions, Subacute, Subacute cutaneous lupus erythematosus, Systemic lupus erythematosus.
Abstract
Recently a number of cases of drug induction or exacerbation of lupus erythematosus (LE) specific skin disease have been described in the literature. Many of the responsible medications are also known for their ability to induce a lichenoid tissue reaction. Aminoquinoline antimalarials are currently the first line of therapy in cutaneous LE specific skin disease. Lichenoid tissue reactions are among the most common cutaneous side effects of aminoquinolone antimalarials. We report three cases of aminoquinolone antimalarial induced or exacerbated LE specific skin disease. We also review the pathophysiology of LE specific skin disease and propose a mechanism by which induction of the lichenoid tissue reaction may result in Koebnerization of LE specific skin lesions.
Url:
DOI: 10.1191/0961203304lu1056cr
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<term>Aminoquinolone antimalarials</term>
<term>Antimalarial</term>
<term>Antimalarial lichenoid tissue reactions</term>
<term>Apoptosis</term>
<term>Arch dermatol</term>
<term>Basal</term>
<term>Basal vacuolopathy</term>
<term>Cutaneous</term>
<term>Cutaneous lupus erythematosus</term>
<term>Dermatitis</term>
<term>Dermatol</term>
<term>Eruption</term>
<term>Erythematosus</term>
<term>Hydroxychloroquine</term>
<term>Idiopathic</term>
<term>Idiopathic cutaneous</term>
<term>Keratinocyte</term>
<term>Keratinocytes</term>
<term>Lesion</term>
<term>Lichenoid</term>
<term>Lichenoid drug eruptions</term>
<term>Lichenoid drug reactions</term>
<term>Lichenoid tissue reaction</term>
<term>Lupus</term>
<term>Medication</term>
<term>Other medications</term>
<term>Paucicellular interface dermatitis</term>
<term>Quinacrine</term>
<term>Skin disease</term>
<term>Skin lesions</term>
<term>Subacute</term>
<term>Subacute cutaneous lupus erythematosus</term>
<term>Systemic lupus erythematosus</term>
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<front><div type="abstract" xml:lang="en">Recently a number of cases of drug induction or exacerbation of lupus erythematosus (LE) specific skin disease have been described in the literature. Many of the responsible medications are also known for their ability to induce a lichenoid tissue reaction. Aminoquinoline antimalarials are currently the first line of therapy in cutaneous LE specific skin disease. Lichenoid tissue reactions are among the most common cutaneous side effects of aminoquinolone antimalarials. We report three cases of aminoquinolone antimalarial induced or exacerbated LE specific skin disease. We also review the pathophysiology of LE specific skin disease and propose a mechanism by which induction of the lichenoid tissue reaction may result in Koebnerization of LE specific skin lesions.</div>
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