Pauci-immune crescentic glomerulonephritis in limited cutaneous systemic sclerosis
Identifieur interne : 001E70 ( Istex/Corpus ); précédent : 001E69; suivant : 001E71Pauci-immune crescentic glomerulonephritis in limited cutaneous systemic sclerosis
Auteurs : P. T. Chan ; C. C. MokSource :
- Clinical Rheumatology [ 0770-3198 ] ; 2012-08-01.
English descriptors
- KwdEn :
Abstract
Abstract: Pauci-immune necrotizing glomerulonephritis is a form of a rapidly progressive glomerulonephritis that is associated with the anti-neutrophil cytoplasmic antibodies (ANCA). ANCA-associated pauci-immune glomerulonephritis may be secondary to rheumatic diseases. We report on a patient with limited cutaneous systemic sclerosis who presented with subacute onset of nephritic syndrome and renal insufficiency. Her myeloperioxidase ANCA titer was elevated and renal biopsy confirmed pauci-immune crescentic glomerulonephritis, which was stabilized with high-dose glucocorticoid and cyclophosphamide. ANCA-associated necrotizing glomerulonephritis is rarely reported in systemic sclerosis and a systematic review is presented.
Url:
DOI: 10.1007/s10067-012-2010-8
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ISTEX:B307FE4C69CBB600642596BF10C63007ED15C1D4Le document en format XML
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T." last="Chan">P. T. Chan</name><affiliation><mods:affiliation>Department of Medicine, Tuen Mun Hospital and Center for Assessment and Treatment of Rheumatic Diseases, Pok Oi Hospital, New Territories, Hong Kong, SAR, China</mods:affiliation></affiliation></author><author><name sortKey="Mok, C C" sort="Mok, C C" uniqKey="Mok C" first="C. C." last="Mok">C. C. 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ANCA-associated pauci-immune glomerulonephritis may be secondary to rheumatic diseases. We report on a patient with limited cutaneous systemic sclerosis who presented with subacute onset of nephritic syndrome and renal insufficiency. Her myeloperioxidase ANCA titer was elevated and renal biopsy confirmed pauci-immune crescentic glomerulonephritis, which was stabilized with high-dose glucocorticoid and cyclophosphamide. ANCA-associated necrotizing glomerulonephritis is rarely reported in systemic sclerosis and a systematic review is presented.</div></front></TEI><istex><corpusName>springer-journals</corpusName><author><json:item><name>P. T. Chan</name><affiliations><json:string>Department of Medicine, Tuen Mun Hospital and Center for Assessment and Treatment of Rheumatic Diseases, Pok Oi Hospital, New Territories, Hong Kong, SAR, China</json:string></affiliations></json:item><json:item><name>C. C. 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ANCA-associated pauci-immune glomerulonephritis may be secondary to rheumatic diseases. We report on a patient with limited cutaneous systemic sclerosis who presented with subacute onset of nephritic syndrome and renal insufficiency. Her myeloperioxidase ANCA titer was elevated and renal biopsy confirmed pauci-immune crescentic glomerulonephritis, which was stabilized with high-dose glucocorticoid and cyclophosphamide. ANCA-associated necrotizing glomerulonephritis is rarely reported in systemic sclerosis and a systematic review is presented.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>ANCA</term></item><item><term>Glomerulonephropathy</term></item><item><term>Nephritis</term></item><item><term>Nephropathy</term></item><item><term>Renal failure</term></item><item><term>Scleroderma</term></item></list></keywords></textClass><textClass><keywords scheme="Journal-Subject-Collection"><list><label>Medicine</label><item><term>SC11</term></item></list></keywords></textClass><textClass><keywords scheme="Journal Subject"><list><head>Medicine & Public Health</head><item><term>Rheumatology</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="2012-04-23">Created</change><change 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DisplayOrder="Western"><GivenName>P.</GivenName><GivenName>T.</GivenName><FamilyName>Chan</FamilyName></AuthorName></Author><Author AffiliationIDS="Aff1 Aff2" CorrespondingAffiliationID="Aff2"><AuthorName DisplayOrder="Western"><GivenName>C.</GivenName><GivenName>C.</GivenName><FamilyName>Mok</FamilyName></AuthorName><Contact><Phone>+852-2468-5386</Phone><Fax>+852-2456-9100</Fax><Email>ccmok2005@yahoo.com</Email></Contact></Author><Affiliation ID="Aff1"><OrgDivision>Department of Medicine, Tuen Mun Hospital and Center for Assessment and Treatment of Rheumatic Diseases</OrgDivision><OrgName>Pok Oi Hospital</OrgName><OrgAddress><Street>New Territories</Street><City>Hong Kong</City><State>SAR</State><Country Code="CN">China</Country></OrgAddress></Affiliation><Affiliation ID="Aff2"><OrgDivision>Department of Medicine</OrgDivision><OrgName>Tuen Mun Hospital</OrgName><OrgAddress><Street>Tsing Chung Koon Road, New Territories</Street><City>Hong Kong</City><State>SAR</State><Country Code="CN">China</Country></OrgAddress></Affiliation></AuthorGroup><Abstract ID="Abs1" Language="En" OutputMedium="All"><Heading>Abstract</Heading><Para>Pauci-immune necrotizing glomerulonephritis is a form of a rapidly progressive glomerulonephritis that is associated with the anti-neutrophil cytoplasmic antibodies (ANCA). ANCA-associated pauci-immune glomerulonephritis may be secondary to rheumatic diseases. We report on a patient with limited cutaneous systemic sclerosis who presented with subacute onset of nephritic syndrome and renal insufficiency. Her myeloperioxidase ANCA titer was elevated and renal biopsy confirmed pauci-immune crescentic glomerulonephritis, which was stabilized with high-dose glucocorticoid and cyclophosphamide. ANCA-associated necrotizing glomerulonephritis is rarely reported in systemic sclerosis and a systematic review is presented.</Para></Abstract><KeywordGroup Language="En" OutputMedium="All"><Heading>Keywords</Heading><Keyword>ANCA</Keyword><Keyword>Glomerulonephropathy</Keyword><Keyword>Nephritis</Keyword><Keyword>Nephropathy</Keyword><Keyword>Renal failure</Keyword><Keyword>Scleroderma</Keyword></KeywordGroup></ArticleHeader><NoBody></NoBody></Article></Issue></Volume></Journal></Publisher></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Pauci-immune crescentic glomerulonephritis in limited cutaneous systemic sclerosis</title></titleInfo><titleInfo type="alternative" contentType="CDATA"><title>Pauci-immune crescentic glomerulonephritis in limited cutaneous systemic sclerosis</title></titleInfo><name type="personal"><namePart type="given">P.</namePart><namePart type="given">T.</namePart><namePart type="family">Chan</namePart><affiliation>Department of Medicine, Tuen Mun Hospital and Center for Assessment and Treatment of Rheumatic Diseases, Pok Oi Hospital, New Territories, Hong Kong, SAR, China</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal" displayLabel="corresp"><namePart type="given">C.</namePart><namePart type="given">C.</namePart><namePart type="family">Mok</namePart><affiliation>Department of Medicine, Tuen Mun Hospital and Center for Assessment and Treatment of Rheumatic Diseases, Pok Oi Hospital, New Territories, Hong Kong, SAR, China</affiliation><affiliation>Department of Medicine, Tuen Mun Hospital, Tsing Chung Koon Road, New Territories, Hong Kong, SAR, China</affiliation><affiliation>E-mail: ccmok2005@yahoo.com</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="review-article" displayLabel="ReviewPaper" authority="ISTEX" authorityURI="https://content-type.data.istex.fr" valueURI="https://content-type.data.istex.fr/ark:/67375/XTP-L5L7X3NF-P">review-article</genre><originInfo><publisher>Springer-Verlag</publisher><place><placeTerm type="text">London</placeTerm></place><dateCreated encoding="w3cdtf">2012-04-23</dateCreated><dateIssued encoding="w3cdtf">2012-08-01</dateIssued><copyrightDate encoding="w3cdtf">2012</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><abstract lang="en">Abstract: Pauci-immune necrotizing glomerulonephritis is a form of a rapidly progressive glomerulonephritis that is associated with the anti-neutrophil cytoplasmic antibodies (ANCA). ANCA-associated pauci-immune glomerulonephritis may be secondary to rheumatic diseases. We report on a patient with limited cutaneous systemic sclerosis who presented with subacute onset of nephritic syndrome and renal insufficiency. Her myeloperioxidase ANCA titer was elevated and renal biopsy confirmed pauci-immune crescentic glomerulonephritis, which was stabilized with high-dose glucocorticoid and cyclophosphamide. ANCA-associated necrotizing glomerulonephritis is rarely reported in systemic sclerosis and a systematic review is presented.</abstract><note>Case Based Review</note><subject lang="en"><genre>Keywords</genre><topic>ANCA</topic><topic>Glomerulonephropathy</topic><topic>Nephritis</topic><topic>Nephropathy</topic><topic>Renal failure</topic><topic>Scleroderma</topic></subject><relatedItem type="host"><titleInfo><title>Clinical Rheumatology</title><subTitle>Journal of the International League of Associations for Rheumatology</subTitle></titleInfo><titleInfo type="abbreviated"><title>Clin Rheumatol</title></titleInfo><genre type="journal" authority="ISTEX" authorityURI="https://publication-type.data.istex.fr" valueURI="https://publication-type.data.istex.fr/ark:/67375/JMC-0GLKJH51-B">journal</genre><originInfo><publisher>Springer</publisher><dateIssued encoding="w3cdtf">2012-07-31</dateIssued><copyrightDate encoding="w3cdtf">2012</copyrightDate></originInfo><subject><genre>Journal-Subject-Collection</genre><topic authority="SpringerSubjectCodes" authorityURI="Medicine">SC11</topic></subject><subject><genre>Medicine & Public Health</genre><topic>Rheumatology</topic></subject><identifier type="ISSN">0770-3198</identifier><identifier type="eISSN">1434-9949</identifier><identifier type="JournalID">10067</identifier><identifier type="IssueArticleCount">20</identifier><identifier type="VolumeIssueCount">12</identifier><part><date>2012</date><detail type="volume"><number>31</number><caption>vol.</caption></detail><detail type="issue"><number>8</number><caption>no.</caption></detail><extent unit="pages"><start>1273</start><end>1277</end></extent></part><recordInfo><recordOrigin>Clinical Rheumatology, 2012</recordOrigin></recordInfo></relatedItem><identifier type="istex">B307FE4C69CBB600642596BF10C63007ED15C1D4</identifier><identifier type="ark">ark:/67375/VQC-L4PKN8Z7-V</identifier><identifier type="DOI">10.1007/s10067-012-2010-8</identifier><identifier type="ArticleID">2010</identifier><identifier type="ArticleID">s10067-012-2010-8</identifier><accessCondition type="use and reproduction" contentType="copyright">Clinical Rheumatology, 2012</accessCondition><recordInfo><recordContentSource authority="ISTEX" authorityURI="https://loaded-corpus.data.istex.fr" valueURI="https://loaded-corpus.data.istex.fr/ark:/67375/XBH-3XSW68JL-F">springer</recordContentSource><recordOrigin>Clinical Rheumatology, 2012</recordOrigin></recordInfo></mods><json:item><extension>json</extension><original>false</original><mimetype>application/json</mimetype><uri>https://api.istex.fr/ark:/67375/VQC-L4PKN8Z7-V/record.json</uri></json:item></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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