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Pulmonary hypertension in a patient with adult-onset stills disease

Identifieur interne : 001678 ( Istex/Corpus ); précédent : 001677; suivant : 001679

Pulmonary hypertension in a patient with adult-onset stills disease

Auteurs : Eisha Mubashir ; M. Mubashir Ahmed ; Samina Hayat ; Maureen Heldmann ; Seth Mark Berney

Source :

RBID : ISTEX:4A0377576EB43DDA444A13ACCF3813D9EE6A8B40

English descriptors

Abstract

Abstract: Pulmonary manifestations of adult-onset Still’s disease (AOSD) include aseptic pneumonitis, pleural effusions, rarely acute respiratory distress syndrome, and restrictive lung disease. Pulmonary arterial hypertension (PAH) occurs with several rheumatologic diseases, however, has only been reported once in AOSD. We describe a 29-year-old woman with a 9-year history of AOSD, who developed PAH without any other obvious cause. Therefore, we conclude that this is likely a result of pulmonary vascular changes related to AOSD.

Url:
DOI: 10.1007/s10067-006-0382-3

Links to Exploration step

ISTEX:4A0377576EB43DDA444A13ACCF3813D9EE6A8B40

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<FamilyName>Mubashir</FamilyName>
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<Para>Pulmonary manifestations of adult-onset Still’s disease (AOSD) include aseptic pneumonitis, pleural effusions, rarely acute respiratory distress syndrome, and restrictive lung disease. Pulmonary arterial hypertension (PAH) occurs with several rheumatologic diseases, however, has only been reported once in AOSD. We describe a 29-year-old woman with a 9-year history of AOSD, who developed PAH without any other obvious cause. Therefore, we conclude that this is likely a result of pulmonary vascular changes related to AOSD.</Para>
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<abstract lang="en">Abstract: Pulmonary manifestations of adult-onset Still’s disease (AOSD) include aseptic pneumonitis, pleural effusions, rarely acute respiratory distress syndrome, and restrictive lung disease. Pulmonary arterial hypertension (PAH) occurs with several rheumatologic diseases, however, has only been reported once in AOSD. We describe a 29-year-old woman with a 9-year history of AOSD, who developed PAH without any other obvious cause. Therefore, we conclude that this is likely a result of pulmonary vascular changes related to AOSD.</abstract>
<note>Case Report</note>
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