Urticarial venulitis
Identifieur interne : 000D17 ( Istex/Corpus ); précédent : 000D16; suivant : 000D18Urticarial venulitis
Auteurs : Nicholas A. SoterSource :
- Dermatologic Therapy [ 1396-0296 ] ; 2000-12.
Abstract
ABSTRACT: Episodes of recurrent urticaria and angioedema may be a clinical manifestation of cutaneous necrotizing venulitis. Known as urticarial vasculitis, this edematous form of necrotizing venulitis occurs in patients with serum sickness, connective tissue disorders, hematologic malignant conditions, an IgMKM component, infections, or physical urticarias after the administration of therapeutic agents, and as an idiopathic disorder. The skin lesions appear as erythematous, occasionally indurated wheals which may contain foci of purpura. Although the individual urticarial lesions may last for less than 24 hours, they often persist up to 3–5 days. The episodes of urticaria are chronic; approximately 70% of the afflicted individuals are women. General features include fever, malaise, and myalgia. Specific organ involvement includes enlargement of the lymph nodes, liver, and spleen and involvement of the synovia, kidneys, gastrointestinal tract, respiratory tract, eyes, and central nervous system. In the treatment of urticarial vasculitis there are no double‐blind, placebo‐controlled trials. Reports exist of the treatment of patients with H1 antihistamines, nonsteroidal anti‐inflammatory agents, colchicine, hydroxychloroquine, dapsone, prednisone, azathioprine, methotrexate, cyclophosphamide, and intramuscular gold therapy.
Url:
DOI: 10.1046/j.1529-8019.2000.00038.x
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ISTEX:415A01EC6476D1078C31B5C09B0599E7E2743469Le document en format XML
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Reports exist of the treatment of patients with H1 antihistamines, nonsteroidal anti‐inflammatory agents, colchicine, hydroxychloroquine, dapsone, prednisone, azathioprine, methotrexate, cyclophosphamide, and intramuscular gold therapy.</div></front></TEI><istex><corpusName>wiley</corpusName><keywords><teeft><json:string>vasculitis</json:string><json:string>urticarial</json:string><json:string>dermatol</json:string><json:string>urticaria</json:string><json:string>urticarial vasculitis</json:string><json:string>venulitis</json:string><json:string>hypocomplementemic</json:string><json:string>hypocomplementemia</json:string><json:string>cutaneous</json:string><json:string>necrotizing</json:string><json:string>urticarial venulitis</json:string><json:string>clin</json:string><json:string>idiopathic</json:string><json:string>soter</json:string><json:string>lupus</json:string><json:string>erythematosus</json:string><json:string>syndrome</json:string><json:string>immune</json:string><json:string>immunol</json:string><json:string>systemic lupus erythematosus</json:string><json:string>acad</json:string><json:string>acad dermatol</json:string><json:string>dapsone</json:string><json:string>colchicine</json:string><json:string>arch dermatol</json:string><json:string>allergy</json:string><json:string>necrotizing vasculitis</json:string><json:string>glomerulonephritis</json:string><json:string>chronic urticaria</json:string><json:string>immune complex</json:string><json:string>hypocomplementemic urticarial vasculitis syndrome</json:string><json:string>skin lesion</json:string><json:string>allergy clin immunol</json:string><json:string>cutaneous vasculitis</json:string><json:string>arthritis rheum</json:string><json:string>lesion</json:string><json:string>renal</json:string><json:string>idiopathic urticarial vasculitis</json:string><json:string>necrotizing venulitis</json:string><json:string>extracutaneous manifestation</json:string><json:string>nonsteroidal agent</json:string><json:string>chronic idiopathic urticaria</json:string><json:string>hypocomplementemic urticarial vasculitis</json:string><json:string>pseudotumor cerebri</json:string><json:string>mayo clin proc</json:string><json:string>serum hypocomplementemia</json:string><json:string>hypocomplementemic vasculitis</json:string><json:string>renal involvement</json:string><json:string>pulmonary disease</json:string><json:string>biopsy specimen</json:string><json:string>respiratory tract</json:string><json:string>cold urticaria</json:string><json:string>physical urticaria</json:string><json:string>laryngeal edema</json:string><json:string>systemic</json:string><json:string>biopsy</json:string></teeft></keywords><author><json:item><name>Nicholas A. 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General features include fever, malaise, and myalgia. Specific organ involvement includes enlargement of the lymph nodes, liver, and spleen and involvement of the synovia, kidneys, gastrointestinal tract, respiratory tract, eyes, and central nervous system. In the treatment of urticarial vasculitis there are no double‐blind, placebo‐controlled trials. Reports exist of the treatment of patients with H<hi rend="subscript">1</hi> antihistamines, nonsteroidal anti‐inflammatory agents, colchicine, hydroxychloroquine, dapsone, prednisone, azathioprine, methotrexate, cyclophosphamide, and intramuscular gold therapy.</p></abstract><textClass><keywords xml:lang="en"><term xml:id="k1">connective tissue disease</term><term xml:id="k2">necrotizing vasculitis</term><term xml:id="k3">obstructive pulmonary disease</term><term xml:id="k4">urticarial vasculitis</term></keywords><keywords rend="tocHeading1"><term>Original Articles</term></keywords></textClass><langUsage><language ident="en"></language></langUsage></profileDesc><revisionDesc><change when="2019-12-20" who="#istex" xml:id="pub2tei">formatting</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/ark:/67375/WNG-J2H45B4P-4/fulltext.txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Science Inc</publisherName><publisherLoc>Boston, MA, USA</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1529-8019</doi><issn type="print">1396-0296</issn><issn type="electronic">1529-8019</issn><idGroup><id type="product" value="DTH"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="DERMATOLOGIC THERAPY">Dermatologic Therapy</title><title type="short">Dermatologic Therapy</title></titleGroup></publicationMeta><publicationMeta level="part" position="12004"><doi origin="wiley">10.1111/dth.2000.13.issue-4</doi><numberingGroup><numbering type="journalVolume" number="13">13</numbering><numbering type="journalIssue" number="4">4</numbering></numberingGroup><coverDate startDate="2000-12">December 2000</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0040000" status="forIssue"><doi origin="wiley">10.1046/j.1529-8019.2000.00038.x</doi><idGroup><id type="unit" value="DTH00038"></id><id type="supplier" value="dth00038"></id></idGroup><countGroup><count type="pageTotal" number="9"></count></countGroup><titleGroup><title type="tocHeading1">Original Articles</title></titleGroup><eventGroup><event type="firstOnline" date="2008-07-07"></event><event type="publishedOnlineFinalForm" date="2008-07-07"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.4 mode:FullText source:FullText result:FullText" date="2010-03-30"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-01-17"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-16"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="400">400</numbering><numbering type="pageLast" number="408">408</numbering></numberingGroup><correspondenceTo>Address correspondence and reprint request to:Nicholas A. Soter, MD, The Ronald O. Perelman Department of Dermatology, New York University Medical Center, 550 First Avenue, New York, New York 10016 USA. 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Although the individual urticarial lesions may last for less than 24 hours, they often persist up to 3–5 days. The episodes of urticaria are chronic; approximately 70% of the afflicted individuals are women. General features include fever, malaise, and myalgia. Specific organ involvement includes enlargement of the lymph nodes, liver, and spleen and involvement of the synovia, kidneys, gastrointestinal tract, respiratory tract, eyes, and central nervous system. In the treatment of urticarial vasculitis there are no double‐blind, placebo‐controlled trials. Reports exist of the treatment of patients with H<sub>1</sub> antihistamines, nonsteroidal anti‐inflammatory agents, colchicine, hydroxychloroquine, dapsone, prednisone, azathioprine, methotrexate, cyclophosphamide, and intramuscular gold therapy.</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Urticarial venulitis</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Urticarial venulitis</title></titleInfo><name type="personal"><namePart type="given">Nicholas A.</namePart><namePart type="family">Soter</namePart><affiliation>Ronald O. Perelman Department of Dermatology, New York University School ofMedicine, New York, New York</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article" authority="ISTEX" authorityURI="https://content-type.data.istex.fr" valueURI="https://content-type.data.istex.fr/ark:/67375/XTP-6N5SZHKN-D">article</genre><originInfo><publisher>Blackwell Science Inc</publisher><place><placeTerm type="text">Boston, MA, USA</placeTerm></place><dateIssued encoding="w3cdtf">2000-12</dateIssued><copyrightDate encoding="w3cdtf">2000</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><extent unit="figures">3</extent><extent unit="tables">5</extent><extent unit="formulas">0</extent><extent unit="references">102</extent><extent unit="linksCrossRef">1</extent><extent unit="words">5202</extent></physicalDescription><abstract lang="en">ABSTRACT: Episodes of recurrent urticaria and angioedema may be a clinical manifestation of cutaneous necrotizing venulitis. Known as urticarial vasculitis, this edematous form of necrotizing venulitis occurs in patients with serum sickness, connective tissue disorders, hematologic malignant conditions, an IgMKM component, infections, or physical urticarias after the administration of therapeutic agents, and as an idiopathic disorder. The skin lesions appear as erythematous, occasionally indurated wheals which may contain foci of purpura. Although the individual urticarial lesions may last for less than 24 hours, they often persist up to 3–5 days. The episodes of urticaria are chronic; approximately 70% of the afflicted individuals are women. General features include fever, malaise, and myalgia. Specific organ involvement includes enlargement of the lymph nodes, liver, and spleen and involvement of the synovia, kidneys, gastrointestinal tract, respiratory tract, eyes, and central nervous system. In the treatment of urticarial vasculitis there are no double‐blind, placebo‐controlled trials. Reports exist of the treatment of patients with H1 antihistamines, nonsteroidal anti‐inflammatory agents, colchicine, hydroxychloroquine, dapsone, prednisone, azathioprine, methotrexate, cyclophosphamide, and intramuscular gold therapy.</abstract><subject lang="en"><genre>keywords</genre><topic>connective tissue disease</topic><topic>necrotizing vasculitis</topic><topic>obstructive pulmonary disease</topic><topic>urticarial vasculitis</topic></subject><relatedItem type="host"><titleInfo><title>Dermatologic Therapy</title></titleInfo><titleInfo type="abbreviated"><title>Dermatologic Therapy</title></titleInfo><genre type="journal" authority="ISTEX" authorityURI="https://publication-type.data.istex.fr" valueURI="https://publication-type.data.istex.fr/ark:/67375/JMC-0GLKJH51-B">journal</genre><identifier type="ISSN">1396-0296</identifier><identifier type="eISSN">1529-8019</identifier><identifier type="DOI">10.1111/(ISSN)1529-8019</identifier><identifier type="PublisherID">DTH</identifier><part><date>2000</date><detail type="volume"><caption>vol.</caption><number>13</number></detail><detail type="issue"><caption>no.</caption><number>4</number></detail><extent unit="pages"><start>400</start><end>408</end><total>9</total></extent></part></relatedItem><relatedItem type="references" displayLabel="cit1"><titleInfo><title>Precipitin reactions of the C1q component of complement with aggregated γ‐globulin and immune complexes in gel diffusion.</title></titleInfo><name type="personal"><namePart type="given">V</namePart><namePart type="family">Agnello</namePart><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">RJ</namePart><namePart type="family">Winchester</namePart><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">HG</namePart><namePart type="family">Kunkel</namePart><role><roleTerm type="text">author</roleTerm></role></name><genre>journal-article</genre><note type="citation/reference">Agnello V, Winchester RJ & Kunkel HG. 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