Necrosing livedo reticularis in a patient with recurrent pulmonary hemorrhage
Identifieur interne : 001721 ( Istex/Checkpoint ); précédent : 001720; suivant : 001722Necrosing livedo reticularis in a patient with recurrent pulmonary hemorrhage
Auteurs : David M. Aronoff ; Jeffrey P. CallenSource :
- Journal of the American Academy of Dermatology [ 0190-9622 ] ; 1997.
English descriptors
- Teeft :
- Alveolar hemorrhage, American academy, Anticardiolipin, Anticoagulant, Antiphospholipid, Antiphospholipid antibodies, Antiphospholipid antibody syndrome, Biopsy specimen, Bland thrombi, Chest examination, Chlorambucil, Cutaneous, Dermatol, Dermatology, Differential diagnosis, Hemorrhage, Hydroxychloroquine, Livedo, Livedo reticularis, Lower extremities, Lung hemorrhage, Lupus, Lupus anticoagulant, Necrosing, Necrosing livedo reticularis, Oral chlorambucil, Pulmonary capillaritis, Pulmonary emboli, Pulmonary hemorrhage, Renal, Reticularis, Rheumatol, Syndrome, Systemic lupus erythematosus, Thrombosis, Ulceration, Venous thrombosis.
Abstract
Abstract: The antiphospholipid antibody (APS) syndrome is characterized by antiphospholipid antibodies (lupus anticoagulant [LA] or anticardiolipin [aCL], a recurrent arterial and venous thrombosis, recurrent fetal loss, and thrombocytopenia. Pulmonary hemorrhage is an unusual complication. We describe a 32-year-old woman with a history of recurrent pulmonary hemorrhage and transient renal dysfunction. Her original diagnosis was Goodpasture’s syndrome, and she was treated with immunosuppressive drugs. Necrosing livedo reticularis of the legs subsequently developed. The presence of aCL and LA in the patient’s serum, the finding of noninflammatory microthrombi in the dermal capillaries, and the lack of laboratory or pathologic features of Goodpasture’s syndrome, confirmed a diagnosis of APS in this patient. (J Am Acad Dermatol 1997;37:300-2.)
Url:
DOI: 10.1016/S0190-9622(97)70010-0
Affiliations:
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<term>American academy</term>
<term>Anticardiolipin</term>
<term>Anticoagulant</term>
<term>Antiphospholipid</term>
<term>Antiphospholipid antibodies</term>
<term>Antiphospholipid antibody syndrome</term>
<term>Biopsy specimen</term>
<term>Bland thrombi</term>
<term>Chest examination</term>
<term>Chlorambucil</term>
<term>Cutaneous</term>
<term>Dermatol</term>
<term>Dermatology</term>
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<term>Hemorrhage</term>
<term>Hydroxychloroquine</term>
<term>Livedo</term>
<term>Livedo reticularis</term>
<term>Lower extremities</term>
<term>Lung hemorrhage</term>
<term>Lupus</term>
<term>Lupus anticoagulant</term>
<term>Necrosing</term>
<term>Necrosing livedo reticularis</term>
<term>Oral chlorambucil</term>
<term>Pulmonary capillaritis</term>
<term>Pulmonary emboli</term>
<term>Pulmonary hemorrhage</term>
<term>Renal</term>
<term>Reticularis</term>
<term>Rheumatol</term>
<term>Syndrome</term>
<term>Systemic lupus erythematosus</term>
<term>Thrombosis</term>
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<term>Venous thrombosis</term>
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<front><div type="abstract" xml:lang="en">Abstract: The antiphospholipid antibody (APS) syndrome is characterized by antiphospholipid antibodies (lupus anticoagulant [LA] or anticardiolipin [aCL], a recurrent arterial and venous thrombosis, recurrent fetal loss, and thrombocytopenia. Pulmonary hemorrhage is an unusual complication. We describe a 32-year-old woman with a history of recurrent pulmonary hemorrhage and transient renal dysfunction. Her original diagnosis was Goodpasture’s syndrome, and she was treated with immunosuppressive drugs. Necrosing livedo reticularis of the legs subsequently developed. The presence of aCL and LA in the patient’s serum, the finding of noninflammatory microthrombi in the dermal capillaries, and the lack of laboratory or pathologic features of Goodpasture’s syndrome, confirmed a diagnosis of APS in this patient. (J Am Acad Dermatol 1997;37:300-2.)</div>
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