Covid-19, ACE2, and the kidney.
Identifieur interne : 000234 ( Main/Exploration ); précédent : 000233; suivant : 000235Covid-19, ACE2, and the kidney.
Auteurs : Jan-Hendrik Hardenberg [Allemagne] ; Friedrich C. Luft [Allemagne]Source :
- Acta physiologica (Oxford, England) [ 1748-1716 ] ; 2020.
Abstract
We are confronted with the most dramatic pandemic world-wide for the past 100 years. We are armed "to-the-teeth" compared to 1918, we know the agent, the genomic sequence, the bodily entry, the proliferation rate, the damage pathogenesis, and the very nature of our enemy. We can identify its bodily presence and our resistance to it in terms of neutralizing antibody production. Nonetheless, the disease has laid lame the great nations of the current world and crippled the less fortunate countries. The primary disease features are not the kidney. However, the entry point has much to do with renal and cardiovascular disease. The kidney is a common target of corona-virus (SARS-CoV2) disease; the longer-term consequences could be as well.
DOI: 10.1111/apha.13539
PubMed: 32662161
Affiliations:
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