Treating type 2 diabetes in COVID-19 patients: the potential benefits of injective therapies.
Identifieur interne : 000019 ( Main/Curation ); précédent : 000018; suivant : 000020Treating type 2 diabetes in COVID-19 patients: the potential benefits of injective therapies.
Auteurs : Miriam Longo [Italie] ; Paola Caruso [Italie] ; Maria Ida Maiorino [Italie] ; Giuseppe Bellastella [Italie] ; Dario Giugliano [Italie] ; Katherine Esposito [Italie]Source :
- Cardiovascular diabetology [ 1475-2840 ] ; 2020.
Descripteurs français
- KwdFr :
- Appréciation des risques (MeSH), Betacoronavirus (pathogénicité), Diabète de type 2 (diagnostic), Diabète de type 2 (sang), Diabète de type 2 (traitement médicamenteux), Diabète de type 2 (épidémiologie), Facteurs de risque (MeSH), Glycémie (effets des médicaments et des substances chimiques), Glycémie (métabolisme), Humains (MeSH), Hypoglycémiants (administration et posologie), Hypoglycémiants (effets indésirables), Incrétines (administration et posologie), Incrétines (effets indésirables), Infections à coronavirus (diagnostic), Infections à coronavirus (thérapie), Infections à coronavirus (virologie), Infections à coronavirus (épidémiologie), Insuline (administration et posologie), Insuline (effets indésirables), Interactions hôte-microbes (MeSH), Marqueurs biologiques (sang), Pandémies (MeSH), Pneumopathie virale (diagnostic), Pneumopathie virale (thérapie), Pneumopathie virale (virologie), Pneumopathie virale (épidémiologie), Prise de décision clinique (MeSH), Récepteur du peptide-1 similaire au glucagon (agonistes), Résultat thérapeutique (MeSH).
- MESH :
- administration et posologie : Hypoglycémiants, Incrétines, Insuline.
- agonistes : Récepteur du peptide-1 similaire au glucagon.
- diagnostic : Diabète de type 2, Infections à coronavirus, Pneumopathie virale.
- effets des médicaments et des substances chimiques : Glycémie.
- effets indésirables : Hypoglycémiants, Incrétines, Insuline.
- métabolisme : Glycémie.
- pathogénicité : Betacoronavirus.
- sang : Diabète de type 2, Marqueurs biologiques.
- thérapie : Infections à coronavirus, Pneumopathie virale.
- traitement médicamenteux : Diabète de type 2.
- virologie : Infections à coronavirus, Pneumopathie virale.
- épidémiologie : Diabète de type 2, Infections à coronavirus, Pneumopathie virale.
- Appréciation des risques, Facteurs de risque, Humains, Interactions hôte-microbes, Pandémies, Prise de décision clinique, Résultat thérapeutique.
English descriptors
- KwdEn :
- Betacoronavirus (pathogenicity), Biomarkers (blood), Blood Glucose (drug effects), Blood Glucose (metabolism), Clinical Decision-Making (MeSH), Coronavirus Infections (diagnosis), Coronavirus Infections (epidemiology), Coronavirus Infections (therapy), Coronavirus Infections (virology), Diabetes Mellitus, Type 2 (blood), Diabetes Mellitus, Type 2 (diagnosis), Diabetes Mellitus, Type 2 (drug therapy), Diabetes Mellitus, Type 2 (epidemiology), Glucagon-Like Peptide-1 Receptor (agonists), Host Microbial Interactions (MeSH), Humans (MeSH), Hypoglycemic Agents (administration & dosage), Hypoglycemic Agents (adverse effects), Incretins (administration & dosage), Incretins (adverse effects), Insulin (administration & dosage), Insulin (adverse effects), Pandemics (MeSH), Pneumonia, Viral (diagnosis), Pneumonia, Viral (epidemiology), Pneumonia, Viral (therapy), Pneumonia, Viral (virology), Risk Assessment (MeSH), Risk Factors (MeSH), Treatment Outcome (MeSH).
- MESH :
- chemical , administration & dosage : Hypoglycemic Agents, Incretins, Insulin.
- chemical , adverse effects : Hypoglycemic Agents, Incretins, Insulin.
- chemical , agonists : Glucagon-Like Peptide-1 Receptor.
- chemical , blood : Biomarkers.
- chemical , drug effects : Blood Glucose.
- chemical , metabolism : Blood Glucose.
- blood : Diabetes Mellitus, Type 2.
- diagnosis : Coronavirus Infections, Diabetes Mellitus, Type 2, Pneumonia, Viral.
- drug therapy : Diabetes Mellitus, Type 2.
- epidemiology : Coronavirus Infections, Diabetes Mellitus, Type 2, Pneumonia, Viral.
- pathogenicity : Betacoronavirus.
- therapy : Coronavirus Infections, Pneumonia, Viral.
- virology : Coronavirus Infections, Pneumonia, Viral.
- Clinical Decision-Making, Host Microbial Interactions, Humans, Pandemics, Risk Assessment, Risk Factors, Treatment Outcome.
Abstract
The coronavirus disease 2019 (COVID-19) has been declared as pandemic by the World Health Organization and is causing substantial morbidity and mortality all over the world. Type 2 diabetes, hypertension, and cardiovascular disease significantly increase the risk for hospitalization and death in COVID-19 patients. Hypoglycemia and hyperglycemia are both predictors for adverse outcomes in hospitalized patients. An optimized glycemic control should be pursued in patients with diabetes and SARS-CoV-2 infection in order to reduce the risk of severe COVID-19 course. Both insulin and GLP-1RAs have shown optimal glucose-lowering and anti-inflammatory effects in type 2 diabetic patients and may represent a valid therapeutic option to treat asymptomatic and non-critically ill COVID-19 diabetic patients.
DOI: 10.1186/s12933-020-01090-9
PubMed: 32698837
PubMed Central: PMC7375203
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<term>Biomarkers (blood)</term>
<term>Blood Glucose (drug effects)</term>
<term>Blood Glucose (metabolism)</term>
<term>Clinical Decision-Making (MeSH)</term>
<term>Coronavirus Infections (diagnosis)</term>
<term>Coronavirus Infections (epidemiology)</term>
<term>Coronavirus Infections (therapy)</term>
<term>Coronavirus Infections (virology)</term>
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<term>Diabetes Mellitus, Type 2 (diagnosis)</term>
<term>Diabetes Mellitus, Type 2 (drug therapy)</term>
<term>Diabetes Mellitus, Type 2 (epidemiology)</term>
<term>Glucagon-Like Peptide-1 Receptor (agonists)</term>
<term>Host Microbial Interactions (MeSH)</term>
<term>Humans (MeSH)</term>
<term>Hypoglycemic Agents (administration & dosage)</term>
<term>Hypoglycemic Agents (adverse effects)</term>
<term>Incretins (administration & dosage)</term>
<term>Incretins (adverse effects)</term>
<term>Insulin (administration & dosage)</term>
<term>Insulin (adverse effects)</term>
<term>Pandemics (MeSH)</term>
<term>Pneumonia, Viral (diagnosis)</term>
<term>Pneumonia, Viral (epidemiology)</term>
<term>Pneumonia, Viral (therapy)</term>
<term>Pneumonia, Viral (virology)</term>
<term>Risk Assessment (MeSH)</term>
<term>Risk Factors (MeSH)</term>
<term>Treatment Outcome (MeSH)</term>
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<term>Betacoronavirus (pathogénicité)</term>
<term>Diabète de type 2 (diagnostic)</term>
<term>Diabète de type 2 (sang)</term>
<term>Diabète de type 2 (traitement médicamenteux)</term>
<term>Diabète de type 2 (épidémiologie)</term>
<term>Facteurs de risque (MeSH)</term>
<term>Glycémie (effets des médicaments et des substances chimiques)</term>
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<term>Hypoglycémiants (effets indésirables)</term>
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<term>Incrétines (effets indésirables)</term>
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<term>Infections à coronavirus (thérapie)</term>
<term>Infections à coronavirus (virologie)</term>
<term>Infections à coronavirus (épidémiologie)</term>
<term>Insuline (administration et posologie)</term>
<term>Insuline (effets indésirables)</term>
<term>Interactions hôte-microbes (MeSH)</term>
<term>Marqueurs biologiques (sang)</term>
<term>Pandémies (MeSH)</term>
<term>Pneumopathie virale (diagnostic)</term>
<term>Pneumopathie virale (thérapie)</term>
<term>Pneumopathie virale (virologie)</term>
<term>Pneumopathie virale (épidémiologie)</term>
<term>Prise de décision clinique (MeSH)</term>
<term>Récepteur du peptide-1 similaire au glucagon (agonistes)</term>
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<term>Insulin</term>
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<term>Incretins</term>
<term>Insulin</term>
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<term>Diabetes Mellitus, Type 2</term>
<term>Pneumonia, Viral</term>
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<term>Pneumopathie virale</term>
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<keywords scheme="MESH" qualifier="effets indésirables" xml:lang="fr"><term>Hypoglycémiants</term>
<term>Incrétines</term>
<term>Insuline</term>
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<keywords scheme="MESH" qualifier="epidemiology" xml:lang="en"><term>Coronavirus Infections</term>
<term>Diabetes Mellitus, Type 2</term>
<term>Pneumonia, Viral</term>
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</keywords>
<keywords scheme="MESH" qualifier="pathogenicity" xml:lang="en"><term>Betacoronavirus</term>
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<term>Marqueurs biologiques</term>
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<term>Pneumonia, Viral</term>
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<keywords scheme="MESH" qualifier="thérapie" xml:lang="fr"><term>Infections à coronavirus</term>
<term>Pneumopathie virale</term>
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<keywords scheme="MESH" qualifier="traitement médicamenteux" xml:lang="fr"><term>Diabète de type 2</term>
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<keywords scheme="MESH" qualifier="virologie" xml:lang="fr"><term>Infections à coronavirus</term>
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<term>Pneumonia, Viral</term>
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<keywords scheme="MESH" qualifier="épidémiologie" xml:lang="fr"><term>Diabète de type 2</term>
<term>Infections à coronavirus</term>
<term>Pneumopathie virale</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Clinical Decision-Making</term>
<term>Host Microbial Interactions</term>
<term>Humans</term>
<term>Pandemics</term>
<term>Risk Assessment</term>
<term>Risk Factors</term>
<term>Treatment Outcome</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Appréciation des risques</term>
<term>Facteurs de risque</term>
<term>Humains</term>
<term>Interactions hôte-microbes</term>
<term>Pandémies</term>
<term>Prise de décision clinique</term>
<term>Résultat thérapeutique</term>
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<front><div type="abstract" xml:lang="en">The coronavirus disease 2019 (COVID-19) has been declared as pandemic by the World Health Organization and is causing substantial morbidity and mortality all over the world. Type 2 diabetes, hypertension, and cardiovascular disease significantly increase the risk for hospitalization and death in COVID-19 patients. Hypoglycemia and hyperglycemia are both predictors for adverse outcomes in hospitalized patients. An optimized glycemic control should be pursued in patients with diabetes and SARS-CoV-2 infection in order to reduce the risk of severe COVID-19 course. Both insulin and GLP-1RAs have shown optimal glucose-lowering and anti-inflammatory effects in type 2 diabetic patients and may represent a valid therapeutic option to treat asymptomatic and non-critically ill COVID-19 diabetic patients.</div>
</front>
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<DateCompleted><Year>2020</Year>
<Month>07</Month>
<Day>29</Day>
</DateCompleted>
<DateRevised><Year>2020</Year>
<Month>07</Month>
<Day>29</Day>
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<PubDate><Year>2020</Year>
<Month>07</Month>
<Day>22</Day>
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<Title>Cardiovascular diabetology</Title>
<ISOAbbreviation>Cardiovasc Diabetol</ISOAbbreviation>
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<ArticleTitle>Treating type 2 diabetes in COVID-19 patients: the potential benefits of injective therapies.</ArticleTitle>
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<Abstract><AbstractText>The coronavirus disease 2019 (COVID-19) has been declared as pandemic by the World Health Organization and is causing substantial morbidity and mortality all over the world. Type 2 diabetes, hypertension, and cardiovascular disease significantly increase the risk for hospitalization and death in COVID-19 patients. Hypoglycemia and hyperglycemia are both predictors for adverse outcomes in hospitalized patients. An optimized glycemic control should be pursued in patients with diabetes and SARS-CoV-2 infection in order to reduce the risk of severe COVID-19 course. Both insulin and GLP-1RAs have shown optimal glucose-lowering and anti-inflammatory effects in type 2 diabetic patients and may represent a valid therapeutic option to treat asymptomatic and non-critically ill COVID-19 diabetic patients.</AbstractText>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Longo</LastName>
<ForeName>Miriam</ForeName>
<Initials>M</Initials>
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<AffiliationInfo><Affiliation>Division of Endocrinology and Metabolic Diseases, Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Piazza Luigi Miraglia 2, 80138, Naples, Italy. miriam.longo@unicampania.it.</Affiliation>
</AffiliationInfo>
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<Author ValidYN="Y"><LastName>Caruso</LastName>
<ForeName>Paola</ForeName>
<Initials>P</Initials>
<AffiliationInfo><Affiliation>Division of Endocrinology and Metabolic Diseases, Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Piazza Luigi Miraglia 2, 80138, Naples, Italy.</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y"><LastName>Maiorino</LastName>
<ForeName>Maria Ida</ForeName>
<Initials>MI</Initials>
<AffiliationInfo><Affiliation>Diabetes Unit, Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Naples, Italy.</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y"><LastName>Bellastella</LastName>
<ForeName>Giuseppe</ForeName>
<Initials>G</Initials>
<AffiliationInfo><Affiliation>Division of Endocrinology and Metabolic Diseases, Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Piazza Luigi Miraglia 2, 80138, Naples, Italy.</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y"><LastName>Giugliano</LastName>
<ForeName>Dario</ForeName>
<Initials>D</Initials>
<AffiliationInfo><Affiliation>Division of Endocrinology and Metabolic Diseases, Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Piazza Luigi Miraglia 2, 80138, Naples, Italy.</Affiliation>
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<ForeName>Katherine</ForeName>
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<AffiliationInfo><Affiliation>Diabetes Unit, Department of Advanced Medical and Surgical Sciences, University of Campania "Luigi Vanvitelli", Naples, Italy.</Affiliation>
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<Day>22</Day>
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<Keyword MajorTopicYN="Y">Insulin</Keyword>
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