Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins
Identifieur interne : 001532 ( Main/Corpus ); précédent : 001531; suivant : 001533Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins
Auteurs : T. Herdegen ; J. D. LeahSource :
- Brain Research Reviews [ 0165-0173 ] ; 1998.
Abstract
This article reviews findings up to the end of 1997 about the inducible transcription factors (ITFs) c-Jun, JunB, JunD, c-Fos, FosB, Fra-1, Fra-2, Krox-20 (Egr-2) and Krox-24 (NGFI-A, Egr-1, Zif268); and the constitutive transcription factors (CTFs) CREB, CREM, ATF-2 and SRF as they pertain to gene expression in the mammalian nervous system. In the first part we consider basic facts about the expression and activity of these transcription factors: the organization of the encoding genes and their promoters, the second messenger cascades converging on their regulatory promoter sites, the control of their transcription, the binding to dimeric partners and to specific DNA sequences, their trans-activation potential, and their posttranslational modifications. In the second part we describe the expression and possible roles of these transcription factors in neural tissue: in the quiescent brain, during pre- and postnatal development, following sensory stimulation, nerve transection (axotomy), neurodegeneration and apoptosis, hypoxia–ischemia, generalized and limbic seizures, long-term potentiation and learning, drug dependence and withdrawal, and following stimulation by neurotransmitters, hormones and neurotrophins. We also describe their expression and possible roles in glial cells. Finally, we discuss the relevance of their expression for nervous system functioning under normal and patho-physiological conditions.
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DOI: 10.1016/S0165-0173(98)00018-6
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Leah</name><affiliation><mods:affiliation>School of Biomedical Sciences, Griffith University, 4111 Nathan, Australia</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:50FA6689CCD0DF2FC37D1A20990311DAA0F92D32</idno><date when="1998" year="1998">1998</date><idno type="doi">10.1016/S0165-0173(98)00018-6</idno><idno type="url">https://api.istex.fr/document/50FA6689CCD0DF2FC37D1A20990311DAA0F92D32/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">001532</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a">Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins</title><author><name sortKey="Herdegen, T" sort="Herdegen, T" uniqKey="Herdegen T" first="T." last="Herdegen">T. Herdegen</name><affiliation><mods:affiliation>Institute of Pharmacology, University of Kiel, Hospitalstrasse 4, 24105 Kiel, Germany</mods:affiliation></affiliation></author><author><name sortKey="Leah, J D" sort="Leah, J D" uniqKey="Leah J" first="J. D." last="Leah">J. D. Leah</name><affiliation><mods:affiliation>School of Biomedical Sciences, Griffith University, 4111 Nathan, Australia</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Brain Research Reviews</title><title level="j" type="abbrev">BRESR</title><idno type="ISSN">0165-0173</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1998">1998</date><biblScope unit="volume">28</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="370">370</biblScope><biblScope unit="page" to="490">490</biblScope></imprint><idno type="ISSN">0165-0173</idno></series><idno type="istex">50FA6689CCD0DF2FC37D1A20990311DAA0F92D32</idno><idno type="DOI">10.1016/S0165-0173(98)00018-6</idno><idno type="PII">S0165-0173(98)00018-6</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0165-0173</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">This article reviews findings up to the end of 1997 about the inducible transcription factors (ITFs) c-Jun, JunB, JunD, c-Fos, FosB, Fra-1, Fra-2, Krox-20 (Egr-2) and Krox-24 (NGFI-A, Egr-1, Zif268); and the constitutive transcription factors (CTFs) CREB, CREM, ATF-2 and SRF as they pertain to gene expression in the mammalian nervous system. In the first part we consider basic facts about the expression and activity of these transcription factors: the organization of the encoding genes and their promoters, the second messenger cascades converging on their regulatory promoter sites, the control of their transcription, the binding to dimeric partners and to specific DNA sequences, their trans-activation potential, and their posttranslational modifications. In the second part we describe the expression and possible roles of these transcription factors in neural tissue: in the quiescent brain, during pre- and postnatal development, following sensory stimulation, nerve transection (axotomy), neurodegeneration and apoptosis, hypoxia–ischemia, generalized and limbic seizures, long-term potentiation and learning, drug dependence and withdrawal, and following stimulation by neurotransmitters, hormones and neurotrophins. We also describe their expression and possible roles in glial cells. Finally, we discuss the relevance of their expression for nervous system functioning under normal and patho-physiological conditions.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>T. Herdegen</name><affiliations><json:string>Institute of Pharmacology, University of Kiel, Hospitalstrasse 4, 24105 Kiel, Germany</json:string></affiliations></json:item><json:item><name>J.D. Leah</name><affiliations><json:string>School of Biomedical Sciences, Griffith University, 4111 Nathan, Australia</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>Transcription factor</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Jun</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Fos</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Krox</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Zif</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>CREB</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>ATF</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>SRF</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Neuron</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Brain</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Review</value></json:item></subject><language><json:string>eng</json:string></language><abstract>This article reviews findings up to the end of 1997 about the inducible transcription factors (ITFs) c-Jun, JunB, JunD, c-Fos, FosB, Fra-1, Fra-2, Krox-20 (Egr-2) and Krox-24 (NGFI-A, Egr-1, Zif268); and the constitutive transcription factors (CTFs) CREB, CREM, ATF-2 and SRF as they pertain to gene expression in the mammalian nervous system. In the first part we consider basic facts about the expression and activity of these transcription factors: the organization of the encoding genes and their promoters, the second messenger cascades converging on their regulatory promoter sites, the control of their transcription, the binding to dimeric partners and to specific DNA sequences, their trans-activation potential, and their posttranslational modifications. In the second part we describe the expression and possible roles of these transcription factors in neural tissue: in the quiescent brain, during pre- and postnatal development, following sensory stimulation, nerve transection (axotomy), neurodegeneration and apoptosis, hypoxia–ischemia, generalized and limbic seizures, long-term potentiation and learning, drug dependence and withdrawal, and following stimulation by neurotransmitters, hormones and neurotrophins. We also describe their expression and possible roles in glial cells. Finally, we discuss the relevance of their expression for nervous system functioning under normal and patho-physiological conditions.</abstract><qualityIndicators><score>7.34</score><pdfVersion>1.2</pdfVersion><pdfPageSize>595 x 758 pts</pdfPageSize><refBibsNative>true</refBibsNative><keywordCount>11</keywordCount><abstractCharCount>1433</abstractCharCount><pdfWordCount>101575</pdfWordCount><pdfCharCount>647616</pdfCharCount><pdfPageCount>121</pdfPageCount><abstractWordCount>195</abstractWordCount></qualityIndicators><title>Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins</title><pii><json:string>S0165-0173(98)00018-6</json:string></pii><genre><json:string>review-article</json:string></genre><host><volume>28</volume><pii><json:string>S0165-0173(00)X0022-7</json:string></pii><pages><last>490</last><first>370</first></pages><issn><json:string>0165-0173</json:string></issn><issue>3</issue><genre></genre><language><json:string>unknown</json:string></language><title>Brain Research Reviews</title><publicationDate>1998</publicationDate></host><categories><wos><json:string>NEUROSCIENCES</json:string></wos></categories><publicationDate>1998</publicationDate><copyrightDate>1998</copyrightDate><doi><json:string>10.1016/S0165-0173(98)00018-6</json:string></doi><id>50FA6689CCD0DF2FC37D1A20990311DAA0F92D32</id><fulltext><json:item><original>true</original><mimetype>application/pdf</mimetype><extension>pdf</extension><uri>https://api.istex.fr/document/50FA6689CCD0DF2FC37D1A20990311DAA0F92D32/fulltext/pdf</uri></json:item><json:item><original>false</original><mimetype>application/zip</mimetype><extension>zip</extension><uri>https://api.istex.fr/document/50FA6689CCD0DF2FC37D1A20990311DAA0F92D32/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/50FA6689CCD0DF2FC37D1A20990311DAA0F92D32/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a">Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>ELSEVIER</publisher><availability><p>Elsevier Science B.V., ©1998</p></availability><date>1998</date></publicationStmt><notesStmt><note type="content">Section title: Full-length review</note><note type="content">Table 1: Formation of dimers between different ITF and CTF proteins in vitro and/or in vivo</note><note type="content">Table 2: Expression of c-Jun and c-Fos mRNAs and proteins in axotomized neurons</note><note type="content">Table 3: Time course of c-Jun expression in axotomized neurons</note><note type="content">Table 4: Summary of the evidence for neuronal genes being regulated by ITFs</note></notesStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a">Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins</title><author><persName><forename type="first">T.</forename><surname>Herdegen</surname></persName><affiliation>Institute of Pharmacology, University of Kiel, Hospitalstrasse 4, 24105 Kiel, Germany</affiliation></author><author><persName><forename type="first">J.D.</forename><surname>Leah</surname></persName><affiliation>School of Biomedical Sciences, Griffith University, 4111 Nathan, Australia</affiliation></author></analytic><monogr><title level="j">Brain Research Reviews</title><title level="j" type="abbrev">BRESR</title><idno type="pISSN">0165-0173</idno><idno type="PII">S0165-0173(00)X0022-7</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1998"></date><biblScope unit="volume">28</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="370">370</biblScope><biblScope unit="page" to="490">490</biblScope></imprint></monogr><idno type="istex">50FA6689CCD0DF2FC37D1A20990311DAA0F92D32</idno><idno type="DOI">10.1016/S0165-0173(98)00018-6</idno><idno type="PII">S0165-0173(98)00018-6</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1998</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>This article reviews findings up to the end of 1997 about the inducible transcription factors (ITFs) c-Jun, JunB, JunD, c-Fos, FosB, Fra-1, Fra-2, Krox-20 (Egr-2) and Krox-24 (NGFI-A, Egr-1, Zif268); and the constitutive transcription factors (CTFs) CREB, CREM, ATF-2 and SRF as they pertain to gene expression in the mammalian nervous system. In the first part we consider basic facts about the expression and activity of these transcription factors: the organization of the encoding genes and their promoters, the second messenger cascades converging on their regulatory promoter sites, the control of their transcription, the binding to dimeric partners and to specific DNA sequences, their trans-activation potential, and their posttranslational modifications. In the second part we describe the expression and possible roles of these transcription factors in neural tissue: in the quiescent brain, during pre- and postnatal development, following sensory stimulation, nerve transection (axotomy), neurodegeneration and apoptosis, hypoxia–ischemia, generalized and limbic seizures, long-term potentiation and learning, drug dependence and withdrawal, and following stimulation by neurotransmitters, hormones and neurotrophins. We also describe their expression and possible roles in glial cells. Finally, we discuss the relevance of their expression for nervous system functioning under normal and patho-physiological conditions.</p></abstract><textClass><keywords scheme="keyword"><list><head>Keywords</head><item><term>Transcription factor</term></item><item><term>Jun</term></item><item><term>Fos</term></item><item><term>Krox</term></item><item><term>Zif</term></item><item><term>CREB</term></item><item><term>ATF</term></item><item><term>SRF</term></item><item><term>Neuron</term></item><item><term>Brain</term></item><item><term>Review</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1998-04-09">Registration</change><change when="1998">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/50FA6689CCD0DF2FC37D1A20990311DAA0F92D32/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier, elements deleted: ce:floats; body; tail"><istex:xmlDeclaration>version="1.0" encoding="utf-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 4.5.2//EN//XML" URI="art452.dtd" name="istex:docType"></istex:docType><istex:document><converted-article version="4.5.2" docsubtype="rev"><item-info><jid>BRESR</jid><aid>90241</aid><ce:pii>S0165-0173(98)00018-6</ce:pii><ce:doi>10.1016/S0165-0173(98)00018-6</ce:doi><ce:copyright year="1998" type="full-transfer">Elsevier Science B.V.</ce:copyright></item-info><head><ce:dochead><ce:textfn>Full-length review</ce:textfn></ce:dochead><ce:title>Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins</ce:title><ce:author-group><ce:author><ce:given-name>T.</ce:given-name><ce:surname>Herdegen</ce:surname><ce:cross-ref refid="AFF1">a</ce:cross-ref><ce:cross-ref refid="FN1"><ce:sup>1</ce:sup></ce:cross-ref><ce:cross-ref refid="FN1"><ce:sup>1</ce:sup></ce:cross-ref></ce:author><ce:author><ce:given-name>J.D.</ce:given-name><ce:surname>Leah</ce:surname><ce:cross-ref refid="AFF2">b</ce:cross-ref><ce:cross-ref refid="CORR1">*</ce:cross-ref></ce:author><ce:affiliation id="AFF1"><ce:label>a</ce:label><ce:textfn>Institute of Pharmacology, University of Kiel, Hospitalstrasse 4, 24105 Kiel, Germany</ce:textfn></ce:affiliation><ce:affiliation id="AFF2"><ce:label>b</ce:label><ce:textfn>School of Biomedical Sciences, Griffith University, 4111 Nathan, Australia</ce:textfn></ce:affiliation><ce:correspondence id="CORR1"><ce:label>*</ce:label><ce:text>Corresponding author. Fax: +61-7-3875-7656; E-mail: j.leah@sct.gu.edu.au</ce:text></ce:correspondence><ce:footnote id="FN1"><ce:label>1</ce:label><ce:note-para>Also corresponding author. Fax: +49-431-5973522; E-mail: t.herdegen@pharmakologie.uni-kiel.de.</ce:note-para></ce:footnote></ce:author-group><ce:date-accepted day="9" month="4" year="1998"></ce:date-accepted><ce:abstract><ce:section-title>Abstract</ce:section-title><ce:abstract-sec><ce:simple-para>This article reviews findings up to the end of 1997 about the inducible transcription factors (ITFs) c-Jun, JunB, JunD, c-Fos, FosB, Fra-1, Fra-2, Krox-20 (Egr-2) and Krox-24 (NGFI-A, Egr-1, Zif268); and the constitutive transcription factors (CTFs) CREB, CREM, ATF-2 and SRF as they pertain to gene expression in the mammalian nervous system. In the first part we consider basic facts about the expression and activity of these transcription factors: the organization of the encoding genes and their promoters, the second messenger cascades converging on their regulatory promoter sites, the control of their transcription, the binding to dimeric partners and to specific DNA sequences, their trans-activation potential, and their posttranslational modifications. In the second part we describe the expression and possible roles of these transcription factors in neural tissue: in the quiescent brain, during pre- and postnatal development, following sensory stimulation, nerve transection (axotomy), neurodegeneration and apoptosis, hypoxia–ischemia, generalized and limbic seizures, long-term potentiation and learning, drug dependence and withdrawal, and following stimulation by neurotransmitters, hormones and neurotrophins. We also describe their expression and possible roles in glial cells. Finally, we discuss the relevance of their expression for nervous system functioning under normal and patho-physiological conditions.</ce:simple-para></ce:abstract-sec></ce:abstract><ce:keywords class="keyword"><ce:section-title>Keywords</ce:section-title><ce:keyword><ce:text>Transcription factor</ce:text></ce:keyword><ce:keyword><ce:text>Jun</ce:text></ce:keyword><ce:keyword><ce:text>Fos</ce:text></ce:keyword><ce:keyword><ce:text>Krox</ce:text></ce:keyword><ce:keyword><ce:text>Zif</ce:text></ce:keyword><ce:keyword><ce:text>CREB</ce:text></ce:keyword><ce:keyword><ce:text>ATF</ce:text></ce:keyword><ce:keyword><ce:text>SRF</ce:text></ce:keyword><ce:keyword><ce:text>Neuron</ce:text></ce:keyword><ce:keyword><ce:text>Brain</ce:text></ce:keyword><ce:keyword><ce:text>Review</ce:text></ce:keyword></ce:keywords></head></converted-article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo><title>Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins</title></titleInfo><titleInfo type="alternative" contentType="CDATA"><title>Inducible and constitutive transcription factors in the mammalian nervous system: control of gene expression by Jun, Fos and Krox, and CREB/ATF proteins</title></titleInfo><name type="personal"><namePart type="given">T.</namePart><namePart type="family">Herdegen</namePart><affiliation>Institute of Pharmacology, University of Kiel, Hospitalstrasse 4, 24105 Kiel, Germany</affiliation><description>Also corresponding author. Fax: +49-431-5973522; E-mail: t.herdegen@pharmakologie.uni-kiel.de.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">J.D.</namePart><namePart type="family">Leah</namePart><affiliation>School of Biomedical Sciences, Griffith University, 4111 Nathan, Australia</affiliation><description>Corresponding author. Fax: +61-7-3875-7656; E-mail: j.leah@sct.gu.edu.au</description><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="review-article">Review article</genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1998</dateIssued><dateValid encoding="w3cdtf">1998-04-09</dateValid><copyrightDate encoding="w3cdtf">1998</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">This article reviews findings up to the end of 1997 about the inducible transcription factors (ITFs) c-Jun, JunB, JunD, c-Fos, FosB, Fra-1, Fra-2, Krox-20 (Egr-2) and Krox-24 (NGFI-A, Egr-1, Zif268); and the constitutive transcription factors (CTFs) CREB, CREM, ATF-2 and SRF as they pertain to gene expression in the mammalian nervous system. In the first part we consider basic facts about the expression and activity of these transcription factors: the organization of the encoding genes and their promoters, the second messenger cascades converging on their regulatory promoter sites, the control of their transcription, the binding to dimeric partners and to specific DNA sequences, their trans-activation potential, and their posttranslational modifications. In the second part we describe the expression and possible roles of these transcription factors in neural tissue: in the quiescent brain, during pre- and postnatal development, following sensory stimulation, nerve transection (axotomy), neurodegeneration and apoptosis, hypoxia–ischemia, generalized and limbic seizures, long-term potentiation and learning, drug dependence and withdrawal, and following stimulation by neurotransmitters, hormones and neurotrophins. We also describe their expression and possible roles in glial cells. Finally, we discuss the relevance of their expression for nervous system functioning under normal and patho-physiological conditions.</abstract><note type="content">Section title: Full-length review</note><note type="content">Table 1: Formation of dimers between different ITF and CTF proteins in vitro and/or in vivo</note><note type="content">Table 2: Expression of c-Jun and c-Fos mRNAs and proteins in axotomized neurons</note><note type="content">Table 3: Time course of c-Jun expression in axotomized neurons</note><note type="content">Table 4: Summary of the evidence for neuronal genes being regulated by ITFs</note><subject><genre>Keywords</genre><topic>Transcription factor</topic><topic>Jun</topic><topic>Fos</topic><topic>Krox</topic><topic>Zif</topic><topic>CREB</topic><topic>ATF</topic><topic>SRF</topic><topic>Neuron</topic><topic>Brain</topic><topic>Review</topic></subject><relatedItem type="host"><titleInfo><title>Brain Research Reviews</title></titleInfo><titleInfo type="abbreviated"><title>BRESR</title></titleInfo><originInfo><dateIssued encoding="w3cdtf">199812</dateIssued></originInfo><identifier type="ISSN">0165-0173</identifier><identifier type="PII">S0165-0173(00)X0022-7</identifier><part><date>199812</date><detail type="volume"><number>28</number><caption>vol.</caption></detail><detail type="issue"><number>3</number><caption>no.</caption></detail><extent unit="issue pages"><start>235</start><end>492</end></extent><extent unit="pages"><start>370</start><end>490</end></extent></part></relatedItem><identifier type="istex">50FA6689CCD0DF2FC37D1A20990311DAA0F92D32</identifier><identifier type="DOI">10.1016/S0165-0173(98)00018-6</identifier><identifier type="PII">S0165-0173(98)00018-6</identifier><recordInfo><recordOrigin>ELSEVIER</recordOrigin><recordContentSource>Elsevier Science B.V., ©1998</recordContentSource></recordInfo></mods></metadata><enrichments><istex:catWosTEI uri="https://api.istex.fr/document/50FA6689CCD0DF2FC37D1A20990311DAA0F92D32/enrichments/catWos"><teiHeader><profileDesc><textClass><classCode scheme="WOS">NEUROSCIENCES</classCode></textClass></profileDesc></teiHeader></istex:catWosTEI></enrichments><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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