Neural regulation of endothelial cell‐mediated inflammation
Identifieur interne : 000C66 ( Istex/Corpus ); précédent : 000C65; suivant : 000C67Neural regulation of endothelial cell‐mediated inflammation
Auteurs : J. C. AnselSource :
- Experimental Dermatology [ 0906-6705 ] ; 2004-09.
English descriptors
- Teeft :
- 2department, 3department, Acth, Activates, Adhesion, Adrenomedullin, Agonist, Allergic dermatitis, Anagen, Antimicrobial, Antioxidant, Apoptosis, Apoptotic, Atopic, Atopic dermatitis, Autoimmune, Basal, Bdnf, Biomedical, Biomedical sciences, Bmdcs, Bohm1, Boltzmann, Calcitonin, Calcitonin peptide, Capsaicin, Cell biology, Cgrp, Charite, Costimulatory molecules, Cutaneous, Cutaneous inflammation, Cytokine, Cytotoxicity, Dendricity, Dendritic, Dendritic cells, Dermal, Dermal microvascular, Dermatitis, Dermatology, Endocrine, Endogenous, Endothelial, Endothelial cells, Eosinophil, Epidermal, Epithelial, Extracellular, Fibre, Fibroblast, Follicle, Functional role, Glutamine, Hacat, Hacat cells, Hair follicle, Hair follicles, Hair growth, Hamburg, Hdmec, Healthy subjects, Homeostasis, Human hair follicle, Human sebocytes, Human skin, Hyperalgesia, Immune, Immune cells, Immune response, Immune system, Immunobiology, Immunohistochemistry, Immunomodulatory, Immunoreactivity, Important role, Inflammation, Inflammatory, Insr, Internal medicine, Intracellular, Intracellular calcium concentration, Keratinocyte, Keratinocytes, Kinase, Ligand, Ludwig boltzmann institute, Luger1, Lymphocyte, Mast cells, Mc1r, Mediator, Melanin, Melanocortin, Melanocortin receptors, Melanocyte, Melanogenesis, Melanoma, Melanoma cells, Melatonin, Microvascular, Mitf, Modulators, Mrna, Msc, Munster, Murine, Naltrexone, Nerve fibers, Nerve fibres, Nerve growth factor, Nervous system, Neuroendocrine, Neurogenic, Neurogenic inflammation, Neuron, Neuropeptide, Neuropeptides, Neurotrophic, Neurotrophic factor, Neurotrophin, Neurotrophins, Neutral endopeptidase, Nk1r, Nmda, Pacap, Papilla, Par2, Pathway, Peptide, Peripheral blood eosinophils, Pigmentation, Plasma membrane, Pomc, Pomc peptides, Present study, Proinflammatory, Pruritus, Receptor, Regulator, Rheumatoid arthritis, Schwann, Sebaceous, Sebaceous gland, Sebocytes, Sensitization, Sensory nerves, Sensory neurons, Somatostatin, Trafficking, Transgenic, Transgenic mice, Trpv1, Tumour, Upregulated, Upregulation, Uremic pruritus, Urocortin, Vanilloid, Vasoactive, Vellus hair follicles, Wound healing.
Abstract
There is much evidence that neurocutaneous interactions may play an important role in modulating a wide variety of biological processes in the skin including inflammation. Perhaps, the key cell type for directing inflammatory processes in the skin is the dermal microvascular endothelial cell (DMEC). All leukocyte trafficking during cutaneous inflammation is mediated by DMEC. This presentation will review various ways in which the cutaneous neurosensory system can modulate DMEC activities, and how this may lead to novel approaches in our understanding and treatment of inflammatory skin disease.
Url:
DOI: 10.1111/j.0906-6705.2004.0212ac.x
Links to Exploration step
ISTEX:78BBBD495204FACFC4678962E67FFFDAEA2B5595Le document en format XML
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Perhaps, the key cell type for directing inflammatory processes in the skin is the dermal microvascular endothelial cell (DMEC). All leukocyte trafficking during cutaneous inflammation is mediated by DMEC. 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C. Ansel</name><affiliations><json:string>Department of Dermatology, Northwestern University, Chicago, IL, USA</json:string></affiliations></json:item></author><articleId><json:string>EXD212AC</json:string></articleId><language><json:string>eng</json:string></language><originalGenre><json:string>abstract</json:string></originalGenre><abstract>There is much evidence that neurocutaneous interactions may play an important role in modulating a wide variety of biological processes in the skin including inflammation. Perhaps, the key cell type for directing inflammatory processes in the skin is the dermal microvascular endothelial cell (DMEC). All leukocyte trafficking during cutaneous inflammation is mediated by DMEC. This presentation will review various ways in which the cutaneous neurosensory system can modulate DMEC activities, and how this may lead to novel approaches in our understanding and treatment of inflammatory skin disease.</abstract><qualityIndicators><score>6.044</score><pdfVersion>1.3</pdfVersion><pdfPageSize>595 x 782 pts</pdfPageSize><refBibsNative>false</refBibsNative><abstractCharCount>600</abstractCharCount><pdfWordCount>23570</pdfWordCount><pdfCharCount>158516</pdfCharCount><pdfPageCount>25</pdfPageCount><abstractWordCount>87</abstractWordCount></qualityIndicators><title>Neural regulation of endothelial cell‐mediated inflammation</title><genre><json:string>abstract</json:string></genre><host><title>Experimental Dermatology</title><language><json:string>unknown</json:string></language><doi><json:string>10.1111/(ISSN)1600-0625</json:string></doi><issn><json:string>0906-6705</json:string></issn><eissn><json:string>1600-0625</json:string></eissn><publisherId><json:string>EXD</json:string></publisherId><volume>13</volume><issue>9</issue><pages><first>574</first><last>574</last><total>1</total></pages><genre><json:string>journal</json:string></genre></host><categories><wos><json:string>science</json:string><json:string>dermatology</json:string></wos><scienceMetrix><json:string>health sciences</json:string><json:string>clinical medicine</json:string><json:string>dermatology & venereal diseases</json:string></scienceMetrix><inist><json:string>sciences appliquees, technologies et medecines</json:string><json:string>sciences biologiques et medicales</json:string><json:string>sciences medicales</json:string><json:string>sarcoidoses. granulomatoses d'etiologie indeterminee. maladies du tissu conjonctif. maladies du tissu elastique. vascularites</json:string></inist></categories><publicationDate>2004</publicationDate><copyrightDate>2004</copyrightDate><doi><json:string>10.1111/j.0906-6705.2004.0212ac.x</json:string></doi><id>78BBBD495204FACFC4678962E67FFFDAEA2B5595</id><score>1</score><fulltext><json:item><extension>pdf</extension><original>true</original><mimetype>application/pdf</mimetype><uri>https://api.istex.fr/document/78BBBD495204FACFC4678962E67FFFDAEA2B5595/fulltext/pdf</uri></json:item><json:item><extension>zip</extension><original>false</original><mimetype>application/zip</mimetype><uri>https://api.istex.fr/document/78BBBD495204FACFC4678962E67FFFDAEA2B5595/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/78BBBD495204FACFC4678962E67FFFDAEA2B5595/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">Neural regulation of endothelial cell‐mediated inflammation</title><respStmt><resp>Références bibliographiques récupérées via GROBID</resp><name resp="ISTEX-API">ISTEX-API (INIST-CNRS)</name></respStmt><respStmt><resp>Références bibliographiques récupérées via GROBID</resp><name resp="ISTEX-API">ISTEX-API (INIST-CNRS)</name></respStmt></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>Blackwell Publishing Ltd/Inc</publisher><pubPlace>Oxford, UK; Malden, USA</pubPlace><availability><p>WILEY</p></availability><date>2004</date></publicationStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Neural regulation of endothelial cell‐mediated inflammation</title><author xml:id="author-1"><persName><forename type="first">J. C.</forename><surname>Ansel</surname></persName><affiliation>Department of Dermatology, Northwestern University, Chicago, IL, USA</affiliation></author></analytic><monogr><title level="j">Experimental Dermatology</title><idno type="pISSN">0906-6705</idno><idno type="eISSN">1600-0625</idno><idno type="DOI">10.1111/(ISSN)1600-0625</idno><imprint><publisher>Blackwell Publishing Ltd/Inc</publisher><pubPlace>Oxford, UK; Malden, USA</pubPlace><date type="published" when="2004-09"></date><biblScope unit="volume">13</biblScope><biblScope unit="issue">9</biblScope><biblScope unit="page" from="574">574</biblScope><biblScope unit="page" to="574">574</biblScope></imprint></monogr><idno type="istex">78BBBD495204FACFC4678962E67FFFDAEA2B5595</idno><idno type="DOI">10.1111/j.0906-6705.2004.0212ac.x</idno><idno type="ArticleID">EXD212AC</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>2004</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>There is much evidence that neurocutaneous interactions may play an important role in modulating a wide variety of biological processes in the skin including inflammation. Perhaps, the key cell type for directing inflammatory processes in the skin is the dermal microvascular endothelial cell (DMEC). All leukocyte trafficking during cutaneous inflammation is mediated by DMEC. 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