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<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Effect of treatment on renal function in severe osteomalacia due to Wilson's disease</title>
<author><name sortKey="Monro, Pauline" sort="Monro, Pauline" uniqKey="Monro P" first="Pauline" last="Monro">Pauline Monro</name>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">PMC</idno>
<idno type="pmid">5312232</idno>
<idno type="pmc">476816</idno>
<idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC476816</idno>
<idno type="RBID">PMC:476816</idno>
<date when="1970">1970</date>
<idno type="wicri:Area/Pmc/Corpus">000165</idno>
<idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Corpus" wicri:corpus="PMC">000165</idno>
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<sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">Effect of treatment on renal function in severe osteomalacia due to Wilson's disease</title>
<author><name sortKey="Monro, Pauline" sort="Monro, Pauline" uniqKey="Monro P" first="Pauline" last="Monro">Pauline Monro</name>
</author>
</analytic>
<series><title level="j">Journal of Clinical Pathology</title>
<idno type="ISSN">0021-9746</idno>
<idno type="eISSN">1472-4146</idno>
<imprint><date when="1970">1970</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc><textClass></textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en"><p>A patient with Wilson's disease presented at the age of 41 with a neurological defect and gross osteomalacia secondary to a defect of renal tubular reabsorption. He also showed the unusual features of a renal stone in the presence of the Fanconi syndrome and a relatively low alkaline phosphatase level, possibly due to the additional inherited defect of hypophosphatasia. During four years of treatment with penicillamine and calciferol clinical improvement was spectacular. Details of amino-acid clearances before and after treatment are given, and the results suggest that, as in the brain and the liver, the function of the distal renal tubules may be restored in Wilson's disease when copper is removed.</p>
<sec sec-type="scanned-figures"><title>Images</title>
<fig id="F1"><graphic xlink:href="jclinpath00086-0030-a" xlink:role="488"></graphic>
</fig>
<fig id="F2"><graphic xlink:href="jclinpath00086-0031-a" xlink:role="489"></graphic>
</fig>
</sec>
</div>
</front>
</TEI>
<pmc article-type="research-article"><pmc-comment>The publisher of this article does not allow downloading of the full text in XML form.</pmc-comment>
<front><journal-meta><journal-id journal-id-type="nlm-ta">J Clin Pathol</journal-id>
<journal-title>Journal of Clinical Pathology</journal-title>
<issn pub-type="ppub">0021-9746</issn>
<issn pub-type="epub">1472-4146</issn>
</journal-meta>
<article-meta><article-id pub-id-type="pmid">5312232</article-id>
<article-id pub-id-type="pmc">476816</article-id>
<article-categories><subj-group subj-group-type="heading"><subject>Articles</subject>
</subj-group>
</article-categories>
<title-group><article-title>Effect of treatment on renal function in severe osteomalacia due to Wilson's disease</article-title>
</title-group>
<contrib-group><contrib contrib-type="author"><name><surname>Monro</surname>
<given-names>Pauline</given-names>
</name>
</contrib>
</contrib-group>
<aff id="af1"><label></label>
Atkinson Morley's Hospital, Wimbledon, London</aff>
<pub-date pub-type="ppub"><month>09</month>
<year>1970</year>
</pub-date>
<volume>23</volume>
<issue>6</issue>
<fpage>487</fpage>
<lpage>491</lpage>
<abstract><p>A patient with Wilson's disease presented at the age of 41 with a neurological defect and gross osteomalacia secondary to a defect of renal tubular reabsorption. He also showed the unusual features of a renal stone in the presence of the Fanconi syndrome and a relatively low alkaline phosphatase level, possibly due to the additional inherited defect of hypophosphatasia. During four years of treatment with penicillamine and calciferol clinical improvement was spectacular. Details of amino-acid clearances before and after treatment are given, and the results suggest that, as in the brain and the liver, the function of the distal renal tubules may be restored in Wilson's disease when copper is removed.</p>
<sec sec-type="scanned-figures"><title>Images</title>
<fig id="F1"><graphic xlink:href="jclinpath00086-0030-a" xlink:role="488"></graphic>
</fig>
<fig id="F2"><graphic xlink:href="jclinpath00086-0031-a" xlink:role="489"></graphic>
</fig>
</sec>
</abstract>
</article-meta>
</front>
</pmc>
</record>
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