Neurology of Volition
Identifieur interne : 001127 ( Pmc/Checkpoint ); précédent : 001126; suivant : 001128Neurology of Volition
Auteurs : Sarah M. Kranick ; Mark HallettSource :
- Experimental brain research [ 0014-4819 ] ; 2013.
Abstract
Neurological disorders of volition may be characterized by deficits in willing and/or agency. When we move our bodies through space, it is the sense that we intended to move (willing) and that our actions were a consequence of this intention (self-agency) that gives us the sense of voluntariness and a general feeling of being “in control.” While it is possible to have movements that share executive machinery ordinarily used for voluntary movement but lack a sense of voluntariness, such as psychogenic movement disorders, it is also possible to claim volition for presumed involuntary movements (early chorea) or even when no movement is produced (anosognosia). The study of such patients should enlighten traditional models of how the percepts of volition are generated in the brain with regards to movement. We discuss volition and its components as multi-leveled processes with feedforward and feedback information flow, and dependence on prior expectations as well as external and internal cues.
Url:
DOI: 10.1007/s00221-013-3399-2
PubMed: 23329204
PubMed Central: 4744643
Affiliations:
Links toward previous steps (curation, corpus...)
Links to Exploration step
PMC:4744643Le document en format XML
<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Neurology of Volition</title>
<author><name sortKey="Kranick, Sarah M" sort="Kranick, Sarah M" uniqKey="Kranick S" first="Sarah M." last="Kranick">Sarah M. Kranick</name>
</author>
<author><name sortKey="Hallett, Mark" sort="Hallett, Mark" uniqKey="Hallett M" first="Mark" last="Hallett">Mark Hallett</name>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">PMC</idno>
<idno type="pmid">23329204</idno>
<idno type="pmc">4744643</idno>
<idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4744643</idno>
<idno type="RBID">PMC:4744643</idno>
<idno type="doi">10.1007/s00221-013-3399-2</idno>
<date when="2013">2013</date>
<idno type="wicri:Area/Pmc/Corpus">001614</idno>
<idno type="wicri:Area/Pmc/Curation">001614</idno>
<idno type="wicri:Area/Pmc/Checkpoint">001127</idno>
</publicationStmt>
<sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">Neurology of Volition</title>
<author><name sortKey="Kranick, Sarah M" sort="Kranick, Sarah M" uniqKey="Kranick S" first="Sarah M." last="Kranick">Sarah M. Kranick</name>
</author>
<author><name sortKey="Hallett, Mark" sort="Hallett, Mark" uniqKey="Hallett M" first="Mark" last="Hallett">Mark Hallett</name>
</author>
</analytic>
<series><title level="j">Experimental brain research</title>
<idno type="ISSN">0014-4819</idno>
<idno type="eISSN">1432-1106</idno>
<imprint><date when="2013">2013</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc><textClass></textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en"><p id="P1">Neurological disorders of volition may be characterized by deficits in willing and/or agency. When we move our bodies through space, it is the sense that we intended to move (willing) and that our actions were a consequence of this intention (self-agency) that gives us the sense of voluntariness and a general feeling of being “in control.” While it is possible to have movements that share executive machinery ordinarily used for voluntary movement but lack a sense of voluntariness, such as psychogenic movement disorders, it is also possible to claim volition for presumed involuntary movements (early chorea) or even when no movement is produced (anosognosia). The study of such patients should enlighten traditional models of how the percepts of volition are generated in the brain with regards to movement. We discuss volition and its components as multi-leveled processes with feedforward and feedback information flow, and dependence on prior expectations as well as external and internal cues.</p>
</div>
</front>
</TEI>
<pmc article-type="research-article"><pmc-comment>The publisher of this article does not allow downloading of the full text in XML form.</pmc-comment>
<pmc-dir>properties manuscript</pmc-dir>
<front><journal-meta><journal-id journal-id-type="nlm-journal-id">0043312</journal-id>
<journal-id journal-id-type="pubmed-jr-id">3641</journal-id>
<journal-id journal-id-type="nlm-ta">Exp Brain Res</journal-id>
<journal-id journal-id-type="iso-abbrev">Exp Brain Res</journal-id>
<journal-title-group><journal-title>Experimental brain research</journal-title>
</journal-title-group>
<issn pub-type="ppub">0014-4819</issn>
<issn pub-type="epub">1432-1106</issn>
</journal-meta>
<article-meta><article-id pub-id-type="pmid">23329204</article-id>
<article-id pub-id-type="pmc">4744643</article-id>
<article-id pub-id-type="doi">10.1007/s00221-013-3399-2</article-id>
<article-id pub-id-type="manuscript">NIHMS756324</article-id>
<article-categories><subj-group subj-group-type="heading"><subject>Article</subject>
</subj-group>
</article-categories>
<title-group><article-title>Neurology of Volition</article-title>
</title-group>
<contrib-group><contrib contrib-type="author"><name><surname>Kranick</surname>
<given-names>Sarah M.</given-names>
</name>
<degrees>MD</degrees>
</contrib>
<contrib contrib-type="author"><name><surname>Hallett</surname>
<given-names>Mark</given-names>
</name>
<degrees>MD</degrees>
</contrib>
<aff id="A1">Human Motor Control Section, Medical Neurology Branch, National Institutes of Neurological Disorders and Stroke, National Institutes of Health, Bethesda MD</aff>
</contrib-group>
<author-notes><corresp id="cor1"><bold>Address correspondence to:</bold>
Sarah Kranick, M.D., National Institutes of Health, Building 10/6-5700, 10 Center Drive, MSC 1430, Bethesda MD 20892-1430, Phone: (301) 451-6784, <email>sarah.kranick@nih.gov</email>
</corresp>
</author-notes>
<pub-date pub-type="nihms-submitted"><day>4</day>
<month>2</month>
<year>2016</year>
</pub-date>
<pub-date pub-type="epub"><day>18</day>
<month>1</month>
<year>2013</year>
</pub-date>
<pub-date pub-type="ppub"><month>9</month>
<year>2013</year>
</pub-date>
<pub-date pub-type="pmc-release"><day>07</day>
<month>2</month>
<year>2016</year>
</pub-date>
<volume>229</volume>
<issue>3</issue>
<fpage>313</fpage>
<lpage>327</lpage>
<pmc-comment>elocation-id from pubmed: 10.1007/s00221-013-3399-2</pmc-comment>
<abstract><p id="P1">Neurological disorders of volition may be characterized by deficits in willing and/or agency. When we move our bodies through space, it is the sense that we intended to move (willing) and that our actions were a consequence of this intention (self-agency) that gives us the sense of voluntariness and a general feeling of being “in control.” While it is possible to have movements that share executive machinery ordinarily used for voluntary movement but lack a sense of voluntariness, such as psychogenic movement disorders, it is also possible to claim volition for presumed involuntary movements (early chorea) or even when no movement is produced (anosognosia). The study of such patients should enlighten traditional models of how the percepts of volition are generated in the brain with regards to movement. We discuss volition and its components as multi-leveled processes with feedforward and feedback information flow, and dependence on prior expectations as well as external and internal cues.</p>
</abstract>
</article-meta>
</front>
</pmc>
<affiliations><list></list>
<tree><noCountry><name sortKey="Hallett, Mark" sort="Hallett, Mark" uniqKey="Hallett M" first="Mark" last="Hallett">Mark Hallett</name>
<name sortKey="Kranick, Sarah M" sort="Kranick, Sarah M" uniqKey="Kranick S" first="Sarah M." last="Kranick">Sarah M. Kranick</name>
</noCountry>
</tree>
</affiliations>
</record>
Pour manipuler ce document sous Unix (Dilib)
EXPLOR_STEP=$WICRI_ROOT/Ticri/CIDE/explor/HapticV1/Data/Pmc/Checkpoint
HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 001127 | SxmlIndent | more
Ou
HfdSelect -h $EXPLOR_AREA/Data/Pmc/Checkpoint/biblio.hfd -nk 001127 | SxmlIndent | more
Pour mettre un lien sur cette page dans le réseau Wicri
{{Explor lien |wiki= Ticri/CIDE |area= HapticV1 |flux= Pmc |étape= Checkpoint |type= RBID |clé= PMC:4744643 |texte= Neurology of Volition }}
Pour générer des pages wiki
HfdIndexSelect -h $EXPLOR_AREA/Data/Pmc/Checkpoint/RBID.i -Sk "pubmed:23329204" \ | HfdSelect -Kh $EXPLOR_AREA/Data/Pmc/Checkpoint/biblio.hfd \ | NlmPubMed2Wicri -a HapticV1
![]() | This area was generated with Dilib version V0.6.23. | ![]() |