Geographic, ethnic, age-related and temporal variation in the incidence of end-stage renal disease in Europe, Canada and the Asia-Pacific region, 1998–2002
Identifieur interne : 001112 ( Istex/Corpus ); précédent : 001111; suivant : 001113Geographic, ethnic, age-related and temporal variation in the incidence of end-stage renal disease in Europe, Canada and the Asia-Pacific region, 1998–2002
Auteurs :Source :
- Nephrology Dialysis Transplantation [ 0931-0509 ] ; 2006-08.
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- KwdEn :
Abstract
Background. Only unbiased estimates of end-stage renal disease (ESRD) incidence and trends are useful for disease control—identification of risk factors and measuring the effect of intervention. Methods. Age- and sex-standardized incidences (with trends) were calculated for all-cause and diabetic/non-diabetic ESRD for persons aged 0–14, 15–29, 30–44 and 45–64 years in 13 populations identified geographically, and six populations identified by ethnicity. Results. The incidence of ESRD varied most with age, ethnicity and prevalence of diabetes. All non-Europid populations had excess ESRD, chiefly due to rates of type 2 diabetic ESRD that were greater than accounted for by community prevalences of diabetes. Their rates of non-diabetic ESRD also were raised, with contributions from most common primary renal diseases except type 1 diabetic nephropathy and polycystic kidney disease. The ESRD rates generally were low, and more similar than different, in Europid populations, except for variable contributions from type 1 (high in Finland, Sweden, Denmark and Canada) and type 2 (high in Austria and Canada) diabetes. In Europid populations during 1998–2002, all-cause ESRD declined by 2% per year in persons aged 0–44 years, and all non-diabetic ESRD by a similar amount in persons aged 45–64 years, in whom diabetic ESRD had increased by 3% per year. Conclusions. Increased susceptibility to type 2 diabetes and to kidney disease progression characterizes excess ESRD in non-Europid peoples. The decline in all-cause ESRD in young persons, and non-diabetic ESRD in the middle-aged, probably reflects improving management of progressive renal disease.
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DOI: 10.1093/ndt/gfl145
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Transplant.</title><idno type="ISSN">0931-0509</idno><idno type="eISSN">1460-2385</idno><imprint><publisher>Oxford University Press</publisher><date type="published" when="2006-08">2006-08</date><biblScope unit="volume">21</biblScope><biblScope unit="issue">8</biblScope><biblScope unit="page" from="2178">2178</biblScope><biblScope unit="page" to="2183">2183</biblScope></imprint><idno type="ISSN">0931-0509</idno></series><idno type="istex">E5FA26A73D3850262190890D8A107552F2B0A4E8</idno><idno type="DOI">10.1093/ndt/gfl145</idno><idno type="local">gfl145</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0931-0509</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Asia-Pacific</term><term>Canada</term><term>Europe</term><term>end-stage renal disease</term><term>incidence</term><term>trends</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Background. Only unbiased estimates of end-stage renal disease (ESRD) incidence and trends are useful for disease control—identification of risk factors and measuring the effect of intervention. Methods. Age- and sex-standardized incidences (with trends) were calculated for all-cause and diabetic/non-diabetic ESRD for persons aged 0–14, 15–29, 30–44 and 45–64 years in 13 populations identified geographically, and six populations identified by ethnicity. Results. The incidence of ESRD varied most with age, ethnicity and prevalence of diabetes. All non-Europid populations had excess ESRD, chiefly due to rates of type 2 diabetic ESRD that were greater than accounted for by community prevalences of diabetes. Their rates of non-diabetic ESRD also were raised, with contributions from most common primary renal diseases except type 1 diabetic nephropathy and polycystic kidney disease. The ESRD rates generally were low, and more similar than different, in Europid populations, except for variable contributions from type 1 (high in Finland, Sweden, Denmark and Canada) and type 2 (high in Austria and Canada) diabetes. In Europid populations during 1998–2002, all-cause ESRD declined by 2% per year in persons aged 0–44 years, and all non-diabetic ESRD by a similar amount in persons aged 45–64 years, in whom diabetic ESRD had increased by 3% per year. Conclusions. Increased susceptibility to type 2 diabetes and to kidney disease progression characterizes excess ESRD in non-Europid peoples. The decline in all-cause ESRD in young persons, and non-diabetic ESRD in the middle-aged, probably reflects improving management of progressive renal disease.</div></front></TEI><istex><corpusName>oup</corpusName><subject><json:item><lang><json:string>eng</json:string></lang><value>Asia-Pacific</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Canada</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>end-stage renal disease</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Europe</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>incidence</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>trends</value></json:item></subject><language><json:string>eng</json:string></language><originalGenre><json:string>other</json:string></originalGenre><abstract>Background. Only unbiased estimates of end-stage renal disease (ESRD) incidence and trends are useful for disease control—identification of risk factors and measuring the effect of intervention. Methods. Age- and sex-standardized incidences (with trends) were calculated for all-cause and diabetic/non-diabetic ESRD for persons aged 0–14, 15–29, 30–44 and 45–64 years in 13 populations identified geographically, and six populations identified by ethnicity. Results. The incidence of ESRD varied most with age, ethnicity and prevalence of diabetes. All non-Europid populations had excess ESRD, chiefly due to rates of type 2 diabetic ESRD that were greater than accounted for by community prevalences of diabetes. Their rates of non-diabetic ESRD also were raised, with contributions from most common primary renal diseases except type 1 diabetic nephropathy and polycystic kidney disease. The ESRD rates generally were low, and more similar than different, in Europid populations, except for variable contributions from type 1 (high in Finland, Sweden, Denmark and Canada) and type 2 (high in Austria and Canada) diabetes. In Europid populations during 1998–2002, all-cause ESRD declined by 2% per year in persons aged 0–44 years, and all non-diabetic ESRD by a similar amount in persons aged 45–64 years, in whom diabetic ESRD had increased by 3% per year. Conclusions. Increased susceptibility to type 2 diabetes and to kidney disease progression characterizes excess ESRD in non-Europid peoples. The decline in all-cause ESRD in young persons, and non-diabetic ESRD in the middle-aged, probably reflects improving management of progressive renal disease.</abstract><qualityIndicators><score>7.046</score><pdfVersion>1.4</pdfVersion><pdfPageSize>595 x 782 pts</pdfPageSize><refBibsNative>false</refBibsNative><keywordCount>6</keywordCount><abstractCharCount>1658</abstractCharCount><pdfWordCount>3642</pdfWordCount><pdfCharCount>25723</pdfCharCount><pdfPageCount>6</pdfPageCount><abstractWordCount>242</abstractWordCount></qualityIndicators><title>Geographic, ethnic, age-related and temporal variation in the incidence of end-stage renal disease in Europe, Canada and the Asia-Pacific region, 1998–2002</title><corporate><json:item><name>The ESRD Incidence Study Group</name><affiliations><json:string>Department of Preventive and Social Medicine, University of Otago, Dunedin, New Zealand, The ERA-EDTA Registry, Department of Medical Informatics, Academic Medical Center, Amsterdam, The Netherlands, CORR, Canadian Institute of Health Information, Toronto, Ontario, Canada and the ANZDATA Registry, Queen Elizabeth Hospital, Adelaide, SA, Australia</json:string></affiliations></json:item></corporate><genre><json:string>other</json:string></genre><host><volume>21</volume><publisherId><json:string>ndt</json:string></publisherId><pages><last>2183</last><first>2178</first></pages><issn><json:string>0931-0509</json:string></issn><issue>8</issue><genre><json:string>journal</json:string></genre><language><json:string>unknown</json:string></language><eissn><json:string>1460-2385</json:string></eissn><title>Nephrology Dialysis Transplantation</title></host><categories><wos><json:string>TRANSPLANTATION</json:string><json:string>UROLOGY & NEPHROLOGY</json:string></wos></categories><publicationDate>2006</publicationDate><copyrightDate>2006</copyrightDate><doi><json:string>10.1093/ndt/gfl145</json:string></doi><id>E5FA26A73D3850262190890D8A107552F2B0A4E8</id><score>0.23751307</score><fulltext><json:item><original>true</original><mimetype>application/pdf</mimetype><extension>pdf</extension><uri>https://api.istex.fr/document/E5FA26A73D3850262190890D8A107552F2B0A4E8/fulltext/pdf</uri></json:item><json:item><original>false</original><mimetype>application/zip</mimetype><extension>zip</extension><uri>https://api.istex.fr/document/E5FA26A73D3850262190890D8A107552F2B0A4E8/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/E5FA26A73D3850262190890D8A107552F2B0A4E8/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">Geographic, ethnic, age-related and temporal variation in the incidence of end-stage renal disease in Europe, Canada and the Asia-Pacific region, 1998–2002</title><respStmt><resp>Références bibliographiques récupérées via GROBID</resp><name resp="ISTEX-API">ISTEX-API (INIST-CNRS)</name></respStmt></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>Oxford University Press</publisher><availability><p>© The Author [2006]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org</p></availability><date>2006-04-04</date></publicationStmt><notesStmt><note>Correspondence and offprint requests to: Dr Margaret McCredie, Department of Preventive and Social Medicine, University of Otago, PO Box 913, Dunedin, New Zealand. Email: margaret.mccredie@stonebow.otago.ac.nz</note></notesStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Geographic, ethnic, age-related and temporal variation in the incidence of end-stage renal disease in Europe, Canada and the Asia-Pacific region, 1998–2002</title><author><orgName>The ESRD Incidence Study Group</orgName><affiliation>Department of Preventive and Social Medicine, University of Otago, Dunedin, New Zealand, The ERA-EDTA Registry, Department of Medical Informatics, Academic Medical Center, Amsterdam, The Netherlands, CORR, Canadian Institute of Health Information, Toronto, Ontario, Canada and the ANZDATA Registry, Queen Elizabeth Hospital, Adelaide, SA, Australia</affiliation></author></analytic><monogr><title level="j">Nephrology Dialysis Transplantation</title><title level="j" type="abbrev">Nephrol. Dial. Transplant.</title><idno type="pISSN">0931-0509</idno><idno type="eISSN">1460-2385</idno><imprint><publisher>Oxford University Press</publisher><date type="published" when="2006-08"></date><biblScope unit="volume">21</biblScope><biblScope unit="issue">8</biblScope><biblScope unit="page" from="2178">2178</biblScope><biblScope unit="page" to="2183">2183</biblScope></imprint></monogr><idno type="istex">E5FA26A73D3850262190890D8A107552F2B0A4E8</idno><idno type="DOI">10.1093/ndt/gfl145</idno><idno type="local">gfl145</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>2006-04-04</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Background. Only unbiased estimates of end-stage renal disease (ESRD) incidence and trends are useful for disease control—identification of risk factors and measuring the effect of intervention. Methods. Age- and sex-standardized incidences (with trends) were calculated for all-cause and diabetic/non-diabetic ESRD for persons aged 0–14, 15–29, 30–44 and 45–64 years in 13 populations identified geographically, and six populations identified by ethnicity. Results. The incidence of ESRD varied most with age, ethnicity and prevalence of diabetes. All non-Europid populations had excess ESRD, chiefly due to rates of type 2 diabetic ESRD that were greater than accounted for by community prevalences of diabetes. Their rates of non-diabetic ESRD also were raised, with contributions from most common primary renal diseases except type 1 diabetic nephropathy and polycystic kidney disease. The ESRD rates generally were low, and more similar than different, in Europid populations, except for variable contributions from type 1 (high in Finland, Sweden, Denmark and Canada) and type 2 (high in Austria and Canada) diabetes. In Europid populations during 1998–2002, all-cause ESRD declined by 2% per year in persons aged 0–44 years, and all non-diabetic ESRD by a similar amount in persons aged 45–64 years, in whom diabetic ESRD had increased by 3% per year. Conclusions. Increased susceptibility to type 2 diabetes and to kidney disease progression characterizes excess ESRD in non-Europid peoples. The decline in all-cause ESRD in young persons, and non-diabetic ESRD in the middle-aged, probably reflects improving management of progressive renal disease.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>KWD</head><item><term>Asia-Pacific</term></item><item><term>Canada</term></item><item><term>end-stage renal disease</term></item><item><term>Europe</term></item><item><term>incidence</term></item><item><term>trends</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="2006-04-04">Created</change><change when="2006-08">Published</change><change xml:id="refBibs-istex" who="#ISTEX-API" when="2016-10-14">References added</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/E5FA26A73D3850262190890D8A107552F2B0A4E8/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="corpus oup" wicri:toSee="no header"><istex:xmlDeclaration>version="1.0" encoding="US-ASCII"</istex:xmlDeclaration><istex:docType PUBLIC="-//NLM//DTD Journal Publishing DTD v2.3 20070202//EN" URI="journalpublishing.dtd" name="istex:docType"></istex:docType><istex:document><article xml:lang="en" article-type="other"><front><journal-meta><journal-id journal-id-type="hwp">ndt</journal-id><journal-id journal-id-type="nlm-ta">Nephrol Dial Transplant</journal-id><journal-id journal-id-type="publisher-id">ndt</journal-id><journal-title>Nephrology Dialysis Transplantation</journal-title><abbrev-journal-title abbrev-type="publisher">Nephrol. Dial. Transplant.</abbrev-journal-title><issn pub-type="ppub">0931-0509</issn><issn pub-type="epub">1460-2385</issn><publisher><publisher-name>Oxford University Press</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="other">gfl145</article-id><article-id pub-id-type="doi">10.1093/ndt/gfl145</article-id><article-categories><subj-group subj-group-type="heading"><subject>II. Scientific Papers</subject><subj-group><subject>Original Articles: Clinical Nephrology</subject></subj-group></subj-group></article-categories><title-group><article-title>Geographic, ethnic, age-related and temporal variation in the incidence of end-stage renal disease in Europe, Canada and the Asia-Pacific region, 1998–2002</article-title></title-group><contrib-group><contrib contrib-type="author"><collab collab-type="author">The ESRD Incidence Study Group</collab></contrib><aff>Department of Preventive and Social Medicine, University of Otago, Dunedin, New Zealand, The ERA-EDTA Registry, Department of Medical Informatics, Academic Medical Center, Amsterdam, The Netherlands, CORR, Canadian Institute of Health Information, Toronto, Ontario, Canada and the ANZDATA Registry, Queen Elizabeth Hospital, Adelaide, SA, Australia</aff></contrib-group><author-notes><corresp><italic>Correspondence and offprint requests to:</italic> Dr Margaret McCredie, Department of Preventive and Social Medicine, University of Otago, PO Box 913, Dunedin, New Zealand. Email: <ext-link xlink:href="margaret.mccredie@stonebow.otago.ac.nz" ext-link-type="email">margaret.mccredie@stonebow.otago.ac.nz</ext-link></corresp></author-notes><pub-date pub-type="epub"><day>4</day><month>4</month><year>2006</year></pub-date><pub-date pub-type="ppub"><month>August</month><year>2006</year></pub-date><volume>21</volume><issue>8</issue><fpage>2178</fpage><lpage>2183</lpage><history><date date-type="accepted"><day>8</day><month>3</month><year>2006</year></date><date date-type="received"><day>20</day><month>2</month><year>2006</year></date></history><permissions><copyright-statement>© The Author [2006]. Published by Oxford University Press on behalf of ERA-EDTA. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org</copyright-statement><copyright-year>2006</copyright-year></permissions><abstract xml:lang="en"><p><bold>Background.</bold> Only unbiased estimates of end-stage renal disease (ESRD) incidence and trends are useful for disease control—identification of risk factors and measuring the effect of intervention.</p><p><bold>Methods.</bold> Age- and sex-standardized incidences (with trends) were calculated for all-cause and diabetic/non-diabetic ESRD for persons aged 0–14, 15–29, 30–44 and 45–64 years in 13 populations identified geographically, and six populations identified by ethnicity.</p><p><bold>Results.</bold> The incidence of ESRD varied most with age, ethnicity and prevalence of diabetes. All non-Europid populations had excess ESRD, chiefly due to rates of type 2 diabetic ESRD that were greater than accounted for by community prevalences of diabetes. Their rates of non-diabetic ESRD also were raised, with contributions from most common primary renal diseases except type 1 diabetic nephropathy and polycystic kidney disease. The ESRD rates generally were low, and more similar than different, in Europid populations, except for variable contributions from type 1 (high in Finland, Sweden, Denmark and Canada) and type 2 (high in Austria and Canada) diabetes. In Europid populations during 1998–2002, all-cause ESRD declined by 2% per year in persons aged 0–44 years, and all non-diabetic ESRD by a similar amount in persons aged 45–64 years, in whom diabetic ESRD had increased by 3% per year.</p><p><bold>Conclusions.</bold> Increased susceptibility to type 2 diabetes and to kidney disease progression characterizes excess ESRD in non-Europid peoples. The decline in all-cause ESRD in young persons, and non-diabetic ESRD in the middle-aged, probably reflects improving management of progressive renal disease.</p></abstract><kwd-group kwd-group-type="KWD" xml:lang="en"><kwd>Asia-Pacific</kwd><kwd>Canada</kwd><kwd>end-stage renal disease</kwd><kwd>Europe</kwd><kwd>incidence</kwd><kwd>trends</kwd></kwd-group><custom-meta-wrap><custom-meta><meta-name>hwp-legacy-fpage</meta-name><meta-value>2178</meta-value></custom-meta><custom-meta><meta-name>cover-date</meta-name><meta-value>August 2006</meta-value></custom-meta><custom-meta><meta-name>hwp-legacy-dochead</meta-name><meta-value>Original Articles: Clinical Nephrology</meta-value></custom-meta></custom-meta-wrap></article-meta></front></article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Geographic, ethnic, age-related and temporal variation in the incidence of end-stage renal disease in Europe, Canada and the Asia-Pacific region, 1998–2002</title></titleInfo><titleInfo type="alternative" lang="en" contentType="CDATA"><title>Geographic, ethnic, age-related and temporal variation in the incidence of end-stage renal disease in Europe, Canada and the Asia-Pacific region, 1998–2002</title></titleInfo><name type="corporate"><namePart>The ESRD Incidence Study Group</namePart><affiliation>Department of Preventive and Social Medicine, University of Otago, Dunedin, New Zealand, The ERA-EDTA Registry, Department of Medical Informatics, Academic Medical Center, Amsterdam, The Netherlands, CORR, Canadian Institute of Health Information, Toronto, Ontario, Canada and the ANZDATA Registry, Queen Elizabeth Hospital, Adelaide, SA, Australia</affiliation></name><typeOfResource>text</typeOfResource><genre type="other" displayLabel="other"></genre><originInfo><publisher>Oxford University Press</publisher><dateIssued encoding="w3cdtf">2006-08</dateIssued><dateCreated encoding="w3cdtf">2006-04-04</dateCreated><copyrightDate encoding="w3cdtf">2006</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">Background. Only unbiased estimates of end-stage renal disease (ESRD) incidence and trends are useful for disease control—identification of risk factors and measuring the effect of intervention. Methods. Age- and sex-standardized incidences (with trends) were calculated for all-cause and diabetic/non-diabetic ESRD for persons aged 0–14, 15–29, 30–44 and 45–64 years in 13 populations identified geographically, and six populations identified by ethnicity. Results. The incidence of ESRD varied most with age, ethnicity and prevalence of diabetes. All non-Europid populations had excess ESRD, chiefly due to rates of type 2 diabetic ESRD that were greater than accounted for by community prevalences of diabetes. Their rates of non-diabetic ESRD also were raised, with contributions from most common primary renal diseases except type 1 diabetic nephropathy and polycystic kidney disease. The ESRD rates generally were low, and more similar than different, in Europid populations, except for variable contributions from type 1 (high in Finland, Sweden, Denmark and Canada) and type 2 (high in Austria and Canada) diabetes. In Europid populations during 1998–2002, all-cause ESRD declined by 2% per year in persons aged 0–44 years, and all non-diabetic ESRD by a similar amount in persons aged 45–64 years, in whom diabetic ESRD had increased by 3% per year. Conclusions. Increased susceptibility to type 2 diabetes and to kidney disease progression characterizes excess ESRD in non-Europid peoples. The decline in all-cause ESRD in young persons, and non-diabetic ESRD in the middle-aged, probably reflects improving management of progressive renal disease.</abstract><note type="author-notes">Correspondence and offprint requests to: Dr Margaret McCredie, Department of Preventive and Social Medicine, University of Otago, PO Box 913, Dunedin, New Zealand. Email: margaret.mccredie@stonebow.otago.ac.nz</note><subject lang="en"><genre>KWD</genre><topic>Asia-Pacific</topic><topic>Canada</topic><topic>end-stage renal disease</topic><topic>Europe</topic><topic>incidence</topic><topic>trends</topic></subject><relatedItem type="host"><titleInfo><title>Nephrology Dialysis Transplantation</title></titleInfo><titleInfo type="abbreviated"><title>Nephrol. Dial. 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