Symposia

Identifieur interne : 000140 ( Main/Exploration ); précédent : 000139; suivant : 000141

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Symposia

Auteurs :

Source :

• Glia [ 0894-1491 ] ; 2013-07.

RBID : ISTEX:919F9667C193D56CE73C361A5A2CC730D23D3ECB

English descriptors

• Teeft :
  • Activates, Activation, Adult brain, Adult neurogenesis, Adult nscs, Allodynia, Amyloid, Animal models, Ankb, Ankg, Apoptosis, Ascl1, Astrocyte, Astrocytic, Astroglial, Autoimmune, Axon, Axon regeneration, Axonal, Axonal regeneration, Bace1, Basal, Basal synaptic transmission, Bdnf, Biomarkers, Brain cells, Brain injury, Cell biology, Cell death, Cell proliferation, Central role, Cerebral ischemia, Cholinergic, Chronic pain, Circadian, Complement activation, Corpus callosum, Cpeb3, Cytokine, Cytosolic, Degeneration, Deletion, Demyelinating, Demyelination, Dendritic, Disease progression, Dorsal, Drosophila, Dysfunction, Emvs, Encephalomyelitis, Endogenous, Endoplasmic, Endoplasmic reticulum, Epilepsy, Epileptogenesis, Exocytosis, Exosomes, Extracellular, Fabp7, Fmrp, Functional hyperemia, Functional maturation, Functional recovery, Functional role, Gaba, Gaba transporters, Ganglion, Gene expression, Gene network, Gfap, Glia, Glia cells, Glial, Glial cells, Gliomedin, Gliotransmission, Glt1, Glucose, Glutamate, Glutamate uptake, Glycolytic, Growth factor, High levels, Hippocampal, Hippocampal neurons, Hippocampus, Hmgb1, Homeostasis, Human brain, Hyperalgesia, Hypersensitivity, Imaging, Immune, Immune cells, Immune response, Important role, Inflammation, Inflammatory, Inhibitor, Intracellular, Ischemia, Ischemic, Ischemic stroke, Kinase, Knockout, Lausanne, Lesion, Lipid, Lipid synthesis, Ller, Ller cells, Ller glia reprogramming, Mac1, Macrophage, Mechanical allodynia, Mediates, Mediator, Membrane proteins, Metabolism, Microglia, Microglial, Microglial activation, Microglial cells, Microglial phagocytosis, Microtubule, Milan, Mitochondrial, Mitochondrion, Molecular mechanisms, Mouse, Mouse model, Mouse models, Mrna, Mtor, Mtorc1, Mtorc2, Multiple sclerosis, Mutant, Mutant mice, Mutation, Myelin, Myelin growth, Myelin sheath, Myelinated, Myelinating, Myelinating glia, Myelination, Nclx, Nerve injury, Nerve repair, Nervous system, Neural, Neural circuits, Neural plasticity, Neurobiology, Neurodegeneration, Neurodegenerative, Neurodegenerative disease, Neurodegenerative diseases, Neurogenesis, Neurogenic, Neuroinflammation, Neurological, Neurology, Neuron, Neuronal, Neuronal activity, Neuronal death, Neuronal excitability, Neuronal loss, Neuropathic, Neuropathic pain, Neuroprotective, Neurosci, Neuroscience, Neurotransmitter, Nmda, Npc, Nrg1, Nrg1 type, Nsc, Oligodendrocyte, Oligodendrocyte development, Oligodendrocyte differentiation, Opcs, Paranodal, Paranodal junctions, Paranodes, Pathological conditions, Pathway, Phagocytosis, Phenotype, Plasma membrane, Plasticity, Polydendrocytes, Postnatal, Precursor, Present evidence, Presynaptic, Primary phagocytosis, Progenitor, Progenitor cells, Pyramidal neurons, Ramified, Ramified microglia, Recent findings, Receptor, Regeneration, Regenerative, Regulator, Remyelination, Reprogramming, Reticulum, Retina, Robust, Schwann, Schwann cell, Schwann cells, Sciatic, Sclerosis, Sgcs, Sodium channels, Spinal, Spinal cord, Spinal cord injury, Subunit, Symposium abstracts, Symposium abstracts symposium, Synapse, Synaptic, Synaptic activity, Synaptic plasticity, Synaptic transmission, Systemic inflammation, Tlr4, Tonic, Tonic inhibition, Trafficking, Transcriptional, Transgenic, Transgenic mice, Translational, Transplantation, Transporter, Traumatic brain injury, Universit, Vesicle, Viable neurons, Vivo, White matter.

Url:

• https://api.istex.fr/document/919F9667C193D56CE73C361A5A2CC730D23D3ECB/fulltext/pdf

DOI: 10.1002/glia.22531

Affiliations:

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• to stream Main, to step Curation: 000140

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