ADRENALINE INFUSION AND ADRENOCORTICOTROPHIN (ACTH) AND CORTISOL RELEASE IN NORMOTENSIVE AND HYPERTENSIVE MAN
Identifieur interne : 000D72 ( Istex/Corpus ); précédent : 000D71; suivant : 000D73ADRENALINE INFUSION AND ADRENOCORTICOTROPHIN (ACTH) AND CORTISOL RELEASE IN NORMOTENSIVE AND HYPERTENSIVE MAN
Auteurs : R. V. Jackson ; A. J. Jackson ; J. E. Grice ; P. J. Penfold ; M. B. Armour ; A. W. BachmannSource :
- Clinical and Experimental Pharmacology and Physiology [ 0305-1870 ] ; 1987-03.
English descriptors
- KwdEn :
- Teeft :
- Acth, Acth release, Acth secretagog, Adrenaline, Adrenaline infusion, Adrenocorticotropin release, Arginine, Arginine vasopressin, Arginine vasopressin pressor, Arterial pressure, Axelrod, Axelrod reisine, Basal, Blood pressure, Cause acth, Clinical chemistry, Cortisol, Cortisol release, Cortisol response, Cortisol rise, Deltoid muscle, Dose adrenaline infusion, Endocrinology, Experimental pharmacology physiology, Greenslopes hospital, Heart rate, Hpaa, Infusion, Intravenous adrenaline infusion, Lower plasma cortisol response, Peak changes, Pituitary corticotrophs, Placebo, Plasma acth, Pressor, Reisine, Rivier, Secretagog, Significant changes, Significant difference, University department, Vasopressin.
Abstract
1. Adrenaline causes ACTH release from cultured rat pituitary corticotrophs (Vale et al. 1983) and there is evidence that it causes ACTH release in rats in vivo (Plotsky et al. 1985). 2. The present study examined the effects of intravenous adrenaline infusion with and without simultaneous administration of the known ACTH secretagog, arginine vasopressin, in normotensive and mild essential hypertensive men on their plasma ACTH and cortisol levels. 3. Low dose adrenaline infusion (0.013 μg/kg per min) does not cause ACTH or cortisol release, but appears to blunt the ACTH and cortisol rise caused by arginine vasopressin (0.14 pressor units/kg, i.m.).
Url:
DOI: 10.1111/j.1440-1681.1987.tb00376.x
Links to Exploration step
ISTEX:844717303AB9A9D13A621BD7044C159BCA5C5451Le document en format XML
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Grice</name><affiliation><mods:affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</mods:affiliation></affiliation></author><author><name sortKey="Penfold, P J" sort="Penfold, P J" uniqKey="Penfold P" first="P. J." last="Penfold">P. J. Penfold</name><affiliation><mods:affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</mods:affiliation></affiliation></author><author><name sortKey="Armour, M B" sort="Armour, M B" uniqKey="Armour M" first="M. B." last="Armour">M. B. Armour</name><affiliation><mods:affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</mods:affiliation></affiliation></author><author><name sortKey="Bachmann, A W" sort="Bachmann, A W" uniqKey="Bachmann A" first="A. W." last="Bachmann">A. W. 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V." last="Jackson">R. V. Jackson</name><affiliation><mods:affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</mods:affiliation></affiliation></author><author><name sortKey="Jackson, A J" sort="Jackson, A J" uniqKey="Jackson A" first="A. J." last="Jackson">A. J. Jackson</name><affiliation><mods:affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</mods:affiliation></affiliation></author><author><name sortKey="Grice, J E" sort="Grice, J E" uniqKey="Grice J" first="J. E." last="Grice">J. E. Grice</name><affiliation><mods:affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</mods:affiliation></affiliation></author><author><name sortKey="Penfold, P J" sort="Penfold, P J" uniqKey="Penfold P" first="P. J." last="Penfold">P. J. Penfold</name><affiliation><mods:affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</mods:affiliation></affiliation></author><author><name sortKey="Armour, M B" sort="Armour, M B" uniqKey="Armour M" first="M. B." last="Armour">M. B. Armour</name><affiliation><mods:affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</mods:affiliation></affiliation></author><author><name sortKey="Bachmann, A W" sort="Bachmann, A W" uniqKey="Bachmann A" first="A. W." last="Bachmann">A. W. Bachmann</name><affiliation><mods:affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Clinical and Experimental Pharmacology and Physiology</title><idno type="ISSN">0305-1870</idno><idno type="eISSN">1440-1681</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1987-03">1987-03</date><biblScope unit="volume">14</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="203">203</biblScope><biblScope unit="page" to="208">208</biblScope></imprint><idno type="ISSN">0305-1870</idno></series><idno type="istex">844717303AB9A9D13A621BD7044C159BCA5C5451</idno><idno type="DOI">10.1111/j.1440-1681.1987.tb00376.x</idno><idno type="ArticleID">CEP203</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0305-1870</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>adrenaline</term><term>adrenocorticotrophin</term><term>blood pressure</term><term>cortisol</term><term>man</term><term>pituitary</term><term>stress</term><term>sympathetic nervous system</term><term>vasopressin</term></keywords><keywords scheme="Teeft" xml:lang="en"><term>Acth</term><term>Acth release</term><term>Acth secretagog</term><term>Adrenaline</term><term>Adrenaline infusion</term><term>Adrenocorticotropin release</term><term>Arginine</term><term>Arginine vasopressin</term><term>Arginine vasopressin pressor</term><term>Arterial pressure</term><term>Axelrod</term><term>Axelrod reisine</term><term>Basal</term><term>Blood pressure</term><term>Cause acth</term><term>Clinical chemistry</term><term>Cortisol</term><term>Cortisol release</term><term>Cortisol response</term><term>Cortisol rise</term><term>Deltoid muscle</term><term>Dose adrenaline infusion</term><term>Endocrinology</term><term>Experimental pharmacology physiology</term><term>Greenslopes hospital</term><term>Heart rate</term><term>Hpaa</term><term>Infusion</term><term>Intravenous adrenaline infusion</term><term>Lower plasma cortisol response</term><term>Peak changes</term><term>Pituitary corticotrophs</term><term>Placebo</term><term>Plasma acth</term><term>Pressor</term><term>Reisine</term><term>Rivier</term><term>Secretagog</term><term>Significant changes</term><term>Significant difference</term><term>University department</term><term>Vasopressin</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">1. Adrenaline causes ACTH release from cultured rat pituitary corticotrophs (Vale et al. 1983) and there is evidence that it causes ACTH release in rats in vivo (Plotsky et al. 1985). 2. The present study examined the effects of intravenous adrenaline infusion with and without simultaneous administration of the known ACTH secretagog, arginine vasopressin, in normotensive and mild essential hypertensive men on their plasma ACTH and cortisol levels. 3. Low dose adrenaline infusion (0.013 μg/kg per min) does not cause ACTH or cortisol release, but appears to blunt the ACTH and cortisol rise caused by arginine vasopressin (0.14 pressor units/kg, i.m.).</div></front></TEI><istex><corpusName>wiley</corpusName><keywords><teeft><json:string>cortisol</json:string><json:string>acth</json:string><json:string>adrenaline</json:string><json:string>basal</json:string><json:string>infusion</json:string><json:string>rivier</json:string><json:string>plasma acth</json:string><json:string>acth release</json:string><json:string>placebo</json:string><json:string>vasopressin</json:string><json:string>secretagog</json:string><json:string>peak changes</json:string><json:string>pressor</json:string><json:string>arginine</json:string><json:string>endocrinology</json:string><json:string>cortisol release</json:string><json:string>hpaa</json:string><json:string>axelrod</json:string><json:string>reisine</json:string><json:string>acth secretagog</json:string><json:string>blood pressure</json:string><json:string>dose adrenaline infusion</json:string><json:string>greenslopes hospital</json:string><json:string>significant difference</json:string><json:string>adrenaline infusion</json:string><json:string>arginine vasopressin pressor</json:string><json:string>axelrod reisine</json:string><json:string>arterial pressure</json:string><json:string>heart rate</json:string><json:string>cortisol rise</json:string><json:string>pituitary corticotrophs</json:string><json:string>intravenous adrenaline infusion</json:string><json:string>deltoid muscle</json:string><json:string>arginine vasopressin</json:string><json:string>significant changes</json:string><json:string>university department</json:string><json:string>cortisol response</json:string><json:string>lower plasma cortisol response</json:string><json:string>experimental pharmacology physiology</json:string><json:string>cause acth</json:string><json:string>adrenocorticotropin release</json:string><json:string>clinical chemistry</json:string></teeft></keywords><author><json:item><name>R. V. Jackson</name><affiliations><json:string>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</json:string></affiliations></json:item><json:item><name>A. J. Jackson</name><affiliations><json:string>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</json:string></affiliations></json:item><json:item><name>J. E. Grice</name><affiliations><json:string>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</json:string></affiliations></json:item><json:item><name>P. J. Penfold</name><affiliations><json:string>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</json:string></affiliations></json:item><json:item><name>M. B. Armour</name><affiliations><json:string>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</json:string></affiliations></json:item><json:item><name>A. W. Bachmann</name><affiliations><json:string>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>adrenaline</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>adrenocorticotrophin</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>blood pressure</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>cortisol</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>man</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>pituitary</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>stress</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>sympathetic nervous system</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>vasopressin</value></json:item></subject><articleId><json:string>CEP203</json:string></articleId><language><json:string>eng</json:string></language><originalGenre><json:string>article</json:string></originalGenre><abstract>1. Adrenaline causes ACTH release from cultured rat pituitary corticotrophs (Vale et al. 1983) and there is evidence that it causes ACTH release in rats in vivo (Plotsky et al. 1985). 2. The present study examined the effects of intravenous adrenaline infusion with and without simultaneous administration of the known ACTH secretagog, arginine vasopressin, in normotensive and mild essential hypertensive men on their plasma ACTH and cortisol levels. 3. Low dose adrenaline infusion (0.013 μg/kg per min) does not cause ACTH or cortisol release, but appears to blunt the ACTH and cortisol rise caused by arginine vasopressin (0.14 pressor units/kg, i.m.).</abstract><qualityIndicators><score>3.541</score><pdfVersion>1.3</pdfVersion><pdfPageSize>468 x 683.759 pts</pdfPageSize><refBibsNative>true</refBibsNative><abstractCharCount>656</abstractCharCount><pdfWordCount>2329</pdfWordCount><pdfCharCount>13495</pdfCharCount><pdfPageCount>6</pdfPageCount><abstractWordCount>101</abstractWordCount></qualityIndicators><title>ADRENALINE INFUSION AND ADRENOCORTICOTROPHIN (ACTH) AND CORTISOL RELEASE IN NORMOTENSIVE AND HYPERTENSIVE MAN</title><genre><json:string>article</json:string></genre><host><title>Clinical and Experimental Pharmacology and Physiology</title><language><json:string>unknown</json:string></language><doi><json:string>10.1111/(ISSN)1440-1681</json:string></doi><issn><json:string>0305-1870</json:string></issn><eissn><json:string>1440-1681</json:string></eissn><publisherId><json:string>CEP</json:string></publisherId><volume>14</volume><issue>3</issue><pages><first>203</first><last>208</last><total>6</total></pages><genre><json:string>journal</json:string></genre></host><categories><wos></wos><scienceMetrix><json:string>health sciences</json:string><json:string>biomedical research</json:string><json:string>physiology</json:string></scienceMetrix><inist><json:string>sciences appliquees, technologies et medecines</json:string><json:string>sciences biologiques et medicales</json:string><json:string>sciences medicales</json:string><json:string>sante publique. hygiene-medecine du travail</json:string></inist></categories><publicationDate>1987</publicationDate><copyrightDate>1987</copyrightDate><doi><json:string>10.1111/j.1440-1681.1987.tb00376.x</json:string></doi><id>844717303AB9A9D13A621BD7044C159BCA5C5451</id><score>1</score><fulltext><json:item><extension>pdf</extension><original>true</original><mimetype>application/pdf</mimetype><uri>https://api.istex.fr/document/844717303AB9A9D13A621BD7044C159BCA5C5451/fulltext/pdf</uri></json:item><json:item><extension>zip</extension><original>false</original><mimetype>application/zip</mimetype><uri>https://api.istex.fr/document/844717303AB9A9D13A621BD7044C159BCA5C5451/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/844717303AB9A9D13A621BD7044C159BCA5C5451/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">ADRENALINE INFUSION AND ADRENOCORTICOTROPHIN (ACTH) AND CORTISOL RELEASE IN NORMOTENSIVE AND HYPERTENSIVE MAN</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><availability><p>WILEY</p></availability><date>1987</date></publicationStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">ADRENALINE INFUSION AND ADRENOCORTICOTROPHIN (ACTH) AND CORTISOL RELEASE IN NORMOTENSIVE AND HYPERTENSIVE MAN</title><author xml:id="author-1"><persName><forename type="first">R. V.</forename><surname>Jackson</surname></persName><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation></author><author xml:id="author-2"><persName><forename type="first">A. J.</forename><surname>Jackson</surname></persName><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation></author><author xml:id="author-3"><persName><forename type="first">J. E.</forename><surname>Grice</surname></persName><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation></author><author xml:id="author-4"><persName><forename type="first">P. J.</forename><surname>Penfold</surname></persName><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation></author><author xml:id="author-5"><persName><forename type="first">M. B.</forename><surname>Armour</surname></persName><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation></author><author xml:id="author-6"><persName><forename type="first">A. W.</forename><surname>Bachmann</surname></persName><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation></author></analytic><monogr><title level="j">Clinical and Experimental Pharmacology and Physiology</title><idno type="pISSN">0305-1870</idno><idno type="eISSN">1440-1681</idno><idno type="DOI">10.1111/(ISSN)1440-1681</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1987-03"></date><biblScope unit="volume">14</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="203">203</biblScope><biblScope unit="page" to="208">208</biblScope></imprint></monogr><idno type="istex">844717303AB9A9D13A621BD7044C159BCA5C5451</idno><idno type="DOI">10.1111/j.1440-1681.1987.tb00376.x</idno><idno type="ArticleID">CEP203</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1987</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>1. Adrenaline causes ACTH release from cultured rat pituitary corticotrophs (Vale et al. 1983) and there is evidence that it causes ACTH release in rats in vivo (Plotsky et al. 1985). 2. The present study examined the effects of intravenous adrenaline infusion with and without simultaneous administration of the known ACTH secretagog, arginine vasopressin, in normotensive and mild essential hypertensive men on their plasma ACTH and cortisol levels. 3. Low dose adrenaline infusion (0.013 μg/kg per min) does not cause ACTH or cortisol release, but appears to blunt the ACTH and cortisol rise caused by arginine vasopressin (0.14 pressor units/kg, i.m.).</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>keywords</head><item><term>adrenaline</term></item><item><term>adrenocorticotrophin</term></item><item><term>blood pressure</term></item><item><term>cortisol</term></item><item><term>man</term></item><item><term>pituitary</term></item><item><term>stress</term></item><item><term>sympathetic nervous system</term></item><item><term>vasopressin</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1987-03">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><extension>txt</extension><original>false</original><mimetype>text/plain</mimetype><uri>https://api.istex.fr/document/844717303AB9A9D13A621BD7044C159BCA5C5451/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1440-1681</doi><issn type="print">0305-1870</issn><issn type="electronic">1440-1681</issn><idGroup><id type="product" value="CEP"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="CLINICAL EXPERIMENTAL PHARMACOLOGY PHYSIOLOGY">Clinical and Experimental Pharmacology and Physiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="03003"><doi origin="wiley">10.1111/cep.1987.14.issue-3</doi><numberingGroup><numbering type="journalVolume" number="14">14</numbering><numbering type="journalIssue" number="3">3</numbering></numberingGroup><coverDate startDate="1987-03">March 1987</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0020300" status="forIssue"><doi origin="wiley">10.1111/j.1440-1681.1987.tb00376.x</doi><idGroup><id type="unit" value="CEP203"></id></idGroup><countGroup><count type="pageTotal" number="6"></count></countGroup><titleGroup><title type="tocHeading1">Article</title></titleGroup><eventGroup><event type="firstOnline" date="2007-06-28"></event><event type="publishedOnlineFinalForm" date="2007-06-28"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.5 mode:FullText source:HeaderRef result:HeaderRef" date="2010-04-07"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-01-09"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-10-16"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="203">203</numbering><numbering type="pageLast" number="208">208</numbering></numberingGroup><linkGroup><link type="toTypesetVersion" href="file:CEP.CEP203.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Received 15 December 1986</unparsedEditorialHistory><countGroup><count type="referenceTotal" number="16"></count><count type="linksCrossRef" number="6"></count></countGroup><titleGroup><title type="main">ADRENALINE INFUSION AND ADRENOCORTICOTROPHIN (ACTH) AND CORTISOL RELEASE IN NORMOTENSIVE AND HYPERTENSIVE MAN</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1"><personName><givenNames>R. V.</givenNames><familyName>Jackson</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1"><personName><givenNames>A. J.</givenNames><familyName>Jackson</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>J. E.</givenNames><familyName>Grice</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a1"><personName><givenNames>P. J.</givenNames><familyName>Penfold</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a1"><personName><givenNames>M. B.</givenNames><familyName>Armour</familyName></personName></creator><creator creatorRole="author" xml:id="cr6" affiliationRef="#a1"><personName><givenNames>A. W.</givenNames><familyName>Bachmann</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="AU"><unparsedAffiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">adrenaline</keyword><keyword xml:id="k2">adrenocorticotrophin</keyword><keyword xml:id="k3">blood pressure</keyword><keyword xml:id="k4">cortisol</keyword><keyword xml:id="k5">man</keyword><keyword xml:id="k6">pituitary</keyword><keyword xml:id="k7">stress</keyword><keyword xml:id="k8">sympathetic nervous system</keyword><keyword xml:id="k9">vasopressin</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><title type="main">SUMMARY</title><p>1. Adrenaline causes ACTH release from cultured rat pituitary corticotrophs (Vale <i>et al.</i> 1983) and there is evidence that it causes ACTH release in rats <i>in vivo</i> (Plotsky <i>et al.</i> 1985).</p><p>2. The present study examined the effects of intravenous adrenaline infusion with and without simultaneous administration of the known ACTH secretagog, arginine vasopressin, in normotensive and mild essential hypertensive men on their plasma ACTH and cortisol levels.</p><p>3. Low dose adrenaline infusion (0.013 μg/kg per min) does not cause ACTH or cortisol release, but appears to blunt the ACTH and cortisol rise caused by arginine vasopressin (0.14 pressor units/kg, i.m.).</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>ADRENALINE INFUSION AND ADRENOCORTICOTROPHIN (ACTH) AND CORTISOL RELEASE IN NORMOTENSIVE AND HYPERTENSIVE MAN</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>ADRENALINE INFUSION AND ADRENOCORTICOTROPHIN (ACTH) AND CORTISOL RELEASE IN NORMOTENSIVE AND HYPERTENSIVE MAN</title></titleInfo><name type="personal"><namePart type="given">R. V.</namePart><namePart type="family">Jackson</namePart><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">A. J.</namePart><namePart type="family">Jackson</namePart><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">J. E.</namePart><namePart type="family">Grice</namePart><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">P. J.</namePart><namePart type="family">Penfold</namePart><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">M. B.</namePart><namePart type="family">Armour</namePart><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">A. W.</namePart><namePart type="family">Bachmann</namePart><affiliation>Neuroendocrine Research Unit, University Department of Medicine, Greenslopes Hospital, Brisbane, Queensland, Australia</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1987-03</dateIssued><edition>Received 15 December 1986</edition><copyrightDate encoding="w3cdtf">1987</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">16</extent></physicalDescription><abstract lang="en">1. Adrenaline causes ACTH release from cultured rat pituitary corticotrophs (Vale et al. 1983) and there is evidence that it causes ACTH release in rats in vivo (Plotsky et al. 1985). 2. The present study examined the effects of intravenous adrenaline infusion with and without simultaneous administration of the known ACTH secretagog, arginine vasopressin, in normotensive and mild essential hypertensive men on their plasma ACTH and cortisol levels. 3. Low dose adrenaline infusion (0.013 μg/kg per min) does not cause ACTH or cortisol release, but appears to blunt the ACTH and cortisol rise caused by arginine vasopressin (0.14 pressor units/kg, i.m.).</abstract><subject lang="en"><genre>keywords</genre><topic>adrenaline</topic><topic>adrenocorticotrophin</topic><topic>blood pressure</topic><topic>cortisol</topic><topic>man</topic><topic>pituitary</topic><topic>stress</topic><topic>sympathetic nervous system</topic><topic>vasopressin</topic></subject><relatedItem type="host"><titleInfo><title>Clinical and Experimental Pharmacology and Physiology</title></titleInfo><genre type="journal">journal</genre><identifier type="ISSN">0305-1870</identifier><identifier type="eISSN">1440-1681</identifier><identifier type="DOI">10.1111/(ISSN)1440-1681</identifier><identifier type="PublisherID">CEP</identifier><part><date>1987</date><detail type="volume"><caption>vol.</caption><number>14</number></detail><detail type="issue"><caption>no.</caption><number>3</number></detail><extent unit="pages"><start>203</start><end>208</end><total>6</total></extent></part></relatedItem><identifier type="istex">844717303AB9A9D13A621BD7044C159BCA5C5451</identifier><identifier type="DOI">10.1111/j.1440-1681.1987.tb00376.x</identifier><identifier type="ArticleID">CEP203</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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