Endocrine correlates of stress vulnerability.
Identifieur interne : 000A27 ( PubMed/Corpus ); précédent : 000A26; suivant : 000A28Endocrine correlates of stress vulnerability.
Auteurs : D H Hellhammer ; S. WadeSource :
- Psychotherapy and psychosomatics [ 0033-3190 ] ; 1993.
English descriptors
- KwdEn :
- Adjustment Disorders (psychology), Adrenocorticotropic Hormone (physiology), Arginine Vasopressin (physiology), Cognition, Corticotropin-Releasing Hormone (physiology), Female, Habituation, Psychophysiologic, Humans, Hypothalamus (metabolism), Hypothalamus (physiology), Male, Pituitary-Adrenal System (physiology), Psychophysiologic Disorders (etiology), Psychophysiologic Disorders (metabolism), Sex Factors, Social Environment, Stress, Psychological (metabolism), Stress, Psychological (psychology), Twins, Monozygotic (psychology).
- MESH :
- chemical , physiology : Adrenocorticotropic Hormone, Arginine Vasopressin, Corticotropin-Releasing Hormone.
- etiology : Psychophysiologic Disorders.
- metabolism : Hypothalamus, Psychophysiologic Disorders, Stress, Psychological.
- physiology : Hypothalamus, Pituitary-Adrenal System.
- psychology : Adjustment Disorders, Stress, Psychological, Twins, Monozygotic.
- Cognition, Female, Habituation, Psychophysiologic, Humans, Male, Sex Factors, Social Environment.
Abstract
Psychological stress is implicated as one element in a constellation of risk factors for the development and maintenance of psychosomatic disease. Endocrine responses to stressful experience vary widely among individuals, and may account for some of the individual variability in vulnerability to development of psychosomatic symptoms under stress. Endocrine stress responsiveness probably has a large genetic component, but is also known to be modifiable by prolonged and/or profound stressful experience. Social and cognitive factors influence the pituitary-adrenal response to stress, and sex differences may also be significant. It may be possible to develop specific tests of endocrine responsiveness, perhaps incorporating psychological and pharmacological challenges, that may serve as markers of chronic stress experience or of vulnerability to psychosomatic disorders.
PubMed: 8234641
Links to Exploration step
pubmed:8234641Le document en format XML
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<author><name sortKey="Hellhammer, D H" sort="Hellhammer, D H" uniqKey="Hellhammer D" first="D H" last="Hellhammer">D H Hellhammer</name>
<affiliation><nlm:affiliation>Center for Psychobiological and Psychosomatic Research, Trier University, FRG.</nlm:affiliation>
</affiliation>
</author>
<author><name sortKey="Wade, S" sort="Wade, S" uniqKey="Wade S" first="S" last="Wade">S. Wade</name>
</author>
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<author><name sortKey="Hellhammer, D H" sort="Hellhammer, D H" uniqKey="Hellhammer D" first="D H" last="Hellhammer">D H Hellhammer</name>
<affiliation><nlm:affiliation>Center for Psychobiological and Psychosomatic Research, Trier University, FRG.</nlm:affiliation>
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<author><name sortKey="Wade, S" sort="Wade, S" uniqKey="Wade S" first="S" last="Wade">S. Wade</name>
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<series><title level="j">Psychotherapy and psychosomatics</title>
<idno type="ISSN">0033-3190</idno>
<imprint><date when="1993" type="published">1993</date>
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Adjustment Disorders (psychology)</term>
<term>Adrenocorticotropic Hormone (physiology)</term>
<term>Arginine Vasopressin (physiology)</term>
<term>Cognition</term>
<term>Corticotropin-Releasing Hormone (physiology)</term>
<term>Female</term>
<term>Habituation, Psychophysiologic</term>
<term>Humans</term>
<term>Hypothalamus (metabolism)</term>
<term>Hypothalamus (physiology)</term>
<term>Male</term>
<term>Pituitary-Adrenal System (physiology)</term>
<term>Psychophysiologic Disorders (etiology)</term>
<term>Psychophysiologic Disorders (metabolism)</term>
<term>Sex Factors</term>
<term>Social Environment</term>
<term>Stress, Psychological (metabolism)</term>
<term>Stress, Psychological (psychology)</term>
<term>Twins, Monozygotic (psychology)</term>
</keywords>
<keywords scheme="MESH" type="chemical" qualifier="physiology" xml:lang="en"><term>Adrenocorticotropic Hormone</term>
<term>Arginine Vasopressin</term>
<term>Corticotropin-Releasing Hormone</term>
</keywords>
<keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Psychophysiologic Disorders</term>
</keywords>
<keywords scheme="MESH" qualifier="metabolism" xml:lang="en"><term>Hypothalamus</term>
<term>Psychophysiologic Disorders</term>
<term>Stress, Psychological</term>
</keywords>
<keywords scheme="MESH" qualifier="physiology" xml:lang="en"><term>Hypothalamus</term>
<term>Pituitary-Adrenal System</term>
</keywords>
<keywords scheme="MESH" qualifier="psychology" xml:lang="en"><term>Adjustment Disorders</term>
<term>Stress, Psychological</term>
<term>Twins, Monozygotic</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Cognition</term>
<term>Female</term>
<term>Habituation, Psychophysiologic</term>
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<term>Male</term>
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<front><div type="abstract" xml:lang="en">Psychological stress is implicated as one element in a constellation of risk factors for the development and maintenance of psychosomatic disease. Endocrine responses to stressful experience vary widely among individuals, and may account for some of the individual variability in vulnerability to development of psychosomatic symptoms under stress. Endocrine stress responsiveness probably has a large genetic component, but is also known to be modifiable by prolonged and/or profound stressful experience. Social and cognitive factors influence the pituitary-adrenal response to stress, and sex differences may also be significant. It may be possible to develop specific tests of endocrine responsiveness, perhaps incorporating psychological and pharmacological challenges, that may serve as markers of chronic stress experience or of vulnerability to psychosomatic disorders.</div>
</front>
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<DateCreated><Year>1993</Year>
<Month>12</Month>
<Day>22</Day>
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<DateCompleted><Year>1993</Year>
<Month>12</Month>
<Day>22</Day>
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<DateRevised><Year>2009</Year>
<Month>11</Month>
<Day>11</Day>
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<Article PubModel="Print"><Journal><ISSN IssnType="Print">0033-3190</ISSN>
<JournalIssue CitedMedium="Print"><Volume>60</Volume>
<Issue>1</Issue>
<PubDate><Year>1993</Year>
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<Title>Psychotherapy and psychosomatics</Title>
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<ArticleTitle>Endocrine correlates of stress vulnerability.</ArticleTitle>
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<Abstract><AbstractText>Psychological stress is implicated as one element in a constellation of risk factors for the development and maintenance of psychosomatic disease. Endocrine responses to stressful experience vary widely among individuals, and may account for some of the individual variability in vulnerability to development of psychosomatic symptoms under stress. Endocrine stress responsiveness probably has a large genetic component, but is also known to be modifiable by prolonged and/or profound stressful experience. Social and cognitive factors influence the pituitary-adrenal response to stress, and sex differences may also be significant. It may be possible to develop specific tests of endocrine responsiveness, perhaps incorporating psychological and pharmacological challenges, that may serve as markers of chronic stress experience or of vulnerability to psychosomatic disorders.</AbstractText>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Hellhammer</LastName>
<ForeName>D H</ForeName>
<Initials>DH</Initials>
<AffiliationInfo><Affiliation>Center for Psychobiological and Psychosomatic Research, Trier University, FRG.</Affiliation>
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<Author ValidYN="Y"><LastName>Wade</LastName>
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<ChemicalList><Chemical><RegistryNumber>113-79-1</RegistryNumber>
<NameOfSubstance UI="D001127">Arginine Vasopressin</NameOfSubstance>
</Chemical>
<Chemical><RegistryNumber>9002-60-2</RegistryNumber>
<NameOfSubstance UI="D000324">Adrenocorticotropic Hormone</NameOfSubstance>
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<Chemical><RegistryNumber>9015-71-8</RegistryNumber>
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<MeshHeading><DescriptorName UI="D000324" MajorTopicYN="N">Adrenocorticotropic Hormone</DescriptorName>
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<MeshHeading><DescriptorName UI="D001127" MajorTopicYN="N">Arginine Vasopressin</DescriptorName>
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<MeshHeading><DescriptorName UI="D003071" MajorTopicYN="N">Cognition</DescriptorName>
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<MeshHeading><DescriptorName UI="D003346" MajorTopicYN="N">Corticotropin-Releasing Hormone</DescriptorName>
<QualifierName UI="Q000502" MajorTopicYN="N">physiology</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D005260" MajorTopicYN="N">Female</DescriptorName>
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<MeshHeading><DescriptorName UI="D006185" MajorTopicYN="N">Habituation, Psychophysiologic</DescriptorName>
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<MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName>
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<MeshHeading><DescriptorName UI="D007031" MajorTopicYN="N">Hypothalamus</DescriptorName>
<QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName>
<QualifierName UI="Q000502" MajorTopicYN="N">physiology</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D010913" MajorTopicYN="N">Pituitary-Adrenal System</DescriptorName>
<QualifierName UI="Q000502" MajorTopicYN="Y">physiology</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D011602" MajorTopicYN="N">Psychophysiologic Disorders</DescriptorName>
<QualifierName UI="Q000209" MajorTopicYN="Y">etiology</QualifierName>
<QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D012737" MajorTopicYN="N">Sex Factors</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D012931" MajorTopicYN="N">Social Environment</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D013315" MajorTopicYN="N">Stress, Psychological</DescriptorName>
<QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName>
<QualifierName UI="Q000523" MajorTopicYN="Y">psychology</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D014430" MajorTopicYN="N">Twins, Monozygotic</DescriptorName>
<QualifierName UI="Q000523" MajorTopicYN="N">psychology</QualifierName>
</MeshHeading>
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<NumberOfReferences>38</NumberOfReferences>
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