UnivTrevesV1, PascalFrancis, Corpus, bibRecord, 000512

Recovery of Upper Limb Function After Cerebellar Stroke Lesion Symptom Mapping and Arm Kinematics

Identifieur interne : 000512 ( PascalFrancis/Corpus ); précédent : 000511; suivant : 000513

Recovery of Upper Limb Function After Cerebellar Stroke Lesion Symptom Mapping and Arm Kinematics

Auteurs : Jürgen Konczak ; Daniela Pierscianek ; Sarah Hirsiger ; Uta Bultmann ; Beate Schoch ; Elke R. Gizewski ; Dagmar Timmann ; Matthias Maschke ; Markus Frings

Source :

Stroke : (1970) [ 0039-2499 ] ; 2010.

RBID : Pascal:10-0476192

Descripteurs français

Pascal (Inist)
- Accident cérébrovasculaire, Ataxie, Pathologie du système nerveux, Pathologie cérébrovasculaire, Membre supérieur, Cervelet, Cartographie, Cinématique, Encéphale, Homme.

English descriptors

KwdEn :
- Ataxia, Cartography, Cerebellum, Cerebrovascular disease, Encephalon, Human, Kinematics, Nervous system diseases, Stroke, Upper limb.

Abstract

Background and Purpose-Loss of movement coordination is the main postacute symptom after cerebellar infarction. Although the course of motor recovery has been described previously, detailed kinematic descriptions of acute stage ataxia are rare and no attempt has been made to link improvements in motor function to measures of neural recovery and lesion location. This study provides a comprehensive assessment of how lesion site and arm dysfunction are associated in the acute stage and outlines the course of upper limb motor recovery for the first 4 months after the infarction. Methods-Sixteen adult patients with cerebellar stroke and 11 age-matched healthy controls participated. Kinematics of goal-directed and unconstrained finger-pointing movements were measured at the acute stage and in 2-week and 3-month follow-ups. MRI data were obtained for the acute and 3-month follow-up sessions. A voxel-based lesion map subtraction analysis was performed to examine the effect of ischemic lesion sites on kinematic performance. Results-In the acute stage, nearly 70% of patients exhibited motor slowing with hand velocity and acceleration maxima below the range of the control group. MRI analysis revealed that in patients with impaired motor performance, lesions were more common in paravermal lobules IVN and affected the deep cerebellar nuclei. Stroke affecting the superior cerebellar artery led to lower motor performance than infractions of the posterior cerebellar artery. By the 2-week-follow-up, hand kinematics had improved dramatically (gains in acceleration up to 86%). Improvements between the 2-week and the 3-month-follow-ups were less pronounced. Conclusion-In the acute stage, arm movements were mainly characterized by abnormal slowness (bradykinesia) and not dyscoordination (ataxia). The motor signs were associated with lesions in paravermal regions of lobules IVN and the deep cerebellar nuclei. Motor recovery was fast, with the majority of gains in upper limb function occurring in the first 2 weeks after the acute phase.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

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A03		`1`		`@0 Stroke : (1970)`
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A08	`01`	`1`	`ENG`	`@1 Recovery of Upper Limb Function After Cerebellar Stroke Lesion Symptom Mapping and Arm Kinematics`
A11	`01`	`1`		`@1 KONCZAK (Jürgen)`
A11	`02`	`1`		`@1 PIERSCIANEK (Daniela)`
A11	`03`	`1`		`@1 HIRSIGER (Sarah)`
A11	`04`	`1`		`@1 BULTMANN (Uta)`
A11	`05`	`1`		`@1 SCHOCH (Beate)`
A11	`06`	`1`		`@1 GIZEWSKI (Elke R.)`
A11	`07`	`1`		`@1 TIMMANN (Dagmar)`
A11	`08`	`1`		`@1 MASCHKE (Matthias)`
A11	`09`	`1`		`@1 FRINGS (Markus)`
A14	`01`			`@1 Human Sensorimotor Control Laboratory, University of Minnesota @2 Minneapolis, Mn @3 USA @Z 1 aut. @Z 3 aut.`
A14	`02`			`@1 Department of Neurology, University of Duisburg-Essen @2 Essen @3 DEU @Z 2 aut. @Z 4 aut. @Z 7 aut. @Z 9 aut.`
A14	`03`			`@1 Institute of Human Movement Sciences and Sport ETH Zürich @2 Zürich @3 CHE @Z 3 aut.`
A14	`04`			`@1 Department of Neurosurgery, University of Duisburg-Essen @2 Essen @3 DEU @Z 5 aut.`
A14	`05`			`@1 Department of Neuroradiology, University of Duisburg-Essen @2 Essen @3 DEU @Z 6 aut.`
A14	`06`			`@1 Department of Neuroradiology, UKGM, Justus-Liebig University @2 Giessen @3 DEU @Z 6 aut.`
A14	`07`			`@1 Department of Neurology, Briiderkrankenhaus @2 Trier @3 DEU @Z 8 aut.`
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A44				`@0 0000 @1 © 2010 INIST-CNRS. All rights reserved.`
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A47	`01`	`1`		`@0 10-0476192`
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C01	`01`		`ENG`	@0 Background and Purpose-Loss of movement coordination is the main postacute symptom after cerebellar infarction. Although the course of motor recovery has been described previously, detailed kinematic descriptions of acute stage ataxia are rare and no attempt has been made to link improvements in motor function to measures of neural recovery and lesion location. This study provides a comprehensive assessment of how lesion site and arm dysfunction are associated in the acute stage and outlines the course of upper limb motor recovery for the first 4 months after the infarction. Methods-Sixteen adult patients with cerebellar stroke and 11 age-matched healthy controls participated. Kinematics of goal-directed and unconstrained finger-pointing movements were measured at the acute stage and in 2-week and 3-month follow-ups. MRI data were obtained for the acute and 3-month follow-up sessions. A voxel-based lesion map subtraction analysis was performed to examine the effect of ischemic lesion sites on kinematic performance. Results-In the acute stage, nearly 70% of patients exhibited motor slowing with hand velocity and acceleration maxima below the range of the control group. MRI analysis revealed that in patients with impaired motor performance, lesions were more common in paravermal lobules IVN and affected the deep cerebellar nuclei. Stroke affecting the superior cerebellar artery led to lower motor performance than infractions of the posterior cerebellar artery. By the 2-week-follow-up, hand kinematics had improved dramatically (gains in acceleration up to 86%). Improvements between the 2-week and the 3-month-follow-ups were less pronounced. Conclusion-In the acute stage, arm movements were mainly characterized by abnormal slowness (bradykinesia) and not dyscoordination (ataxia). The motor signs were associated with lesions in paravermal regions of lobules IVN and the deep cerebellar nuclei. Motor recovery was fast, with the majority of gains in upper limb function occurring in the first 2 weeks after the acute phase.
C02	`01`	`X`		`@0 002B17C`
C02	`02`	`X`		`@0 002B17A03`
C03	`01`	`X`	`FRE`	`@0 Accident cérébrovasculaire @5 01`
C03	`01`	`X`	`ENG`	`@0 Stroke @5 01`
C03	`01`	`X`	`SPA`	`@0 Accidente cerebrovascular @5 01`
C03	`02`	`X`	`FRE`	`@0 Ataxie @5 02`
C03	`02`	`X`	`ENG`	`@0 Ataxia @5 02`
C03	`02`	`X`	`SPA`	`@0 Ataxia @5 02`
C03	`03`	`X`	`FRE`	`@0 Pathologie du système nerveux @5 03`
C03	`03`	`X`	`ENG`	`@0 Nervous system diseases @5 03`
C03	`03`	`X`	`SPA`	`@0 Sistema nervioso patología @5 03`
C03	`04`	`X`	`FRE`	`@0 Pathologie cérébrovasculaire @5 04`
C03	`04`	`X`	`ENG`	`@0 Cerebrovascular disease @5 04`
C03	`04`	`X`	`SPA`	`@0 Vaso sanguíneo encéfalo patología @5 04`
C03	`05`	`X`	`FRE`	`@0 Membre supérieur @5 09`
C03	`05`	`X`	`ENG`	`@0 Upper limb @5 09`
C03	`05`	`X`	`SPA`	`@0 Miembro superior @5 09`
C03	`06`	`X`	`FRE`	`@0 Cervelet @5 10`
C03	`06`	`X`	`ENG`	`@0 Cerebellum @5 10`
C03	`06`	`X`	`SPA`	`@0 Cerebelo @5 10`
C03	`07`	`X`	`FRE`	`@0 Cartographie @5 11`
C03	`07`	`X`	`ENG`	`@0 Cartography @5 11`
C03	`07`	`X`	`SPA`	`@0 Cartografía @5 11`
C03	`08`	`X`	`FRE`	`@0 Cinématique @5 12`
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C03	`09`	`X`	`FRE`	`@0 Encéphale @5 13`
C03	`09`	`X`	`ENG`	`@0 Encephalon @5 13`
C03	`09`	`X`	`SPA`	`@0 Encéfalo @5 13`
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C03	`10`	`X`	`ENG`	`@0 Human @5 14`
C03	`10`	`X`	`SPA`	`@0 Hombre @5 14`
C07	`01`	`X`	`FRE`	`@0 Pathologie de l'appareil circulatoire @5 37`
C07	`01`	`X`	`ENG`	`@0 Cardiovascular disease @5 37`
C07	`01`	`X`	`SPA`	`@0 Aparato circulatorio patología @5 37`
C07	`02`	`X`	`FRE`	`@0 Pathologie de l'encéphale @5 38`
C07	`02`	`X`	`ENG`	`@0 Cerebral disorder @5 38`
C07	`02`	`X`	`SPA`	`@0 Encéfalo patología @5 38`
C07	`03`	`X`	`FRE`	`@0 Pathologie du système nerveux central @5 39`
C07	`03`	`X`	`ENG`	`@0 Central nervous system disease @5 39`
C07	`03`	`X`	`SPA`	`@0 Sistema nervosio central patología @5 39`
C07	`04`	`X`	`FRE`	`@0 Pathologie des vaisseaux sanguins @5 41`
C07	`04`	`X`	`ENG`	`@0 Vascular disease @5 41`
C07	`04`	`X`	`SPA`	`@0 Vaso sanguíneo patología @5 41`
C07	`05`	`X`	`FRE`	`@0 Trouble neurologique @5 42`
C07	`05`	`X`	`ENG`	`@0 Neurological disorder @5 42`
C07	`05`	`X`	`SPA`	`@0 Trastorno neurológico @5 42`
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N44	`01`			`@1 OTO`
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Format Inist (serveur)

NO :	PASCAL 10-0476192 INIST
ET :	Recovery of Upper Limb Function After Cerebellar Stroke Lesion Symptom Mapping and Arm Kinematics
AU :	KONCZAK (Jürgen); PIERSCIANEK (Daniela); HIRSIGER (Sarah); BULTMANN (Uta); SCHOCH (Beate); GIZEWSKI (Elke R.); TIMMANN (Dagmar); MASCHKE (Matthias); FRINGS (Markus)
AF :	Human Sensorimotor Control Laboratory, University of Minnesota/Minneapolis, Mn/Etats-Unis (1 aut., 3 aut.); Department of Neurology, University of Duisburg-Essen/Essen/Allemagne (2 aut., 4 aut., 7 aut., 9 aut.); Institute of Human Movement Sciences and Sport ETH Zürich/Zürich/Suisse (3 aut.); Department of Neurosurgery, University of Duisburg-Essen/Essen/Allemagne (5 aut.); Department of Neuroradiology, University of Duisburg-Essen/Essen/Allemagne (6 aut.); Department of Neuroradiology, UKGM, Justus-Liebig University/Giessen/Allemagne (6 aut.); Department of Neurology, Briiderkrankenhaus/Trier/Allemagne (8 aut.)
DT :	Publication en série; Niveau analytique
SO :	Stroke : (1970); ISSN 0039-2499; Coden SJCCA7; Etats-Unis; Da. 2010; Vol. 41; No. 10; Pp. 2191-2200; Bibl. 37 ref.
LA :	Anglais
EA :	Background and Purpose-Loss of movement coordination is the main postacute symptom after cerebellar infarction. Although the course of motor recovery has been described previously, detailed kinematic descriptions of acute stage ataxia are rare and no attempt has been made to link improvements in motor function to measures of neural recovery and lesion location. This study provides a comprehensive assessment of how lesion site and arm dysfunction are associated in the acute stage and outlines the course of upper limb motor recovery for the first 4 months after the infarction. Methods-Sixteen adult patients with cerebellar stroke and 11 age-matched healthy controls participated. Kinematics of goal-directed and unconstrained finger-pointing movements were measured at the acute stage and in 2-week and 3-month follow-ups. MRI data were obtained for the acute and 3-month follow-up sessions. A voxel-based lesion map subtraction analysis was performed to examine the effect of ischemic lesion sites on kinematic performance. Results-In the acute stage, nearly 70% of patients exhibited motor slowing with hand velocity and acceleration maxima below the range of the control group. MRI analysis revealed that in patients with impaired motor performance, lesions were more common in paravermal lobules IVN and affected the deep cerebellar nuclei. Stroke affecting the superior cerebellar artery led to lower motor performance than infractions of the posterior cerebellar artery. By the 2-week-follow-up, hand kinematics had improved dramatically (gains in acceleration up to 86%). Improvements between the 2-week and the 3-month-follow-ups were less pronounced. Conclusion-In the acute stage, arm movements were mainly characterized by abnormal slowness (bradykinesia) and not dyscoordination (ataxia). The motor signs were associated with lesions in paravermal regions of lobules IVN and the deep cerebellar nuclei. Motor recovery was fast, with the majority of gains in upper limb function occurring in the first 2 weeks after the acute phase.
CC :	002B17C; 002B17A03
FD :	Accident cérébrovasculaire; Ataxie; Pathologie du système nerveux; Pathologie cérébrovasculaire; Membre supérieur; Cervelet; Cartographie; Cinématique; Encéphale; Homme
FG :	Pathologie de l'appareil circulatoire; Pathologie de l'encéphale; Pathologie du système nerveux central; Pathologie des vaisseaux sanguins; Trouble neurologique
ED :	Stroke; Ataxia; Nervous system diseases; Cerebrovascular disease; Upper limb; Cerebellum; Cartography; Kinematics; Encephalon; Human
EG :	Cardiovascular disease; Cerebral disorder; Central nervous system disease; Vascular disease; Neurological disorder
SD :	Accidente cerebrovascular; Ataxia; Sistema nervioso patología; Vaso sanguíneo encéfalo patología; Miembro superior; Cerebelo; Cartografía; Cinemática; Encéfalo; Hombre
LO :	INIST-4004.354000192450920100
ID :	10-0476192

Links to Exploration step

Pascal:10-0476192

Le document en format XML

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<author><name sortKey="Maschke, Matthias" sort="Maschke, Matthias" uniqKey="Maschke M" first="Matthias" last="Maschke">Matthias Maschke</name>
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<author><name sortKey="Frings, Markus" sort="Frings, Markus" uniqKey="Frings M" first="Markus" last="Frings">Markus Frings</name>
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<sourceDesc><biblStruct><analytic><title xml:lang="en" level="a">Recovery of Upper Limb Function After Cerebellar Stroke Lesion Symptom Mapping and Arm Kinematics</title>
<author><name sortKey="Konczak, Jurgen" sort="Konczak, Jurgen" uniqKey="Konczak J" first="Jürgen" last="Konczak">Jürgen Konczak</name>
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<author><name sortKey="Pierscianek, Daniela" sort="Pierscianek, Daniela" uniqKey="Pierscianek D" first="Daniela" last="Pierscianek">Daniela Pierscianek</name>
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<sZ>4 aut.</sZ>
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<author><name sortKey="Hirsiger, Sarah" sort="Hirsiger, Sarah" uniqKey="Hirsiger S" first="Sarah" last="Hirsiger">Sarah Hirsiger</name>
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<author><name sortKey="Bultmann, Uta" sort="Bultmann, Uta" uniqKey="Bultmann U" first="Uta" last="Bultmann">Uta Bultmann</name>
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<s2>Essen</s2>
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<author><name sortKey="Schoch, Beate" sort="Schoch, Beate" uniqKey="Schoch B" first="Beate" last="Schoch">Beate Schoch</name>
<affiliation><inist:fA14 i1="04"><s1>Department of Neurosurgery, University of Duisburg-Essen</s1>
<s2>Essen</s2>
<s3>DEU</s3>
<sZ>5 aut.</sZ>
</inist:fA14>
</affiliation>
</author>
<author><name sortKey="Gizewski, Elke R" sort="Gizewski, Elke R" uniqKey="Gizewski E" first="Elke R." last="Gizewski">Elke R. Gizewski</name>
<affiliation><inist:fA14 i1="05"><s1>Department of Neuroradiology, University of Duisburg-Essen</s1>
<s2>Essen</s2>
<s3>DEU</s3>
<sZ>6 aut.</sZ>
</inist:fA14>
</affiliation>
<affiliation><inist:fA14 i1="06"><s1>Department of Neuroradiology, UKGM, Justus-Liebig University</s1>
<s2>Giessen</s2>
<s3>DEU</s3>
<sZ>6 aut.</sZ>
</inist:fA14>
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</author>
<author><name sortKey="Timmann, Dagmar" sort="Timmann, Dagmar" uniqKey="Timmann D" first="Dagmar" last="Timmann">Dagmar Timmann</name>
<affiliation><inist:fA14 i1="02"><s1>Department of Neurology, University of Duisburg-Essen</s1>
<s2>Essen</s2>
<s3>DEU</s3>
<sZ>2 aut.</sZ>
<sZ>4 aut.</sZ>
<sZ>7 aut.</sZ>
<sZ>9 aut.</sZ>
</inist:fA14>
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</author>
<author><name sortKey="Maschke, Matthias" sort="Maschke, Matthias" uniqKey="Maschke M" first="Matthias" last="Maschke">Matthias Maschke</name>
<affiliation><inist:fA14 i1="07"><s1>Department of Neurology, Briiderkrankenhaus</s1>
<s2>Trier</s2>
<s3>DEU</s3>
<sZ>8 aut.</sZ>
</inist:fA14>
</affiliation>
</author>
<author><name sortKey="Frings, Markus" sort="Frings, Markus" uniqKey="Frings M" first="Markus" last="Frings">Markus Frings</name>
<affiliation><inist:fA14 i1="02"><s1>Department of Neurology, University of Duisburg-Essen</s1>
<s2>Essen</s2>
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<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Ataxia</term>
<term>Cartography</term>
<term>Cerebellum</term>
<term>Cerebrovascular disease</term>
<term>Encephalon</term>
<term>Human</term>
<term>Kinematics</term>
<term>Nervous system diseases</term>
<term>Stroke</term>
<term>Upper limb</term>
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<term>Ataxie</term>
<term>Pathologie du   système nerveux</term>
<term>Pathologie cérébrovasculaire</term>
<term>Membre supérieur</term>
<term>Cervelet</term>
<term>Cartographie</term>
<term>Cinématique</term>
<term>Encéphale</term>
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<front><div type="abstract" xml:lang="en">Background and Purpose-Loss of movement coordination is the main postacute symptom after cerebellar infarction. Although the course of motor recovery has been described previously, detailed kinematic descriptions of acute stage ataxia are rare and no attempt has been made to link improvements in motor function to measures of neural recovery and lesion location. This study provides a comprehensive assessment of how lesion site and arm dysfunction are associated in the acute stage and outlines the course of upper limb motor recovery for the first 4 months after the infarction. Methods-Sixteen adult patients with cerebellar stroke and 11 age-matched healthy controls participated. Kinematics of goal-directed and unconstrained finger-pointing movements were measured at the acute stage and in 2-week and 3-month follow-ups. MRI data were obtained for the acute and 3-month follow-up sessions. A voxel-based lesion map subtraction analysis was performed to examine the effect of ischemic lesion sites on kinematic performance. Results-In the acute stage, nearly 70% of patients exhibited motor slowing with hand velocity and acceleration maxima below the range of the control group. MRI analysis revealed that in patients with impaired motor performance, lesions were more common in paravermal lobules IVN and affected the deep cerebellar nuclei. Stroke affecting the superior cerebellar artery led to lower motor performance than infractions of the posterior cerebellar artery. By the 2-week-follow-up, hand kinematics had improved dramatically (gains in acceleration up to 86%). Improvements between the 2-week and the 3-month-follow-ups were less pronounced. Conclusion-In the acute stage, arm movements were mainly characterized by abnormal slowness (bradykinesia) and not dyscoordination (ataxia). The motor signs were associated with lesions in paravermal regions of lobules IVN and the deep cerebellar nuclei. Motor recovery was fast, with the majority of gains in upper limb function occurring in the first 2 weeks after the acute phase.</div>
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<sZ>8 aut.</sZ>
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<server><NO>PASCAL 10-0476192 INIST</NO>
<ET>Recovery of Upper Limb Function After Cerebellar Stroke Lesion Symptom Mapping and Arm Kinematics</ET>
<AU>KONCZAK (Jürgen); PIERSCIANEK (Daniela); HIRSIGER (Sarah); BULTMANN (Uta); SCHOCH (Beate); GIZEWSKI (Elke R.); TIMMANN (Dagmar); MASCHKE (Matthias); FRINGS (Markus)</AU>
<AF>Human Sensorimotor Control Laboratory, University of Minnesota/Minneapolis, Mn/Etats-Unis (1 aut., 3 aut.); Department of Neurology, University of Duisburg-Essen/Essen/Allemagne (2 aut., 4 aut., 7 aut., 9 aut.); Institute of Human Movement Sciences and Sport ETH Zürich/Zürich/Suisse (3 aut.); Department of Neurosurgery, University of Duisburg-Essen/Essen/Allemagne (5 aut.); Department of Neuroradiology, University of Duisburg-Essen/Essen/Allemagne (6 aut.); Department of Neuroradiology, UKGM, Justus-Liebig University/Giessen/Allemagne (6 aut.); Department of Neurology, Briiderkrankenhaus/Trier/Allemagne (8 aut.)</AF>
<DT>Publication en série; Niveau analytique</DT>
<SO>Stroke : (1970); ISSN 0039-2499; Coden SJCCA7; Etats-Unis; Da. 2010; Vol. 41; No. 10; Pp. 2191-2200; Bibl. 37 ref.</SO>
<LA>Anglais</LA>
<EA>Background and Purpose-Loss of movement coordination is the main postacute symptom after cerebellar infarction. Although the course of motor recovery has been described previously, detailed kinematic descriptions of acute stage ataxia are rare and no attempt has been made to link improvements in motor function to measures of neural recovery and lesion location. This study provides a comprehensive assessment of how lesion site and arm dysfunction are associated in the acute stage and outlines the course of upper limb motor recovery for the first 4 months after the infarction. Methods-Sixteen adult patients with cerebellar stroke and 11 age-matched healthy controls participated. Kinematics of goal-directed and unconstrained finger-pointing movements were measured at the acute stage and in 2-week and 3-month follow-ups. MRI data were obtained for the acute and 3-month follow-up sessions. A voxel-based lesion map subtraction analysis was performed to examine the effect of ischemic lesion sites on kinematic performance. Results-In the acute stage, nearly 70% of patients exhibited motor slowing with hand velocity and acceleration maxima below the range of the control group. MRI analysis revealed that in patients with impaired motor performance, lesions were more common in paravermal lobules IVN and affected the deep cerebellar nuclei. Stroke affecting the superior cerebellar artery led to lower motor performance than infractions of the posterior cerebellar artery. By the 2-week-follow-up, hand kinematics had improved dramatically (gains in acceleration up to 86%). Improvements between the 2-week and the 3-month-follow-ups were less pronounced. Conclusion-In the acute stage, arm movements were mainly characterized by abnormal slowness (bradykinesia) and not dyscoordination (ataxia). The motor signs were associated with lesions in paravermal regions of lobules IVN and the deep cerebellar nuclei. Motor recovery was fast, with the majority of gains in upper limb function occurring in the first 2 weeks after the acute phase.</EA>
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<FD>Accident cérébrovasculaire; Ataxie; Pathologie du système nerveux; Pathologie cérébrovasculaire; Membre supérieur; Cervelet; Cartographie; Cinématique; Encéphale; Homme</FD>
<FG>Pathologie de l'appareil circulatoire; Pathologie de l'encéphale; Pathologie du   système nerveux central; Pathologie des vaisseaux sanguins; Trouble neurologique</FG>
<ED>Stroke; Ataxia; Nervous system diseases; Cerebrovascular disease; Upper limb; Cerebellum; Cartography; Kinematics; Encephalon; Human</ED>
<EG>Cardiovascular disease; Cerebral disorder; Central nervous system disease; Vascular disease; Neurological disorder</EG>
<SD>Accidente cerebrovascular; Ataxia; Sistema nervioso patología; Vaso sanguíneo encéfalo patología; Miembro superior; Cerebelo; Cartografía; Cinemática; Encéfalo; Hombre</SD>
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Recovery of Upper Limb Function After Cerebellar Stroke Lesion Symptom Mapping and Arm Kinematics

Recovery of Upper Limb Function After Cerebellar Stroke Lesion Symptom Mapping and Arm Kinematics

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