Gastrointestinal Tumours
Identifieur interne : 001630 ( Istex/Corpus ); précédent : 001629; suivant : 001631Gastrointestinal Tumours
Auteurs :Source :
- Annals of Oncology [ 0923-7534 ] ; 1998.
Url:
DOI: 10.1093/annonc/9.suppl_4.46
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ISTEX:DAF603482F5488941081E06A840AF363B9FC5944Le document en format XML
<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title>Gastrointestinal Tumours</title></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:DAF603482F5488941081E06A840AF363B9FC5944</idno><date when="1998" year="1998">1998</date><idno type="doi">10.1093/annonc/9.suppl_4.46</idno><idno type="url">https://api.istex.fr/document/DAF603482F5488941081E06A840AF363B9FC5944/fulltext/pdf</idno><idno type="wicri:Area/Istex/Corpus">001630</idno><idno type="wicri:explorRef" wicri:stream="Istex" wicri:step="Corpus" wicri:corpus="ISTEX">001630</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a">Gastrointestinal Tumours</title></analytic><monogr></monogr><series><title level="j">Annals of Oncology</title><idno type="ISSN">0923-7534</idno><idno type="eISSN">1569-8041</idno><imprint><publisher>Oxford University Press</publisher><date type="published" when="1998">1998</date><biblScope unit="volume">9</biblScope><biblScope unit="supplement">suppl_4</biblScope><biblScope unit="page" from="46">46</biblScope><biblScope unit="page" to="56">56</biblScope></imprint><idno type="ISSN">0923-7534</idno></series><idno type="istex">DAF603482F5488941081E06A840AF363B9FC5944</idno><idno type="DOI">10.1093/annonc/9.suppl_4.46</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0923-7534</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader></TEI><istex><corpusName>oup</corpusName><subject><json:item><value>Abstracts</value></json:item></subject><language><json:string>unknown</json:string></language><originalGenre><json:string>abstract</json:string></originalGenre><qualityIndicators><score>7.012</score><pdfVersion>1.5</pdfVersion><pdfPageSize>595.44 x 841.68 pts</pdfPageSize><refBibsNative>false</refBibsNative><keywordCount>1</keywordCount><abstractCharCount>0</abstractCharCount><pdfWordCount>15633</pdfWordCount><pdfCharCount>93310</pdfCharCount><pdfPageCount>11</pdfPageCount><abstractWordCount>1</abstractWordCount></qualityIndicators><title>Gastrointestinal Tumours</title><refBibs><json:item><author><json:item><name>Ico </name></json:item><json:item><name>H,C Valencia</name></json:item><json:item><name>S,H Servet</name></json:item><json:item><name>H,C Zaragoza ; 6</name></json:item><json:item><name> Lozano Blesa</name></json:item><json:item><name> Zaragoza</name></json:item><json:item><name>C Spain</name></json:item></author><host><volume>1</volume><pages><last>1</last><first>1</first></pages><issue>1</issue><author></author><title>M.A. Satolll</title></host></json:item><json:item><author><json:item><name> Serv</name></json:item><json:item><name> Rt-Az</name></json:item><json:item><name>O I R M S Osp</name></json:item><json:item><name> Anna</name></json:item><json:item><name>Italy,J To</name></json:item><json:item><name>R Salvador</name></json:item><json:item><name>R Bemabe</name></json:item><json:item><name>E Lasso</name></json:item><json:item><name>J,J Garcla-Fdez</name></json:item><json:item><name> Reina</name></json:item><json:item><name> Secctdn Oncologla</name></json:item><json:item><name>Hospital M6dlca</name></json:item><json:item><name>R Juan</name></json:item><json:item><name> Jimenez</name></json:item><json:item><name> Huelva</name></json:item></author><host><volume>50504331</volume><pages><last>95</last><first>22</first></pages><issue>95</issue><author></author><title>9% Cl Cl</title></host><title>Spain Introduction: The treatment of gastrointestinal tract cancer is expanding with the development of new, more active, 5FU based regimens and the discovery of new cytotoxlc agents with activity In these diseases The direct thymydilate synthase(TS) inhibitors have been developed in order to reduce toxicity compared with indirect TS inhibitors, such as 5FU or tegafur. Purpose: To investigate the toxicity of three thymidylate synthase inhibitor regimens. Patients and methods: 30 patients(pts) have been treated with: F.A: 20 mg/m2 followed by 5-FU-425 mg/m2 I.V on days 1-5 q 3 weeks (10 pts: 50 cycles was administered), Raltitrexed 3 mg/m2 I.V. for 1 day each 21 days (9 pts: 43 cycles) and tegafur 1200 mg/day oral on days 1-21 q. 4 weeks. (11 pts: 31 cycles) 124 treatment cycles were evaluated for WHO toxicity cnteria Results: Toxicity was as follows: Arm F.A. plus 5-FU: Leucopenia 11 grade IV, 1 grade III, 3 grade II y 6 grade.l). Mucositis and nauseas 10/50: 20%, Cl 95%'10%-33%. Vomiting and liver toxicity ocurred in 8 Arm Raltltrexed: The main liver toxicity was rising In serum hepatic transminase levels grade l-lll (12 Non-haematological toxicity grade III-IV ocurred. Arm Tegafur The main toxicity was vomltlng:5</title></json:item><json:item><author><json:item><name>J,T Hartmann</name></json:item><json:item><name>J,P Nichols</name></json:item><json:item><name>A Droz</name></json:item><json:item><name> Horwlch</name></json:item><json:item><name>S,D Geri</name></json:item><json:item><name>J Fossa</name></json:item><json:item><name>J Beyer</name></json:item><json:item><name>H,J Pont</name></json:item><json:item><name>C Schmdl</name></json:item></author><host><pages><last>2</last><first>17</first></pages><author></author><title>Indianapolis-USA</title></host><title>Germany Introduction: (1) To evaluate the frequency and types of hematologlc disorders In EGGCT, and (2) to study the clinical behavior and outcome of those patients (pts) Method: The clinical courses of 637 pts with EGGCT, subsequently treated during clsplabn (P)-area in 12 European and American cancer centers, were 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