Cardiorespiratory responses of white sturgeon to environmental hypercapnia
Identifieur interne : 000356 ( PascalFrancis/Corpus ); précédent : 000355; suivant : 000357Cardiorespiratory responses of white sturgeon to environmental hypercapnia
Auteurs : C. E. Crocker ; A. P. Farrell ; A. K. Gamperl ; J. J. Jr CechSource :
- American journal of physiology. Regulatory, integrative and comparative physiology [ 0363-6119 ] ; 2000.
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- Pascal (Inist)
English descriptors
- KwdEn :
Abstract
Cardioventilatory variables and blood-gas, acid-base status were measured in cannulated white sturgeon (Acipenser transmontanus) maintained at 19°C during normocapnic and hypercapnic (PwCO2 ∼20 Torr) water conditions and after the injection of adrenergic analogs. Hypercapnia produced significant increases in arterial PCO2, ventilatory frequency, and plasma concentration of cortisol and epinephrine, and it produced significant decreases in arterial pH and plasma concentration of glucose but no change in arterial PO2, hematocrit, and concentration of lactate or norepinephrine. Hypercapnia significantly increased cardiac output (Q) by 22%, mean arterial pressure (MAP) by 8%, and heart rate (HR) by 8%. However, gut blood flow (GBF) remained constant. In normocapnic fish, phenylephrine significantly constricted the splanchnic circulation, whereas isoproterenol significantly increased Q and produced a systemic vasodilation. During hypercapnia, propranolol significantly decreased Q, GBF, MAP, and HR, whereas phentolamine significantly decreased MAP and increased GBF. These changes suggest that cardiovascular function in the white sturgeon is sensitive to both α- and (3-adrenergic modulation. We found microspheres to be unreliable in predicting GBF on the basis of our comparisons with simultaneous direct measurements of GBF. Overall, our results demonstrate that environmental hypercapnia (e.g., as is experienced in high-intensity culture situations) elicits stress responses in white sturgeon that significantly elevate steady-state cardiovascular and ventilatory activity levels.
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Pour connaître la documentation sur le format Inist Standard.
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Format Inist (serveur)
| NO : | PASCAL 00-0512579 INIST |
|---|---|
| ET : | Cardiorespiratory responses of white sturgeon to environmental hypercapnia |
| AU : | CROCKER (C. E.); FARRELL (A. P.); GAMPERL (A. K.); CECH (J. J. JR) |
| AF : | Department of Wildlife, Fish, and Conservation Biology, University of California/Davis, California 95616-8751/Etats-Unis (1 aut., 4 aut.); Department of Biological Sciences, Simon Fraser University/Burnaby, British Columbia V5A 1S6/Canada (2 aut., 3 aut.) |
| DT : | Publication en série; Niveau analytique |
| SO : | American journal of physiology. Regulatory, integrative and comparative physiology; ISSN 0363-6119; Coden AJPRDO; Etats-Unis; Da. 2000; Vol. 48; No. 2; R617-R628; Bibl. 42 ref. |
| LA : | Anglais |
| EA : | Cardioventilatory variables and blood-gas, acid-base status were measured in cannulated white sturgeon (Acipenser transmontanus) maintained at 19°C during normocapnic and hypercapnic (PwCO2 ∼20 Torr) water conditions and after the injection of adrenergic analogs. Hypercapnia produced significant increases in arterial PCO2, ventilatory frequency, and plasma concentration of cortisol and epinephrine, and it produced significant decreases in arterial pH and plasma concentration of glucose but no change in arterial PO2, hematocrit, and concentration of lactate or norepinephrine. Hypercapnia significantly increased cardiac output (Q) by 22%, mean arterial pressure (MAP) by 8%, and heart rate (HR) by 8%. However, gut blood flow (GBF) remained constant. In normocapnic fish, phenylephrine significantly constricted the splanchnic circulation, whereas isoproterenol significantly increased Q and produced a systemic vasodilation. During hypercapnia, propranolol significantly decreased Q, GBF, MAP, and HR, whereas phentolamine significantly decreased MAP and increased GBF. These changes suggest that cardiovascular function in the white sturgeon is sensitive to both α- and (3-adrenergic modulation. We found microspheres to be unreliable in predicting GBF on the basis of our comparisons with simultaneous direct measurements of GBF. Overall, our results demonstrate that environmental hypercapnia (e.g., as is experienced in high-intensity culture situations) elicits stress responses in white sturgeon that significantly elevate steady-state cardiovascular and ventilatory activity levels. |
| CC : | 002A25E |
| FD : | Rythme cardiaque; Gaz sanguin; Pression artérielle; Equilibre acidobasique; Carbone dioxyde; Hypercapnie; Noradrénaline; Récepteur adrénergique; Contrôle cardiorespiratoire; Contrôle cardiovasculaire; Respiration; Acipenser transmontanus |
| FG : | Hémodynamique; Pisces; Vertebrata |
| ED : | Heart rate; Blood gas; Arterial pressure; Acid base balance; Carbon dioxide; Hypercapnia; Norepinephrine; Adrenergic receptor; Cardiorespiratory control; Cardiovascular control; Respiration; Acipenser transmontanus |
| EG : | Hemodynamics; Pisces; Vertebrata |
| SD : | Ritmo cardíaco; Gas sanguíneo; Presión arterial; Equilibrio acido-base; Carbono dióxido; Hipercapnia; Noradrenalina; Receptor adrenérgico; Control cardiorespiratorio; Control cardiovascular; Respiración; Acipenser transmontanus |
| LO : | INIST-670E.354000091005050330 |
| ID : | 00-0512579 |
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Pascal:00-0512579Le document en format XML
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Gamperl</name><affiliation><inist:fA14 i1="02"><s1>Department of Biological Sciences, Simon Fraser University</s1><s2>Burnaby, British Columbia V5A 1S6</s2><s3>CAN</s3><sZ>2 aut.</sZ><sZ>3 aut.</sZ></inist:fA14></affiliation></author><author><name sortKey="Cech, J J Jr" sort="Cech, J J Jr" uniqKey="Cech J" first="J. J. Jr" last="Cech">J. J. Jr Cech</name><affiliation><inist:fA14 i1="01"><s1>Department of Wildlife, Fish, and Conservation Biology, University of California</s1><s2>Davis, California 95616-8751</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>4 aut.</sZ></inist:fA14></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">INIST</idno><idno type="inist">00-0512579</idno><date when="2000">2000</date><idno type="stanalyst">PASCAL 00-0512579 INIST</idno><idno type="RBID">Pascal:00-0512579</idno><idno type="wicri:Area/PascalFrancis/Corpus">000356</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en" level="a">Cardiorespiratory responses of white sturgeon to environmental hypercapnia</title><author><name sortKey="Crocker, C E" sort="Crocker, C E" uniqKey="Crocker C" first="C. E." last="Crocker">C. E. Crocker</name><affiliation><inist:fA14 i1="01"><s1>Department of Wildlife, Fish, and Conservation Biology, University of California</s1><s2>Davis, California 95616-8751</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>4 aut.</sZ></inist:fA14></affiliation></author><author><name sortKey="Farrell, A P" sort="Farrell, A P" uniqKey="Farrell A" first="A. P." last="Farrell">A. P. Farrell</name><affiliation><inist:fA14 i1="02"><s1>Department of Biological Sciences, Simon Fraser University</s1><s2>Burnaby, British Columbia V5A 1S6</s2><s3>CAN</s3><sZ>2 aut.</sZ><sZ>3 aut.</sZ></inist:fA14></affiliation></author><author><name sortKey="Gamperl, A K" sort="Gamperl, A K" uniqKey="Gamperl A" first="A. K." last="Gamperl">A. K. Gamperl</name><affiliation><inist:fA14 i1="02"><s1>Department of Biological Sciences, Simon Fraser University</s1><s2>Burnaby, British Columbia V5A 1S6</s2><s3>CAN</s3><sZ>2 aut.</sZ><sZ>3 aut.</sZ></inist:fA14></affiliation></author><author><name sortKey="Cech, J J Jr" sort="Cech, J J Jr" uniqKey="Cech J" first="J. J. Jr" last="Cech">J. J. Jr Cech</name><affiliation><inist:fA14 i1="01"><s1>Department of Wildlife, Fish, and Conservation Biology, University of California</s1><s2>Davis, California 95616-8751</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>4 aut.</sZ></inist:fA14></affiliation></author></analytic><series><title level="j" type="main">American journal of physiology. Regulatory, integrative and comparative physiology</title><title level="j" type="abbreviated">Am. j. physiol., Regul. integr. comp. physiol.</title><idno type="ISSN">0363-6119</idno><imprint><date when="2000">2000</date></imprint></series></biblStruct></sourceDesc><seriesStmt><title level="j" type="main">American journal of physiology. Regulatory, integrative and comparative physiology</title><title level="j" type="abbreviated">Am. j. physiol., Regul. integr. comp. physiol.</title><idno type="ISSN">0363-6119</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Acid base balance</term><term>Acipenser transmontanus</term><term>Adrenergic receptor</term><term>Arterial pressure</term><term>Blood gas</term><term>Carbon dioxide</term><term>Cardiorespiratory control</term><term>Cardiovascular control</term><term>Heart rate</term><term>Hypercapnia</term><term>Norepinephrine</term><term>Respiration</term></keywords><keywords scheme="Pascal" xml:lang="fr"><term>Rythme cardiaque</term><term>Gaz sanguin</term><term>Pression artérielle</term><term>Equilibre acidobasique</term><term>Carbone dioxyde</term><term>Hypercapnie</term><term>Noradrénaline</term><term>Récepteur adrénergique</term><term>Contrôle cardiorespiratoire</term><term>Contrôle cardiovasculaire</term><term>Respiration</term><term>Acipenser transmontanus</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Cardioventilatory variables and blood-gas, acid-base status were measured in cannulated white sturgeon (Acipenser transmontanus) maintained at 19°C during normocapnic and hypercapnic (Pw<sub>CO2</sub> ∼20 Torr) water conditions and after the injection of adrenergic analogs. Hypercapnia produced significant increases in arterial PCO<sub>2</sub>, ventilatory frequency, and plasma concentration of cortisol and epinephrine, and it produced significant decreases in arterial pH and plasma concentration of glucose but no change in arterial PO<sub>2</sub>, hematocrit, and concentration of lactate or norepinephrine. Hypercapnia significantly increased cardiac output (Q) by 22%, mean arterial pressure (MAP) by 8%, and heart rate (HR) by 8%. However, gut blood flow (GBF) remained constant. In normocapnic fish, phenylephrine significantly constricted the splanchnic circulation, whereas isoproterenol significantly increased Q and produced a systemic vasodilation. During hypercapnia, propranolol significantly decreased Q, GBF, MAP, and HR, whereas phentolamine significantly decreased MAP and increased GBF. These changes suggest that cardiovascular function in the white sturgeon is sensitive to both α- and (3-adrenergic modulation. We found microspheres to be unreliable in predicting GBF on the basis of our comparisons with simultaneous direct measurements of GBF. Overall, our results demonstrate that environmental hypercapnia (e.g., as is experienced in high-intensity culture situations) elicits stress responses in white sturgeon that significantly elevate steady-state cardiovascular and ventilatory activity levels.</div></front></TEI><inist><standard h6="B"><pA><fA01 i1="01" i2="1"><s0>0363-6119</s0></fA01><fA02 i1="01"><s0>AJPRDO</s0></fA02><fA03 i2="1"><s0>Am. j. physiol., Regul. integr. comp. physiol.</s0></fA03><fA05><s2>48</s2></fA05><fA06><s2>2</s2></fA06><fA08 i1="01" i2="1" l="ENG"><s1>Cardiorespiratory responses of white sturgeon to environmental hypercapnia</s1></fA08><fA11 i1="01" i2="1"><s1>CROCKER (C. E.)</s1></fA11><fA11 i1="02" i2="1"><s1>FARRELL (A. P.)</s1></fA11><fA11 i1="03" i2="1"><s1>GAMPERL (A. K.)</s1></fA11><fA11 i1="04" i2="1"><s1>CECH (J. J. JR)</s1></fA11><fA14 i1="01"><s1>Department of Wildlife, Fish, and Conservation Biology, University of California</s1><s2>Davis, California 95616-8751</s2><s3>USA</s3><sZ>1 aut.</sZ><sZ>4 aut.</sZ></fA14><fA14 i1="02"><s1>Department of Biological Sciences, Simon Fraser University</s1><s2>Burnaby, British Columbia V5A 1S6</s2><s3>CAN</s3><sZ>2 aut.</sZ><sZ>3 aut.</sZ></fA14><fA20><s2>R617-R628</s2></fA20><fA21><s1>2000</s1></fA21><fA23 i1="01"><s0>ENG</s0></fA23><fA43 i1="01"><s1>INIST</s1><s2>670E</s2><s5>354000091005050330</s5></fA43><fA44><s0>0000</s0><s1>© 2000 INIST-CNRS. All rights reserved.</s1></fA44><fA45><s0>42 ref.</s0></fA45><fA47 i1="01" i2="1"><s0>00-0512579</s0></fA47><fA60><s1>P</s1></fA60><fA61><s0>A</s0></fA61><fA64 i1="01" i2="1"><s0>American journal of physiology. Regulatory, integrative and comparative physiology</s0></fA64><fA66 i1="01"><s0>USA</s0></fA66><fC01 i1="01" l="ENG"><s0>Cardioventilatory variables and blood-gas, acid-base status were measured in cannulated white sturgeon (Acipenser transmontanus) maintained at 19°C during normocapnic and hypercapnic (Pw<sub>CO2</sub> ∼20 Torr) water conditions and after the injection of adrenergic analogs. Hypercapnia produced significant increases in arterial PCO<sub>2</sub>, ventilatory frequency, and plasma concentration of cortisol and epinephrine, and it produced significant decreases in arterial pH and plasma concentration of glucose but no change in arterial PO<sub>2</sub>, hematocrit, and concentration of lactate or norepinephrine. Hypercapnia significantly increased cardiac output (Q) by 22%, mean arterial pressure (MAP) by 8%, and heart rate (HR) by 8%. However, gut blood flow (GBF) remained constant. In normocapnic fish, phenylephrine significantly constricted the splanchnic circulation, whereas isoproterenol significantly increased Q and produced a systemic vasodilation. During hypercapnia, propranolol significantly decreased Q, GBF, MAP, and HR, whereas phentolamine significantly decreased MAP and increased GBF. These changes suggest that cardiovascular function in the white sturgeon is sensitive to both α- and (3-adrenergic modulation. We found microspheres to be unreliable in predicting GBF on the basis of our comparisons with simultaneous direct measurements of GBF. 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E.); FARRELL (A. P.); GAMPERL (A. K.); CECH (J. J. JR)</AU><AF>Department of Wildlife, Fish, and Conservation Biology, University of California/Davis, California 95616-8751/Etats-Unis (1 aut., 4 aut.); Department of Biological Sciences, Simon Fraser University/Burnaby, British Columbia V5A 1S6/Canada (2 aut., 3 aut.)</AF><DT>Publication en série; Niveau analytique</DT><SO>American journal of physiology. Regulatory, integrative and comparative physiology; ISSN 0363-6119; Coden AJPRDO; Etats-Unis; Da. 2000; Vol. 48; No. 2; R617-R628; Bibl. 42 ref.</SO><LA>Anglais</LA><EA>Cardioventilatory variables and blood-gas, acid-base status were measured in cannulated white sturgeon (Acipenser transmontanus) maintained at 19°C during normocapnic and hypercapnic (Pw<sub>CO2</sub> ∼20 Torr) water conditions and after the injection of adrenergic analogs. Hypercapnia produced significant increases in arterial PCO<sub>2</sub>, ventilatory frequency, and plasma concentration of cortisol and epinephrine, and it produced significant decreases in arterial pH and plasma concentration of glucose but no change in arterial PO<sub>2</sub>, hematocrit, and concentration of lactate or norepinephrine. Hypercapnia significantly increased cardiac output (Q) by 22%, mean arterial pressure (MAP) by 8%, and heart rate (HR) by 8%. However, gut blood flow (GBF) remained constant. In normocapnic fish, phenylephrine significantly constricted the splanchnic circulation, whereas isoproterenol significantly increased Q and produced a systemic vasodilation. During hypercapnia, propranolol significantly decreased Q, GBF, MAP, and HR, whereas phentolamine significantly decreased MAP and increased GBF. These changes suggest that cardiovascular function in the white sturgeon is sensitive to both α- and (3-adrenergic modulation. We found microspheres to be unreliable in predicting GBF on the basis of our comparisons with simultaneous direct measurements of GBF. Overall, our results demonstrate that environmental hypercapnia (e.g., as is experienced in high-intensity culture situations) elicits stress responses in white sturgeon that significantly elevate steady-state cardiovascular and ventilatory activity levels.</EA><CC>002A25E</CC><FD>Rythme cardiaque; Gaz sanguin; Pression artérielle; Equilibre acidobasique; Carbone dioxyde; Hypercapnie; Noradrénaline; Récepteur adrénergique; Contrôle cardiorespiratoire; Contrôle cardiovasculaire; Respiration; Acipenser transmontanus</FD><FG>Hémodynamique; Pisces; Vertebrata</FG><ED>Heart rate; Blood gas; Arterial pressure; Acid base balance; Carbon dioxide; Hypercapnia; Norepinephrine; Adrenergic receptor; Cardiorespiratory control; Cardiovascular control; Respiration; Acipenser transmontanus</ED><EG>Hemodynamics; Pisces; Vertebrata</EG><SD>Ritmo cardíaco; Gas sanguíneo; Presión arterial; Equilibrio acido-base; Carbono dióxido; Hipercapnia; Noradrenalina; Receptor adrenérgico; Control cardiorespiratorio; Control cardiovascular; Respiración; Acipenser transmontanus</SD><LO>INIST-670E.354000091005050330</LO><ID>00-0512579</ID></server></inist></record>Pour manipuler ce document sous Unix (Dilib)
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