The effect of luteolin-7-O-beta-D-glucuronopyranoside on gastritis and esophagitis in rats.
Identifieur interne : 001B43 ( Main/Curation ); précédent : 001B42; suivant : 001B44The effect of luteolin-7-O-beta-D-glucuronopyranoside on gastritis and esophagitis in rats.
Auteurs : Young Sil Min [Corée du Sud] ; Ki Lyong Bai ; Sung Hyuk Yim ; Young Joo Lee ; Hyun Ju Song ; Jin Hak Kim ; Inhye Ham ; Wan Kyun Whang ; Uy Dong SohnSource :
- Archives of pharmacal research [ 0253-6269 ] ; 2006.
Descripteurs français
- KwdFr :
- Acide gastrique (métabolisme), Animaux (MeSH), Antioxydants (isolement et purification), Antioxydants (pharmacologie), Feuilles de plante (composition chimique), Gastrite (induit chimiquement), Gastrite (métabolisme), Gastrite (prévention et contrôle), Glucosides (isolement et purification), Glucosides (pharmacologie), Indométacine (MeSH), Lutéoline (isolement et purification), Lutéoline (pharmacologie), Malonaldéhyde (métabolisme), Mesure de l'acidité gastrique (MeSH), Mâle (MeSH), Oesophagite peptique (métabolisme), Oesophagite peptique (prévention et contrôle), Oesophagite peptique (étiologie), Oméprazole (pharmacologie), Peroxydation lipidique (MeSH), Pylore (chirurgie), Rat Sprague-Dawley (MeSH), Rats (MeSH), Relation dose-effet des médicaments (MeSH), Salix (composition chimique).
- MESH :
- chirurgie : Pylore.
- composition chimique : Feuilles de plante, Salix.
- induit chimiquement : Gastrite.
- isolement et purification : Antioxydants, Glucosides, Lutéoline.
- métabolisme : Acide gastrique, Gastrite, Malonaldéhyde, Oesophagite peptique.
- pharmacologie : Antioxydants, Glucosides, Lutéoline, Oméprazole.
- prévention et contrôle : Gastrite, Oesophagite peptique.
- étiologie : Oesophagite peptique.
- Animaux, Indométacine, Mesure de l'acidité gastrique, Mâle, Peroxydation lipidique, Rat Sprague-Dawley, Rats, Relation dose-effet des médicaments.
English descriptors
- KwdEn :
- Animals (MeSH), Antioxidants (isolation & purification), Antioxidants (pharmacology), Dose-Response Relationship, Drug (MeSH), Esophagitis, Peptic (etiology), Esophagitis, Peptic (metabolism), Esophagitis, Peptic (prevention & control), Gastric Acid (metabolism), Gastric Acidity Determination (MeSH), Gastritis (chemically induced), Gastritis (metabolism), Gastritis (prevention & control), Glucosides (isolation & purification), Glucosides (pharmacology), Indomethacin (MeSH), Lipid Peroxidation (MeSH), Luteolin (isolation & purification), Luteolin (pharmacology), Male (MeSH), Malondialdehyde (metabolism), Omeprazole (pharmacology), Plant Leaves (chemistry), Pylorus (surgery), Rats (MeSH), Rats, Sprague-Dawley (MeSH), Salix (chemistry).
- MESH :
- chemical , isolation & purification : Antioxidants, Glucosides, Luteolin.
- chemical , metabolism : Malondialdehyde.
- chemical , pharmacology : Antioxidants, Glucosides, Luteolin, Omeprazole.
- chemically induced : Gastritis.
- chemistry : Plant Leaves, Salix.
- etiology : Esophagitis, Peptic.
- metabolism : Esophagitis, Peptic, Gastric Acid, Gastritis.
- prevention & control : Esophagitis, Peptic, Gastritis.
- surgery : Pylorus.
- Animals, Dose-Response Relationship, Drug, Gastric Acidity Determination, Indomethacin, Lipid Peroxidation, Male, Rats, Rats, Sprague-Dawley.
Abstract
This study evaluated the inhibitory action of luteolin-7-O-beta-D-glucuronopyranoside, luteolin which was isolated from Salix gilgiana leaves, and omeprazole on reflux esophagitis and gastritis in rats. Reflux esophagitis and gastritis were induced surgically and by the administration of indomethacin, respectively. The intraduodenal administration of luteolin-7-O-beta-D-glucuronopyranoside decreased the ulcer index, injury area, gastric volume and acid output, and increased the gastric pH compared with luteolin. Luteolin-7-O-beta-D-glucuronopyranoside significantly decreased the size of the gastric lesions that had been induced by exposing the gastric mucosa to indomethacin. The malondialdehyde content, which is the end product of lipid peroxidation, was increased significantly after inducing of reflux esophagitis. The malondialdehyde content was decreased by Luteolin-7-O-beta-D-glucuronopyranoside but not luteolin or omeprazole. Luteolin-7-O-beta-D-glucuronopyranoside has a more potent antioxidative effect than luteolin. Luteolin-7-O-beta-D-glucuronopyranoside is a promising drug for the treatment of reflux esophagitis and gastritis.
DOI: 10.1007/BF02969421
PubMed: 16833016
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Sung Hyuk" uniqKey="Yim S" first="Sung Hyuk" last="Yim">Sung Hyuk Yim</name></author><author><name sortKey="Lee, Young Joo" sort="Lee, Young Joo" uniqKey="Lee Y" first="Young Joo" last="Lee">Young Joo Lee</name></author><author><name sortKey="Song, Hyun Ju" sort="Song, Hyun Ju" uniqKey="Song H" first="Hyun Ju" last="Song">Hyun Ju Song</name></author><author><name sortKey="Kim, Jin Hak" sort="Kim, Jin Hak" uniqKey="Kim J" first="Jin Hak" last="Kim">Jin Hak Kim</name></author><author><name sortKey="Ham, Inhye" sort="Ham, Inhye" uniqKey="Ham I" first="Inhye" last="Ham">Inhye Ham</name></author><author><name sortKey="Whang, Wan Kyun" sort="Whang, Wan Kyun" uniqKey="Whang W" first="Wan Kyun" last="Whang">Wan Kyun Whang</name></author><author><name sortKey="Sohn, Uy Dong" sort="Sohn, Uy Dong" uniqKey="Sohn U" first="Uy Dong" last="Sohn">Uy Dong Sohn</name></author></analytic><series><title level="j">Archives of pharmacal research</title><idno type="ISSN">0253-6269</idno><imprint><date when="2006" type="published">2006</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Animals (MeSH)</term><term>Antioxidants (isolation & purification)</term><term>Antioxidants (pharmacology)</term><term>Dose-Response Relationship, Drug (MeSH)</term><term>Esophagitis, Peptic (etiology)</term><term>Esophagitis, Peptic (metabolism)</term><term>Esophagitis, Peptic (prevention & control)</term><term>Gastric Acid (metabolism)</term><term>Gastric Acidity Determination (MeSH)</term><term>Gastritis (chemically induced)</term><term>Gastritis (metabolism)</term><term>Gastritis (prevention & control)</term><term>Glucosides (isolation & purification)</term><term>Glucosides (pharmacology)</term><term>Indomethacin (MeSH)</term><term>Lipid Peroxidation (MeSH)</term><term>Luteolin (isolation & purification)</term><term>Luteolin (pharmacology)</term><term>Male (MeSH)</term><term>Malondialdehyde (metabolism)</term><term>Omeprazole (pharmacology)</term><term>Plant Leaves (chemistry)</term><term>Pylorus (surgery)</term><term>Rats (MeSH)</term><term>Rats, Sprague-Dawley (MeSH)</term><term>Salix (chemistry)</term></keywords><keywords scheme="KwdFr" xml:lang="fr"><term>Acide gastrique (métabolisme)</term><term>Animaux (MeSH)</term><term>Antioxydants (isolement et purification)</term><term>Antioxydants (pharmacologie)</term><term>Feuilles de plante (composition chimique)</term><term>Gastrite (induit chimiquement)</term><term>Gastrite (métabolisme)</term><term>Gastrite (prévention et contrôle)</term><term>Glucosides (isolement et purification)</term><term>Glucosides (pharmacologie)</term><term>Indométacine (MeSH)</term><term>Lutéoline (isolement et purification)</term><term>Lutéoline (pharmacologie)</term><term>Malonaldéhyde (métabolisme)</term><term>Mesure de l'acidité gastrique (MeSH)</term><term>Mâle (MeSH)</term><term>Oesophagite peptique (métabolisme)</term><term>Oesophagite peptique (prévention et contrôle)</term><term>Oesophagite peptique (étiologie)</term><term>Oméprazole (pharmacologie)</term><term>Peroxydation lipidique (MeSH)</term><term>Pylore (chirurgie)</term><term>Rat Sprague-Dawley (MeSH)</term><term>Rats (MeSH)</term><term>Relation dose-effet des médicaments (MeSH)</term><term>Salix (composition chimique)</term></keywords><keywords scheme="MESH" type="chemical" qualifier="isolation & purification" xml:lang="en"><term>Antioxidants</term><term>Glucosides</term><term>Luteolin</term></keywords><keywords scheme="MESH" type="chemical" qualifier="metabolism" xml:lang="en"><term>Malondialdehyde</term></keywords><keywords scheme="MESH" type="chemical" qualifier="pharmacology" xml:lang="en"><term>Antioxidants</term><term>Glucosides</term><term>Luteolin</term><term>Omeprazole</term></keywords><keywords scheme="MESH" qualifier="chemically induced" xml:lang="en"><term>Gastritis</term></keywords><keywords scheme="MESH" qualifier="chemistry" xml:lang="en"><term>Plant Leaves</term><term>Salix</term></keywords><keywords scheme="MESH" qualifier="chirurgie" xml:lang="fr"><term>Pylore</term></keywords><keywords scheme="MESH" qualifier="composition chimique" xml:lang="fr"><term>Feuilles de plante</term><term>Salix</term></keywords><keywords scheme="MESH" qualifier="etiology" xml:lang="en"><term>Esophagitis, Peptic</term></keywords><keywords scheme="MESH" qualifier="induit chimiquement" xml:lang="fr"><term>Gastrite</term></keywords><keywords scheme="MESH" qualifier="isolement et purification" xml:lang="fr"><term>Antioxydants</term><term>Glucosides</term><term>Lutéoline</term></keywords><keywords scheme="MESH" qualifier="metabolism" xml:lang="en"><term>Esophagitis, Peptic</term><term>Gastric Acid</term><term>Gastritis</term></keywords><keywords scheme="MESH" qualifier="métabolisme" xml:lang="fr"><term>Acide gastrique</term><term>Gastrite</term><term>Malonaldéhyde</term><term>Oesophagite peptique</term></keywords><keywords scheme="MESH" qualifier="pharmacologie" xml:lang="fr"><term>Antioxydants</term><term>Glucosides</term><term>Lutéoline</term><term>Oméprazole</term></keywords><keywords scheme="MESH" qualifier="prevention & control" xml:lang="en"><term>Esophagitis, Peptic</term><term>Gastritis</term></keywords><keywords scheme="MESH" qualifier="prévention et contrôle" xml:lang="fr"><term>Gastrite</term><term>Oesophagite peptique</term></keywords><keywords scheme="MESH" qualifier="surgery" xml:lang="en"><term>Pylorus</term></keywords><keywords scheme="MESH" qualifier="étiologie" xml:lang="fr"><term>Oesophagite peptique</term></keywords><keywords scheme="MESH" xml:lang="en"><term>Animals</term><term>Dose-Response Relationship, Drug</term><term>Gastric Acidity Determination</term><term>Indomethacin</term><term>Lipid Peroxidation</term><term>Male</term><term>Rats</term><term>Rats, Sprague-Dawley</term></keywords><keywords scheme="MESH" xml:lang="fr"><term>Animaux</term><term>Indométacine</term><term>Mesure de l'acidité gastrique</term><term>Mâle</term><term>Peroxydation lipidique</term><term>Rat Sprague-Dawley</term><term>Rats</term><term>Relation dose-effet des médicaments</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">This study evaluated the inhibitory action of luteolin-7-O-beta-D-glucuronopyranoside, luteolin which was isolated from Salix gilgiana leaves, and omeprazole on reflux esophagitis and gastritis in rats. Reflux esophagitis and gastritis were induced surgically and by the administration of indomethacin, respectively. The intraduodenal administration of luteolin-7-O-beta-D-glucuronopyranoside decreased the ulcer index, injury area, gastric volume and acid output, and increased the gastric pH compared with luteolin. Luteolin-7-O-beta-D-glucuronopyranoside significantly decreased the size of the gastric lesions that had been induced by exposing the gastric mucosa to indomethacin. The malondialdehyde content, which is the end product of lipid peroxidation, was increased significantly after inducing of reflux esophagitis. The malondialdehyde content was decreased by Luteolin-7-O-beta-D-glucuronopyranoside but not luteolin or omeprazole. Luteolin-7-O-beta-D-glucuronopyranoside has a more potent antioxidative effect than luteolin. Luteolin-7-O-beta-D-glucuronopyranoside is a promising drug for the treatment of reflux esophagitis and gastritis.</div></front></TEI><pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">16833016</PMID><DateCompleted><Year>2006</Year><Month>09</Month><Day>25</Day></DateCompleted><DateRevised><Year>2019</Year><Month>09</Month><Day>17</Day></DateRevised><Article PubModel="Print"><Journal><ISSN IssnType="Print">0253-6269</ISSN><JournalIssue CitedMedium="Print"><Volume>29</Volume><Issue>6</Issue><PubDate><Year>2006</Year><Month>Jun</Month></PubDate></JournalIssue><Title>Archives of pharmacal research</Title><ISOAbbreviation>Arch Pharm Res</ISOAbbreviation></Journal><ArticleTitle>The effect of luteolin-7-O-beta-D-glucuronopyranoside on gastritis and esophagitis in rats.</ArticleTitle><Pagination><MedlinePgn>484-9</MedlinePgn></Pagination><Abstract><AbstractText>This study evaluated the inhibitory action of luteolin-7-O-beta-D-glucuronopyranoside, luteolin which was isolated from Salix gilgiana leaves, and omeprazole on reflux esophagitis and gastritis in rats. Reflux esophagitis and gastritis were induced surgically and by the administration of indomethacin, respectively. The intraduodenal administration of luteolin-7-O-beta-D-glucuronopyranoside decreased the ulcer index, injury area, gastric volume and acid output, and increased the gastric pH compared with luteolin. Luteolin-7-O-beta-D-glucuronopyranoside significantly decreased the size of the gastric lesions that had been induced by exposing the gastric mucosa to indomethacin. The malondialdehyde content, which is the end product of lipid peroxidation, was increased significantly after inducing of reflux esophagitis. The malondialdehyde content was decreased by Luteolin-7-O-beta-D-glucuronopyranoside but not luteolin or omeprazole. Luteolin-7-O-beta-D-glucuronopyranoside has a more potent antioxidative effect than luteolin. Luteolin-7-O-beta-D-glucuronopyranoside is a promising drug for the treatment of reflux esophagitis and gastritis.</AbstractText></Abstract><AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>Min</LastName><ForeName>Young Sil</ForeName><Initials>YS</Initials><AffiliationInfo><Affiliation>Department of Pharmacology, College of Pharmacy, Chung Ang University, Seoul, Korea.</Affiliation></AffiliationInfo></Author><Author ValidYN="Y"><LastName>Bai</LastName><ForeName>Ki Lyong</ForeName><Initials>KL</Initials></Author><Author ValidYN="Y"><LastName>Yim</LastName><ForeName>Sung Hyuk</ForeName><Initials>SH</Initials></Author><Author ValidYN="Y"><LastName>Lee</LastName><ForeName>Young Joo</ForeName><Initials>YJ</Initials></Author><Author ValidYN="Y"><LastName>Song</LastName><ForeName>Hyun Ju</ForeName><Initials>HJ</Initials></Author><Author ValidYN="Y"><LastName>Kim</LastName><ForeName>Jin Hak</ForeName><Initials>JH</Initials></Author><Author ValidYN="Y"><LastName>Ham</LastName><ForeName>Inhye</ForeName><Initials>I</Initials></Author><Author ValidYN="Y"><LastName>Whang</LastName><ForeName>Wan Kyun</ForeName><Initials>WK</Initials></Author><Author ValidYN="Y"><LastName>Sohn</LastName><ForeName>Uy Dong</ForeName><Initials>UD</Initials></Author></AuthorList><Language>eng</Language><PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType><PublicationType UI="D013485">Research Support, Non-U.S. Gov't</PublicationType></PublicationTypeList></Article><MedlineJournalInfo><Country>Korea (South)</Country><MedlineTA>Arch Pharm Res</MedlineTA><NlmUniqueID>8000036</NlmUniqueID><ISSNLinking>0253-6269</ISSNLinking></MedlineJournalInfo><ChemicalList><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D000975">Antioxidants</NameOfSubstance></Chemical><Chemical><RegistryNumber>0</RegistryNumber><NameOfSubstance UI="D005960">Glucosides</NameOfSubstance></Chemical><Chemical><RegistryNumber>4Y8F71G49Q</RegistryNumber><NameOfSubstance UI="D008315">Malondialdehyde</NameOfSubstance></Chemical><Chemical><RegistryNumber>98J6XDS46I</RegistryNumber><NameOfSubstance UI="C066408">luteolin-7-glucoside</NameOfSubstance></Chemical><Chemical><RegistryNumber>KG60484QX9</RegistryNumber><NameOfSubstance UI="D009853">Omeprazole</NameOfSubstance></Chemical><Chemical><RegistryNumber>KUX1ZNC9J2</RegistryNumber><NameOfSubstance UI="D047311">Luteolin</NameOfSubstance></Chemical><Chemical><RegistryNumber>XXE1CET956</RegistryNumber><NameOfSubstance UI="D007213">Indomethacin</NameOfSubstance></Chemical></ChemicalList><CitationSubset>IM</CitationSubset><MeshHeadingList><MeshHeading><DescriptorName UI="D000818" MajorTopicYN="N">Animals</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D000975" MajorTopicYN="N">Antioxidants</DescriptorName><QualifierName UI="Q000302" MajorTopicYN="N">isolation & purification</QualifierName><QualifierName UI="Q000494" MajorTopicYN="Y">pharmacology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D004305" MajorTopicYN="N">Dose-Response Relationship, Drug</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D004942" MajorTopicYN="N">Esophagitis, Peptic</DescriptorName><QualifierName UI="Q000209" MajorTopicYN="N">etiology</QualifierName><QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName><QualifierName UI="Q000517" MajorTopicYN="Y">prevention & control</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005744" MajorTopicYN="N">Gastric Acid</DescriptorName><QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005745" MajorTopicYN="N">Gastric Acidity Determination</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D005756" MajorTopicYN="N">Gastritis</DescriptorName><QualifierName UI="Q000139" MajorTopicYN="N">chemically induced</QualifierName><QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName><QualifierName UI="Q000517" MajorTopicYN="Y">prevention & control</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D005960" MajorTopicYN="N">Glucosides</DescriptorName><QualifierName UI="Q000302" MajorTopicYN="N">isolation & purification</QualifierName><QualifierName UI="Q000494" MajorTopicYN="Y">pharmacology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D007213" MajorTopicYN="N">Indomethacin</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D015227" MajorTopicYN="N">Lipid Peroxidation</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D047311" MajorTopicYN="N">Luteolin</DescriptorName><QualifierName UI="Q000302" MajorTopicYN="N">isolation & purification</QualifierName><QualifierName UI="Q000494" MajorTopicYN="Y">pharmacology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D008297" MajorTopicYN="N">Male</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D008315" MajorTopicYN="N">Malondialdehyde</DescriptorName><QualifierName UI="Q000378" MajorTopicYN="N">metabolism</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D009853" MajorTopicYN="N">Omeprazole</DescriptorName><QualifierName UI="Q000494" MajorTopicYN="N">pharmacology</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D018515" MajorTopicYN="N">Plant Leaves</DescriptorName><QualifierName UI="Q000737" MajorTopicYN="N">chemistry</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D011708" MajorTopicYN="N">Pylorus</DescriptorName><QualifierName UI="Q000601" MajorTopicYN="N">surgery</QualifierName></MeshHeading><MeshHeading><DescriptorName UI="D051381" MajorTopicYN="N">Rats</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D017207" MajorTopicYN="N">Rats, Sprague-Dawley</DescriptorName></MeshHeading><MeshHeading><DescriptorName UI="D032108" MajorTopicYN="N">Salix</DescriptorName><QualifierName UI="Q000737" MajorTopicYN="Y">chemistry</QualifierName></MeshHeading></MeshHeadingList></MedlineCitation><PubmedData><History><PubMedPubDate PubStatus="pubmed"><Year>2006</Year><Month>7</Month><Day>13</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="medline"><Year>2006</Year><Month>9</Month><Day>26</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate><PubMedPubDate PubStatus="entrez"><Year>2006</Year><Month>7</Month><Day>13</Day><Hour>9</Hour><Minute>0</Minute></PubMedPubDate></History><PublicationStatus>ppublish</PublicationStatus><ArticleIdList><ArticleId IdType="pubmed">16833016</ArticleId><ArticleId IdType="doi">10.1007/BF02969421</ArticleId></ArticleIdList></PubmedData></pubmed></record>Pour manipuler ce document sous Unix (Dilib)
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