AustralieFrV1, PubMed, Corpus, bibRecord, 000790

Control of Metastasis by NK Cells.

Identifieur interne : 000790 ( PubMed/Corpus ); précédent : 000789; suivant : 000791

Control of Metastasis by NK Cells.

Auteurs : Alejandro L Pez-Soto ; Segundo Gonzalez ; Mark J. Smyth ; Lorenzo Galluzzi

Source :

Cancer cell [ 1878-3686 ] ; 2017.

RBID : pubmed:28810142

English descriptors

KwdEn :
- Animals, Humans, Immunologic Surveillance, Immunotherapy, Killer Cells, Natural (immunology), Neoplasm Metastasis (immunology), Neoplasm Metastasis (pathology), Neoplasm Metastasis (prevention & control), Neoplasms (immunology), Neoplasms (pathology), Neoplasms (prevention & control), Tumor Escape, Tumor Microenvironment.
MESH :
- immunology : Killer Cells, Natural, Neoplasm Metastasis, Neoplasms.
- pathology : Neoplasm Metastasis, Neoplasms.
- prevention & control : Neoplasm Metastasis, Neoplasms.
- Animals, Humans, Immunologic Surveillance, Immunotherapy, Tumor Escape, Tumor Microenvironment.

Abstract

The metastatic spread of malignant cells to distant anatomical locations is a prominent cause of cancer-related death. Metastasis is governed by cancer-cell-intrinsic mechanisms that enable neoplastic cells to invade the local microenvironment, reach the circulation, and colonize distant sites, including the so-called epithelial-to-mesenchymal transition. Moreover, metastasis is regulated by microenvironmental and systemic processes, such as immunosurveillance. Here, we outline the cancer-cell-intrinsic and -extrinsic factors that regulate metastasis, discuss the key role of natural killer (NK) cells in the control of metastatic dissemination, and present potential therapeutic approaches to prevent or target metastatic disease by harnessing NK cells.

DOI: 10.1016/j.ccell.2017.06.009
PubMed: 28810142

Links to Exploration step

pubmed:28810142

Le document en format XML

<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Control of Metastasis by NK Cells.</title>
<author><name sortKey="L Pez Soto, Alejandro" sort="L Pez Soto, Alejandro" uniqKey="L Pez Soto A" first="Alejandro" last="L Pez-Soto">Alejandro L Pez-Soto</name>
<affiliation><nlm:affiliation>Departamento de Biología Funcional, Área de Inmunología, Universidad de Oviedo, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), 33006 Oviedo, Asturias, Spain. Electronic address: lopezsalejandro@uniovi.es.</nlm:affiliation>
</affiliation>
</author>
<author><name sortKey="Gonzalez, Segundo" sort="Gonzalez, Segundo" uniqKey="Gonzalez S" first="Segundo" last="Gonzalez">Segundo Gonzalez</name>
<affiliation><nlm:affiliation>Departamento de Biología Funcional, Área de Inmunología, Universidad de Oviedo, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), 33006 Oviedo, Asturias, Spain.</nlm:affiliation>
</affiliation>
</author>
<author><name sortKey="Smyth, Mark J" sort="Smyth, Mark J" uniqKey="Smyth M" first="Mark J" last="Smyth">Mark J. Smyth</name>
<affiliation><nlm:affiliation>Immunology of Cancer and Infection Laboratory, QIMR Berghofer Medical Research Institute, Herston, QLD 4006, Australia.</nlm:affiliation>
</affiliation>
</author>
<author><name sortKey="Galluzzi, Lorenzo" sort="Galluzzi, Lorenzo" uniqKey="Galluzzi L" first="Lorenzo" last="Galluzzi">Lorenzo Galluzzi</name>
<affiliation><nlm:affiliation>Department of Radiation Oncology, Weill Cornell Medical College, New York, NY 10065, USA; Sandra and Edward Meyer Cancer Center, New York, NY 10065, USA; Université Paris Descartes/Paris V, 75006 Paris, France. Electronic address: deadoc@vodafone.it.</nlm:affiliation>
</affiliation>
</author>
</titleStmt>
<publicationStmt><idno type="wicri:source">PubMed</idno>
<date when="2017">2017</date>
<idno type="RBID">pubmed:28810142</idno>
<idno type="pmid">28810142</idno>
<idno type="doi">10.1016/j.ccell.2017.06.009</idno>
<idno type="wicri:Area/PubMed/Corpus">000790</idno>
<idno type="wicri:explorRef" wicri:stream="PubMed" wicri:step="Corpus" wicri:corpus="PubMed">000790</idno>
</publicationStmt>
<sourceDesc><biblStruct><analytic><title xml:lang="en">Control of Metastasis by NK Cells.</title>
<author><name sortKey="L Pez Soto, Alejandro" sort="L Pez Soto, Alejandro" uniqKey="L Pez Soto A" first="Alejandro" last="L Pez-Soto">Alejandro L Pez-Soto</name>
<affiliation><nlm:affiliation>Departamento de Biología Funcional, Área de Inmunología, Universidad de Oviedo, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), 33006 Oviedo, Asturias, Spain. Electronic address: lopezsalejandro@uniovi.es.</nlm:affiliation>
</affiliation>
</author>
<author><name sortKey="Gonzalez, Segundo" sort="Gonzalez, Segundo" uniqKey="Gonzalez S" first="Segundo" last="Gonzalez">Segundo Gonzalez</name>
<affiliation><nlm:affiliation>Departamento de Biología Funcional, Área de Inmunología, Universidad de Oviedo, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), 33006 Oviedo, Asturias, Spain.</nlm:affiliation>
</affiliation>
</author>
<author><name sortKey="Smyth, Mark J" sort="Smyth, Mark J" uniqKey="Smyth M" first="Mark J" last="Smyth">Mark J. Smyth</name>
<affiliation><nlm:affiliation>Immunology of Cancer and Infection Laboratory, QIMR Berghofer Medical Research Institute, Herston, QLD 4006, Australia.</nlm:affiliation>
</affiliation>
</author>
<author><name sortKey="Galluzzi, Lorenzo" sort="Galluzzi, Lorenzo" uniqKey="Galluzzi L" first="Lorenzo" last="Galluzzi">Lorenzo Galluzzi</name>
<affiliation><nlm:affiliation>Department of Radiation Oncology, Weill Cornell Medical College, New York, NY 10065, USA; Sandra and Edward Meyer Cancer Center, New York, NY 10065, USA; Université Paris Descartes/Paris V, 75006 Paris, France. Electronic address: deadoc@vodafone.it.</nlm:affiliation>
</affiliation>
</author>
</analytic>
<series><title level="j">Cancer cell</title>
<idno type="eISSN">1878-3686</idno>
<imprint><date when="2017" type="published">2017</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
</fileDesc>
<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Animals</term>
<term>Humans</term>
<term>Immunologic Surveillance</term>
<term>Immunotherapy</term>
<term>Killer Cells, Natural (immunology)</term>
<term>Neoplasm Metastasis (immunology)</term>
<term>Neoplasm Metastasis (pathology)</term>
<term>Neoplasm Metastasis (prevention & control)</term>
<term>Neoplasms (immunology)</term>
<term>Neoplasms (pathology)</term>
<term>Neoplasms (prevention & control)</term>
<term>Tumor Escape</term>
<term>Tumor Microenvironment</term>
</keywords>
<keywords scheme="MESH" qualifier="immunology" xml:lang="en"><term>Killer Cells, Natural</term>
<term>Neoplasm Metastasis</term>
<term>Neoplasms</term>
</keywords>
<keywords scheme="MESH" qualifier="pathology" xml:lang="en"><term>Neoplasm Metastasis</term>
<term>Neoplasms</term>
</keywords>
<keywords scheme="MESH" qualifier="prevention & control" xml:lang="en"><term>Neoplasm Metastasis</term>
<term>Neoplasms</term>
</keywords>
<keywords scheme="MESH" xml:lang="en"><term>Animals</term>
<term>Humans</term>
<term>Immunologic Surveillance</term>
<term>Immunotherapy</term>
<term>Tumor Escape</term>
<term>Tumor Microenvironment</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">The metastatic spread of malignant cells to distant anatomical locations is a prominent cause of cancer-related death. Metastasis is governed by cancer-cell-intrinsic mechanisms that enable neoplastic cells to invade the local microenvironment, reach the circulation, and colonize distant sites, including the so-called epithelial-to-mesenchymal transition. Moreover, metastasis is regulated by microenvironmental and systemic processes, such as immunosurveillance. Here, we outline the cancer-cell-intrinsic and -extrinsic factors that regulate metastasis, discuss the key role of natural killer (NK) cells in the control of metastatic dissemination, and present potential therapeutic approaches to prevent or target metastatic disease by harnessing NK cells.</div>
</front>
</TEI>
<pubmed><MedlineCitation Status="MEDLINE" Owner="NLM"><PMID Version="1">28810142</PMID>
<DateCreated><Year>2017</Year>
<Month>08</Month>
<Day>15</Day>
</DateCreated>
<DateCompleted><Year>2017</Year>
<Month>09</Month>
<Day>11</Day>
</DateCompleted>
<DateRevised><Year>2017</Year>
<Month>09</Month>
<Day>11</Day>
</DateRevised>
<Article PubModel="Print"><Journal><ISSN IssnType="Electronic">1878-3686</ISSN>
<JournalIssue CitedMedium="Internet"><Volume>32</Volume>
<Issue>2</Issue>
<PubDate><Year>2017</Year>
<Month>Aug</Month>
<Day>14</Day>
</PubDate>
</JournalIssue>
<Title>Cancer cell</Title>
<ISOAbbreviation>Cancer Cell</ISOAbbreviation>
</Journal>
<ArticleTitle>Control of Metastasis by NK Cells.</ArticleTitle>
<Pagination><MedlinePgn>135-154</MedlinePgn>
</Pagination>
<ELocationID EIdType="pii" ValidYN="Y">S1535-6108(17)30260-X</ELocationID>
<ELocationID EIdType="doi" ValidYN="Y">10.1016/j.ccell.2017.06.009</ELocationID>
<Abstract><AbstractText>The metastatic spread of malignant cells to distant anatomical locations is a prominent cause of cancer-related death. Metastasis is governed by cancer-cell-intrinsic mechanisms that enable neoplastic cells to invade the local microenvironment, reach the circulation, and colonize distant sites, including the so-called epithelial-to-mesenchymal transition. Moreover, metastasis is regulated by microenvironmental and systemic processes, such as immunosurveillance. Here, we outline the cancer-cell-intrinsic and -extrinsic factors that regulate metastasis, discuss the key role of natural killer (NK) cells in the control of metastatic dissemination, and present potential therapeutic approaches to prevent or target metastatic disease by harnessing NK cells.</AbstractText>
<CopyrightInformation>Copyright © 2017 Elsevier Inc. All rights reserved.</CopyrightInformation>
</Abstract>
<AuthorList CompleteYN="Y"><Author ValidYN="Y"><LastName>López-Soto</LastName>
<ForeName>Alejandro</ForeName>
<Initials>A</Initials>
<AffiliationInfo><Affiliation>Departamento de Biología Funcional, Área de Inmunología, Universidad de Oviedo, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), 33006 Oviedo, Asturias, Spain. Electronic address: lopezsalejandro@uniovi.es.</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y"><LastName>Gonzalez</LastName>
<ForeName>Segundo</ForeName>
<Initials>S</Initials>
<AffiliationInfo><Affiliation>Departamento de Biología Funcional, Área de Inmunología, Universidad de Oviedo, Instituto Universitario de Oncología del Principado de Asturias (IUOPA), 33006 Oviedo, Asturias, Spain.</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y"><LastName>Smyth</LastName>
<ForeName>Mark J</ForeName>
<Initials>MJ</Initials>
<AffiliationInfo><Affiliation>Immunology of Cancer and Infection Laboratory, QIMR Berghofer Medical Research Institute, Herston, QLD 4006, Australia.</Affiliation>
</AffiliationInfo>
</Author>
<Author ValidYN="Y"><LastName>Galluzzi</LastName>
<ForeName>Lorenzo</ForeName>
<Initials>L</Initials>
<AffiliationInfo><Affiliation>Department of Radiation Oncology, Weill Cornell Medical College, New York, NY 10065, USA; Sandra and Edward Meyer Cancer Center, New York, NY 10065, USA; Université Paris Descartes/Paris V, 75006 Paris, France. Electronic address: deadoc@vodafone.it.</Affiliation>
</AffiliationInfo>
</Author>
</AuthorList>
<Language>eng</Language>
<PublicationTypeList><PublicationType UI="D016428">Journal Article</PublicationType>
<PublicationType UI="D016454">Review</PublicationType>
</PublicationTypeList>
</Article>
<MedlineJournalInfo><Country>United States</Country>
<MedlineTA>Cancer Cell</MedlineTA>
<NlmUniqueID>101130617</NlmUniqueID>
<ISSNLinking>1535-6108</ISSNLinking>
</MedlineJournalInfo>
<CitationSubset>IM</CitationSubset>
<MeshHeadingList><MeshHeading><DescriptorName UI="D000818" MajorTopicYN="N">Animals</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D006801" MajorTopicYN="N">Humans</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D007157" MajorTopicYN="N">Immunologic Surveillance</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D007167" MajorTopicYN="N">Immunotherapy</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D007694" MajorTopicYN="N">Killer Cells, Natural</DescriptorName>
<QualifierName UI="Q000276" MajorTopicYN="Y">immunology</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D009362" MajorTopicYN="N">Neoplasm Metastasis</DescriptorName>
<QualifierName UI="Q000276" MajorTopicYN="Y">immunology</QualifierName>
<QualifierName UI="Q000473" MajorTopicYN="N">pathology</QualifierName>
<QualifierName UI="Q000517" MajorTopicYN="N">prevention & control</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D009369" MajorTopicYN="N">Neoplasms</DescriptorName>
<QualifierName UI="Q000276" MajorTopicYN="N">immunology</QualifierName>
<QualifierName UI="Q000473" MajorTopicYN="N">pathology</QualifierName>
<QualifierName UI="Q000517" MajorTopicYN="Y">prevention & control</QualifierName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D019139" MajorTopicYN="N">Tumor Escape</DescriptorName>
</MeshHeading>
<MeshHeading><DescriptorName UI="D059016" MajorTopicYN="Y">Tumor Microenvironment</DescriptorName>
</MeshHeading>
</MeshHeadingList>
<KeywordList Owner="NOTNLM"><Keyword MajorTopicYN="N">ADCC</Keyword>
<Keyword MajorTopicYN="N">CTLA4</Keyword>
<Keyword MajorTopicYN="N">DNAM-1</Keyword>
<Keyword MajorTopicYN="N">NKG2D</Keyword>
<Keyword MajorTopicYN="N">PD-1</Keyword>
<Keyword MajorTopicYN="N">TGFB1</Keyword>
<Keyword MajorTopicYN="N">circulating tumor cells</Keyword>
<Keyword MajorTopicYN="N">disseminated tumor cells</Keyword>
<Keyword MajorTopicYN="N">immune checkpoint blockers</Keyword>
<Keyword MajorTopicYN="N">metastatic cascade</Keyword>
</KeywordList>
</MedlineCitation>
<PubmedData><History><PubMedPubDate PubStatus="received"><Year>2017</Year>
<Month>04</Month>
<Day>19</Day>
</PubMedPubDate>
<PubMedPubDate PubStatus="revised"><Year>2017</Year>
<Month>05</Month>
<Day>21</Day>
</PubMedPubDate>
<PubMedPubDate PubStatus="accepted"><Year>2017</Year>
<Month>06</Month>
<Day>22</Day>
</PubMedPubDate>
<PubMedPubDate PubStatus="entrez"><Year>2017</Year>
<Month>8</Month>
<Day>16</Day>
<Hour>6</Hour>
<Minute>0</Minute>
</PubMedPubDate>
<PubMedPubDate PubStatus="pubmed"><Year>2017</Year>
<Month>8</Month>
<Day>16</Day>
<Hour>6</Hour>
<Minute>0</Minute>
</PubMedPubDate>
<PubMedPubDate PubStatus="medline"><Year>2017</Year>
<Month>9</Month>
<Day>12</Day>
<Hour>6</Hour>
<Minute>0</Minute>
</PubMedPubDate>
</History>
<PublicationStatus>ppublish</PublicationStatus>
<ArticleIdList><ArticleId IdType="pubmed">28810142</ArticleId>
<ArticleId IdType="pii">S1535-6108(17)30260-X</ArticleId>
<ArticleId IdType="doi">10.1016/j.ccell.2017.06.009</ArticleId>
</ArticleIdList>
</PubmedData>
</pubmed>
</record>

Pour manipuler ce document sous Unix (Dilib)

EXPLOR_STEP=$WICRI_ROOT/Wicri/Asie/explor/AustralieFrV1/Data/PubMed/Corpus

HfdSelect -h $EXPLOR_STEP/biblio.hfd -nk 000790 | SxmlIndent | more

HfdSelect -h $EXPLOR_AREA/Data/PubMed/Corpus/biblio.hfd -nk 000790 | SxmlIndent | more

Pour mettre un lien sur cette page dans le réseau Wicri

{{Explor lien
   |wiki=    Wicri/Asie
   |area=    AustralieFrV1
   |flux=    PubMed
   |étape=   Corpus
   |type=    RBID
   |clé=     pubmed:28810142
   |texte=   Control of Metastasis by NK Cells.
}}

Pour générer des pages wiki

HfdIndexSelect -h $EXPLOR_AREA/Data/PubMed/Corpus/RBID.i   -Sk "pubmed:28810142" \
       | HfdSelect -Kh $EXPLOR_AREA/Data/PubMed/Corpus/biblio.hfd   \
       | NlmPubMed2Wicri -a AustralieFrV1

This area was generated with Dilib version V0.6.33.
Data generation: Tue Dec 5 10:43:12 2017. Site generation: Tue Mar 5 14:07:20 2024

	Serveur d'exploration sur les relations entre la France et l'Australie
	Attention, ce site est en cours de développement ! Attention, site généré par des moyens informatiques à partir de corpus bruts. Les informations ne sont donc pas validées.

Serveur d'exploration sur les relations entre la France et l'Australie

Control of Metastasis by NK Cells.

Control of Metastasis by NK Cells.

Source :

English descriptors

Abstract

Links to Exploration step

Le document en format XML

Pour manipuler ce document sous Unix (Dilib)

Pour mettre un lien sur cette page dans le réseau Wicri

Pour générer des pages wiki