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<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">The differing tempo of growth in bone size, mass, and density in girls is region-specific</title><author><name sortKey="Bass, Shona" sort="Bass, Shona" uniqKey="Bass S" first="Shona" last="Bass">Shona Bass</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author><author><name sortKey="Delmas, Pierre D" sort="Delmas, Pierre D" uniqKey="Delmas P" first="Pierre D." last="Delmas">Pierre D. Delmas</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">INSERM Research Unit 403, E. Herriot Hospital, cedex 0369437, Lyon, France</nlm:aff></affiliation></author><author><name sortKey="Pearce, Georgina" sort="Pearce, Georgina" uniqKey="Pearce G" first="Georgina" last="Pearce">Georgina Pearce</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author><author><name sortKey="Hendrich, Elke" sort="Hendrich, Elke" uniqKey="Hendrich E" first="Elke" last="Hendrich">Elke Hendrich</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author><author><name sortKey="Tabensky, Aaron" sort="Tabensky, Aaron" uniqKey="Tabensky A" first="Aaron" last="Tabensky">Aaron Tabensky</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author><author><name sortKey="Seeman, Ego" sort="Seeman, Ego" uniqKey="Seeman E" first="Ego" last="Seeman">Ego Seeman</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">PMC</idno><idno type="pmid">10491415</idno><idno type="pmc">408435</idno><idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC408435</idno><idno type="RBID">PMC:408435</idno><date when="1999">1999</date><idno type="wicri:Area/Pmc/Corpus">000A03</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Corpus" wicri:corpus="PMC">000A03</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">The differing tempo of growth in bone size, mass, and density in girls is region-specific</title><author><name sortKey="Bass, Shona" sort="Bass, Shona" uniqKey="Bass S" first="Shona" last="Bass">Shona Bass</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author><author><name sortKey="Delmas, Pierre D" sort="Delmas, Pierre D" uniqKey="Delmas P" first="Pierre D." last="Delmas">Pierre D. Delmas</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">INSERM Research Unit 403, E. Herriot Hospital, cedex 0369437, Lyon, France</nlm:aff></affiliation></author><author><name sortKey="Pearce, Georgina" sort="Pearce, Georgina" uniqKey="Pearce G" first="Georgina" last="Pearce">Georgina Pearce</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author><author><name sortKey="Hendrich, Elke" sort="Hendrich, Elke" uniqKey="Hendrich E" first="Elke" last="Hendrich">Elke Hendrich</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author><author><name sortKey="Tabensky, Aaron" sort="Tabensky, Aaron" uniqKey="Tabensky A" first="Aaron" last="Tabensky">Aaron Tabensky</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author><author><name sortKey="Seeman, Ego" sort="Seeman, Ego" uniqKey="Seeman E" first="Ego" last="Seeman">Ego Seeman</name><affiliation><nlm:aff id="N0x95997f8.0x9c703d0">Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia</nlm:aff></affiliation></author></analytic><series><title level="j">Journal of Clinical Investigation</title><idno type="ISSN">0021-9738</idno><imprint><date when="1999">1999</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en"><p>The differing tempo and direction of growth of the periosteal and endocortical surfaces, and the differing tempo of growth of the axial and appendicular skeleton, may predispose to regional deficits in bone size, bone mineral content (BMC), and volumetric bone mineral density (vBMD). These traits were measured during 2 years by dual x-ray absorptiometry in 109 girls. By 7 years of age, bone size was approximately 80% of its maturational peak, and BMC was approximately 40% of its peak. Before puberty, the legs grew more rapidly than the trunk. During puberty, the growth spurt was truncal. Between 7 and 17 years, femoral and lumbar spine BMC increased by 50–150% because bone size increased. vBMD increased by 10–30%. Thus, growth builds a bigger, but only moderately denser, skeleton. Regions growing rapidly, or distant from their peak, may be more severely affected by illness than those growing slowly or nearer completion of growth. Depending on the age of exposure to disease, deficits may occur in limb dimensions (prepuberty), spine dimensions (early puberty), or vBMD by interference with mineral accrual (late puberty). As vBMD is independent of age before puberty, the position of an individual’s vBMD in the population distribution is established early in life. Bone fragility in old age may have its foundations in growth.</p></div></front></TEI><pmc article-type="research-article"><pmc-comment>The publisher of this article does not allow downloading of the full text in XML form.</pmc-comment>
<front><journal-meta><journal-id journal-id-type="nlm-ta">J Clin Invest</journal-id><journal-id journal-id-type="publisher-id">J CLIN INVEST</journal-id><journal-title>Journal of Clinical Investigation</journal-title><issn pub-type="ppub">0021-9738</issn><publisher><publisher-name>American Society for Clinical Investigation</publisher-name></publisher></journal-meta><article-meta><article-id pub-id-type="pmid">10491415</article-id><article-id pub-id-type="pmc">408435</article-id><article-id pub-id-type="publisher-id">07060</article-id><article-categories><subj-group subj-group-type="heading"><subject>Article</subject><subj-group><subject>Hormones and Metabolism</subject></subj-group></subj-group></article-categories><title-group><article-title>The differing tempo of growth in bone size, mass, and density in girls is region-specific</article-title></title-group><contrib-group><contrib contrib-type="author"><name><surname>Bass</surname><given-names>Shona</given-names></name><xref ref-type="aff" rid="N0x95997f8.0x9c703d0">1</xref></contrib><contrib contrib-type="author"><name><surname>Delmas</surname><given-names>Pierre D.</given-names></name><xref ref-type="aff" rid="N0x95997f8.0x9c703d0">2</xref></contrib><contrib contrib-type="author"><name><surname>Pearce</surname><given-names>Georgina</given-names></name><xref ref-type="aff" rid="N0x95997f8.0x9c703d0">1</xref></contrib><contrib contrib-type="author"><name><surname>Hendrich</surname><given-names>Elke</given-names></name><xref ref-type="aff" rid="N0x95997f8.0x9c703d0">1</xref></contrib><contrib contrib-type="author"><name><surname>Tabensky</surname><given-names>Aaron</given-names></name><xref ref-type="aff" rid="N0x95997f8.0x9c703d0">1</xref></contrib><contrib contrib-type="author"><name><surname>Seeman</surname><given-names>Ego</given-names></name><xref ref-type="aff" rid="N0x95997f8.0x9c703d0">1</xref></contrib></contrib-group><aff id="N0x95997f8.0x9c703d0"><label>1</label>Endocrine Unit and Department of Medicine, Austin and Repatriation Medical Centre, University of Melbourne, Melbourne 3084, Australia<label>2</label>INSERM Research Unit 403, E. Herriot Hospital, cedex 0369437, Lyon, France</aff><author-notes><fn><p>Address correspondence to: Ego Seeman, Endocrine Department, Austin and Repatriation Medical Centre, Heidelberg, Melbourne 3084, Australia. Phone: 61-3-9496-5489; Fax: 61-3-9496-3365; E-mail: <email>ego@austin.unimelb.edu.au</email>.</p></fn></author-notes><pub-date pub-type="ppub"><day>15</day><month>9</month><year>1999</year></pub-date><volume>104</volume><issue>6</issue><fpage>795</fpage><lpage>804</lpage><history><date date-type="received"><day>12</day><month>4</month><year>1999</year></date><date date-type="accepted"><day>5</day><month>8</month><year>1999</year></date></history><copyright-statement>Copyright © 1999, American Society for Clinical Investigation</copyright-statement><copyright-year>1999</copyright-year><abstract><p>The differing tempo and direction of growth of the periosteal and endocortical surfaces, and the differing tempo of growth of the axial and appendicular skeleton, may predispose to regional deficits in bone size, bone mineral content (BMC), and volumetric bone mineral density (vBMD). These traits were measured during 2 years by dual x-ray absorptiometry in 109 girls. By 7 years of age, bone size was approximately 80% of its maturational peak, and BMC was approximately 40% of its peak. Before puberty, the legs grew more rapidly than the trunk. During puberty, the growth spurt was truncal. Between 7 and 17 years, femoral and lumbar spine BMC increased by 50–150% because bone size increased. vBMD increased by 10–30%. Thus, growth builds a bigger, but only moderately denser, skeleton. Regions growing rapidly, or distant from their peak, may be more severely affected by illness than those growing slowly or nearer completion of growth. Depending on the age of exposure to disease, deficits may occur in limb dimensions (prepuberty), spine dimensions (early puberty), or vBMD by interference with mineral accrual (late puberty). As vBMD is independent of age before puberty, the position of an individual’s vBMD in the population distribution is established early in life. Bone fragility in old age may have its foundations in growth.</p></abstract></article-meta></front></pmc></record>Pour manipuler ce document sous Unix (Dilib)
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