Impact of substance use on the onset and course of early psychosis
Identifieur interne :
001A03 ( PascalFrancis/Curation );
précédent :
001A02;
suivant :
001A04
Impact of substance use on the onset and course of early psychosis
Auteurs : Hélène Verdoux [
France] ;
Marie Toumier [
France] ;
Audrey Cougnard [
France]
Source :
-
Schizophrenia research [ 0920-9964 ] ; 2005.
RBID : Pascal:05-0468970
Descripteurs français
- Pascal (Inist)
- Toxicomanie,
Précoce,
Psychose,
Cannabis,
Facteur risque,
Schizophrénie,
Pronostic,
Association morbide,
Article synthèse,
Homme,
Abus de substance.
- Wicri :
English descriptors
- KwdEn :
- Cannabis,
Concomitant disease,
Drug addiction,
Early,
Human,
Prognosis,
Psychosis,
Review,
Risk factor,
Schizophrenia,
Substance abuse.
Abstract
The strong comorbidity between psychosis and substance use is already identifiable in early psychosis, raising the question of the direction of the association between substance use and psychosis onset. It has long been considered that this association was explained by the self-medication hypothesis. This hypothesis has been recently challenged by several prospective studies carried out in population-based samples, showing a dose-response relationship between cannabis exposure and risk of psychosis. This association was independent from potential confounding factors such as exposure to other drugs and preexistence of psychotic symptoms. As a large percentage of subjects from the general population is now exposed to this drug, even a small increase in the risk of adverse effects may have significant deleterious consequences for the health of the population. Hence, reducing exposure to cannabis may contribute to prevention of some incident cases of psychosis. Regarding prognosis, persistent substance misuse after the onset of psychosis has a deleterious impact on clinical outcome. Therapeutic programs for subjects with dual diagnosis should be implemented early in the course of psychosis to maximise their impact on the course of illness.
pA |
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A11 | 01 | 1 | | @1 VERDOUX (Hélène) |
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A11 | 02 | 1 | | @1 TOUMIER (Marie) |
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A11 | 03 | 1 | | @1 COUGNARD (Audrey) |
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A12 | 01 | 1 | | @1 ADDINGTON (Jean) @9 ed. |
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A14 | 01 | | | @1 EA 3676, IFR of Public Health, University Victor Segalen Bordeaux 2 @2 Bordeaux @3 FRA @Z 1 aut. @Z 2 aut. @Z 3 aut. |
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A15 | 01 | | | @1 University of Toronto, Psychiatry, CAMH, 250 College Street @2 Toronto, M5T 1R8 @3 CAN @Z 1 aut. |
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C01 | 01 | | ENG | @0 The strong comorbidity between psychosis and substance use is already identifiable in early psychosis, raising the question of the direction of the association between substance use and psychosis onset. It has long been considered that this association was explained by the self-medication hypothesis. This hypothesis has been recently challenged by several prospective studies carried out in population-based samples, showing a dose-response relationship between cannabis exposure and risk of psychosis. This association was independent from potential confounding factors such as exposure to other drugs and preexistence of psychotic symptoms. As a large percentage of subjects from the general population is now exposed to this drug, even a small increase in the risk of adverse effects may have significant deleterious consequences for the health of the population. Hence, reducing exposure to cannabis may contribute to prevention of some incident cases of psychosis. Regarding prognosis, persistent substance misuse after the onset of psychosis has a deleterious impact on clinical outcome. Therapeutic programs for subjects with dual diagnosis should be implemented early in the course of psychosis to maximise their impact on the course of illness. |
---|
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C03 | 03 | X | FRE | @0 Psychose @5 03 |
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C03 | 03 | X | ENG | @0 Psychosis @5 03 |
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C03 | 04 | X | FRE | @0 Cannabis @2 NS @5 04 |
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C03 | 07 | X | SPA | @0 Pronóstico @5 07 |
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C03 | 08 | X | FRE | @0 Association morbide @5 08 |
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C03 | 08 | X | ENG | @0 Concomitant disease @5 08 |
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C03 | 09 | X | FRE | @0 Article synthèse @5 09 |
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C03 | 09 | X | SPA | @0 Artículo síntesis @5 09 |
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C03 | 10 | X | FRE | @0 Homme @5 18 |
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C03 | 10 | X | ENG | @0 Human @5 18 |
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C03 | 10 | X | SPA | @0 Hombre @5 18 |
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C03 | 11 | X | FRE | @0 Abus de substance @4 CD @5 96 |
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C03 | 11 | X | ENG | @0 Substance abuse @4 CD @5 96 |
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C03 | 11 | X | SPA | @0 Abuso de sustancias @4 CD @5 96 |
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C07 | 01 | X | FRE | @0 Cannabidaceae @2 NS |
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C07 | 01 | X | ENG | @0 Cannabidaceae @2 NS |
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C07 | 01 | X | SPA | @0 Cannabidaceae @2 NS |
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C07 | 02 | X | FRE | @0 Dicotyledones @2 NS |
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C07 | 02 | X | ENG | @0 Dicotyledones @2 NS |
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C07 | 02 | X | SPA | @0 Dicotyledones @2 NS |
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C07 | 04 | X | FRE | @0 Spermatophyta @2 NS |
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C07 | 04 | X | ENG | @0 Spermatophyta @2 NS |
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C07 | 04 | X | SPA | @0 Spermatophyta @2 NS |
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N21 | | | | @1 332 |
---|
|
pR |
A30 | 01 | 1 | ENG | @1 International conference on Early Psychosis @2 4 @3 Vancouver CAN @4 2004-09-28 |
---|
|
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Le document en format XML
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<fC07 i1="04" i2="X" l="FRE"><s0>Spermatophyta</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="04" i2="X" l="ENG"><s0>Spermatophyta</s0>
<s2>NS</s2>
</fC07>
<fC07 i1="04" i2="X" l="SPA"><s0>Spermatophyta</s0>
<s2>NS</s2>
</fC07>
<fN21><s1>332</s1>
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</pA>
<pR><fA30 i1="01" i2="1" l="ENG"><s1>International conference on Early Psychosis</s1>
<s2>4</s2>
<s3>Vancouver CAN</s3>
<s4>2004-09-28</s4>
</fA30>
</pR>
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