AustralieFrV1, PascalFrancis, Corpus, bibRecord, 004774

Plasma lipids predict myocardial infarction, but not stroke, in patients with established cerebrovascular disease

Identifieur interne : 004774 ( PascalFrancis/Corpus ); précédent : 004773; suivant : 004775

Plasma lipids predict myocardial infarction, but not stroke, in patients with established cerebrovascular disease

Auteurs : Pierre Amarenco ; Anushka Patel ; Mark Woodward ; Duncan J. Campbell ; David R. Sullivan ; Samuel Colman ; John Chalmers ; Bruce Neal ; Stephen Macmahon

Source :

European heart journal [ 0195-668X ] ; 2005.

RBID : Pascal:05-0404764

Descripteurs français

Pascal (Inist)
- Infarctus myocarde, Accident cérébrovasculaire, Cérébrovasculaire pathologie, Ischémie, Plasma sanguin, Lipide, Prédiction, Homme, Etude cas témoin, Hémorragie, Insuffisance cardiaque, Appareil circulatoire, Cardiologie, Phlébologie.

English descriptors

KwdEn :
- Blood plasma, Cardiology, Case control study, Cerebrovascular disease, Circulatory system, Heart failure, Hemorrhage, Human, Ischemia, Lipids, Myocardial infarction, Phlebology, Prediction, Stroke.

Abstract

Aims To evaluate the role of plasma lipids in recurrent vascular events, including stroke, among individuals with established cerebrovascular disease. Methods and results Plasma total cholesterol, HDL cholesterol, and triglycerides were measured at baseline among individuals participating in the Perindopril Protection Against Recurrent Stroke (PROGRESS) study, a randomized clinical trial of blood pressure lowering among patients with previous stroke or transient ischaemic attack. A series of nested case-control studies were used to investigate the association between each of these lipid variables and the risk of subsequent haemorrhagic stroke, ischaemic stroke, myocardial infarction (MI), and heart failure. A total of 895 patients were selected as cases (83 haemorrhagic stroke, 472 ischaemic stroke, 206 MI, and 258 heart failure) and each was matched with one to three controls. After adjustment for other major cardiovascular risk factors, none of the lipid variables was associated with the risk of either stroke subtype. There were significant positive and negative associations for total cholesterol and HDL, respectively, with the risk of MI; the odds ratio comparing the highest and lowest thirds of each of these lipid variables was 2.00 (95% CI: 1.30-3.09) for total cholesterol and 0.58 (95% CI: 0.37-0.90) for HDL. HDL was inversely associated with the risk of heart failure; however, this result was of borderline statistical significance (P=0.05). Conclusion Lipid variables are associated with the risk of MI, but not recurrent stroke, in patients with established cerebrovascular disease.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

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A11	`02`	`1`		`@1 PATEL (Anushka)`
A11	`03`	`1`		`@1 WOODWARD (Mark)`
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A14	`04`			`@1 Department of Medicine, University of Melbourne @2 Victoria @3 AUS @Z 4 aut.`
A14	`05`			`@1 Department of Clinical Biochemistry, Royal Prince Alfred Hospital @2 Sydney @3 AUS @Z 5 aut.`
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C01	`01`		`ENG`	@0 Aims To evaluate the role of plasma lipids in recurrent vascular events, including stroke, among individuals with established cerebrovascular disease. Methods and results Plasma total cholesterol, HDL cholesterol, and triglycerides were measured at baseline among individuals participating in the Perindopril Protection Against Recurrent Stroke (PROGRESS) study, a randomized clinical trial of blood pressure lowering among patients with previous stroke or transient ischaemic attack. A series of nested case-control studies were used to investigate the association between each of these lipid variables and the risk of subsequent haemorrhagic stroke, ischaemic stroke, myocardial infarction (MI), and heart failure. A total of 895 patients were selected as cases (83 haemorrhagic stroke, 472 ischaemic stroke, 206 MI, and 258 heart failure) and each was matched with one to three controls. After adjustment for other major cardiovascular risk factors, none of the lipid variables was associated with the risk of either stroke subtype. There were significant positive and negative associations for total cholesterol and HDL, respectively, with the risk of MI; the odds ratio comparing the highest and lowest thirds of each of these lipid variables was 2.00 (95% CI: 1.30-3.09) for total cholesterol and 0.58 (95% CI: 0.37-0.90) for HDL. HDL was inversely associated with the risk of heart failure; however, this result was of borderline statistical significance (P=0.05). Conclusion Lipid variables are associated with the risk of MI, but not recurrent stroke, in patients with established cerebrovascular disease.
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Format Inist (serveur)

NO :	PASCAL 05-0404764 INIST
ET :	Plasma lipids predict myocardial infarction, but not stroke, in patients with established cerebrovascular disease
AU :	AMARENCO (Pierre); PATEL (Anushka); WOODWARD (Mark); CAMPBELL (Duncan J.); SULLIVAN (David R.); COLMAN (Samuel); CHALMERS (John); NEAL (Bruce); MACMAHON (Stephen)
AF :	Department of Neurology and Stroke Centre, Bichat Hospital and Medical School, Denis Diderot University/Paris/France (1 aut., 2 aut., 3 aut., 6 aut., 7 aut., 8 aut., 9 aut.); The George Institute for International Health, University of Sydney, PO Box M201, Missenden Road/Sydney, NSW 2050/Australie (4 aut.); St Vincent's Institute of Medical Research/Victoria/Australie (4 aut.); Department of Medicine, University of Melbourne/Victoria/Australie (4 aut.); Department of Clinical Biochemistry, Royal Prince Alfred Hospital/Sydney/Australie (5 aut.)
DT :	Publication en série; Article; Editorial; Niveau analytique
SO :	European heart journal; ISSN 0195-668X; Royaume-Uni; Da. 2005; Vol. 26; No. 18; 1818-1819,1910-1915 [8 p.]; Bibl. 33 ref.
LA :	Anglais
EA :	Aims To evaluate the role of plasma lipids in recurrent vascular events, including stroke, among individuals with established cerebrovascular disease. Methods and results Plasma total cholesterol, HDL cholesterol, and triglycerides were measured at baseline among individuals participating in the Perindopril Protection Against Recurrent Stroke (PROGRESS) study, a randomized clinical trial of blood pressure lowering among patients with previous stroke or transient ischaemic attack. A series of nested case-control studies were used to investigate the association between each of these lipid variables and the risk of subsequent haemorrhagic stroke, ischaemic stroke, myocardial infarction (MI), and heart failure. A total of 895 patients were selected as cases (83 haemorrhagic stroke, 472 ischaemic stroke, 206 MI, and 258 heart failure) and each was matched with one to three controls. After adjustment for other major cardiovascular risk factors, none of the lipid variables was associated with the risk of either stroke subtype. There were significant positive and negative associations for total cholesterol and HDL, respectively, with the risk of MI; the odds ratio comparing the highest and lowest thirds of each of these lipid variables was 2.00 (95% CI: 1.30-3.09) for total cholesterol and 0.58 (95% CI: 0.37-0.90) for HDL. HDL was inversely associated with the risk of heart failure; however, this result was of borderline statistical significance (P=0.05). Conclusion Lipid variables are associated with the risk of MI, but not recurrent stroke, in patients with established cerebrovascular disease.
CC :	002B12A03; 002B17C; 002B12A01
FD :	Infarctus myocarde; Accident cérébrovasculaire; Cérébrovasculaire pathologie; Ischémie; Plasma sanguin; Lipide; Prédiction; Homme; Etude cas témoin; Hémorragie; Insuffisance cardiaque; Appareil circulatoire; Cardiologie; Phlébologie
FG :	Appareil circulatoire pathologie; Myocarde pathologie; Encéphale pathologie; Système nerveux central pathologie; Système nerveux pathologie; Vaisseau sanguin pathologie; Cardiopathie
ED :	Myocardial infarction; Stroke; Cerebrovascular disease; Ischemia; Blood plasma; Lipids; Prediction; Human; Case control study; Hemorrhage; Heart failure; Circulatory system; Cardiology; Phlebology
EG :	Cardiovascular disease; Myocardial disease; Cerebral disorder; Central nervous system disease; Nervous system diseases; Vascular disease; Heart disease
SD :	Infarto miocardio; Accidente cerebrovascular; Vaso sanguíneo encéfalo patología; Isquemia; Plasma sanguíneo; Lípido; Predicción; Hombre; Estudio caso control; Hemorragia; Insuficiencia cardíaca; Aparato circulatorio; Cardiología; Flebología
LO :	INIST-18785.354000132653440140
ID :	05-0404764

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Pascal:05-0404764

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<author><name sortKey="Sullivan, David R" sort="Sullivan, David R" uniqKey="Sullivan D" first="David R." last="Sullivan">David R. Sullivan</name>
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</inist:fA14>
</affiliation>
</author>
<author><name sortKey="Colman, Samuel" sort="Colman, Samuel" uniqKey="Colman S" first="Samuel" last="Colman">Samuel Colman</name>
<affiliation><inist:fA14 i1="01"><s1>Department of Neurology and Stroke Centre, Bichat Hospital and Medical School, Denis Diderot University</s1>
<s2>Paris</s2>
<s3>FRA</s3>
<sZ>1 aut.</sZ>
<sZ>2 aut.</sZ>
<sZ>3 aut.</sZ>
<sZ>6 aut.</sZ>
<sZ>7 aut.</sZ>
<sZ>8 aut.</sZ>
<sZ>9 aut.</sZ>
</inist:fA14>
</affiliation>
</author>
<author><name sortKey="Chalmers, John" sort="Chalmers, John" uniqKey="Chalmers J" first="John" last="Chalmers">John Chalmers</name>
<affiliation><inist:fA14 i1="01"><s1>Department of Neurology and Stroke Centre, Bichat Hospital and Medical School, Denis Diderot University</s1>
<s2>Paris</s2>
<s3>FRA</s3>
<sZ>1 aut.</sZ>
<sZ>2 aut.</sZ>
<sZ>3 aut.</sZ>
<sZ>6 aut.</sZ>
<sZ>7 aut.</sZ>
<sZ>8 aut.</sZ>
<sZ>9 aut.</sZ>
</inist:fA14>
</affiliation>
</author>
<author><name sortKey="Neal, Bruce" sort="Neal, Bruce" uniqKey="Neal B" first="Bruce" last="Neal">Bruce Neal</name>
<affiliation><inist:fA14 i1="01"><s1>Department of Neurology and Stroke Centre, Bichat Hospital and Medical School, Denis Diderot University</s1>
<s2>Paris</s2>
<s3>FRA</s3>
<sZ>1 aut.</sZ>
<sZ>2 aut.</sZ>
<sZ>3 aut.</sZ>
<sZ>6 aut.</sZ>
<sZ>7 aut.</sZ>
<sZ>8 aut.</sZ>
<sZ>9 aut.</sZ>
</inist:fA14>
</affiliation>
</author>
<author><name sortKey="Macmahon, Stephen" sort="Macmahon, Stephen" uniqKey="Macmahon S" first="Stephen" last="Macmahon">Stephen Macmahon</name>
<affiliation><inist:fA14 i1="01"><s1>Department of Neurology and Stroke Centre, Bichat Hospital and Medical School, Denis Diderot University</s1>
<s2>Paris</s2>
<s3>FRA</s3>
<sZ>1 aut.</sZ>
<sZ>2 aut.</sZ>
<sZ>3 aut.</sZ>
<sZ>6 aut.</sZ>
<sZ>7 aut.</sZ>
<sZ>8 aut.</sZ>
<sZ>9 aut.</sZ>
</inist:fA14>
</affiliation>
</author>
</analytic>
<series><title level="j" type="main">European heart journal</title>
<title level="j" type="abbreviated">Eur. heart j.</title>
<idno type="ISSN">0195-668X</idno>
<imprint><date when="2005">2005</date>
</imprint>
</series>
</biblStruct>
</sourceDesc>
<seriesStmt><title level="j" type="main">European heart journal</title>
<title level="j" type="abbreviated">Eur. heart j.</title>
<idno type="ISSN">0195-668X</idno>
</seriesStmt>
</fileDesc>
<profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Blood plasma</term>
<term>Cardiology</term>
<term>Case control study</term>
<term>Cerebrovascular disease</term>
<term>Circulatory system</term>
<term>Heart failure</term>
<term>Hemorrhage</term>
<term>Human</term>
<term>Ischemia</term>
<term>Lipids</term>
<term>Myocardial infarction</term>
<term>Phlebology</term>
<term>Prediction</term>
<term>Stroke</term>
</keywords>
<keywords scheme="Pascal" xml:lang="fr"><term>Infarctus myocarde</term>
<term>Accident cérébrovasculaire</term>
<term>Cérébrovasculaire pathologie</term>
<term>Ischémie</term>
<term>Plasma sanguin</term>
<term>Lipide</term>
<term>Prédiction</term>
<term>Homme</term>
<term>Etude cas témoin</term>
<term>Hémorragie</term>
<term>Insuffisance cardiaque</term>
<term>Appareil circulatoire</term>
<term>Cardiologie</term>
<term>Phlébologie</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">Aims To evaluate the role of plasma lipids in recurrent vascular events, including stroke, among individuals with established cerebrovascular disease. Methods and results Plasma total cholesterol, HDL cholesterol, and triglycerides were measured at baseline among individuals participating in the Perindopril Protection Against Recurrent Stroke (PROGRESS) study, a randomized clinical trial of blood pressure lowering among patients with previous stroke or transient ischaemic attack. A series of nested case-control studies were used to investigate the association between each of these lipid variables and the risk of subsequent haemorrhagic stroke, ischaemic stroke, myocardial infarction (MI), and heart failure. A total of 895 patients were selected as cases (83 haemorrhagic stroke, 472 ischaemic stroke, 206 MI, and 258 heart failure) and each was matched with one to three controls. After adjustment for other major cardiovascular risk factors, none of the lipid variables was associated with the risk of either stroke subtype. There were significant positive and negative associations for total cholesterol and HDL, respectively, with the risk of MI; the odds ratio comparing the highest and lowest thirds of each of these lipid variables was 2.00 (95% CI: 1.30-3.09) for total cholesterol and 0.58 (95% CI: 0.37-0.90) for HDL. HDL was inversely associated with the risk of heart failure; however, this result was of borderline statistical significance (P=0.05). Conclusion Lipid variables are associated with the risk of MI, but not recurrent stroke, in patients with established cerebrovascular disease.</div>
</front>
</TEI>
<inist><standard h6="B"><pA><fA01 i1="01" i2="1"><s0>0195-668X</s0>
</fA01>
<fA03 i2="1"><s0>Eur. heart j.</s0>
</fA03>
<fA05><s2>26</s2>
</fA05>
<fA06><s2>18</s2>
</fA06>
<fA08 i1="01" i2="1" l="ENG"><s1>Plasma lipids predict myocardial infarction, but not stroke, in patients with established cerebrovascular disease</s1>
</fA08>
<fA11 i1="01" i2="1"><s1>AMARENCO (Pierre)</s1>
</fA11>
<fA11 i1="02" i2="1"><s1>PATEL (Anushka)</s1>
</fA11>
<fA11 i1="03" i2="1"><s1>WOODWARD (Mark)</s1>
</fA11>
<fA11 i1="04" i2="1"><s1>CAMPBELL (Duncan J.)</s1>
</fA11>
<fA11 i1="05" i2="1"><s1>SULLIVAN (David R.)</s1>
</fA11>
<fA11 i1="06" i2="1"><s1>COLMAN (Samuel)</s1>
</fA11>
<fA11 i1="07" i2="1"><s1>CHALMERS (John)</s1>
</fA11>
<fA11 i1="08" i2="1"><s1>NEAL (Bruce)</s1>
</fA11>
<fA11 i1="09" i2="1"><s1>MACMAHON (Stephen)</s1>
</fA11>
<fA14 i1="01"><s1>Department of Neurology and Stroke Centre, Bichat Hospital and Medical School, Denis Diderot University</s1>
<s2>Paris</s2>
<s3>FRA</s3>
<sZ>1 aut.</sZ>
<sZ>2 aut.</sZ>
<sZ>3 aut.</sZ>
<sZ>6 aut.</sZ>
<sZ>7 aut.</sZ>
<sZ>8 aut.</sZ>
<sZ>9 aut.</sZ>
</fA14>
<fA14 i1="02"><s1>The George Institute for International Health, University of Sydney, PO Box M201, Missenden Road</s1>
<s2>Sydney, NSW 2050</s2>
<s3>AUS</s3>
<sZ>4 aut.</sZ>
</fA14>
<fA14 i1="03"><s1>St Vincent's Institute of Medical Research</s1>
<s2>Victoria</s2>
<s3>AUS</s3>
<sZ>4 aut.</sZ>
</fA14>
<fA14 i1="04"><s1>Department of Medicine, University of Melbourne</s1>
<s2>Victoria</s2>
<s3>AUS</s3>
<sZ>4 aut.</sZ>
</fA14>
<fA14 i1="05"><s1>Department of Clinical Biochemistry, Royal Prince Alfred Hospital</s1>
<s2>Sydney</s2>
<s3>AUS</s3>
<sZ>5 aut.</sZ>
</fA14>
<fA20><s2>1818-1819,1910-1915 [8 p.]</s2>
</fA20>
<fA21><s1>2005</s1>
</fA21>
<fA23 i1="01"><s0>ENG</s0>
</fA23>
<fA43 i1="01"><s1>INIST</s1>
<s2>18785</s2>
<s5>354000132653440140</s5>
</fA43>
<fA44><s0>0000</s0>
<s1>© 2005 INIST-CNRS. All rights reserved.</s1>
</fA44>
<fA45><s0>33 ref.</s0>
</fA45>
<fA47 i1="01" i2="1"><s0>05-0404764</s0>
</fA47>
<fA60><s1>P</s1>
<s3>AR</s3>
<s3>ED</s3>
</fA60>
<fA61><s0>A</s0>
</fA61>
<fA64 i1="01" i2="1"><s0>European heart journal</s0>
</fA64>
<fA66 i1="01"><s0>GBR</s0>
</fA66>
<fC01 i1="01" l="ENG"><s0>Aims To evaluate the role of plasma lipids in recurrent vascular events, including stroke, among individuals with established cerebrovascular disease. Methods and results Plasma total cholesterol, HDL cholesterol, and triglycerides were measured at baseline among individuals participating in the Perindopril Protection Against Recurrent Stroke (PROGRESS) study, a randomized clinical trial of blood pressure lowering among patients with previous stroke or transient ischaemic attack. A series of nested case-control studies were used to investigate the association between each of these lipid variables and the risk of subsequent haemorrhagic stroke, ischaemic stroke, myocardial infarction (MI), and heart failure. A total of 895 patients were selected as cases (83 haemorrhagic stroke, 472 ischaemic stroke, 206 MI, and 258 heart failure) and each was matched with one to three controls. After adjustment for other major cardiovascular risk factors, none of the lipid variables was associated with the risk of either stroke subtype. There were significant positive and negative associations for total cholesterol and HDL, respectively, with the risk of MI; the odds ratio comparing the highest and lowest thirds of each of these lipid variables was 2.00 (95% CI: 1.30-3.09) for total cholesterol and 0.58 (95% CI: 0.37-0.90) for HDL. HDL was inversely associated with the risk of heart failure; however, this result was of borderline statistical significance (P=0.05). Conclusion Lipid variables are associated with the risk of MI, but not recurrent stroke, in patients with established cerebrovascular disease.</s0>
</fC01>
<fC02 i1="01" i2="X"><s0>002B12A03</s0>
</fC02>
<fC02 i1="02" i2="X"><s0>002B17C</s0>
</fC02>
<fC02 i1="03" i2="X"><s0>002B12A01</s0>
</fC02>
<fC03 i1="01" i2="X" l="FRE"><s0>Infarctus myocarde</s0>
<s2>NM</s2>
<s5>01</s5>
</fC03>
<fC03 i1="01" i2="X" l="ENG"><s0>Myocardial infarction</s0>
<s2>NM</s2>
<s5>01</s5>
</fC03>
<fC03 i1="01" i2="X" l="SPA"><s0>Infarto miocardio</s0>
<s2>NM</s2>
<s5>01</s5>
</fC03>
<fC03 i1="02" i2="X" l="FRE"><s0>Accident cérébrovasculaire</s0>
<s5>02</s5>
</fC03>
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<s5>02</s5>
</fC03>
<fC03 i1="02" i2="X" l="SPA"><s0>Accidente cerebrovascular</s0>
<s5>02</s5>
</fC03>
<fC03 i1="03" i2="X" l="FRE"><s0>Cérébrovasculaire pathologie</s0>
<s5>03</s5>
</fC03>
<fC03 i1="03" i2="X" l="ENG"><s0>Cerebrovascular disease</s0>
<s5>03</s5>
</fC03>
<fC03 i1="03" i2="X" l="SPA"><s0>Vaso sanguíneo encéfalo patología</s0>
<s5>03</s5>
</fC03>
<fC03 i1="04" i2="X" l="FRE"><s0>Ischémie</s0>
<s5>04</s5>
</fC03>
<fC03 i1="04" i2="X" l="ENG"><s0>Ischemia</s0>
<s5>04</s5>
</fC03>
<fC03 i1="04" i2="X" l="SPA"><s0>Isquemia</s0>
<s5>04</s5>
</fC03>
<fC03 i1="05" i2="X" l="FRE"><s0>Plasma sanguin</s0>
<s5>09</s5>
</fC03>
<fC03 i1="05" i2="X" l="ENG"><s0>Blood plasma</s0>
<s5>09</s5>
</fC03>
<fC03 i1="05" i2="X" l="SPA"><s0>Plasma sanguíneo</s0>
<s5>09</s5>
</fC03>
<fC03 i1="06" i2="X" l="FRE"><s0>Lipide</s0>
<s5>10</s5>
</fC03>
<fC03 i1="06" i2="X" l="ENG"><s0>Lipids</s0>
<s5>10</s5>
</fC03>
<fC03 i1="06" i2="X" l="SPA"><s0>Lípido</s0>
<s5>10</s5>
</fC03>
<fC03 i1="07" i2="X" l="FRE"><s0>Prédiction</s0>
<s5>11</s5>
</fC03>
<fC03 i1="07" i2="X" l="ENG"><s0>Prediction</s0>
<s5>11</s5>
</fC03>
<fC03 i1="07" i2="X" l="SPA"><s0>Predicción</s0>
<s5>11</s5>
</fC03>
<fC03 i1="08" i2="X" l="FRE"><s0>Homme</s0>
<s5>12</s5>
</fC03>
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<s5>12</s5>
</fC03>
<fC03 i1="08" i2="X" l="SPA"><s0>Hombre</s0>
<s5>12</s5>
</fC03>
<fC03 i1="09" i2="X" l="FRE"><s0>Etude cas témoin</s0>
<s5>13</s5>
</fC03>
<fC03 i1="09" i2="X" l="ENG"><s0>Case control study</s0>
<s5>13</s5>
</fC03>
<fC03 i1="09" i2="X" l="SPA"><s0>Estudio caso control</s0>
<s5>13</s5>
</fC03>
<fC03 i1="10" i2="X" l="FRE"><s0>Hémorragie</s0>
<s5>14</s5>
</fC03>
<fC03 i1="10" i2="X" l="ENG"><s0>Hemorrhage</s0>
<s5>14</s5>
</fC03>
<fC03 i1="10" i2="X" l="SPA"><s0>Hemorragia</s0>
<s5>14</s5>
</fC03>
<fC03 i1="11" i2="X" l="FRE"><s0>Insuffisance cardiaque</s0>
<s5>15</s5>
</fC03>
<fC03 i1="11" i2="X" l="ENG"><s0>Heart failure</s0>
<s5>15</s5>
</fC03>
<fC03 i1="11" i2="X" l="SPA"><s0>Insuficiencia cardíaca</s0>
<s5>15</s5>
</fC03>
<fC03 i1="12" i2="X" l="FRE"><s0>Appareil circulatoire</s0>
<s5>17</s5>
</fC03>
<fC03 i1="12" i2="X" l="ENG"><s0>Circulatory system</s0>
<s5>17</s5>
</fC03>
<fC03 i1="12" i2="X" l="SPA"><s0>Aparato circulatorio</s0>
<s5>17</s5>
</fC03>
<fC03 i1="13" i2="X" l="FRE"><s0>Cardiologie</s0>
<s5>18</s5>
</fC03>
<fC03 i1="13" i2="X" l="ENG"><s0>Cardiology</s0>
<s5>18</s5>
</fC03>
<fC03 i1="13" i2="X" l="SPA"><s0>Cardiología</s0>
<s5>18</s5>
</fC03>
<fC03 i1="14" i2="X" l="FRE"><s0>Phlébologie</s0>
<s5>19</s5>
</fC03>
<fC03 i1="14" i2="X" l="ENG"><s0>Phlebology</s0>
<s5>19</s5>
</fC03>
<fC03 i1="14" i2="X" l="SPA"><s0>Flebología</s0>
<s5>19</s5>
</fC03>
<fC07 i1="01" i2="X" l="FRE"><s0>Appareil circulatoire pathologie</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="ENG"><s0>Cardiovascular disease</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="SPA"><s0>Aparato circulatorio patología</s0>
<s5>37</s5>
</fC07>
<fC07 i1="02" i2="X" l="FRE"><s0>Myocarde pathologie</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="ENG"><s0>Myocardial disease</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="SPA"><s0>Miocardio patología</s0>
<s5>38</s5>
</fC07>
<fC07 i1="03" i2="X" l="FRE"><s0>Encéphale pathologie</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="ENG"><s0>Cerebral disorder</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="SPA"><s0>Encéfalo patología</s0>
<s5>39</s5>
</fC07>
<fC07 i1="04" i2="X" l="FRE"><s0>Système nerveux central pathologie</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="ENG"><s0>Central nervous system disease</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="SPA"><s0>Sistema nervosio central patología</s0>
<s5>40</s5>
</fC07>
<fC07 i1="05" i2="X" l="FRE"><s0>Système nerveux pathologie</s0>
<s5>41</s5>
</fC07>
<fC07 i1="05" i2="X" l="ENG"><s0>Nervous system diseases</s0>
<s5>41</s5>
</fC07>
<fC07 i1="05" i2="X" l="SPA"><s0>Sistema nervioso patología</s0>
<s5>41</s5>
</fC07>
<fC07 i1="06" i2="X" l="FRE"><s0>Vaisseau sanguin pathologie</s0>
<s5>42</s5>
</fC07>
<fC07 i1="06" i2="X" l="ENG"><s0>Vascular disease</s0>
<s5>42</s5>
</fC07>
<fC07 i1="06" i2="X" l="SPA"><s0>Vaso sanguíneo patología</s0>
<s5>42</s5>
</fC07>
<fC07 i1="07" i2="X" l="FRE"><s0>Cardiopathie</s0>
<s5>43</s5>
</fC07>
<fC07 i1="07" i2="X" l="ENG"><s0>Heart disease</s0>
<s5>43</s5>
</fC07>
<fC07 i1="07" i2="X" l="SPA"><s0>Cardiopatía</s0>
<s5>43</s5>
</fC07>
<fN21><s1>283</s1>
</fN21>
<fN44 i1="01"><s1>OTO</s1>
</fN44>
<fN82><s1>OTO</s1>
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<server><NO>PASCAL 05-0404764 INIST</NO>
<ET>Plasma lipids predict myocardial infarction, but not stroke, in patients with established cerebrovascular disease</ET>
<AU>AMARENCO (Pierre); PATEL (Anushka); WOODWARD (Mark); CAMPBELL (Duncan J.); SULLIVAN (David R.); COLMAN (Samuel); CHALMERS (John); NEAL (Bruce); MACMAHON (Stephen)</AU>
<AF>Department of Neurology and Stroke Centre, Bichat Hospital and Medical School, Denis Diderot University/Paris/France (1 aut., 2 aut., 3 aut., 6 aut., 7 aut., 8 aut., 9 aut.); The George Institute for International Health, University of Sydney, PO Box M201, Missenden Road/Sydney, NSW 2050/Australie (4 aut.); St Vincent's Institute of Medical Research/Victoria/Australie (4 aut.); Department of Medicine, University of Melbourne/Victoria/Australie (4 aut.); Department of Clinical Biochemistry, Royal Prince Alfred Hospital/Sydney/Australie (5 aut.)</AF>
<DT>Publication en série; Article; Editorial; Niveau analytique</DT>
<SO>European heart journal; ISSN 0195-668X; Royaume-Uni; Da. 2005; Vol. 26; No. 18; 1818-1819,1910-1915 [8 p.]; Bibl. 33 ref.</SO>
<LA>Anglais</LA>
<EA>Aims To evaluate the role of plasma lipids in recurrent vascular events, including stroke, among individuals with established cerebrovascular disease. Methods and results Plasma total cholesterol, HDL cholesterol, and triglycerides were measured at baseline among individuals participating in the Perindopril Protection Against Recurrent Stroke (PROGRESS) study, a randomized clinical trial of blood pressure lowering among patients with previous stroke or transient ischaemic attack. A series of nested case-control studies were used to investigate the association between each of these lipid variables and the risk of subsequent haemorrhagic stroke, ischaemic stroke, myocardial infarction (MI), and heart failure. A total of 895 patients were selected as cases (83 haemorrhagic stroke, 472 ischaemic stroke, 206 MI, and 258 heart failure) and each was matched with one to three controls. After adjustment for other major cardiovascular risk factors, none of the lipid variables was associated with the risk of either stroke subtype. There were significant positive and negative associations for total cholesterol and HDL, respectively, with the risk of MI; the odds ratio comparing the highest and lowest thirds of each of these lipid variables was 2.00 (95% CI: 1.30-3.09) for total cholesterol and 0.58 (95% CI: 0.37-0.90) for HDL. HDL was inversely associated with the risk of heart failure; however, this result was of borderline statistical significance (P=0.05). Conclusion Lipid variables are associated with the risk of MI, but not recurrent stroke, in patients with established cerebrovascular disease.</EA>
<CC>002B12A03; 002B17C; 002B12A01</CC>
<FD>Infarctus myocarde; Accident cérébrovasculaire; Cérébrovasculaire pathologie; Ischémie; Plasma sanguin; Lipide; Prédiction; Homme; Etude cas témoin; Hémorragie; Insuffisance cardiaque; Appareil circulatoire; Cardiologie; Phlébologie</FD>
<FG>Appareil circulatoire pathologie; Myocarde pathologie; Encéphale pathologie; Système nerveux central pathologie; Système nerveux pathologie; Vaisseau sanguin pathologie; Cardiopathie</FG>
<ED>Myocardial infarction; Stroke; Cerebrovascular disease; Ischemia; Blood plasma; Lipids; Prediction; Human; Case control study; Hemorrhage; Heart failure; Circulatory system; Cardiology; Phlebology</ED>
<EG>Cardiovascular disease; Myocardial disease; Cerebral disorder; Central nervous system disease; Nervous system diseases; Vascular disease; Heart disease</EG>
<SD>Infarto miocardio; Accidente cerebrovascular; Vaso sanguíneo encéfalo patología; Isquemia; Plasma sanguíneo; Lípido; Predicción; Hombre; Estudio caso control; Hemorragia; Insuficiencia cardíaca; Aparato circulatorio; Cardiología; Flebología</SD>
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<ID>05-0404764</ID>
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   |area=    AustralieFrV1
   |flux=    PascalFrancis
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   |clé=     Pascal:05-0404764
   |texte=   Plasma lipids predict myocardial infarction, but not stroke, in patients with established cerebrovascular disease
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	Serveur d'exploration sur les relations entre la France et l'Australie
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Plasma lipids predict myocardial infarction, but not stroke, in patients with established cerebrovascular disease

Plasma lipids predict myocardial infarction, but not stroke, in patients with established cerebrovascular disease

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