High-intensity statin therapy alters the natural history of diabetic coronary atherosclerosis: insights from SATURN.
Identifieur interne : 001D73 ( Ncbi/Curation ); précédent : 001D72; suivant : 001D74High-intensity statin therapy alters the natural history of diabetic coronary atherosclerosis: insights from SATURN.
Auteurs : Brian Stegman [États-Unis] ; Rishi Puri [États-Unis] ; Leslie Cho [États-Unis] ; Mingyuan Shao [États-Unis] ; Christie M. Ballantyne [États-Unis] ; Phillip J. Barter [Australie] ; M John Chapman [France] ; Raimund Erbel [Allemagne] ; Peter Libby [États-Unis] ; Joel S. Raichlen [États-Unis] ; Kiyoko Uno [États-Unis] ; Yu Kataoka [Australie] ; Steven E. Nissen [États-Unis] ; Stephen J. Nicholls [Oman]Source :
- Diabetes care [ 1935-5548 ] ; 2014.
Descripteurs français
- KwdFr :
- Acides heptanoïques (usage thérapeutique), Adulte d'âge moyen, Atorvastatine de calcium, Cholestérol HDL (sang), Cholestérol LDL (sang), Diabète (traitement médicamenteux), Femelle, Fluorobenzènes (usage thérapeutique), Humains, Inhibiteurs de l'hydroxyméthylglutaryl-CoA réductase (usage thérapeutique), Maladie des artères coronaires (traitement médicamenteux), Mâle, Plaque d'athérosclérose (traitement médicamenteux), Plaque d'athérosclérose (étiologie), Pyrimidines (usage thérapeutique), Pyrroles (usage thérapeutique), Rosuvastatine de calcium, Sujet âgé, Sulfonamides (usage thérapeutique), Échographie interventionnelle.
- MESH :
- sang : Cholestérol HDL, Cholestérol LDL.
- traitement médicamenteux : Diabète, Maladie des artères coronaires, Plaque d'athérosclérose.
- usage thérapeutique : Acides heptanoïques, Fluorobenzènes, Inhibiteurs de l'hydroxyméthylglutaryl-CoA réductase, Pyrimidines, Pyrroles, Sulfonamides.
- étiologie : Plaque d'athérosclérose.
- Adulte d'âge moyen, Atorvastatine de calcium, Femelle, Humains, Mâle, Rosuvastatine de calcium, Sujet âgé, Échographie interventionnelle.
English descriptors
- KwdEn :
- Aged, Atorvastatin Calcium, Cholesterol, HDL (blood), Cholesterol, LDL (blood), Coronary Artery Disease (drug therapy), Diabetes Mellitus (drug therapy), Female, Fluorobenzenes (therapeutic use), Heptanoic Acids (therapeutic use), Humans, Hydroxymethylglutaryl-CoA Reductase Inhibitors (therapeutic use), Male, Middle Aged, Plaque, Atherosclerotic (drug therapy), Plaque, Atherosclerotic (etiology), Pyrimidines (therapeutic use), Pyrroles (therapeutic use), Rosuvastatin Calcium, Sulfonamides (therapeutic use), Ultrasonography, Interventional.
- MESH :
- chemical , blood : Cholesterol, HDL, Cholesterol, LDL.
- chemical , therapeutic use : Fluorobenzenes, Heptanoic Acids, Hydroxymethylglutaryl-CoA Reductase Inhibitors, Pyrimidines, Pyrroles, Sulfonamides.
- chemical : Atorvastatin Calcium, Rosuvastatin Calcium.
- drug therapy : Coronary Artery Disease, Diabetes Mellitus, Plaque, Atherosclerotic.
- etiology : Plaque, Atherosclerotic.
- Aged, Female, Humans, Male, Middle Aged, Ultrasonography, Interventional.
Abstract
Although statins can induce coronary atheroma regression, this benefit has yet to be demonstrated in diabetic individuals. We tested the hypothesis that high-intensity statin therapy may promote coronary atheroma regression in patients with diabetes.
DOI: 10.2337/dc14-1121
PubMed: 25190674
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pubmed:25190674Le document en format XML
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<author><name sortKey="Libby, Peter" sort="Libby, Peter" uniqKey="Libby P" first="Peter" last="Libby">Peter Libby</name>
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<author><name sortKey="Raichlen, Joel S" sort="Raichlen, Joel S" uniqKey="Raichlen J" first="Joel S" last="Raichlen">Joel S. Raichlen</name>
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<author><name sortKey="Uno, Kiyoko" sort="Uno, Kiyoko" uniqKey="Uno K" first="Kiyoko" last="Uno">Kiyoko Uno</name>
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<placeName><region type="state">Ohio</region>
</placeName>
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<author><name sortKey="Kataoka, Yu" sort="Kataoka, Yu" uniqKey="Kataoka Y" first="Yu" last="Kataoka">Yu Kataoka</name>
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<country xml:lang="fr">Australie</country>
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<author><name sortKey="Nissen, Steven E" sort="Nissen, Steven E" uniqKey="Nissen S" first="Steven E" last="Nissen">Steven E. Nissen</name>
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<term>Atorvastatin Calcium</term>
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<term>Cholesterol, LDL (blood)</term>
<term>Coronary Artery Disease (drug therapy)</term>
<term>Diabetes Mellitus (drug therapy)</term>
<term>Female</term>
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<term>Heptanoic Acids (therapeutic use)</term>
<term>Humans</term>
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<term>Male</term>
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<term>Plaque, Atherosclerotic (etiology)</term>
<term>Pyrimidines (therapeutic use)</term>
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<term>Rosuvastatin Calcium</term>
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<term>Ultrasonography, Interventional</term>
</keywords>
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<term>Adulte d'âge moyen</term>
<term>Atorvastatine de calcium</term>
<term>Cholestérol HDL (sang)</term>
<term>Cholestérol LDL (sang)</term>
<term>Diabète (traitement médicamenteux)</term>
<term>Femelle</term>
<term>Fluorobenzènes (usage thérapeutique)</term>
<term>Humains</term>
<term>Inhibiteurs de l'hydroxyméthylglutaryl-CoA réductase (usage thérapeutique)</term>
<term>Maladie des artères coronaires (traitement médicamenteux)</term>
<term>Mâle</term>
<term>Plaque d'athérosclérose (traitement médicamenteux)</term>
<term>Plaque d'athérosclérose (étiologie)</term>
<term>Pyrimidines (usage thérapeutique)</term>
<term>Pyrroles (usage thérapeutique)</term>
<term>Rosuvastatine de calcium</term>
<term>Sujet âgé</term>
<term>Sulfonamides (usage thérapeutique)</term>
<term>Échographie interventionnelle</term>
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<term>Cholesterol, LDL</term>
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<term>Heptanoic Acids</term>
<term>Hydroxymethylglutaryl-CoA Reductase Inhibitors</term>
<term>Pyrimidines</term>
<term>Pyrroles</term>
<term>Sulfonamides</term>
</keywords>
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<term>Rosuvastatin Calcium</term>
</keywords>
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<term>Diabetes Mellitus</term>
<term>Plaque, Atherosclerotic</term>
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<term>Male</term>
<term>Middle Aged</term>
<term>Ultrasonography, Interventional</term>
</keywords>
<keywords scheme="MESH" xml:lang="fr"><term>Adulte d'âge moyen</term>
<term>Atorvastatine de calcium</term>
<term>Femelle</term>
<term>Humains</term>
<term>Mâle</term>
<term>Rosuvastatine de calcium</term>
<term>Sujet âgé</term>
<term>Échographie interventionnelle</term>
</keywords>
</textClass>
</profileDesc>
</teiHeader>
<front><div type="abstract" xml:lang="en">Although statins can induce coronary atheroma regression, this benefit has yet to be demonstrated in diabetic individuals. We tested the hypothesis that high-intensity statin therapy may promote coronary atheroma regression in patients with diabetes.</div>
</front>
</TEI>
</record>
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