Interrelationships between the kinetics of VLDL subspecies and HDL catabolism in abdominal obesity: a multicenter tracer kinetic study.
Identifieur interne : 003780 ( Main/Curation ); précédent : 003779; suivant : 003781Interrelationships between the kinetics of VLDL subspecies and HDL catabolism in abdominal obesity: a multicenter tracer kinetic study.
Auteurs : Bruno Vergès [Finlande] ; Martin Adiels ; Jan Boren ; Peter Hugh Barrett ; Gerald F. Watts ; Dick Chan ; Laurence Duvillard ; Sanni Söderlund ; Niina Matikainen ; Juhani Kahri ; Isabelle Robin ; Marja-Riitta TaskinenSource :
- The Journal of clinical endocrinology and metabolism [ 1945-7197 ] ; 2014.
Descripteurs français
- KwdFr :
- MESH :
- métabolisme : Apolipoprotéines, Foie, Lipoprotéines HDL, Lipoprotéines VLDL, Obésité abdominale, Tissu adipeux.
- Adulte, Adulte d'âge moyen, Femelle, Humains, Mâle, Sujet âgé.
English descriptors
- KwdEn :
- MESH :
- chemical , metabolism : Apolipoproteins, Lipoproteins, HDL, Lipoproteins, VLDL.
- metabolism : Adipose Tissue, Liver, Obesity, Abdominal.
- Adult, Aged, Female, Humans, Male, Middle Aged.
Abstract
Low plasma high-density lipoprotein (HDL) cholesterol is a major abnormality in abdominal obesity. This relates due to accelerated HDL catabolism, but the underlying mechanism requires further elucidation. The relationships between HDL catabolism and other variables that may be modified in abdominal obesity, such as very low-density lipoprotein (VLDL) subspecies (VLDL1, VLDL2) kinetics, liver fat, or visceral adiposity, remain to be investigated.
DOI: 10.1210/jc.2014-2365
PubMed: 25077901
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pubmed:25077901Le document en format XML
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<term>Aged</term>
<term>Apolipoproteins (metabolism)</term>
<term>Female</term>
<term>Humans</term>
<term>Lipoproteins, HDL (metabolism)</term>
<term>Lipoproteins, VLDL (metabolism)</term>
<term>Liver (metabolism)</term>
<term>Male</term>
<term>Middle Aged</term>
<term>Obesity, Abdominal (metabolism)</term>
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<term>Adulte d'âge moyen</term>
<term>Apolipoprotéines (métabolisme)</term>
<term>Femelle</term>
<term>Foie (métabolisme)</term>
<term>Humains</term>
<term>Lipoprotéines HDL (métabolisme)</term>
<term>Lipoprotéines VLDL (métabolisme)</term>
<term>Mâle</term>
<term>Obésité abdominale (métabolisme)</term>
<term>Sujet âgé</term>
<term>Tissu adipeux (métabolisme)</term>
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<keywords scheme="MESH" type="chemical" qualifier="metabolism" xml:lang="en"><term>Apolipoproteins</term>
<term>Lipoproteins, HDL</term>
<term>Lipoproteins, VLDL</term>
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<term>Liver</term>
<term>Obesity, Abdominal</term>
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<keywords scheme="MESH" qualifier="métabolisme" xml:lang="fr"><term>Apolipoprotéines</term>
<term>Foie</term>
<term>Lipoprotéines HDL</term>
<term>Lipoprotéines VLDL</term>
<term>Obésité abdominale</term>
<term>Tissu adipeux</term>
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<term>Aged</term>
<term>Female</term>
<term>Humans</term>
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<term>Middle Aged</term>
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<term>Adulte d'âge moyen</term>
<term>Femelle</term>
<term>Humains</term>
<term>Mâle</term>
<term>Sujet âgé</term>
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<front><div type="abstract" xml:lang="en">Low plasma high-density lipoprotein (HDL) cholesterol is a major abnormality in abdominal obesity. This relates due to accelerated HDL catabolism, but the underlying mechanism requires further elucidation. The relationships between HDL catabolism and other variables that may be modified in abdominal obesity, such as very low-density lipoprotein (VLDL) subspecies (VLDL1, VLDL2) kinetics, liver fat, or visceral adiposity, remain to be investigated.</div>
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