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Does diabetes mellitus‐induced bladder remodeling affect lower urinary tract function?: ICI‐RS 2011

Identifieur interne : 008323 ( Main/Merge ); précédent : 008322; suivant : 008324

Does diabetes mellitus‐induced bladder remodeling affect lower urinary tract function?: ICI‐RS 2011

Auteurs : Ruth Kirschner-Hermanns [Allemagne] ; Firouz Daneshgari ; Bahareh Vahabi [Royaume-Uni] ; Lori Birder [États-Unis] ; Matthias Oelke [Allemagne] ; Samuel Chacko

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RBID : ISTEX:8700A55EE88A1ECF62174D4C68E271F38BCF851C

Descripteurs français

English descriptors

Abstract

Due to an increase in aging population and changing eating habits diabetes mellitus (DM) type II is a rapidly increasing condition worldwide. Although not so detrimental as other co‐morbidities, uropathy contributes to a significantly reduced quality‐of‐life in those affected. The purpose of this ICS‐RS report is to highlight clinical and basic research data to outline directions for further research and possible treatment approaches.

Url:
DOI: 10.1002/nau.22228

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ISTEX:8700A55EE88A1ECF62174D4C68E271F38BCF851C

Le document en format XML

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<name sortKey="Daneshgari, Firouz" sort="Daneshgari, Firouz" uniqKey="Daneshgari F" first="Firouz" last="Daneshgari">Firouz Daneshgari</name>
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<title level="j" type="main">Neurourology and Urodynamics</title>
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<term>Aldose</term>
<term>Aldose reductase</term>
<term>Animal models</term>
<term>Asymptomatic bacteriuria</term>
<term>Benign prostatic hyperplasia</term>
<term>Bladder</term>
<term>Bladder capacity</term>
<term>Bladder dysfunction</term>
<term>Bladder sensation</term>
<term>Boopathi chacko</term>
<term>Complication</term>
<term>Contractility</term>
<term>Cystopathy</term>
<term>Detrusor</term>
<term>Detrusor muscle</term>
<term>Detrusor overactivity</term>
<term>Detrusor underactivity</term>
<term>Diabetes</term>
<term>Diabetes mellitus</term>
<term>Diabetic</term>
<term>Diabetic bladder</term>
<term>Diabetic bladder dysfunction</term>
<term>Diabetic complications</term>
<term>Diabetic cystopathy</term>
<term>Diabetic patients</term>
<term>Diabetic rabbits</term>
<term>Diabetic uropathy</term>
<term>Dysfunction</term>
<term>Efferent pathways</term>
<term>Force generation</term>
<term>Glucose</term>
<term>High glucose increases</term>
<term>Hyperglycemia</term>
<term>Incontinence</term>
<term>Lipid</term>
<term>Lipid peroxidation</term>
<term>Luts</term>
<term>Mellitus</term>
<term>Metabolic syndrome</term>
<term>Microvascular damage</term>
<term>Mitochondrial</term>
<term>Mitochondrial dysfunction</term>
<term>Muscle cells</term>
<term>Ndings</term>
<term>Nerve growth factor</term>
<term>Neuropathy</term>
<term>Neurourol</term>
<term>Neurourol urodyn</term>
<term>Overactive bladder</term>
<term>Overactivity</term>
<term>Oxidative</term>
<term>Oxidative stress</term>
<term>Pathogenesis</term>
<term>Pathway</term>
<term>Peroxidation</term>
<term>Polyol pathway</term>
<term>Reactive oxygen species</term>
<term>Reductase</term>
<term>Tract function</term>
<term>Tract symptoms</term>
<term>Unpublished data</term>
<term>Urinary</term>
<term>Urinary incontinence</term>
<term>Urinary tract infection</term>
<term>Urodyn</term>
<term>Urodynamic</term>
<term>Urol</term>
<term>Urothelium</term>
<term>Voiding</term>
<term>Voiding dysfunction</term>
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<term>Aldose</term>
<term>Aldose reductase</term>
<term>Animal models</term>
<term>Asymptomatic bacteriuria</term>
<term>Benign prostatic hyperplasia</term>
<term>Bladder</term>
<term>Bladder capacity</term>
<term>Bladder dysfunction</term>
<term>Bladder sensation</term>
<term>Boopathi chacko</term>
<term>Complication</term>
<term>Contractility</term>
<term>Cystopathy</term>
<term>Detrusor</term>
<term>Detrusor muscle</term>
<term>Detrusor overactivity</term>
<term>Detrusor underactivity</term>
<term>Diabetes</term>
<term>Diabetes mellitus</term>
<term>Diabetic</term>
<term>Diabetic bladder</term>
<term>Diabetic bladder dysfunction</term>
<term>Diabetic complications</term>
<term>Diabetic cystopathy</term>
<term>Diabetic patients</term>
<term>Diabetic rabbits</term>
<term>Diabetic uropathy</term>
<term>Dysfunction</term>
<term>Efferent pathways</term>
<term>Force generation</term>
<term>Glucose</term>
<term>High glucose increases</term>
<term>Hyperglycemia</term>
<term>Incontinence</term>
<term>Lipid</term>
<term>Lipid peroxidation</term>
<term>Luts</term>
<term>Mellitus</term>
<term>Metabolic syndrome</term>
<term>Microvascular damage</term>
<term>Mitochondrial</term>
<term>Mitochondrial dysfunction</term>
<term>Muscle cells</term>
<term>Ndings</term>
<term>Nerve growth factor</term>
<term>Neuropathy</term>
<term>Neurourol</term>
<term>Neurourol urodyn</term>
<term>Overactive bladder</term>
<term>Overactivity</term>
<term>Oxidative</term>
<term>Oxidative stress</term>
<term>Pathogenesis</term>
<term>Pathway</term>
<term>Peroxidation</term>
<term>Polyol pathway</term>
<term>Reactive oxygen species</term>
<term>Reductase</term>
<term>Tract function</term>
<term>Tract symptoms</term>
<term>Unpublished data</term>
<term>Urinary</term>
<term>Urinary incontinence</term>
<term>Urinary tract infection</term>
<term>Urodyn</term>
<term>Urodynamic</term>
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<term>Urothelium</term>
<term>Voiding</term>
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<front>
<div type="abstract">Due to an increase in aging population and changing eating habits diabetes mellitus (DM) type II is a rapidly increasing condition worldwide. Although not so detrimental as other co‐morbidities, uropathy contributes to a significantly reduced quality‐of‐life in those affected. The purpose of this ICS‐RS report is to highlight clinical and basic research data to outline directions for further research and possible treatment approaches.</div>
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