List of bibliographic references
Number of relevant bibliographic references: 27.
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Ident. | Authors (with country if any) | Title |
---|
000131 (2016) |
Naomi P. Visanji [Canada] ; Jonathan M. Brotchie [Canada] ; Lorraine V. Kalia [Canada] ; James B. Koprich [Canada] ; Anurag Tandon [Canada] ; Joel C. Watts [Canada] ; Anthony E. Lang [Canada] | α-Synuclein-Based Animal Models of Parkinson's Disease: Challenges and Opportunities in a New Era. |
000241 (2016) |
Qing He [République populaire de Chine] ; James B. Koprich [Canada] ; Ying Wang [République populaire de Chine] ; Wen-Bo Yu [République populaire de Chine] ; Bao-Guo Xiao [République populaire de Chine] ; Jonathan M. Brotchie [Canada] ; Jian Wang [République populaire de Chine] | Treatment with Trehalose Prevents Behavioral and Neurochemical Deficits Produced in an AAV α-Synuclein Rat Model of Parkinson's Disease. |
000455 (2015) |
Yilong Ma [États-Unis] ; Tom H. Johnston [Canada] ; Shichun Peng [États-Unis] ; Chuantao Zuo [République populaire de Chine] ; James B. Koprich [Canada] ; Susan H. Fox [Canada] ; Yihui Guan [République populaire de Chine] ; David Eidelberg [États-Unis] ; Jonathan M. Brotchie [Canada] | Reproducibility of a Parkinsonism-related metabolic brain network in non-human primates: A descriptive pilot study with FDG PET. |
000553 (2015) |
Sujoy Phookan [États-Unis] ; Alexander C. Sutton [États-Unis] ; Ian Walling [États-Unis] ; Autumn Smith [États-Unis] ; Katherine A. O'Connor [États-Unis] ; Joannalee C. Campbell [États-Unis] ; Megan Calos [États-Unis] ; Wilson Yu [États-Unis] ; Julie G. Pilitsis [États-Unis] ; Jonathan M. Brotchie [Canada] ; Damian S. Shin [États-Unis] | Gap junction blockers attenuate beta oscillations and improve forelimb function in hemiparkinsonian rats. |
000653 (2014) |
Sherri L. Thiele [Canada] ; Betty Chen [Canada] ; Charlotte Lo [Canada] ; Tracey S. Gertler [États-Unis] ; Ruth Warre [Canada] ; James D. Surmeier [États-Unis] ; Jonathan M. Brotchie [Canada] ; Joanne E. Nash [Canada] | Selective loss of bi-directional synaptic plasticity in the direct and indirect striatal output pathways accompanies generation of parkinsonism and l-DOPA induced dyskinesia in mouse models. |
000727 (2014) |
Philippe Huot [Canada] ; Tom H. Johnston [Canada] ; Katie D. Lewis [Australie] ; James B. Koprich [Canada] ; M Gabriela Reyes [Canada] ; Susan H. Fox [Canada] ; Matthew J. Piggott [Australie] ; Jonathan M. Brotchie [Canada] | UWA-121, a mixed dopamine and serotonin re-uptake inhibitor, enhances L-DOPA anti-parkinsonian action without worsening dyskinesia or psychosis-like behaviours in the MPTP-lesioned common marmoset. |
000905 (2013) |
James B. Koprich [Canada] ; Philippe Huot ; Susan H. Fox ; Keith Jarvie ; Anthony E. Lang ; Philip Seeman ; Jonathan M. Brotchie | The effects of fast-off-D2 receptor antagonism on L-DOPA-induced dyskinesia and psychosis in parkinsonian macaques. |
000959 (2013) |
Lorraine V. Kalia [Canada] ; Jonathan M. Brotchie ; Susan H. Fox | Novel nondopaminergic targets for motor features of Parkinson's disease: review of recent trials. |
000968 (2013) |
Philippe Huot [Canada] ; Tom H. Johnston ; James B. Koprich ; Susan H. Fox ; Jonathan M. Brotchie | The pharmacology of L-DOPA-induced dyskinesia in Parkinson's disease. |
000A39 (2012) |
Philippe Huot [Canada] ; Tom H. Johnston ; James B. Koprich ; Ahmed Aman ; Susan H. Fox ; Jonathan M. Brotchie | L-745,870 reduces L-DOPA-induced dyskinesia in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-lesioned macaque model of Parkinson's disease. |
000A71 (2012) |
Tom H. Johnston [Canada] ; Zak Millar ; Philippe Huot ; Keith Wagg ; Sherri Thiele ; Danielle Salomonczyk ; Christopher J. Yong-Kee ; Michael N. Gandy ; Matthew Mcildowie ; Katie D. Lewis ; Jordi Gomez-Ramirez ; Joohyung Lee ; Susan H. Fox ; Mathew Martin-Iverson ; Joanne E. Nash ; Matthew J. Piggott ; Jonathan M. Brotchie | A novel MDMA analogue, UWA-101, that lacks psychoactivity and cytotoxicity, enhances L-DOPA benefit in parkinsonian primates. |
000A75 (2012) |
Philippe Huot [Canada] ; Tom H. Johnston ; Naomi P. Visanji ; Tayyeba Darr ; Donna Pires ; Lili-Naz Hazrati ; Jonathan M. Brotchie ; Susan H. Fox | Increased levels of 5-HT1A receptor binding in ventral visual pathways in Parkinson's disease. |
000B70 (2011) |
Philippe Huot [Canada] ; Jonathan M. Brotchie | 5-HT(1A) receptor stimulation and L-DOPA-induced dyskinesia in Parkinson's disease: bridging the gap between serotonergic and glutamatergic mechanisms. |
000B73 (2011) |
Philippe Huot [Canada] ; Susan H. Fox ; Adrian Newman-Tancredi ; Jonathan M. Brotchie | Anatomically selective serotonergic type 1A and serotonergic type 2A therapies for Parkinson's disease: an approach to reducing dyskinesia without exacerbating parkinsonism? |
000B76 (2011) |
Philippe Huot [Canada] ; Susan H. Fox ; Jonathan M. Brotchie | The serotonergic system in Parkinson's disease. |
000C35 (2011) |
James B. Koprich [Canada] ; Tom H. Johnston ; Philippe Huot ; M Gabriela Reyes ; Maria Espinosa ; Jonathan M. Brotchie | Progressive neurodegeneration or endogenous compensation in an animal model of Parkinson's disease produced by decreasing doses of alpha-synuclein. |
000D45 (2010) |
Tom H. Johnston [Canada] ; Susan H. Fox ; Matthew J. Mcildowie ; Matthew J. Piggott ; Jonathan M. Brotchie | Reduction of L-DOPA-induced dyskinesia by the selective metabotropic glutamate receptor 5 antagonist 3-[(2-methyl-1,3-thiazol-4-yl)ethynyl]pyridine in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-lesioned macaque model of Parkinson's disease. |
000D99 (2010) |
Susan H. Fox [Canada] ; Jonathan M. Brotchie | The MPTP-lesioned non-human primate models of Parkinson's disease. Past, present, and future. |
000E54 (2009) |
Susan H. Fox [Canada] ; Rosalind Chuang ; Jonathan M. Brotchie | Serotonin and Parkinson's disease: On movement, mood, and madness. |
000F44 (2008) |
Susan H. Fox [Canada] ; Jonathan M. Brotchie ; Anthony E. Lang | Non-dopaminergic treatments in development for Parkinson's disease. |
001029 (2008) |
Susan H. Fox [Canada] ; Rosalind Chuang ; Jonathan M. Brotchie | Parkinson's disease--opportunities for novel therapeutics to reduce the problems of levodopa therapy. |
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