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<record><TEI><teiHeader><fileDesc><titleStmt><title xml:lang="en">Macroautophagy</title><author><name sortKey="Hannigan, Adrienne M" sort="Hannigan, Adrienne M" uniqKey="Hannigan A" first="Adrienne M." last="Hannigan">Adrienne M. Hannigan</name><affiliation><nlm:aff id="A1"> The Genome Sciences Centre; British Columbia Cancer Agency; Vancouver, British Columbia, Canada</nlm:aff></affiliation></author><author><name sortKey="Gorski, Sharon M" sort="Gorski, Sharon M" uniqKey="Gorski S" first="Sharon M." last="Gorski">Sharon M. Gorski</name><affiliation><nlm:aff id="A1"> The Genome Sciences Centre; British Columbia Cancer Agency; Vancouver, British Columbia, Canada</nlm:aff></affiliation><affiliation><nlm:aff id="A2"> Department of Molecular Biology and Biochemistry; Simon Fraser University; Burnaby, British Columbia, Canada</nlm:aff></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">PMC</idno><idno type="pmid">19098459</idno><idno type="pmc">3135626</idno><idno type="url">http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3135626</idno><idno type="RBID">PMC:3135626</idno><date when="2009">2009</date><idno type="wicri:Area/Pmc/Corpus">000406</idno><idno type="wicri:explorRef" wicri:stream="Pmc" wicri:step="Corpus" wicri:corpus="PMC">000406</idno></publicationStmt><sourceDesc><biblStruct><analytic><title xml:lang="en" level="a" type="main">Macroautophagy</title><author><name sortKey="Hannigan, Adrienne M" sort="Hannigan, Adrienne M" uniqKey="Hannigan A" first="Adrienne M." last="Hannigan">Adrienne M. Hannigan</name><affiliation><nlm:aff id="A1"> The Genome Sciences Centre; British Columbia Cancer Agency; Vancouver, British Columbia, Canada</nlm:aff></affiliation></author><author><name sortKey="Gorski, Sharon M" sort="Gorski, Sharon M" uniqKey="Gorski S" first="Sharon M." last="Gorski">Sharon M. Gorski</name><affiliation><nlm:aff id="A1"> The Genome Sciences Centre; British Columbia Cancer Agency; Vancouver, British Columbia, Canada</nlm:aff></affiliation><affiliation><nlm:aff id="A2"> Department of Molecular Biology and Biochemistry; Simon Fraser University; Burnaby, British Columbia, Canada</nlm:aff></affiliation></author></analytic><series><title level="j">Autophagy</title><idno type="ISSN">1554-8627</idno><idno type="eISSN">1554-8635</idno><imprint><date when="2009">2009</date></imprint></series></biblStruct></sourceDesc></fileDesc><profileDesc><textClass></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en"><p id="P1">Dietary compounds can influence the risk of cancer and other diseases through diverse mechanisms which include the activation or inhibition of macroautophagy. Macroautophagy is a catabolic process for the lysosomal degradation and recycling of cytoplasmic constituents which has been implicated in several pathologies, including cancer and neurodegeneration. In some instances, macroautophagy acts to suppress tumor formation and neural degeneration. Thus, it may be feasible to design diets, supplements or therapeutics that can alter the level of macroautophagy within cells to prevent or treat disease. While critical questions still need to be answered before we can safely and effectively implement such a strategy, we provide here a review of the literature regarding dietary constituents that have a demonstrated macroautophagy-modulating function.</p></div></front></TEI><pmc article-type="research-article" xml:lang="EN"><pmc-comment>The publisher of this article does not allow downloading of the full text in XML form.</pmc-comment>
<pmc-dir>properties manuscript</pmc-dir>
<front><journal-meta><journal-id journal-id-type="nlm-journal-id">101265188</journal-id><journal-id journal-id-type="pubmed-jr-id">33022</journal-id><journal-id journal-id-type="nlm-ta">Autophagy</journal-id><journal-title>Autophagy</journal-title><issn pub-type="ppub">1554-8627</issn><issn pub-type="epub">1554-8635</issn></journal-meta><article-meta><article-id pub-id-type="pmid">19098459</article-id><article-id pub-id-type="pmc">3135626</article-id><article-id pub-id-type="manuscript">CAMS1839</article-id><article-categories><subj-group subj-group-type="heading"><subject>Article</subject></subj-group></article-categories><title-group><article-title>Macroautophagy</article-title><subtitle>The key ingredient to a healthy diet?</subtitle></title-group><contrib-group><contrib contrib-type="author"><name><surname>Hannigan</surname><given-names>Adrienne M.</given-names></name><xref rid="A1" ref-type="aff">1</xref></contrib><contrib contrib-type="author"><name><surname>Gorski</surname><given-names>Sharon M.</given-names></name><xref rid="A1" ref-type="aff">1</xref><xref rid="A2" ref-type="aff">2</xref><xref rid="FN1" ref-type="author-notes">*</xref></contrib></contrib-group><aff id="A1"><label>1</label> The Genome Sciences Centre; British Columbia Cancer Agency; Vancouver, British Columbia, Canada</aff><aff id="A2"><label>2</label> Department of Molecular Biology and Biochemistry; Simon Fraser University; Burnaby, British Columbia, Canada</aff><author-notes><corresp id="FN1"><label>*</label>Correspondence to: Sharon M. Gorski; The Genome Sciences Centre; BC Cancer Research Centre; 675 West 10th Avenue; Vancouver, British Columbia V5Z 1L3 Canada; Tel.: 604.675.8113; Fax: 604.675.8178; <email>sgorski@bcgsc.ca</email></corresp></author-notes><pub-date pub-type="nihms-submitted"><day>6</day><month>7</month><year>2011</year></pub-date><pub-date pub-type="epub"><day>3</day><month>2</month><year>2009</year></pub-date><pub-date pub-type="ppub"><month>2</month><year>2009</year></pub-date><pub-date pub-type="pmc-release"><day>13</day><month>7</month><year>2011</year></pub-date><volume>5</volume><issue>2</issue><fpage>140</fpage><lpage>151</lpage><abstract><p id="P1">Dietary compounds can influence the risk of cancer and other diseases through diverse mechanisms which include the activation or inhibition of macroautophagy. Macroautophagy is a catabolic process for the lysosomal degradation and recycling of cytoplasmic constituents which has been implicated in several pathologies, including cancer and neurodegeneration. In some instances, macroautophagy acts to suppress tumor formation and neural degeneration. Thus, it may be feasible to design diets, supplements or therapeutics that can alter the level of macroautophagy within cells to prevent or treat disease. While critical questions still need to be answered before we can safely and effectively implement such a strategy, we provide here a review of the literature regarding dietary constituents that have a demonstrated macroautophagy-modulating function.</p></abstract><kwd-group><kwd>macroautophagy</kwd><kwd>autophagy</kwd><kwd>diet</kwd><kwd>nutrition</kwd><kwd>natural compounds</kwd><kwd>LC3</kwd><kwd>cancer</kwd><kwd>prevention</kwd></kwd-group><contract-num rid="GG5">78882
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