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La maladie de Parkinson au Canada (serveur d'exploration)

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Behavior and cognition in corticobasal degeneration and progressive supranuclear palsy

Identifieur interne : 000458 ( PascalFrancis/Corpus ); précédent : 000457; suivant : 000459

Behavior and cognition in corticobasal degeneration and progressive supranuclear palsy

Auteurs : Andrew Kertesz ; Paul Mcmonagle

Source :

RBID : Pascal:10-0131742

Descripteurs français

English descriptors

Abstract

Progressive supranuclear palsy (PSP) and corticobasal degeneration (CBD), previously described as Parkinsonian syndromes are also cognitive disorders, and biologically related to the frontotemporal dementia or Pick's disease. PSP and CBD overlap clinically, pathologically and genetically, sharing tau haplotypes and mutations. In our series of CBD/PSP patients with cognitive presentation (n=36), primary progressive aphasia (PPA) was particularly common, but behavioral onset occurred also. CBD or PSP as motor presentations developed significant language disorder in 17/19. The underlying pathology is predictably tau positive in these clinical combinations, regardless of the presentation. Other cognitive features of CBDS include apraxia, alien hand and apathy, but often frontal lobe dementia with disinhibition develops also. CBDS also has visuospatial deficit, because of the parietal involvement. PSP was considered the prototype of subcortical dementia, with bradyphrenia, poor recall and executive deficit, but cortical features were recognized to be important also. Language testing and a behavioral inventory should be part of neuropsychological tests to facilitate diagnosis and to quantify the deficit. The clinical, genetic and pathological relationship is strong between CBD /PSP and the aphasic and behavioral components of the Pick complex.

Notice en format standard (ISO 2709)

Pour connaître la documentation sur le format Inist Standard.

pA  
A01 01  1    @0 0022-510X
A02 01      @0 JNSCAG
A03   1    @0 J. neurol. sci.
A05       @2 289
A06       @2 1-2
A08 01  1  ENG  @1 Behavior and cognition in corticobasal degeneration and progressive supranuclear palsy
A09 01  1  ENG  @1 Mental Dysfunction in Parkinson's Disease: Proceedings of the 9th International Congress on Mental Dysfunction and other Non-Motor Features in Parkinson's Disease and Related Disorders, Dresden, Germany, 16-19 October 2008
A11 01  1    @1 KERTESZ (Andrew)
A11 02  1    @1 MCMONAGLE (Paul)
A12 01  1    @1 REICHMANN (Heinz) @9 ed.
A12 02  1    @1 KORCZYN (Amos D.) @9 ed.
A14 01      @1 Dept of Cognitive Neurology, University of Western Ontario, St Joseph's Hospital, 268 Grosvenor St @2 London, Ontario, N6A 4V2 @3 CAN @Z 1 aut.
A14 02      @1 Dept of Neurology, The Royal Victoria Hospital, 274 Grosvenor Road @2 Belfast BT12 6BA @3 GBR @Z 2 aut.
A15 01      @1 Department of Neurology, Dresden University Medical School @2 Dresden @3 DEU @Z 1 aut.
A15 02      @1 Department of Neurology, Tel Aviv University Medical School @2 Ramat Aviv @3 ISR @Z 2 aut.
A20       @1 138-143
A21       @1 2010
A23 01      @0 ENG
A43 01      @1 INIST @2 12185 @5 354000190042590260
A44       @0 0000 @1 © 2010 INIST-CNRS. All rights reserved.
A45       @0 104 ref.
A47 01  1    @0 10-0131742
A60       @1 P @2 C
A61       @0 A
A64 01  1    @0 Journal of the neurological sciences
A66 01      @0 IRL
C01 01    ENG  @0 Progressive supranuclear palsy (PSP) and corticobasal degeneration (CBD), previously described as Parkinsonian syndromes are also cognitive disorders, and biologically related to the frontotemporal dementia or Pick's disease. PSP and CBD overlap clinically, pathologically and genetically, sharing tau haplotypes and mutations. In our series of CBD/PSP patients with cognitive presentation (n=36), primary progressive aphasia (PPA) was particularly common, but behavioral onset occurred also. CBD or PSP as motor presentations developed significant language disorder in 17/19. The underlying pathology is predictably tau positive in these clinical combinations, regardless of the presentation. Other cognitive features of CBDS include apraxia, alien hand and apathy, but often frontal lobe dementia with disinhibition develops also. CBDS also has visuospatial deficit, because of the parietal involvement. PSP was considered the prototype of subcortical dementia, with bradyphrenia, poor recall and executive deficit, but cortical features were recognized to be important also. Language testing and a behavioral inventory should be part of neuropsychological tests to facilitate diagnosis and to quantify the deficit. The clinical, genetic and pathological relationship is strong between CBD /PSP and the aphasic and behavioral components of the Pick complex.
C02 01  X    @0 002B17
C02 02  X    @0 002B17G
C03 01  X  FRE  @0 Dégénérescence @5 01
C03 01  X  ENG  @0 Degeneration @5 01
C03 01  X  SPA  @0 Degeneración @5 01
C03 02  X  FRE  @0 Démence frontotemporale @2 NM @5 02
C03 02  X  ENG  @0 Frontotemporal dementia @2 NM @5 02
C03 02  X  SPA  @0 Demencia frontotemporal @2 NM @5 02
C03 03  X  FRE  @0 Aphasie @5 03
C03 03  X  ENG  @0 Aphasia @5 03
C03 03  X  SPA  @0 Afasia @5 03
C03 04  X  FRE  @0 Apraxie @5 04
C03 04  X  ENG  @0 Apraxia @5 04
C03 04  X  SPA  @0 Apraxia @5 04
C03 05  X  FRE  @0 Pathologie du système nerveux @5 05
C03 05  X  ENG  @0 Nervous system diseases @5 05
C03 05  X  SPA  @0 Sistema nervioso patología @5 05
C03 06  X  FRE  @0 Comportement @5 09
C03 06  X  ENG  @0 Behavior @5 09
C03 06  X  SPA  @0 Conducta @5 09
C03 07  X  FRE  @0 Cognition @5 10
C03 07  X  ENG  @0 Cognition @5 10
C03 07  X  SPA  @0 Cognición @5 10
C03 08  X  FRE  @0 Ophtalmoplégie supranucléaire @5 11
C03 08  X  ENG  @0 Supranuclear ophthalmoplegia @5 11
C03 08  X  SPA  @0 Oftalmoplejía supranuclear @5 11
C07 01  X  FRE  @0 Pathologie de l'encéphale @5 37
C07 01  X  ENG  @0 Cerebral disorder @5 37
C07 01  X  SPA  @0 Encéfalo patología @5 37
C07 02  X  FRE  @0 Maladie dégénérative @5 38
C07 02  X  ENG  @0 Degenerative disease @5 38
C07 02  X  SPA  @0 Enfermedad degenerativa @5 38
C07 03  X  FRE  @0 Syndrome oculomoteur @5 39
C07 03  X  ENG  @0 Oculomotor syndrome @5 39
C07 03  X  SPA  @0 Oculomotricidad síndrome @5 39
C07 04  X  FRE  @0 Pathologie de l'oeil @5 40
C07 04  X  ENG  @0 Eye disease @5 40
C07 04  X  SPA  @0 Ojo patología @5 40
C07 05  X  FRE  @0 Pathologie du système nerveux central @5 41
C07 05  X  ENG  @0 Central nervous system disease @5 41
C07 05  X  SPA  @0 Sistema nervosio central patología @5 41
C07 06  X  FRE  @0 Syndrome du tronc cérébral @5 43
C07 06  X  ENG  @0 Brain stem syndrome @5 43
C07 06  X  SPA  @0 Tallo encefalico sindrome @5 43
C07 07  X  FRE  @0 Trouble de la communication @5 44
C07 07  X  ENG  @0 Communication disorder @5 44
C07 07  X  SPA  @0 Trastorno comunicación @5 44
C07 08  X  FRE  @0 Trouble du langage @5 45
C07 08  X  ENG  @0 Language disorder @5 45
C07 08  X  SPA  @0 Trastorno lenguaje @5 45
C07 09  X  FRE  @0 Trouble neurologique @5 46
C07 09  X  ENG  @0 Neurological disorder @5 46
C07 09  X  SPA  @0 Trastorno neurológico @5 46
N21       @1 081
N44 01      @1 OTO
N82       @1 OTO
pR  
A30 01  1  ENG  @1 International Congress on Mental Dysfunction and other non-motor features in Parkinson's Disease and Related Disorders @2 9 @3 Dresden DEU @4 2008-10-16

Format Inist (serveur)

NO : PASCAL 10-0131742 INIST
ET : Behavior and cognition in corticobasal degeneration and progressive supranuclear palsy
AU : KERTESZ (Andrew); MCMONAGLE (Paul); REICHMANN (Heinz); KORCZYN (Amos D.)
AF : Dept of Cognitive Neurology, University of Western Ontario, St Joseph's Hospital, 268 Grosvenor St/London, Ontario, N6A 4V2/Canada (1 aut.); Dept of Neurology, The Royal Victoria Hospital, 274 Grosvenor Road/Belfast BT12 6BA/Royaume-Uni (2 aut.); Department of Neurology, Dresden University Medical School/Dresden/Allemagne (1 aut.); Department of Neurology, Tel Aviv University Medical School/Ramat Aviv/Israël (2 aut.)
DT : Publication en série; Congrès; Niveau analytique
SO : Journal of the neurological sciences; ISSN 0022-510X; Coden JNSCAG; Irlande; Da. 2010; Vol. 289; No. 1-2; Pp. 138-143; Bibl. 104 ref.
LA : Anglais
EA : Progressive supranuclear palsy (PSP) and corticobasal degeneration (CBD), previously described as Parkinsonian syndromes are also cognitive disorders, and biologically related to the frontotemporal dementia or Pick's disease. PSP and CBD overlap clinically, pathologically and genetically, sharing tau haplotypes and mutations. In our series of CBD/PSP patients with cognitive presentation (n=36), primary progressive aphasia (PPA) was particularly common, but behavioral onset occurred also. CBD or PSP as motor presentations developed significant language disorder in 17/19. The underlying pathology is predictably tau positive in these clinical combinations, regardless of the presentation. Other cognitive features of CBDS include apraxia, alien hand and apathy, but often frontal lobe dementia with disinhibition develops also. CBDS also has visuospatial deficit, because of the parietal involvement. PSP was considered the prototype of subcortical dementia, with bradyphrenia, poor recall and executive deficit, but cortical features were recognized to be important also. Language testing and a behavioral inventory should be part of neuropsychological tests to facilitate diagnosis and to quantify the deficit. The clinical, genetic and pathological relationship is strong between CBD /PSP and the aphasic and behavioral components of the Pick complex.
CC : 002B17; 002B17G
FD : Dégénérescence; Démence frontotemporale; Aphasie; Apraxie; Pathologie du système nerveux; Comportement; Cognition; Ophtalmoplégie supranucléaire
FG : Pathologie de l'encéphale; Maladie dégénérative; Syndrome oculomoteur; Pathologie de l'oeil; Pathologie du système nerveux central; Syndrome du tronc cérébral; Trouble de la communication; Trouble du langage; Trouble neurologique
ED : Degeneration; Frontotemporal dementia; Aphasia; Apraxia; Nervous system diseases; Behavior; Cognition; Supranuclear ophthalmoplegia
EG : Cerebral disorder; Degenerative disease; Oculomotor syndrome; Eye disease; Central nervous system disease; Brain stem syndrome; Communication disorder; Language disorder; Neurological disorder
SD : Degeneración; Demencia frontotemporal; Afasia; Apraxia; Sistema nervioso patología; Conducta; Cognición; Oftalmoplejía supranuclear
LO : INIST-12185.354000190042590260
ID : 10-0131742

Links to Exploration step

Pascal:10-0131742

Le document en format XML

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<fC03 i1="08" i2="X" l="SPA">
<s0>Oftalmoplejía supranuclear</s0>
<s5>11</s5>
</fC03>
<fC07 i1="01" i2="X" l="FRE">
<s0>Pathologie de l'encéphale</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="ENG">
<s0>Cerebral disorder</s0>
<s5>37</s5>
</fC07>
<fC07 i1="01" i2="X" l="SPA">
<s0>Encéfalo patología</s0>
<s5>37</s5>
</fC07>
<fC07 i1="02" i2="X" l="FRE">
<s0>Maladie dégénérative</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="ENG">
<s0>Degenerative disease</s0>
<s5>38</s5>
</fC07>
<fC07 i1="02" i2="X" l="SPA">
<s0>Enfermedad degenerativa</s0>
<s5>38</s5>
</fC07>
<fC07 i1="03" i2="X" l="FRE">
<s0>Syndrome oculomoteur</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="ENG">
<s0>Oculomotor syndrome</s0>
<s5>39</s5>
</fC07>
<fC07 i1="03" i2="X" l="SPA">
<s0>Oculomotricidad síndrome</s0>
<s5>39</s5>
</fC07>
<fC07 i1="04" i2="X" l="FRE">
<s0>Pathologie de l'oeil</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="ENG">
<s0>Eye disease</s0>
<s5>40</s5>
</fC07>
<fC07 i1="04" i2="X" l="SPA">
<s0>Ojo patología</s0>
<s5>40</s5>
</fC07>
<fC07 i1="05" i2="X" l="FRE">
<s0>Pathologie du système nerveux central</s0>
<s5>41</s5>
</fC07>
<fC07 i1="05" i2="X" l="ENG">
<s0>Central nervous system disease</s0>
<s5>41</s5>
</fC07>
<fC07 i1="05" i2="X" l="SPA">
<s0>Sistema nervosio central patología</s0>
<s5>41</s5>
</fC07>
<fC07 i1="06" i2="X" l="FRE">
<s0>Syndrome du tronc cérébral</s0>
<s5>43</s5>
</fC07>
<fC07 i1="06" i2="X" l="ENG">
<s0>Brain stem syndrome</s0>
<s5>43</s5>
</fC07>
<fC07 i1="06" i2="X" l="SPA">
<s0>Tallo encefalico sindrome</s0>
<s5>43</s5>
</fC07>
<fC07 i1="07" i2="X" l="FRE">
<s0>Trouble de la communication</s0>
<s5>44</s5>
</fC07>
<fC07 i1="07" i2="X" l="ENG">
<s0>Communication disorder</s0>
<s5>44</s5>
</fC07>
<fC07 i1="07" i2="X" l="SPA">
<s0>Trastorno comunicación</s0>
<s5>44</s5>
</fC07>
<fC07 i1="08" i2="X" l="FRE">
<s0>Trouble du langage</s0>
<s5>45</s5>
</fC07>
<fC07 i1="08" i2="X" l="ENG">
<s0>Language disorder</s0>
<s5>45</s5>
</fC07>
<fC07 i1="08" i2="X" l="SPA">
<s0>Trastorno lenguaje</s0>
<s5>45</s5>
</fC07>
<fC07 i1="09" i2="X" l="FRE">
<s0>Trouble neurologique</s0>
<s5>46</s5>
</fC07>
<fC07 i1="09" i2="X" l="ENG">
<s0>Neurological disorder</s0>
<s5>46</s5>
</fC07>
<fC07 i1="09" i2="X" l="SPA">
<s0>Trastorno neurológico</s0>
<s5>46</s5>
</fC07>
<fN21>
<s1>081</s1>
</fN21>
<fN44 i1="01">
<s1>OTO</s1>
</fN44>
<fN82>
<s1>OTO</s1>
</fN82>
</pA>
<pR>
<fA30 i1="01" i2="1" l="ENG">
<s1>International Congress on Mental Dysfunction and other non-motor features in Parkinson's Disease and Related Disorders</s1>
<s2>9</s2>
<s3>Dresden DEU</s3>
<s4>2008-10-16</s4>
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<server>
<NO>PASCAL 10-0131742 INIST</NO>
<ET>Behavior and cognition in corticobasal degeneration and progressive supranuclear palsy</ET>
<AU>KERTESZ (Andrew); MCMONAGLE (Paul); REICHMANN (Heinz); KORCZYN (Amos D.)</AU>
<AF>Dept of Cognitive Neurology, University of Western Ontario, St Joseph's Hospital, 268 Grosvenor St/London, Ontario, N6A 4V2/Canada (1 aut.); Dept of Neurology, The Royal Victoria Hospital, 274 Grosvenor Road/Belfast BT12 6BA/Royaume-Uni (2 aut.); Department of Neurology, Dresden University Medical School/Dresden/Allemagne (1 aut.); Department of Neurology, Tel Aviv University Medical School/Ramat Aviv/Israël (2 aut.)</AF>
<DT>Publication en série; Congrès; Niveau analytique</DT>
<SO>Journal of the neurological sciences; ISSN 0022-510X; Coden JNSCAG; Irlande; Da. 2010; Vol. 289; No. 1-2; Pp. 138-143; Bibl. 104 ref.</SO>
<LA>Anglais</LA>
<EA>Progressive supranuclear palsy (PSP) and corticobasal degeneration (CBD), previously described as Parkinsonian syndromes are also cognitive disorders, and biologically related to the frontotemporal dementia or Pick's disease. PSP and CBD overlap clinically, pathologically and genetically, sharing tau haplotypes and mutations. In our series of CBD/PSP patients with cognitive presentation (n=36), primary progressive aphasia (PPA) was particularly common, but behavioral onset occurred also. CBD or PSP as motor presentations developed significant language disorder in 17/19. The underlying pathology is predictably tau positive in these clinical combinations, regardless of the presentation. Other cognitive features of CBDS include apraxia, alien hand and apathy, but often frontal lobe dementia with disinhibition develops also. CBDS also has visuospatial deficit, because of the parietal involvement. PSP was considered the prototype of subcortical dementia, with bradyphrenia, poor recall and executive deficit, but cortical features were recognized to be important also. Language testing and a behavioral inventory should be part of neuropsychological tests to facilitate diagnosis and to quantify the deficit. The clinical, genetic and pathological relationship is strong between CBD /PSP and the aphasic and behavioral components of the Pick complex.</EA>
<CC>002B17; 002B17G</CC>
<FD>Dégénérescence; Démence frontotemporale; Aphasie; Apraxie; Pathologie du système nerveux; Comportement; Cognition; Ophtalmoplégie supranucléaire</FD>
<FG>Pathologie de l'encéphale; Maladie dégénérative; Syndrome oculomoteur; Pathologie de l'oeil; Pathologie du système nerveux central; Syndrome du tronc cérébral; Trouble de la communication; Trouble du langage; Trouble neurologique</FG>
<ED>Degeneration; Frontotemporal dementia; Aphasia; Apraxia; Nervous system diseases; Behavior; Cognition; Supranuclear ophthalmoplegia</ED>
<EG>Cerebral disorder; Degenerative disease; Oculomotor syndrome; Eye disease; Central nervous system disease; Brain stem syndrome; Communication disorder; Language disorder; Neurological disorder</EG>
<SD>Degeneración; Demencia frontotemporal; Afasia; Apraxia; Sistema nervioso patología; Conducta; Cognición; Oftalmoplejía supranuclear</SD>
<LO>INIST-12185.354000190042590260</LO>
<ID>10-0131742</ID>
</server>
</inist>
</record>

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