Dopamine, reward, and frontostriatal circuitry in impulse control disorders in Parkinson's disease: insights from functional imaging.
Identifieur interne : 000C77 ( Ncbi/Merge ); précédent : 000C76; suivant : 000C78Dopamine, reward, and frontostriatal circuitry in impulse control disorders in Parkinson's disease: insights from functional imaging.
Auteurs : Nicola Ray [Canada] ; Antonio P. StrafellaSource :
- Clinical EEG and neuroscience [ 1550-0594 ] ; 2010.
English descriptors
- KwdEn :
- Animals, Brain Mapping (methods), Corpus Striatum (physiopathology), Disruptive, Impulse Control, and Conduct Disorders (complications), Disruptive, Impulse Control, and Conduct Disorders (physiopathology), Dopamine (metabolism), Frontal Lobe (physiopathology), Humans, Magnetic Resonance Imaging (methods), Nerve Net (physiopathology), Parkinson Disease (complications), Parkinson Disease (physiopathology), Reward.
- MESH :
- chemical , metabolism : Dopamine.
- complications : Disruptive, Impulse Control, and Conduct Disorders, Parkinson Disease.
- methods : Brain Mapping, Magnetic Resonance Imaging.
- physiopathology : Corpus Striatum, Disruptive, Impulse Control, and Conduct Disorders, Frontal Lobe, Nerve Net, Parkinson Disease.
- Animals, Humans, Reward.
Abstract
Dopamine agonists have been implicated in the development of impulse control disorders (ICDs). This may be due to the ability of agonists to tonically stimulate dopamine receptors. Recent neuroimaging data provided evidence that dopamine agonists induce significant changes in those frontostriatal circuits that process reward and mediate our ability to control impulses. Tonic stimulation of dopamine receptors via agonists may impair reward processing and inhibitory control mechanisms in ways that promote pathological repetition of behaviors. We will provide an overview of the current understanding of the neurobiology underlying ICDs in Parkinson's disease (PD).
DOI: 10.1177/155005941004100208
PubMed: 20521491
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pubmed:20521491Le document en format XML
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<front><div type="abstract" xml:lang="en">Dopamine agonists have been implicated in the development of impulse control disorders (ICDs). This may be due to the ability of agonists to tonically stimulate dopamine receptors. Recent neuroimaging data provided evidence that dopamine agonists induce significant changes in those frontostriatal circuits that process reward and mediate our ability to control impulses. Tonic stimulation of dopamine receptors via agonists may impair reward processing and inhibitory control mechanisms in ways that promote pathological repetition of behaviors. We will provide an overview of the current understanding of the neurobiology underlying ICDs in Parkinson's disease (PD).</div>
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<Abstract><AbstractText>Dopamine agonists have been implicated in the development of impulse control disorders (ICDs). This may be due to the ability of agonists to tonically stimulate dopamine receptors. Recent neuroimaging data provided evidence that dopamine agonists induce significant changes in those frontostriatal circuits that process reward and mediate our ability to control impulses. Tonic stimulation of dopamine receptors via agonists may impair reward processing and inhibitory control mechanisms in ways that promote pathological repetition of behaviors. We will provide an overview of the current understanding of the neurobiology underlying ICDs in Parkinson's disease (PD).</AbstractText>
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